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- SPRED1 Gene Sprouty Related EVH1 Domain Containing 1
- BRD4 Connects Enhancer Remodeling to Senescence Immune Surveillance
- Micrornas in Medullary Thyroid Carcinoma: a State of the Art Review of the Regulatory Mechanisms and Future Perspectives
- REVIEW Molecular Genetics of Medullary Thyroid Carcinoma: The
- Deletions of IKZF1 and SPRED1 Are Associated
- Supplemental Table-1
- A Functional Genetic Screen Deines the AKT-Induced Senescence
- University of Cincinnati
- Src Acts with WNT/FGFRL Signaling to Pattern the Planarian Anteroposterior Axis 3 4 5 Authors and Affiliations
- Neurofibromatosis Type 1-Legius Syndrome
- Cutting the Brakes on Ras—Cytoplasmic Gaps As Targets of Inactivation in Cancer
- SPRED Proteins and Their Roles in Signal Transduction, Development, and Malignancy
- Patient History for Deletion/Duplication Analysis by Mlpa
- The Neurofibromin Recruitment Factor Spred1 Binds to the GAP Related Domain Without Affecting Ras Inactivation
- Mutation Profile of Primary Subungual Melanomas in Caucasians
- SPRED1, a RAS MAPK Pathway Inhibitor That Causes Legius Syndrome, Is a Tumour Suppressor Downregulated in Paediatric Acute Myeloblastic Leukaemia
- SUPPLEMENTARY APPENDIX Tumor Suppressors BTG1 and BTG2 Regulate Early Mouse B-Cell Development
- Acknowledgements
- REVIEW Sprouty Proteins
- Simultaneous Detection of NF1, SPRED1, LZTR1, and NF2 Gene Mutations by Targeted NGS in an Italian Cohort of Suspected NF1 Patients
- Towards Clinical Decision Support in Oncology: Identifying Driver Alterations and Therapeutic Options
- SPRED1 Mutations (Legius Syndrome): Another Clinically Useful
- Supplemental Table 1 Alung Cancer NOS (2%), Carcinoid (0.7%)
- Oncogenic KRAS Engages an RSK1/NF1 Pathway to Inhibit Wild-Type RAS Signaling in Pancreatic Cancer
- Evaluation of the Role of Kras Gene in Colon Cancer Pathway Using String and Cytoscape Software
- Metabolic Network-Based Stratification of Hepatocellular Carcinoma Reveals Three Distinct Tumor Subtypes
- MOMA Cancerpanel V4 (2020-10-01), 119 Genes
- The Protein Tyrosine Phosphatase, Shp2, Functions in Multiple
- Boards' Fodder
- Pathogenic Mutations Associated with Legius Syndrome Modify the Spred1 Surface and Are Involved in Direct Binding to the Ras Inactivator Neurofibromin
- Supp Tables.Pdf
- The Transcriptional Landscape of Plasmacytoid Dendritic Cell Differentiation
- Rapid Mendelian Sequencing Gene List December 2018
- 2021 Pathology & Lab CPT Updates
- Inflammation and Hras Signaling Control Epithelial–Mesenchymal Transition During Skin Tumor Progression
- Diagnostic Difficulties and Possibilities of NF1-Like Syndromes in Childhood
- Oncogenic KRAS Engages an RSK1/NF1 Complex in Pancreatic Cancer
- Cooperative Regulation of Ras/MAPK Pathway Signaling by Spredl and Neurofibromin: Insights Into the Moiecuiar Basis of Legius Syndrome
- The Neurofibromin Recruitment Factor Spred1 Binds to the GAP Related Domain Without Affecting Ras Inactivation
- A Novel Kindred with Familial Gastrointestinal Stromal Tumors Caused by a Rare KIT Germline Mutation (N655K): Clinico-Pathological Presentation and TKI Sensitivity