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SCYL3
A Computational Approach for Defining a Signature of Β-Cell Golgi Stress in Diabetes Mellitus
Inhibiting TRK Proteins in Clinical Cancer Therapy
Overlapping Role of SCYL1 and SCYL3 in Maintaining Motor Neuron Viability
Recount Brain Example with Data from SRP027383
Long Non-Coding RNA Landscape in Prostate Cancer Molecular Subtypes: a Feature Selection Approach
Supplementary Tables S1-S3
WO 2016/040794 Al 17 March 2016 (17.03.2016) P O P C T
Dual Specificity Phosphatases from Molecular Mechanisms to Biological Function
Page 1 Exploring the Understudied Human Kinome For
Dynamics of Dual Specificity Phosphatases and Their Interplay with Protein Kinases in Immune Signaling Yashwanth Subbannayya1,2, Sneha M
Predict AID Targeting in Non-Ig Genes Multiple Transcription Factor
Using Regulatory Genomics Data to Interpret the Function of Disease Variants and Prioritise Genes from Expression Studies[Versio
Overlapping Role of SCYL1 and SCYL3 in Maintaining Motor Neuron Viability
A Resource for Exploring the Understudied Human Kinome for Research and Therapeutic
Supplementary Information
Affymetrix Probe ID Gene Symbol 1007 S at DDR1 1494 F At
Homo Sapiens, Homo Neanderthalensis and the Denisova Specimen: New Insights on Their Evolutionary Histories Using Whole-Genome Comparisons
Tracing the Origin and Evolution of Pseudokinases Across the Tree of Life
Top View
SCYL3 (G-8): Sc-398328
The Glomerular Transcriptome and a Predicted Protein–Protein Interaction Network
Gprofiler2 -- an R Package for Gene List Functional Enrichment Analysis and Namespace Conversion Toolset G:Profiler [Version 2; Peer Review: 2 Approved]
Reproducible Copy Number Variation Patterns Among Single Circulating Tumor Cells of Lung Cancer Patients
Supplementary Materials: Small-Molecule Inhibition of Rho
1 Supplemental Table 1. Comparison Among Q-RT-PCR, ISH-TMA And
Identification and Characterization of a Novel SCYL3-NTRK1 Rearrangement in a Colorectal Cancer Patient
Genetic Variations in Evolutionary Accelerated Regions Disrupt Cognition in Schizophrenia
Identification of Potential Synthetic Lethal Genes to P53 Using a Computational Biology Approach Xiaosheng Wang1* and Richard Simon2*
Using Sequence-, Structure- and Receptor-Site Similarities To
Differential Analysis of RNA-Seq Data at the Gene Level Using the Deseq2
Section 1: Introduction
UNIVERSITY of CALIFORNIA, SAN DIEGO Accurate Prediction Of
Reduced Expression of the Kinesin-Associated Protein 3 (KIFAP3) Gene Increases Survival in Sporadic Amyotrophic Lateral Sclerosis
Supplementary Information
Verification of Immunology-Related Genetic Associations in BPD
Supplemental Material