หมายเลข 9 ICD10 Coding
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Natural History of Odontogenic Infection
Natural History of Odontogenic Infection The usual cause of odontogenic infections is necrosis of the pulp of the tooth, which is followed by bacterial invasion through the pulp chamber and into the deeper tissues. Necrosis of the pulp is the result of deep caries in the tooth, to which the pulp responds with a typical inflammatory reaction. Vasodilation and edema cause pressure in the tooth and severe pain as the rigid walls of the tooth prevent swelling. If left untreated the pressure leads to strangulation of the blood supply to the tooth through the apex and consequent necrosis. The necrotic pulp then provides a perfect setting for bacterial invasion into the bone tissue. Once the bacteria have invaded the bone, the infection spreads equally in all directions until a cortical plate is encountered. During the time of intrabony spread, the patient usually experiences sufficient pain to seek treatment. Extraction of the tooth (or removal of the necrotic pulp by an endodontic procedure) results in resolution of the infection. Direction of Spread of Infection The direction of the infection's spread from the tooth apex depends on the thickness of the overlying bone and the relationship of the bone's perforation site to the muscle attachments of the jaws. If no treatment is provided for it, the infection erodes through the thinnest, nearest cortical plate of bone and into the overlying soft tissue. If the root apex is centrally located, the infection erodes through the thinnest bone first. In the maxilla the thinner bone is the labial-buccal side; the palatal cortex is thicker. -
Unlikely Case of Submasseteric Abscess Originating from a Maxillary Molar: the Skipping Lesion
Submasseteric abscess Unlikely case of submasseteric abscess originating from a maxillary molar: The skipping lesion Abstract Objective Min Jim Lima & Alauddin Muhamad Husinb We report a case of submasseteric abscess originating from a maxillary tooth, complicated by underlying diabetes mellitus and a multidrug- a Oral Maxillofacial Surgery Department, Hospital Tanah Merah, Tanah Merah, Kelantan, Malaysia resistant organism. b Oral Maxillofacial Surgery Department, Hospital Sultanah Nur Zahirah, Kuala Terengganu, Terengganu, Malaysia Materials and methods Corresponding author: A 61-year-old male patient with uncontrolled diabetes mellitus presented with swelling on the left cheek of 2 weeks in duration with rapid Dr. Min Jim Lim Oral Maxillofacial Surgery Department progression to trismus, dysphagia and rupture of swelling with pus Hospital Tanah Merah discharge. Culture and sensitivity testing revealed the presence of 17500 Tanah Merah Klebsiella pneumoniae Kelantan multidrug- resistant . Based on the patient’s history Malaysia and clinical presentation, a diagnosis of submasseteric abscess originat- [email protected] ing from the maxillary molar was made. Antibiotic administration, con- trol of systemic disease and wound dressing were done as treatment. How to cite this article: Result Lim MJ, Muhamad Husin A. Unlikely case of submasseteric abscess originating from a maxillary The patient made a full recovery, with scarring on the ruptured region. molar: The skipping lesion. J Oral Science Rehabilitation. 2018 Dec;4(4):52–55. Conclusion Submasseteric abscess is a rare case of infection that can occur in the submasseteric space. As is commonly known, infection of the submas- seteric space originates from mandibular third molars; hence, maxillary molars seem to be an unlikely source of infection. -
Deep Neck Infections 55
