List of ICD Codes (V.2009 and V.2012 ICD-10-CA¹)
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Diagnostic Code Descriptions (ICD9)
INFECTIONS AND PARASITIC DISEASES INTESTINAL AND INFECTIOUS DISEASES (001 – 009.3) 001 CHOLERA 001.0 DUE TO VIBRIO CHOLERAE 001.1 DUE TO VIBRIO CHOLERAE EL TOR 001.9 UNSPECIFIED 002 TYPHOID AND PARATYPHOID FEVERS 002.0 TYPHOID FEVER 002.1 PARATYPHOID FEVER 'A' 002.2 PARATYPHOID FEVER 'B' 002.3 PARATYPHOID FEVER 'C' 002.9 PARATYPHOID FEVER, UNSPECIFIED 003 OTHER SALMONELLA INFECTIONS 003.0 SALMONELLA GASTROENTERITIS 003.1 SALMONELLA SEPTICAEMIA 003.2 LOCALIZED SALMONELLA INFECTIONS 003.8 OTHER 003.9 UNSPECIFIED 004 SHIGELLOSIS 004.0 SHIGELLA DYSENTERIAE 004.1 SHIGELLA FLEXNERI 004.2 SHIGELLA BOYDII 004.3 SHIGELLA SONNEI 004.8 OTHER 004.9 UNSPECIFIED 005 OTHER FOOD POISONING (BACTERIAL) 005.0 STAPHYLOCOCCAL FOOD POISONING 005.1 BOTULISM 005.2 FOOD POISONING DUE TO CLOSTRIDIUM PERFRINGENS (CL.WELCHII) 005.3 FOOD POISONING DUE TO OTHER CLOSTRIDIA 005.4 FOOD POISONING DUE TO VIBRIO PARAHAEMOLYTICUS 005.8 OTHER BACTERIAL FOOD POISONING 005.9 FOOD POISONING, UNSPECIFIED 006 AMOEBIASIS 006.0 ACUTE AMOEBIC DYSENTERY WITHOUT MENTION OF ABSCESS 006.1 CHRONIC INTESTINAL AMOEBIASIS WITHOUT MENTION OF ABSCESS 006.2 AMOEBIC NONDYSENTERIC COLITIS 006.3 AMOEBIC LIVER ABSCESS 006.4 AMOEBIC LUNG ABSCESS 006.5 AMOEBIC BRAIN ABSCESS 006.6 AMOEBIC SKIN ULCERATION 006.8 AMOEBIC INFECTION OF OTHER SITES 006.9 AMOEBIASIS, UNSPECIFIED 007 OTHER PROTOZOAL INTESTINAL DISEASES 007.0 BALANTIDIASIS 007.1 GIARDIASIS 007.2 COCCIDIOSIS 007.3 INTESTINAL TRICHOMONIASIS 007.8 OTHER PROTOZOAL INTESTINAL DISEASES 007.9 UNSPECIFIED 008 INTESTINAL INFECTIONS DUE TO OTHER ORGANISMS -
Molecular Detection of Human Parasitic Pathogens
MOLECULAR DETECTION OF HUMAN PARASITIC PATHOGENS MOLECULAR DETECTION OF HUMAN PARASITIC PATHOGENS EDITED BY DONGYOU LIU Boca Raton London New York CRC Press is an imprint of the Taylor & Francis Group, an informa business CRC Press Taylor & Francis Group 6000 Broken Sound Parkway NW, Suite 300 Boca Raton, FL 33487-2742 © 2013 by Taylor & Francis Group, LLC CRC Press is an imprint of Taylor & Francis Group, an Informa business No claim to original U.S. Government works Version Date: 20120608 International Standard Book Number-13: 978-1-4398-1243-3 (eBook - PDF) This book contains information obtained from authentic and highly regarded sources. Reasonable efforts have been made to publish reliable data and information, but the author and publisher cannot assume responsibility for the validity of all materials or the consequences of their use. The authors and publishers have attempted to trace the copyright holders of all material reproduced in this publication and apologize to copyright holders if permission to publish in this form has not been obtained. If any copyright material has not been acknowledged please write and let us know so we may rectify in any future reprint. Except as permitted under U.S. Copyright Law, no part of this book may be reprinted, reproduced, transmitted, or utilized in any form by any electronic, mechanical, or other means, now known or hereafter invented, including photocopying, microfilming, and recording, or in any information storage or retrieval system, without written permission from the publishers. For permission to photocopy or use material electronically from this work, please access www.copyright.com (http://www.copyright.com/) or contact the Copyright Clearance Center, Inc. -
