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ATP5L
Establishing the Pathogenicity of Novel Mitochondrial DNA Sequence Variations: a Cell and Molecular Biology Approach
Molecular Profile of Tumor-Specific CD8+ T Cell Hypofunction in a Transplantable Murine Cancer Model
A Computational Approach for Defining a Signature of Β-Cell Golgi Stress in Diabetes Mellitus
Low Abundance of the Matrix Arm of Complex I in Mitochondria Predicts Longevity in Mice
Supplementary Table S4. FGA Co-Expressed Gene List in LUAD
Single-Cell RNA Sequencing Reveals Renal Endothelium Heterogeneity and Metabolic Adaptation to Water Deprivation
Novel KMT2A-ATP5L Gene Fusion in a Young Adult with Rapidly Progressive Ph-Like T(9;12) Acute B Lymphoblastic Leukemia
ATP5D Shrna (M) Lentiviral Particles: Sc-141346-V
Supplemental Table 1A. Differential Gene Expression Profile of Adehcd40l and Adehnull Treated Cells Vs Untreated Cells
C6orf203 Controls OXPHOS Function Through Modulation of Mitochondrial Protein Biosynthesis
Molecular Architecture Underlying Fluid Absorption by the Developing Inner
Supplemental Figure 1. Protein-Protein Interaction Network with Increased Expression in Fteb During the Luteal Phase
Transcriptomic and Proteomic Landscape of Mitochondrial
Mitochondrial Structure and Bioenergetics in Normal and Disease Conditions
Online Appendix 3. Supplemental Information Methods the Complete Methods Used in These Studies Are Described Below. Subjects. Le
Supplemental Figure 1: Immunoelectron Microscopy of Ribeye on the Harvested Sucrose Pellet from the Gradient Centrifugation Shown in Multiple Panels
Adipose Gene Expression Profiles Reveal Novel Insights Into the Adaptation of Northern Eurasian Semi-Domestic Reindeer (Rangifer
A Genomic View of Estrogen Actions in Human Breast Cancer Cells by Expression Profiling of the Hormone-Responsive Transcriptome
Top View
Supplemental Figures 04 12 2017
ATP5L CRISPR/Cas9 KO Plasmid (H): Sc-411494
Eriocitrin Ameliorates Diet-Induced Hepatic Steatosis with Activation of Mitochondrial Biogenesis
Impaired Mitochondrial Biogenesis in Adipose Tissue in Acquired Obesity
Assembly of the Membrane Domain of ATP Synthase in Human Mitochondria
Mass Spectrometry-Based Global Proteomic Analysis of Endoplasmic Reticulum and Mitochondria Contact Sites
SUPPLEMENTARY APPENDIX Post-Transplant Molecularly Defined Burkitt Lymphomas Are Frequently MYC -Negative and Characterized by the 11Q-Gain/Loss Pattern
Genomic Profiling of Primary Histiocytic Sarcoma Reveals Two Molecular
Membranes of Human Neutrophils Secretory Vesicle Membranes And
Proteomic Landscape of the Human Choroid–Retinal Pigment Epithelial Complex
Supplemental Materials Integrative Metabolomic and Transcriptomic
The MYCL and MXD1 Transcription Factors Regulate the Fitness of Murine Dendritic Cells
Data Set 1. Biological Analysis of the Genes Found to Be Significant in the Endotoxin Study
Supplementary Table 1 - Transcriptomics Analysis
Gene Symbol Probe Set Fold Change Parametric Description Map Genbank Unigene AI/AD P-Value Accession Cluster
You Can Check If Genes Are Captured by the Agilent Sureselect V5 Exome
A Thermodynamic Based Interpretation of Protein Expression Heterogeneity in Different
Distinction in Gene Expression Profiles Demonstrated in Parathyroid
ORAOV1 Is a Probable Target Within the 11Q13.3 Amplicon in Lymph Node Metastases from Gastric Adenocarcinoma
Detecting Gene Modules Differentially Expressed in Multiple Human Brain
The Genetic Evidence of Burn-Induced Cardiac Mitochondrial Metabolism Dysfunction
Mitochondria and Disease: Mutation and Expression Landscape of Mitochondri-Associated Genes in Different Disease Types
Investigating the Role of BEST1 Protein in Best Vitelliform Macular
Cancer Discovery MANUSCRIPT
Investigating the Influence of Perinatal Nicotine and Alcohol Exposure On
Mass-Spectrometry Based Proteomics Reveals Mitochondrial
Subcellular Proteomics Reveals a Role for Nucleocytoplasmic Shuttling At
Dichloroacetate Prevents Cisplatin-Induced Nephrotoxicity Without Compromising Cisplatin Anticancer Properties
Table S1. the Four TAZ Mutations Examined in This Study. Patients Were Diagnosed with Barth
In Vivo Cell-Autonomous Transcriptional Abnormalities Revealed in Mice Expressing Mutant Huntingtin in Striatal but Not Cortical Neurons
Mitochondrial-Nuclear Crosstalk, Haplotype and Copy Number