Deep Neck Infections 55 Behrad B. Aynehchi Gady Har-El Deep neck space infections (DNSIs) are a relatively penetrating trauma, surgical instrument trauma, spread infrequent entity in the postpenicillin era. Their occur- from superfi cial infections, necrotic malignant nodes, rence, however, poses considerable challenges in diagnosis mastoiditis with resultant Bezold abscess, and unknown and treatment and they may result in potentially serious causes (3–5). In inner cities, where intravenous drug or even fatal complications in the absence of timely rec- abuse (IVDA) is more common, there is a higher preva- ognition. The advent of antibiotics has led to a continu- lence of infections of the jugular vein and carotid sheath ing evolution in etiology, presentation, clinical course, and from contaminated needles (6–8). The emerging practice antimicrobial resistance patterns. These trends combined of “shotgunning” crack cocaine has been associated with with the complex anatomy of the head and neck under- retropharyngeal abscesses as well (9). These purulent col- score the importance of clinical suspicion and thorough lections from direct inoculation, however, seem to have a diagnostic evaluation. Proper management of a recog- more benign clinical course compared to those spreading nized DNSI begins with securing the airway. Despite recent from infl amed tissue (10). Congenital anomalies includ- advances in imaging and conservative medical manage- ing thyroglossal duct cysts and branchial cleft anomalies ment, surgical drainage remains a mainstay in the treat- must also be considered, particularly in cases where no ment in many cases. apparent source can be readily identifi ed. Regardless of the etiology, infection and infl ammation can spread through- Q1 ETIOLOGY out the various regions via arteries, veins, lymphatics, or direct extension along fascial planes. -
Methodical Complex on Gross Anatomy for Ii Course
MINISTRY OF HIGHER AND SECONDARY SPECIAL EDUCATION OF UZBEKISTAN BUKHARA STATE MEDICAL INSTITUTE NAMED AFTER ABU ALI IBN SINO DEPARTMENT OF ANATOMY "APPROVED" by Vice-Rector for Academic and educational work, Associate prof. G.J.Jarilkasinova ________________________________ "_____" ________________ 2020 Area of knowledge: 500000 - Health and social care Education field: 510000 - Healthcare Educational direction: 5510100 - Medical business 5111000 - Professional education (5510100 - Medicine business) 5510200 - Pediatric Medicine 5510300 - Medico-prophylactic business 5510400 – Dentistry (by directions) 5510900 – Medico-biological business EDUCATIONAL - METHODICAL COMPLEX ON GROSS ANATOMY FOR II COURSE Bukhara 2020 The scientific program was approved by the Resolution of the Coordination Council No. ___ of August ___, 2020 on the activities of educational and methodological associations in the areas of higher and secondary special and vocational education. The teaching and methodical complex was developed by order of the Ministry of Higher and Secondary Special Education of the Republic of Uzbekistan dated March 1, 2017 No. 107. Compilers: Radjabov A.B. - Head of the Department of Anatomy, Associate Professor Khasanova D.A. - Assistant of the Department of Anatomy, PhD Bobomurodov N.L. - Associate Professor of the Department of Anatomy Reviewers: Davronov R.D. - Head of the Department Histology and Medical biology, Associate Professor Djuraeva G.B. - Head of the Department of the Department of Pathological Anatomy and Judicial Medicine, Associate Professor The working educational program for anatomy is compiled on the basis of working educational curriculum and educational program for the areas of 5510100 - Medical business. This is discussed and approved at the department Protocol № ______ of "____" _______________2020 Head of the chair, associate professor: Radjabov A.B. -
Post-Operative Computed Tomography Scans in Severe Cervicofacial
Post-operative computed tomography scans in severe cervicofacial infections Yanga Ngcwama 9508272 Supervisor: Prof JA Morkel 1 CONTENTS Title 3 Declaration 4 Acknowledgements 5 Dedication 6 List of abbreviations 7 Key words 7 Abstract 8 List of tables 9 List of figures 10 1. Introduction 11 2. Literature review 12 3. Aims and objectives 24 4. Materials and methods 25 5. Results 28 6. Discussion 34 7. Conclusion 37 8. References 38 9. Annexures Annexure 1: Patient Information Letter 41 Annexure 2: Consent Form 42 Annexure 3: Patient Consent to Clinical Photography 43 Annexure 4: Data Capturing Sheet 44 2 TITLE Post-operative computed tomography scans in severe cervicofacial