CMS PMB ICD-10 Coding
CMS July 2005 PMB Coding 4th Character Final Dcoument July 2005 Page 1 of 69 Code Diagnosis Treatment CMS PMB ICD-10 Coding 1. Annexure A of the General Regulations made in terms of the Medical Schemes Act, 131 of 1998 provides a schedule of “diagnosis and treatment pairs”, which cumulatively comprise the prescribed minimum benefits (PMBs) to be provided to beneficiaries of medical schemes in terms of section 29(1)(o) of the Act. 2. The attached ICD10 codes represent the Council for Medical Schemes’ interpretation of the “diagnosis” portion of these PMBs. 3. In the event of conflict between this interpretation and the definition of conditions set out in Annexure A to the regulations, the definition of conditions contained in the regulations will prevail. 4. In this schedule: a. where only the primary code in the form of a dagger code has been used, all asterisk codes listed under that specific code as per the ICD-10 set of books form part of the PMB diagnosis; b. where a dagger code has been used in conjunction with specific asterisk codes, omitted asterisk codes relevant to that dagger code do not form part of the PMB diagnosis. 5. The ICD-10 codes have been coded up to the 4th character. A draft document containing 5th character codes where applicable, will be released for comments within the next month. After due consideration, a final document containing up to the 5th character will conclude the PMB coding process. 906A Acute generalised Medical management; A80.0 Acute paralytic poliomyelitis, vaccine-associated paralysis, including -
2011/109440 Al
(12) INTERNATIONAL APPLICATION PUBLISHED UNDER THE PATENT COOPERATION TREATY (PCT) (19) World Intellectual Property Organization International Bureau (10) International Publication Number (43) International Publication Date _ . 9 September 2011 (09.09.2011) 2011/109440 Al (51) International Patent Classification: [CH/CH]; Chemin Des Chevreuils 1, 1188 Gimel (CH). C12Q 1/68 (2006.01) G01N 33/53 (2006.01) HOLTERMAN, Daniel [US/US]; 14465 North 14th St., Phoenix, AZ 85022 (US). (21) International Application Number: PCT/US201 1/026750 (74) Agent: AKHAVAN, Ramin; Caris Life Sciences, Inc., 6655 N. MacArthur Blvd., Irving, TX 75039 (US). (22) International Filing Date: 1 March 201 1 (01 .03.201 1) (81) Designated States (unless otherwise indicated, for every kind of national protection available): AE, AG, AL, AM, English (25) Filing Language: AO, AT, AU, AZ, BA, BB, BG, BH, BR, BW, BY, BZ, (26) Publication Language: English CA, CH, CL, CN, CO, CR, CU, CZ, DE, DK, DM, DO, DZ, EC, EE, EG, ES, FI, GB, GD, GE, GH, GM, GT, (30) Priority Data: HN, HR, HU, ID, IL, IN, IS, JP, KE, KG, KM, KN, KP, 61/274,124 1 March 2010 (01 .03.2010) US KR, KZ, LA, LC, LK, LR, LS, LT, LU, LY, MA, MD, 61/357,5 17 22 June 2010 (22.06.2010) US ME, MG, MK, MN, MW, MX, MY, MZ, NA, NG, NI, 61/364,785 15 July 2010 (15.07.2010) us NO, NZ, OM, PE, PG, PH, PL, PT, RO, RS, RU, SC, SD, (71) Applicant (for all designated States except US): CARIS SE, SG, SK, SL, SM, ST, SV, SY, TH, TJ, TM, TN, TR, LIFE SCIENCES LUXEMBOURG HOLDINGS [LU/ TT, TZ, UA, UG, US, UZ, VC, VN, ZA, ZM, ZW. -
Annex 1: List of Medical Case Rates
