Annex 1: List of Medical Case Rates
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Natural Remedies of Common Human Parasites and Pathogens
ACTA SCIENTIFIC MICROBIOLOGY (ISSN: 2581-3226) Volume 2 Issue 11 November 2019 Investigation Paper Natural Remedies of Common Human Parasites and Pathogens Omar M Amin* Parasitology Center, Scottsdale, Arizona *Corresponding Author: Omar M Amin, Parasitology Center, Scottsdale, Arizona. Received: July 12, 2019; Published: October 16, 2019 DOI: 10.31080/ASMI.2019.02.0401 • Bleeding. • Appetite changes. • Malabsorption. • Mucus. • Rectal itching. • Gut leakage. • Poor digestion. • Systemic/other symptoms • Fatigue. • Skin rash. • Dry cough. • Brain fog/memory loss. • Lymph blockage. Figure 1 • Allergies. • Nausea. Diagnosis and management of: • Muscle or joint pain. • Parasitic organisms and agents of medical and public • Dermatitis. health importance in fecal, blood, skin, urine specimens. • Headaches. • Toxicities related to Neurocutaneous Syndrome (NCS). • Insomnia. Development of anti-parasitic herbal products (F/C/R) Edu- How we get infected cational services: workshops, seminars, training and publications Drinking water or juice: Giardia, Cryptosporidium. provided. 1. 2. Skin contact with contaminated water: Schistosomi- Consultations and protocols for herbal and allopathic treat- asis, swimmers itch. ments. Research: over 220 publications on parasites from all con- Food (fecal-oral infections): most protozoans, ex., tinents. 3. Blastocysts, Entamoeba spp. and worms: Ascaris. Why test? 4. Arthropods: Lyme disease, plague, typhus, etc. You need to be tested if you have one or more of these symp- 5. Air: Upper respiratory tract infections (viruses, bac- toms: teria), ex GI symptoms 6. Pets: Hydatid., flu, Valleycyst disease,fever, Hanta heart virus. worm, larva mi- grans (dogs), Toxoplasma (cats), Taenia (beef, swine. • Diarrhea/constipation. People (contagious diseases): AIDS, herpes. • Irritable bowel 7. Soil: hook worms, thread worms. • Cramps 8. -
Ten Leading Causes of Death by Age Group,Race/Ethnicity, and Gender
Ten Leading Causes of Death by Age Group, Race/Ethnicity, and Gender Suburban Cook County 2006-2008 Suggested Citation: Cook County Department of Public Health. (2013). Ten Leading Causes of Death Tables 2008. Oak Forest, IL: Community Epidemiology and Health Planning Unit Table of Contents All Suburban Cook County: All Deaths By Gender: Females Males By Race/Ethnicity and Gender: Hispanic Hispanic Females Hispanic Males Non Hispanic Asian/Pacific Islander Non Hispanic Asian/Pacific Islander Females Non Hispanic Asian/Pacific Islander Males Non Hispanic Black Non Hispanic Black Females Non Hispanic Black Males Non Hispanic White Non Hispanic White Females Non Hispanic White Males By District: Cook County Department of Public Health Jurisdiction North District West District Southwest District South District Resources: District Map Rankable Causes of Death Rankable Causes of Infant Death 10 Leading Causes of Death by Age Group, Suburban Cook County, 2006-2008 (Counts/Percent of Total Deaths) All Deaths Rank <1 1-4 5-17 18-24 25-34 35-44 45-54 55-64 65+ Total Unintentional Unintentional Unintentional Unintentional Malignant Malignant Malignant Short Gestation Heart Disease Heart Disease 1 Injury Injury Injury Injury Neoplasms Neoplasms Neoplasms 170 24.9% 24 23.8% 57 24.6% 196 37.0% 281 34.4% 300 19.2% 1,251 31.7% 2,489 38.9% 13,422 28.2% 16,345 26.4% Congenital Congenital Unintentional Malignant Malignant Homicide Homicide Homicide Heart Disease Heart Disease 2 Anomalies Anomalies Injury Neoplasms Neoplasms 127 18.6% 9 8.9% 44 19.0% 135 -
