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Securin
Patronus Is the Elusive Plant Securin, Preventing Chromosome Separation By
Regulation of the Cell Cycle and DNA Damage-Induced Checkpoint Activation
Control of the Cell Cycle During Meiotic Maturation of Mouse Oocytes
APC/C-Cdh1-Dependent Anaphase and Telophase Progression During
Functional Interaction Between Bubr1 and Securin in an Anaphase- Promoting Complex/Cyclosomecdc20–Independent Manner
CDK Regulation of Meiosis: Lessons from S. Cerevisiae and S. Pombe
Structural Analysis of Human Cdc20 Supports Multisite Degron Recognition by APC/C
Inhibitory Factors Associated with Anaphase-Promoting Complex/Cylosome in Mitotic Checkpoint
RCS1, a Substrate of APC/C, Controls the Metaphase to Anaphase Transition
DNA Damage-Induced Mitotic Catastrophe Is Mediated by the Chk1-Dependent Mitotic Exit DNA Damage Checkpoint
Regulation of Mitotic Exit by Cell Cycle Checkpoints: Lessons from Saccharomyces Cerevisiae
Structural Analysis of Bub3 Interactions in the Mitotic Spindle Checkpoint
Bimodal Activation of Bubr1 by Bub3 Sustains Mitotic Checkpoint Signaling
Securin Promotes the Identification of Favourable Outcome in Invasive Breast Cancer
Ubiquitination of Cdc20 by the APC Occurs Through an Intramolecular Mechanism
Mitotic Cdks Control the Metaphase–Anaphase Transition and Trigger Spindle Elongation
Ribonucleic Acid Export 1 Is a Kinetochore-Associated Protein That Participates in Chromosome Alignment in Mouse Oocytes
Patronus Is the Elusive Plant Securin, Preventing Chromosome Separation by Antagonizing Separase
Top View
Cell Division and the Anaphase-Promoting Complex
The Spindle Assembly Checkpoint Functions During Early Development in Non-Chordate Embryos
The Anaphase Promoting Complex/ Cyclosome: a Machine Designed to Destroy
Bubr1 Blocks Substrate Recruitment to the APC/C in a KEN-Box-Dependent Manner
Securin Is Not Required for Cellular Viability, but Is Required for Normal Growth of Mouse Embryonic Fibroblasts Junjie Mei*‡, Xingxu Huang* and Pumin Zhang*†
HBV X Protein Targets Hbubr1, Which Induces Dysregulation of the Mitotic Checkpoint
The Mitotic Exit Network Integrates Temporal and Spatial Signals By
Mad2 Is Required for Inhibiting Securin and Cyclin B Degradation Following Spindle Depolymerisation in Meiosis I Mouse Oocytes
Human Securin Proteolysis Is Controlled by the Spindle Checkpoint and Reveals When the APC/C Switches from Activation by Cdc20 to Cdh1
Nek2a Destruction Marks APC/C Activation at the Prophase-To- Prometaphase Transition by Spindle-Checkpoint-Restricted Cdc20
Pttg1/Securin Is Required for the Branching Morphogenesis of the Mammary Gland and Suppresses Mammary Tumorigenesis
Role of Securin, Separase and Cohesins in Female Meiosis and Polar Body Formation in Drosophila
Interplay Between Phosphatases and the Anaphase-Promoting Complex/Cyclosome in Mitosis
Emi1 Regulates the Anaphase-Promoting Complex by a Different Mechanism Than Mad2 Proteins
Role of Securin, Separase and Cohesins in Female Meiosis And
Cdc20 and Securin Overexpression Predict Short-Term Breast Cancer Survival
Cyclin Dependent Kinase 1-Dependent Activation of APC/C Ubiquitin Ligase
A Prometaphase Mechanism of Securin Destruction Is Essential for Meiotic Progression in Mouse Oocytes ✉ Christopher Thomas 1,3 , Benjamin Wetherall 1, Mark D
How the Overexpressed Hpttg1 Gene Promotes Metastasis in Breast Cancer
Cell Cycle Adaptations of Embryonic Stem Cells
Snapshot: Cell-Cycle Regulators I David O