infections By Yanga Ngcwama Submitted in partial fulfillment (mini-thesis) for the Magister Chirurgiae Dentium (Maxillo-Facial and Oral Surgery) Department of Maxillo-Facial and Oral Surgery at the Faculty of Dentistry University of the Western Cape June 2015 3 DECLARATION I, Yanga Ngcwama, declare that this mini-thesis is my own work, that all sources I have quoted have been indicated and acknowledged by means of references, and that it has not been presented for any other degree at any university: Signed: Date: 08 October 2015. Department of Maxillo-Facial and Oral Surgery Faculty of Dentistry University of the Western Cape South Africa 4 ACKNOWLEDGEMENTS I wish to acknowledge my sincere gratitude to the following individuals for their assistance in this research project. (1) Professor J.A. Morkel, for going more than an extra mile in assisting his registrars with their training and their research projects. Long Live. (2) Professor G. -
Studies of the Function of the Human Pylorus : and Its Role in The
+.1 Studúes OlTlæ Ftrnctíon OJTIrc Humanfolonts And,Iß R.ole InTlæ Riegulø;tíon OÍ Cústríß Drnptging David R. Fone Departments of Medicine and Gastroenterology, Royal Adelaide Hospital University of Adelaide August 1990 Table of Contents TABLE OF CONTENTS . SUMMARY vil DECLARATION...... X DED|CAT|ON.. .. ... xt ACKNOWLEDGMENTS xil CHAPTER 1 ANATOMY OF THE PYLORUS 1.1 INTRODUCTION.. 1 1.2 MUSCULAR ANATOMY 2 1.3 MUCOSAL ANATOMY 4 1.4 NEURALANATOMY 1.4.1 Extrinsic lnnervation of the Pylorus 5 1.4.2 lntrinsic lnnervation of the Pylorus 7 1.5 INTERSTITIAL CELLS OF CAJAL 8 1.6 CONCLUSTON 9 CHAPTER 2 MEASUREMENT OF PYLORIC MOTILITY 2.1 INTRODUCTION 10 2.2 METHODOLOG ICAL CHALLENGES 2.2.1 The Anatomical Mobility of the Pylorus . 10 2.2.2 The Narrowness of the Zone of Pyloric Contraction 12 2.3 METHODS USED TO MEASURE PYLORIC MOTILITY 2.3.1 lntraluminal Techniques 2.3.1.1 Balloon Measurements. 12 t 2.3 1.2 lntraluminal Side-hole Manometry . 13 2.3 1.9 The Sleeve Sensor 14 2.3 1.4 Endoscopy. 16 2.3 1.5 Measurements of Transpyloric Flow . 16 2.3 'I .6 lmpedance Electrodes 16 2.3.2 Extraluminal Techniques For Recording Pyl;'; l'¡"r¡iit¡l 2.3.2.'t Strain Gauges . 17 2.3.2.2 lnduction Coils . 17 2.3.2.3 Electromyography 17 2.3.3 Non-lnvasive Approaches For Recording 2.3.3.1 Radiology :ï:: Y:1":'1 18 2.3.9.2 Ultrasonography . 1B 2.3.3.3 Electrogastrography 19 2.3.4 ln Vitro Studies of Pyloric Muscle 19 2.4 CONCLUSTON. -
Nbde Part 2 Decks and Remembed-Arroz Con Mango
ARROZ CON MANGO Dear friends, these are remembered/repeated questions (RQs) and answers I COPIED and PASTED from different discussions on Facebook. I feel sorry because I couldn’t organize the file the way I wanted but I hope it helps. Probably you’ll find some wrong answers in this file, but PLEASE … DO NOT CRITICIZE! Find out the right answer, learn it, share it, PASS your test and BE HAPPY J I wish you all the best GOD BLESS YOU! PAITO 1. All of the following are adverse effects of opioids except? diarrhea and somnolence 2. Advantage of osteogenesis distraction is? less relapse, large movements 3. An investigation that is not accurate but consistent is: reliability 4. Remineralized enamel is rough and cavitation? Dark hard and opaque 5. Characteristics of a child with autism - repetitive action, sensitive to light and noise 6. S,z,che sounds : Teeth barely touching – True 7. Something about bio-transformation, more polar and less lipid soluble? - True 8. How much of he population has herpes? 80% - (65-90% worldwide; 80-85% USA) More than 3.7 billion people under the age of 50 – or 67% of the population – are infected with herpes simplex virus type 1 (HSV-1), according to WHO's first global estimates of HSV-1 infection published today in the journal PLOS ONE. 9. Steps of plaque formation: pellicle, biofilm, materia alba, plaque 10. Dose of hydrocortisone taken per year that will indicate have adrenal insufficiency and need supplement dose for surgery - 20 mg 2 weeks for 2 years 11. Rpd clasp breakage due to what? Work hardening 12. -
Student to Student Guides