Cutaneous Balamuthia Mandrillaris Infection As a Precursor To
Volume 23 Number 7 | July 2017 Dermatology Online Journal || Case Report DOJ 23 (7): 4 Cutaneous Balamuthia mandrillaris infection as a precursor to Balamuthia amoebic encephalitis (BAE) in a healthy 84-year-old Californian Larisa M Lehmer, Gabriel E Ulibarri, Bruce D Ragsdale, James Kunkle Affiliations: University of California Irvine Health, Department of Dermatology, Irvine, California, Central Coast Pathology Laboratory, San Luis Obispo, California, Western Diagnostic Services Laboratory, San Luis Obispo, California, Central Coast Dermatology, Arroyo Grande, California Corresponding Author: Larisa M Lehmer, Department of Dermatology, UC Irvine Health, 118 Med Surg 1, Irvine, CA 92697-2400, Email: [email protected] Abstract Keywords: Balamuthia mandrillaris; cutaneous balamuthiasis; granulomatous amoebic encephalitis; Soil and freshwater-dwelling amoebae may GAE; balamuthia amoebic encephalitis; BAE; opportunistically infect the skin and evoke a immunocompromise; granulomatous dermatitis; granulomatous dermatitis that camouflages their immune senescence. underlying morphology. Amoebic infestations are incredibly rare in the U.S., predominantly occurring in the young, elderly, and immunocompromised. Introduction Sadly, because diagnosis is difficult and unsuspected, Balamuthia mandrillaris (B. mandrillaris), along most cases are diagnosed at autopsy. The following with Naegleria, Sappinia and several species of case is of a healthy 84-year-old man with a non- Acanthomeba, are free-living amoebae found in fresh healing nodulo-ulcerative cutaneous lesion on his water and soil worldwide that may opportunistically left forearm that appeared following a gardening infect the skin and/or central nervous system (CNS) injury. Lesional punch biopsies repeatedly showed of humans. The first sign of infection ranges from non-specific granulomatous inflammation with a stubborn indurated plaque or ulceration on the no pathogens evident histologically or by culture. -
Die Prinzipien Der Chirurgischen Therapie Beim Fortgeschrittenen
Aus der Chirurgischen Klinik und Poliklinik - Innenstadt, der Ludwig-Maximilian- Universität-München Direktor: Prof. Dr. med. Wolf Mutschler Die Prinzipien der chirurgischen Therapie beim fortgeschrittenen Pyoderma gangränosum Dissertation zum Erwerb des Doktorgrades der Medizin an der Medizinischen Fakultät der Ludwig-Maximilians-Universität zu München vorgelegt von Christoph Hendrik Volkering aus Groß-Gerau 2008 Mit Genehmigung der Medizinischen Fakultät der Universität München Berichterstatter: Prof. Dr. Sigurd Keßler Mitberichterstatter: Prof. Dr. Hans C. Korting Priv. Doz. Dr. Martin K. Angele Dekan: Prof. Dr. med. Dr. h.c. Maximilian Reiser, FACR Tag der mündlichen Prüfung: 20.11.2008 - 2 - INHALT 1. Einleitung: ........................................................................................................... - 6 - 1.1. Das Pyoderma gangränosum: ..................................................................... - 6 - 1.1.1. Geschichte: ......................................................................................... - 6 - 1.1.2. Inzidenz: ............................................................................................. - 6 - 1.1.3. Assoziierte Erkrankungen: .................................................................. - 7 - 1.1.4. Typen des Pyoderma gangränosum: .................................................. - 9 - 1.1.5. Histopathologie: ................................................................................ - 12 - 1.1.6. Pathogenese: ................................................................................... -