ANNEX 1. LIST OF MEDICAL CASE RATES FIRST CASE RATE ICD CODE DESCRIPTION GROUP Professional Health Care Case Rate Fee Institution Fee P91.3 Neonatal cerebral irritability ABNORMAL SENSORIUM IN THE NEWBORN 12,000 3,600 8,400 P91.4 Neonatal cerebral depression ABNORMAL SENSORIUM IN THE NEWBORN 12,000 3,600 8,400 P91.6 Hypoxic ischemic encephalopathy of newborn ABNORMAL SENSORIUM IN THE NEWBORN 12,000 3,600 8,400 P91.8 Other specified disturbances of cerebral status of newborn ABNORMAL SENSORIUM IN THE NEWBORN 12,000 3,600 8,400 P91.9 Disturbance of cerebral status of newborn, unspecified ABNORMAL SENSORIUM IN THE NEWBORN 12,000 3,600 8,400 Peritonsillar abscess; Abscess of tonsil; Peritonsillar J36 ABSCESS OF RESPIRATORY TRACT 10,000 3,000 7,000 cellulitis; Quinsy Other diseases of larynx; Abscess of larynx; Cellulitis of larynx; Disease NOS of larynx; Necrosis of larynx; J38.7 ABSCESS OF RESPIRATORY TRACT 10,000 3,000 7,000 Pachyderma of larynx; Perichondritis of larynx; Ulcer of larynx Retropharyngeal and parapharyngeal abscess; J39.0 ABSCESS OF RESPIRATORY TRACT 10,000 3,000 7,000 Peripharyngeal abscess Other abscess of pharynx; Cellulitis of pharynx; J39.1 ABSCESS OF RESPIRATORY TRACT 10,000 3,000 7,000 Nasopharyngeal abscess Other diseases of pharynx; Cyst of pharynx or nasopharynx; J39.2 ABSCESS OF RESPIRATORY TRACT 10,000 3,000 7,000 Oedema of pharynx or nasopharynx J85.1 Abscess of lung with pneumonia ABSCESS OF RESPIRATORY TRACT 10,000 3,000 7,000 J85.2 Abscess of lung without pneumonia; Abscess of lung NOS ABSCESS OF RESPIRATORY -
Natural Remedies of Common Human Parasites and Pathogens
ACTA SCIENTIFIC MICROBIOLOGY (ISSN: 2581-3226) Volume 2 Issue 11 November 2019 Investigation Paper Natural Remedies of Common Human Parasites and Pathogens Omar M Amin* Parasitology Center, Scottsdale, Arizona *Corresponding Author: Omar M Amin, Parasitology Center, Scottsdale, Arizona. Received: July 12, 2019; Published: October 16, 2019 DOI: 10.31080/ASMI.2019.02.0401 • Bleeding. • Appetite changes. • Malabsorption. • Mucus. • Rectal itching. • Gut leakage. • Poor digestion. • Systemic/other symptoms • Fatigue. • Skin rash. • Dry cough. • Brain fog/memory loss. • Lymph blockage. Figure 1 • Allergies. • Nausea. Diagnosis and management of: • Muscle or joint pain. • Parasitic organisms and agents of medical and public • Dermatitis. health importance in fecal, blood, skin, urine specimens. • Headaches. • Toxicities related to Neurocutaneous Syndrome (NCS). • Insomnia. Development of anti-parasitic herbal products (F/C/R) Edu- How we get infected cational services: workshops, seminars, training and publications Drinking water or juice: Giardia, Cryptosporidium. provided. 1. 2. Skin contact with contaminated water: Schistosomi- Consultations and protocols for herbal and allopathic treat- asis, swimmers itch. ments. Research: over 220 publications on parasites from all con- Food (fecal-oral infections): most protozoans, ex., tinents. 3. Blastocysts, Entamoeba spp. and worms: Ascaris. Why test? 4. Arthropods: Lyme disease, plague, typhus, etc. You need to be tested if you have one or more of these symp- 5. Air: Upper respiratory tract infections (viruses, bac- toms: teria), ex GI symptoms 6. Pets: Hydatid., flu, Valleycyst disease,fever, Hanta heart virus. worm, larva mi- grans (dogs), Toxoplasma (cats), Taenia (beef, swine. • Diarrhea/constipation. People (contagious diseases): AIDS, herpes. • Irritable bowel 7. Soil: hook worms, thread worms. • Cramps 8. -
Cutaneous Balamuthia Mandrillaris Infection As a Precursor To