Harvard School of Dental Medicine Student-to-Student Guide to Clinic: How to Excel in Third Year 2010-2011 Edition Adam Donnell Mindy Gil Brandon Grunes Sharon Jin Aram Kim Michelle Mian Tracy Pogal-Sussman Kim Whippy 1999 – Blaine Langberg & Justine Tompkins 2000 – Blaine Langberg & Justine Tompkins 2001 – Blaine Langberg & Justine Tompkins 2002 – Mark Abel & David Halmos 2003 – Ketan Amin 2004 – Rishita Saraiya & Vanessa Yu 2005 – Prathima Prasanna & Amy Crystal 2006 – Seenu Susarla & Brooke Blicher 2007 – Deepak Gupta & Daniel Cassarella 2008 – Bryan Limmer & Josh Kristiansen 2009 – Byran Limmer & Josh Kristiansen 2010 – Adam Donnell, Tracy Pogal-Sussman, Kim Whippy, Mindy Gil, Sharon Jin, Brandon Grunes, Aram Kim, Michelle Mian 1 2 Foreword Dear Class of 2012, We present the 12th edition of this guide to you to assist your transition from the medical school to the HSDM clinic. You have accomplished an enormous amount thus far, but the transformation to come is beyond expectation. Third year is challenging, but fun; you‘ll look back a year from now with amazement at the material you‘ve learned, the skills you‘ve acquired, and the new language that gradually becomes second nature. To ease this process, we would like to share with you the material in this guide, starting with lessons from our own experience. Course material is the bedrock of third year. Without knowing and fully understanding prevention, disease control, and the basics of dentistry, even the most technically skilled dental student can not provide patients with successful treatment. Be on time to lectures, don‘t be afraid to ask questions, and take some time to review your notes in the evening. -
Surgical Anatomy of the Infratemporal Fossa Surgical Anatomy of the Infratemporal Fossa
Surgical Anatomy of the Infratemporal Fossa Surgical Anatomy of the Infratemporal Fossa John D.Langdon Professor and Head of Department Department of Oral and Maxillofacial Surgery King’s College London, UK Barry K.B.Berkovitz Reader in Anatomy Division of Anatomy, Cell and Human Biology King’s College London, UK Bernard J.Moxham Professor of Anatomy and Head of Teaching in Biosciences Cardiff School of Biosciences Cardiff University, UK MARTIN DUNITZ © 2003 Martin Dunitz, a member of the Taylor & Francis Group First published in the United Kingdom in 2003 by Martin Dunitz, Taylor & Francis Group plc, 11 New Fetter Lane, London EC4P 4EE Tel.: +44 (0) 20 7583 9855 Fax.: +44 (0) 20 7842 2298 E-mail: [email protected] Website: http://www.dunitz.co.uk This edition published in the Taylor & Francis e-Library, 2005. “To purchase your own copy of this or any of Taylor & Francis or Routledge’s collection of thousands of eBooks please go to www.eBookstore.tandf.co.uk.” All rights reserved. No part of this publication may be reproduced, stored in a retrieval system, or transmitted, in any form or by any means, electronic, mechanical, photocopying, recording, or otherwise, without the prior permission of the publisher or in accordance with the provisions of the Copyright, Designs and Patents Act 1988 or under the terms of any licence permitting limited copying issued by the Copyright Licensing Agency, 90 Tottenham Court Road, London W1P OLP. Although every effort has been made to ensure that all owners of copyright material have been acknowledged in this publication, we would be glad to acknowledge in subsequent reprints or editions any omissions brought to our attention. -
ODONTOGENTIC INFECTIONS Infection Spread Determinants
ODONTOGENTIC INFECTIONS The Host The Organism The Environment In a state of homeostasis, there is Peter A. Vellis, D.D.S. a balance between the three. PROGRESSION OF ODONTOGENIC Infection Spread Determinants INFECTIONS • Location, location , location 1. Source 2. Bone density 3. Muscle attachment 4. Fascial planes “The Path of Least Resistance” Odontogentic Infections Progression of Odontogenic Infections • Common occurrences • Periapical due primarily to caries • Periodontal and periodontal • Soft tissue involvement disease. – Determined by perforation of the cortical bone in relation to the muscle attachments • Odontogentic infections • Cellulitis‐ acute, painful, diffuse borders can extend to potential • fascial spaces. Abscess‐ chronic, localized pain, fluctuant, well circumscribed. INFECTIONS Severity of the Infection Classic signs and symptoms: • Dolor- Pain Complete Tumor- Swelling History Calor- Warmth – Chief Complaint Rubor- Redness – Onset Loss of function – Duration Trismus – Symptoms Difficulty in breathing, swallowing, chewing Severity of the Infection Physical Examination • Vital Signs • How the patient – Temperature‐ feels‐ Malaise systemic involvement >101 F • Previous treatment – Blood Pressure‐ mild • Self treatment elevation • Past Medical – Pulse‐ >100 History – Increased Respiratory • Review of Systems Rate‐ normal 14‐16 – Lymphadenopathy Fascial Planes/Spaces Fascial Planes/Spaces • Potential spaces for • Primary spaces infectious spread – Canine between loose – Buccal connective tissue – Submandibular – Submental -
Aetio-Pathogenesis and Clinical Pattern of Orofacial Infections
2 Aetio-Pathogenesis and Clinical Pattern of Orofacial Infections Babatunde O. Akinbami Department of Oral and Maxillofacial Surgery, University of Port Harcourt Teaching Hospital, Rivers State, Nigeria 1. Introduction Microbial induced inflammatory disease in the orofacial/head and neck region which commonly arise from odontogenic tissues, should be handled with every sense of urgency, otherwise within a short period of time, they will result in acute emergency situations.1,2 The outcome of the management of the conditions are greatly affected by the duration of the disease and extent of spread before presentation in the hospital, severity(virulence of causative organisms) of these infections as well as the presence and control of local and systemic diseases. Odontogenic tissues include 1. Hard tooth tissue 2. Periodontium 2. Predisposing factors of orofacial infections Local factors and systemic conditions that are associated with orofacial infections are listed below. Local factors Systemic factors 1. Caries, impaction, pericoronitis Human immunodeficiency virus 2. Poor oral hygiene, periodontitis Alcoholism 3. Trauma Measles, chronic malaria, tuberculosis Diabetis mellitus, hypo- and 4. Foreign body, calculi hyperthyroidism 5. Local fungal and viral infections Liver disease, renal failure, heart failure 6. Post extraction/surgery Blood dyscrasias 7. Irradiation Steroid therapy 8. Failed root canal therapy Cytotoxic drugs 9. Needle injections Excessive antibiotics, 10. Secondary infection of tumors, cyst, Malnutrition fractures 11. -
Description Concept ID Synonyms Definition
Description Concept ID Synonyms Definition Category ABNORMALITIES OF TEETH 426390 Subcategory Cementum Defect 399115 Cementum aplasia 346218 Absence or paucity of cellular cementum (seen in hypophosphatasia) Cementum hypoplasia 180000 Hypocementosis Disturbance in structure of cementum, often seen in Juvenile periodontitis Florid cemento-osseous dysplasia 958771 Familial multiple cementoma; Florid osseous dysplasia Diffuse, multifocal cementosseous dysplasia Hypercementosis (Cementation 901056 Cementation hyperplasia; Cementosis; Cementum An idiopathic, non-neoplastic condition characterized by the excessive hyperplasia) hyperplasia buildup of normal cementum (calcified tissue) on the roots of one or more teeth Hypophosphatasia 976620 Hypophosphatasia mild; Phosphoethanol-aminuria Cementum defect; Autosomal recessive hereditary disease characterized by deficiency of alkaline phosphatase Odontohypophosphatasia 976622 Hypophosphatasia in which dental findings are the predominant manifestations of the disease Pulp sclerosis 179199 Dentin sclerosis Dentinal reaction to aging OR mild irritation Subcategory Dentin Defect 515523 Dentinogenesis imperfecta (Shell Teeth) 856459 Dentin, Hereditary Opalescent; Shell Teeth Dentin Defect; Autosomal dominant genetic disorder of tooth development Dentinogenesis Imperfecta - Shield I 977473 Dentin, Hereditary Opalescent; Shell Teeth Dentin Defect; Autosomal dominant genetic disorder of tooth development Dentinogenesis Imperfecta - Shield II 976722 Dentin, Hereditary Opalescent; Shell Teeth Dentin Defect;