Helping Mothers Defend Their Decision to Breastfeed
University of Central Florida STARS Electronic Theses and Dissertations, 2004-2019 2015 Helping Mothers Defend their Decision to Breastfeed Kandis Natoli University of Central Florida Part of the Nursing Commons Find similar works at: https://stars.library.ucf.edu/etd University of Central Florida Libraries http://library.ucf.edu This Doctoral Dissertation (Open Access) is brought to you for free and open access by STARS. It has been accepted for inclusion in Electronic Theses and Dissertations, 2004-2019 by an authorized administrator of STARS. For more information, please contact [email protected]. STARS Citation Natoli, Kandis, "Helping Mothers Defend their Decision to Breastfeed" (2015). Electronic Theses and Dissertations, 2004-2019. 1392. https://stars.library.ucf.edu/etd/1392 HELPING MOTHERS DEFEND THEIR DECISION TO BREASTFEED by KANDIS M. NATOLI M.S.N. University of Central Florida, 2006 B.S.N. University of Central Florida, 2005 A dissertation submitted in partial fulfillment of the requirements for the Degree of Doctor of Philosophy in Nursing in the College of Nursing at the University of Central Florida Orlando Florida Fall Term 2015 Major Professor: Karen Arioan © 2015 Kandis M. Natoli ii ABSTRACT The United States has established breastfeeding as an important health indicator within the Healthy People agenda. Healthy People target goals for breastfeeding initiation, duration, and exclusivity remain unmet. The US Surgeon General’s Office reports that lack of knowledge and widespread misinformation about breastfeeding are barriers to meeting Healthy People goals. Breastfeeding mothers are vulnerable to messages that cast doubt on their ability to breastfeed. Very little research has examined specific approaches to help people resist negative messages about health beliefs and behaviors. -
Perineal Amoebiasis
Arch Dis Child: first published as 10.1136/adc.55.3.234 on 1 March 1980. Downloaded from 234 Yoshioka and Miyata damage in our patient, as speculated by Vachon logical tests in our patient showed any abnormality, et al.,5 because liver function was normal or border- nor was there any evidence of other autoimmune line, and HBV antigenaemia persisted even after the disease. anaemia improved and the DAGT had become negative. The role of HBV in the pathogenesis of References AIHA has thus been unclear. Dacie J V. The autoimmune haemolytic anaemia. In: From many recent studies of type B hepatitis6 it is The haemolytic anaemias; congenital and acquired. Part 2. suggested that a defect of cell-mediated immunity London: Churchill, 1962: 539. may yield the carrier state ofHBV. This hypothesis is 2 Zuelzer W W, Mastrangelo R, Stulberg C S, Poulik M D, consistent with the fact that newborn babies may be Page R H, Thompson R I. Autoimmune hemolytic anemia. Natural history and viral-immunologic inter- infected from their HBV-carrier mother, resulting in actions in childhood. Am JMed 1970; 49: 80-93. the persistent carrier state.7 Likewise, the role of 3 Barrett-Connor E. Anemia and infection. Am J Med cellular immunity in the pathogenesis of AIHA has 1972; 52: 242-53. become clearer. Kruger et al.8 suggested that an 4 Habibi B, Homberg J-C, Schaison G, Salmon C. Auto- immune hemolytic anemia in children. A review of 80 imbalance between reduced T-cells and increased but cases. Am JMed 1974; 56: 61-9. -