Volume 23 Number 7 | July 2017 Dermatology Online Journal || Case Report DOJ 23 (7): 4 Cutaneous Balamuthia mandrillaris infection as a precursor to Balamuthia amoebic encephalitis (BAE) in a healthy 84-year-old Californian Larisa M Lehmer, Gabriel E Ulibarri, Bruce D Ragsdale, James Kunkle Affiliations: University of California Irvine Health, Department of Dermatology, Irvine, California, Central Coast Pathology Laboratory, San Luis Obispo, California, Western Diagnostic Services Laboratory, San Luis Obispo, California, Central Coast Dermatology, Arroyo Grande, California Corresponding Author: Larisa M Lehmer, Department of Dermatology, UC Irvine Health, 118 Med Surg 1, Irvine, CA 92697-2400, Email: [email protected] Abstract Keywords: Balamuthia mandrillaris; cutaneous balamuthiasis; granulomatous amoebic encephalitis; Soil and freshwater-dwelling amoebae may GAE; balamuthia amoebic encephalitis; BAE; opportunistically infect the skin and evoke a immunocompromise; granulomatous dermatitis; granulomatous dermatitis that camouflages their immune senescence. underlying morphology. Amoebic infestations are incredibly rare in the U.S., predominantly occurring in the young, elderly, and immunocompromised. Introduction Sadly, because diagnosis is difficult and unsuspected, Balamuthia mandrillaris (B. mandrillaris), along most cases are diagnosed at autopsy. The following with Naegleria, Sappinia and several species of case is of a healthy 84-year-old man with a non- Acanthomeba, are free-living amoebae found in fresh healing nodulo-ulcerative cutaneous lesion on his water and soil worldwide that may opportunistically left forearm that appeared following a gardening infect the skin and/or central nervous system (CNS) injury. Lesional punch biopsies repeatedly showed of humans. The first sign of infection ranges from non-specific granulomatous inflammation with a stubborn indurated plaque or ulceration on the no pathogens evident histologically or by culture. -
Die Prinzipien Der Chirurgischen Therapie Beim Fortgeschrittenen
Aus der Chirurgischen Klinik und Poliklinik - Innenstadt, der Ludwig-Maximilian- Universität-München Direktor: Prof. Dr. med. Wolf Mutschler Die Prinzipien der chirurgischen Therapie beim fortgeschrittenen Pyoderma gangränosum Dissertation zum Erwerb des Doktorgrades der Medizin an der Medizinischen Fakultät der Ludwig-Maximilians-Universität zu München vorgelegt von Christoph Hendrik Volkering aus Groß-Gerau 2008 Mit Genehmigung der Medizinischen Fakultät der Universität München Berichterstatter: Prof. Dr. Sigurd Keßler Mitberichterstatter: Prof. Dr. Hans C. Korting Priv. Doz. Dr. Martin K. Angele Dekan: Prof. Dr. med. Dr. h.c. Maximilian Reiser, FACR Tag der mündlichen Prüfung: 20.11.2008 - 2 - INHALT 1. Einleitung: ........................................................................................................... - 6 - 1.1. Das Pyoderma gangränosum: ..................................................................... - 6 - 1.1.1. Geschichte: ......................................................................................... - 6 - 1.1.2. Inzidenz: ............................................................................................. - 6 - 1.1.3. Assoziierte Erkrankungen: .................................................................. - 7 - 1.1.4. Typen des Pyoderma gangränosum: .................................................. - 9 - 1.1.5. Histopathologie: ................................................................................ - 12 - 1.1.6. Pathogenese: ................................................................................... -