XI. COMPLICATIONS of PREGNANCY, Childbffith and the PUERPERIUM 630 Hydatidiform Mole Trophoblastic Disease NOS Vesicular Mole Ex
XI. COMPLICATIONS OF PREGNANCY, CHILDBffiTH AND THE PUERPERIUM PREGNANCY WITH ABORTIVE OUTCOME (630-639) 630 Hydatidiform mole Trophoblastic disease NOS Vesicular mole Excludes: chorionepithelioma (181) 631 Other abnormal product of conception Blighted ovum Mole: NOS carneous fleshy Excludes: with mention of conditions in 630 (630) 632 Missed abortion Early fetal death with retention of dead fetus Retained products of conception, not following spontaneous or induced abortion or delivery Excludes: failed induced abortion (638) missed delivery (656.4) with abnormal product of conception (630, 631) 633 Ectopic pregnancy Includes: ruptured ectopic pregnancy 633.0 Abdominal pregnancy 633.1 Tubalpregnancy Fallopian pregnancy Rupture of (fallopian) tube due to pregnancy Tubal abortion 633.2 Ovarian pregnancy 633.8 Other ectopic pregnancy Pregnancy: Pregnancy: cervical intraligamentous combined mesometric cornual mural - 355- 356 TABULAR LIST 633.9 Unspecified The following fourth-digit subdivisions are for use with categories 634-638: .0 Complicated by genital tract and pelvic infection [any condition listed in 639.0] .1 Complicated by delayed or excessive haemorrhage [any condition listed in 639.1] .2 Complicated by damage to pelvic organs and tissues [any condi- tion listed in 639.2] .3 Complicated by renal failure [any condition listed in 639.3] .4 Complicated by metabolic disorder [any condition listed in 639.4] .5 Complicated by shock [any condition listed in 639.5] .6 Complicated by embolism [any condition listed in 639.6] .7 With other -
Glossary of Icd-10 Codes
glossary of icd-10 codes List of 113 Causes of Death (1999-present) Cause of Death ICD-10 Codes Salmonella infections A01-A02 Shigellosis and amebiasis A03, A06 Certain other intestinal infections A04, A07-A09 Tuberculosis A16-A19 Respiratory Tuberculosis A16 Other Tuberculosis A17-A19 Whooping cough A37 Scarlet fever and erysipelas A38, A46 Meningococcal infection A39 Septicemia A40-A41 Syphilis A50-A53 Acute poliomyelitis A80 Arthropod-borne viral encephalitis A83-A84, A85.2 Measles B05 Viral Hepatitis B15-B19 Human immunodeficiency virus (HIV) disease B20-B24 Malaria B50-B54 Other and unspecified infections and parasitic diseases and their sequelae A00, A05, A20-A36, A42-A44, A48-A49, A54-A79, A81-A82, A85.0-A85.1, A85.8, A86-B04, B06-B09, B25-B49, B55-B99 Malignant neoplasm C00-C97 Malignant neoplasm of lip, oral cavity and pharynx C00-C14 Malignant neoplasm of esophagus C15 Malignant neoplasm of stomach C16 Malignant neoplasm of colon, rectum, anus C18-C21 Malignant neoplasm of liver and intrahepatic bile ducts C22 Malignant neoplasm of pancreas C25 Malignant neoplasm of larynx C32 Malignant neoplasm of trachea, bronchus and lung C33-C34 Malignant melanoma of skin C43 Malignant neoplasm of breast C50 Malignant neoplasm of cervix uteri C53 Malignant neoplasm of corpus uteri and uterus, part unspecified C54-C55 Malignant neoplasm of ovary C56 Malignant neoplasm of prostate C61 Malignant neoplasm of kidney and renal pelvis C64-C65 Malignant neoplasm of bladder C67 Malignant neoplasm of meninges, brain, and other parts of central nervous -
5D4181239216c.Pdf