CNS Infections: LP Is an Important Test in the Diagnosis of Meningitis and Encephalitis
Bacterial meningitis: bacterial infection of the Leptomeninges. • The most common cause of bacterial meningitis in neonates is B group streptococcus and Gram negative bacteria (E.coli and Citrobacter)-ampicillin and gentamycin or third generation cephalosporines. Citrobacter is associated with high incidence of multiple brain abscesses. The most common cause of bacterial meningitis in age group 3 months-5 years are H.influenzae and streptococcus pneumoniae-penecillinnes and third generation cephalosporines. The most common cause in older children and adults are strept. Pneumoniae and Neisseria meningitides-penecillinnes and cephalosporines. The most common cause of viral meningitis is enteroviruses (echo and Cocksakie viruses). • The clinical picture depends on the age: neonates and children present with sepsis. Adults and old children (fever, headaches, vomiting, neck stiffness and decreased level of consciousness. Hemorrhagic rash in case of Neisseria meningitides). Patients should be started on high dose Abs and in the absence of increased ICP , LP should be performed (high protein, decreased glucose, pleocytosis. Neutrophils in thousands. In the presence of raised ICP start Abs and get CT head. Duration of treatment 10-14 days iv Abs. • Individuals exposed to N.meningidites infection should be given Rifampicin for 3-5 days. Vaccines are available. • Talk about TB meningitis (basal meningitis with multiple cranial nerve palsies and sometimes hydrocephalus) and • Discuss the indications and contraindications of LP in CNS infections: LP is an important test in the diagnosis of meningitis and encephalitis. It is contraindicated in the presence signs of increased ICP due to space occupying lesion such as abscess, subdural and epidural empyema (papilloedema, decreased level of consciousness). -
Perineal Amoebiasis
Arch Dis Child: first published as 10.1136/adc.55.3.234 on 1 March 1980. Downloaded from 234 Yoshioka and Miyata damage in our patient, as speculated by Vachon logical tests in our patient showed any abnormality, et al.,5 because liver function was normal or border- nor was there any evidence of other autoimmune line, and HBV antigenaemia persisted even after the disease. anaemia improved and the DAGT had become negative. The role of HBV in the pathogenesis of References AIHA has thus been unclear. Dacie J V. The autoimmune haemolytic anaemia. In: From many recent studies of type B hepatitis6 it is The haemolytic anaemias; congenital and acquired. Part 2. suggested that a defect of cell-mediated immunity London: Churchill, 1962: 539. may yield the carrier state ofHBV. This hypothesis is 2 Zuelzer W W, Mastrangelo R, Stulberg C S, Poulik M D, consistent with the fact that newborn babies may be Page R H, Thompson R I. Autoimmune hemolytic anemia. Natural history and viral-immunologic inter- infected from their HBV-carrier mother, resulting in actions in childhood. Am JMed 1970; 49: 80-93. the persistent carrier state.7 Likewise, the role of 3 Barrett-Connor E. Anemia and infection. Am J Med cellular immunity in the pathogenesis of AIHA has 1972; 52: 242-53. become clearer. Kruger et al.8 suggested that an 4 Habibi B, Homberg J-C, Schaison G, Salmon C. Auto- immune hemolytic anemia in children. A review of 80 imbalance between reduced T-cells and increased but cases. Am JMed 1974; 56: 61-9. -
Management of Brain Abscesses
ORIGINAL ARTICLE Management of Brain Abscesses Riaz ur Rehman, Azmatullah khattak, Mian Iftihar Ul Huq, Mewat Shah, Mushtaq ABSTRACT Objective To find the etiology and outcome of treatment of brain abscesses. Study design Descriptive case series. Place & Department of Neurosurgery, Hayatabad Medical complex Peshawar, from October 2008 Duration of to January 2010. study Methodology This study was carried out on patients of brain abscesses of all ages and both genders. Patients having fungal brain abscess, amoebic brain abscess and tuberculous brain abscess were excluded. Brain abscess was diagnosed on contrast CT scan. Cases of early cerebritis were treated using parenteral antibiotics for six to eight weeks. Surgical treatment consisted of either burr hole aspiration with the help of brain cannula, re-aspiration or craniotomy and excision of abscess capsule. Therapeutic outcome was assessed with CT scan on follow up. Procedure related complications and mortality were also recorded. Results A total of 73 patients were managed. The commonest age group was from 11-20 year. The mean age was 26.36 ± 14.1 year (range - 0.16 - 67 year). There were 46 (63.01%) male and 27 (36.99%) female patients. The majority of brain abscesses were supratentorial (n=65 - 89.04%). In 8 (11.96%) cases abscess was in infratentorial region. Contiguous focus of infection was responsible for brain abscess in 29 (39.72%) patients, Majority of patients presented with headache (n=30 - 41.09%) and vomiting (n=25 - 34.24%). Surgical drainage was performed in 70 (95.89%) patients where as 3 (4.11%) patients were treated conservatively. Initially only burr hole aspiration was done in all surgically treated patients. -
5D4181239216c.Pdf
http://www.shamela.ws مت إعداد هذا امللف آليا بواسطة املكتبة الشاملة الكتاب: اﻷمراض اجللدية لﻷطفال املؤلف: الدكتور حممود حجازي ]موافق للمطبوع[ مﻻحظة: ]هذا الكتاب من كتب املستودع مبوقع املكتبة الشاملة[ اﻷمراض اجللدية لﻷطفال املؤلف / الدكتور ـ حممود حجازى ----------------- الفصل اﻷول مركبات اجللد الفهرس الفصل التايل فهرس الصور حبث اخلط املائل يف هذا الكتاب ميثل رأي وخربة املؤلف ****** مركبات اجللد جلد اجلنني: يف اﻷايم اﻷوىل من حياة اجلنني تتكون الطبقة السطحية للجلد من طبقة واحدة من اخلﻻاي حيث تتحول إىل طبقتني بني اﻷسبوع اخلامس والسادس، السطحية هي البشرة والسفلية هي الطبقة النامية من اجللد. إن الطبقة النامية هي املسؤولة عن تكوين معظم املكوانت الظهارية للجلد مثل الطبقة القاعدة والغدد العرقية. أما اخلﻻاي الظهارية النامية فإهنا مسؤولة عن تكوين الغدد الدهنية والغدد العرقية وغدد »أبو كراين« العرقية وجراب الشعر أما طبقة مالييجى فتتكون يف الشهر الرابع من عمر اجلنني. جلد الطفل: جلد الطفل له ملمس انعم ويشبه جلد البالغ من الناحية التشرحيية لﻷنسجة مع بعض الفروقات البسيطة. إن طبقة البشرة من اجللد يف اﻷطفال هي نفسها يف البالغني إﻻ أهنا أقل متاسكاً والفرق الرئيسي واهلام هو عدم النضوج الكامل لنسيج الكوﻻجني وجراب الشعر والغدد الدهنية يف اﻷطفال. كما أن البشرة السطحية مع طبقة اﻷدمة السفلية أقل متاسكاً يف اﻷطفال وهذا يفسر حدوث اﻷثر اﻷكثر تفاعﻻً نتيجة املؤثرات البسيطة يف اﻷطفال، حيث أن لدغة احلشرات قد حتدث فأليل جلدية. كما وأن التغريات نتيجة اﻻختﻻف يف نسبة مساحة اجللد إىل جسم اﻷطفال وكذلك نسبة نشاط اﻷوعية الدموية وكذلك القابلية للتعرق الزائد، كل ذلك له عﻻقة هامة على تنظيم حرارة جسم اﻷطفال، حيث أن بعض املؤثرات البسيطة قد تؤدي إىل املزيد من فقدان حرارة جسم اﻷطفال.