http://www.shamela.ws مت إعداد هذا امللف آليا بواسطة املكتبة الشاملة الكتاب: اﻷمراض اجللدية لﻷطفال املؤلف: الدكتور حممود حجازي ]موافق للمطبوع[ مﻻحظة: ]هذا الكتاب من كتب املستودع مبوقع املكتبة الشاملة[ اﻷمراض اجللدية لﻷطفال املؤلف / الدكتور ـ حممود حجازى ----------------- الفصل اﻷول مركبات اجللد الفهرس الفصل التايل فهرس الصور حبث اخلط املائل يف هذا الكتاب ميثل رأي وخربة املؤلف ****** مركبات اجللد جلد اجلنني: يف اﻷايم اﻷوىل من حياة اجلنني تتكون الطبقة السطحية للجلد من طبقة واحدة من اخلﻻاي حيث تتحول إىل طبقتني بني اﻷسبوع اخلامس والسادس، السطحية هي البشرة والسفلية هي الطبقة النامية من اجللد. إن الطبقة النامية هي املسؤولة عن تكوين معظم املكوانت الظهارية للجلد مثل الطبقة القاعدة والغدد العرقية. أما اخلﻻاي الظهارية النامية فإهنا مسؤولة عن تكوين الغدد الدهنية والغدد العرقية وغدد »أبو كراين« العرقية وجراب الشعر أما طبقة مالييجى فتتكون يف الشهر الرابع من عمر اجلنني. جلد الطفل: جلد الطفل له ملمس انعم ويشبه جلد البالغ من الناحية التشرحيية لﻷنسجة مع بعض الفروقات البسيطة. إن طبقة البشرة من اجللد يف اﻷطفال هي نفسها يف البالغني إﻻ أهنا أقل متاسكاً والفرق الرئيسي واهلام هو عدم النضوج الكامل لنسيج الكوﻻجني وجراب الشعر والغدد الدهنية يف اﻷطفال. كما أن البشرة السطحية مع طبقة اﻷدمة السفلية أقل متاسكاً يف اﻷطفال وهذا يفسر حدوث اﻷثر اﻷكثر تفاعﻻً نتيجة املؤثرات البسيطة يف اﻷطفال، حيث أن لدغة احلشرات قد حتدث فأليل جلدية. كما وأن التغريات نتيجة اﻻختﻻف يف نسبة مساحة اجللد إىل جسم اﻷطفال وكذلك نسبة نشاط اﻷوعية الدموية وكذلك القابلية للتعرق الزائد، كل ذلك له عﻻقة هامة على تنظيم حرارة جسم اﻷطفال، حيث أن بعض املؤثرات البسيطة قد تؤدي إىل املزيد من فقدان حرارة جسم اﻷطفال. -
Guidebook On
Government of the People's Republic of Bangladesh Ministry of Health and Family Welfare Guidebook on ICD 10 ICD-10 Coding January 2015 Third edition Management Information System (MIS) Directorate General of Health Services (DGHS) Mohakhali, Dhaka-1212 www.dghs.gov.bd in collaboration with Government of the People's Republic of Bangladesh Ministry of Health and Family Welfare Guidebook on ICD 10 ICD-10 Coding January 2015 Third edition Management Information System (MIS) Directorate General of Health Services (DGHS) Mohakhali, Dhaka-1212 www.dghs.gov.bd in collaboration with Special Acknowledgement: Professor Dr. Deen Mohammad Noorul Huq Director General of Health Services Dr. N. Paranietharan WHO Representative to Bangladesh Editorial Board For This Edition Editor: Professor Dr. Abul Kalam Azad ADG (Planning & development) & Director-MIS-Health, DGHS Contributors: Dr. Rashidun Nessa Deputy Director, Management Information System, DGHS Professor Dr. Md. Ayub Ali Chowdhury Professor of Nephrology, National Institute of Kidney Diseases & Urology, Dhaka Professor Dr. MAK Azad Chowdhury Professor, Dept. of Neonatology, Dhaka Shishu Hospital Professor Dr. Ismail Hossain Associate Professor, Dept. of Medicine Shaheed Suhrawardy Medical College Hospital Dr. Md. Habibullah Talukder Raskin Associate Professor, Dept. of Cancer Epidemiology, NICR & H Dr. Mahmudul Haque Assistant Professor, Dept. of Community Medicine, NIPSOM Dr. Motlabur Rahman Assistant Professor, Dept. of Medicine, DMCH Dr. Ashish Kumar Saha Assistant Director, MIS, DGHS Dr. Lokman Hakim Program Manager (HIS & eHealth), MIS, DGHS Dr. Gowsal Azam Deputy Chief (Medical), MIS, DGHS Dr. Sultan Shamiul Bashar Medical Officer, MIS, DGHS Dr. Jeenat Maitry Medical Officer, MIS, DGHS Contributors of Second Edition Editor: Professor Dr. Abul Kalam Azad Additional Director General (Planning & Development) & Director, MIS-Health, DGHS Contributors: Dr. -
Meningitis, Encephalitis and Neonatal Sepsis 1 GBD 2017 1 WHO
The global burden of meningitis in children: Challenges with interpreting global health estimates Supplementary Appendix Contents Mortality modelling methods – Meningitis, encephalitis and neonatal sepsis 1 GBD 2017 1 WHO-MCEE 2000-2017 3 Pathogen specific meningitis mortality and incidence modelling methods 4 GBD 2017 4 MCEE/JHSPH 5 Tables and Figures Table S1: ICD10 codes mapped to meningitis, encephalitis and neonatal sepsis according to model 7 Table S2: Definitions of IHME’s GBD 2017 data quality star rating 12 Table S3: Location level covariates according to model 13 Table S4: Covariates used in WHO-MCEE’s multinomial logistic regression by age group, model and cause 14 Table S5: MCEE Final cause of death list 15 Table S6: MCEE modelled cause of death categories according to age 16 Figure S1: Quality of underlying cause of death data and modelling methods used to generate death estimates according to model. 17 Mortality modelling methods – Meningitis, encephalitis and neonatal sepsis This section provides a brief overview on how the different models derive meningitis, encephalitis and neonatal sepsis mortality estimates. However, full methodology is provided by the modellers elsewhere. Both GBD 2017 and MCEE 2000-2017 models used cause of death data from vital registration (VR) systems, sample registration systems (SR) and verbal autopsy (VA) studies to assign causes of death ensuring that the total number of deaths matches other estimates for the age-specific all-cause mortality. This involves the generation of data where data are The global burden of meningitis in children: Challenges with interpreting global health estimates Supplementary Appendix incomplete or completely missing. -
Review Article
DOI: 10.14260/jemds/2014/2146 REVIEW ARTICLE SKIN MANIFESTATIONS OF GASTROINTESTINAL DISEASES: A REVIEW Manisha Nijhawan1, Puneet Agarwal2, Sandeep Nijhawan3, Prashant4, Abhishek Saini5 HOW TO CITE THIS ARTICLE: Manisha Nijhawan, Puneet Agarwal, Sandeep Nijhawan, Prashant, Abhishek Saini. “Skin Manifestations of Gastrointestinal Diseases: A Review”. Journal of Evolution of Medical and Dental Sciences 2014; Vol. 3, Issue 09, March 3; Page: 2357-2372, DOI: 10.14260/jemds/2014/2146 ABSTRACT: Skin is the largest organ of human body and stands as a guard for our internal organs. It can be regarded as a mirror giving a reflection of metabolic, biochemical and functional status of our internal organs. Dermatologists/Gastroenterologist should be aware of the dermatological manifestations as these change may be the first clue that a patient has underlying gastrointestinal (GI) or liver disease. Recognizing these signs is important in early and appropriate diagnosis. This article reviews the important dermatological manifestation of various GI and liver diseases. KEYWORDS: Skin and GI. Different dermatological manifestation in gastrointestinal diseases can be classified as:- 1) Specific skin manifestations 2) Reactive skin manifestations 3) Skin manifestations secondary to the deficiency of nutrients due to GI disease 4) Skin manifestations secondary to the treatment For clinician skin manifestation can be simply classified as- 1. Dermatological manifestations in benign GI diseases 2. Dermatological manifestations in malignant GI disease J of Evolution of Med and Dent Sci/ eISSN- 2278-4802, pISSN- 2278-4748/ Vol. 3/ Issue 09/ Mar 3, 2014 Page 2357 DOI: 10.14260/jemds/2014/2146 REVIEW ARTICLE A: Skin manifestations in esophageal diseases: Dysphagia: Esophageal webs This is a developmental abnormality with one or more horizontal membrane in upper esophagus.