865 Abdominal Aortic Aneurysm 503, 667 Endoleaks 668 Abdominal
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Cardiothoracic Fellowship Program
Cardiothoracic Fellowship Program Table of Contents Program Contact ............................................................................................ 3 Other contact numbers .................................................................................. 4 Introduction ........................................................................................................... 5 Goals and Objectives of Fellowship: ..................................................................... 6 Rotation Schedule: ........................................................................................ 7 Core Curriculum .................................................................................................... 8 Fellow’s Responsibilities ..................................................................................... 22 Resources ........................................................................................................... 23 Facilities ....................................................................................................... 23 Educational Program .......................................................................................... 26 Duty Hours .......................................................................................................... 29 Evaluation ........................................................................................................... 30 Table of Appendices .................................................................................... 31 Appendix A -
Screening Chest X-Ray Interpretations and Radiographic Techniques IOM GUIDELINES FIRST EDITION Iii
FIRST EDITION 2015 Screening Chest X-Ray Interpretations and Radiographic Techniques IOM GUIDELINES Global Radiology Coordination and Teleradiology Centre Migration Health Division International Organization for Migration (Manila Administrative Centre) 24th floor Citibank Tower, Paseo De Roxas 8741, Makati city 1226 Metro Manila, Philippines Email: [email protected] • [email protected] Tel: +632 230 1674 The opinions expressed in the report are those of the authors and do not necessarily reflect the views of the International Organization for Migration (IOM). The designations employed and the presentation of material throughout the report do not imply the expression of any opinion whatsoever on the part of IOM concerning the legal status of any country, territory, city or area, or of its authorities, or concerning its frontiers or boundaries. IOM is committed to the principle that humane and orderly migration benefits migrants and society. As an intergovernmental organization, IOM acts with its partners in the international community to: assist in meeting the operational challenges of migration; advance understanding of migration issues; encourage social and economic development through migration; and uphold the human dignity and well-being of migrants. Author Sifrash Meseret GELAW, MD Radiologist, MPH; Global Radiology Coordinator IOM, Manila Administrative Centre, Manila, Philippines Major Contributor Anthony MACDERMOTT, MD former Global HAP Quality Coordinator, IOM, Regional Office for Asia and the Pacific, Bangkok, Thailand Additional -
Pediatrics-EOR-Outline.Pdf
DERMATOLOGY – 15% Acne Vulgaris Inflammatory skin condition assoc. with papules & pustules involving pilosebaceous units Pathophysiology: • 4 main factors – follicular hyperkeratinization with plugging of sebaceous ducts, increased sebum production, Propionibacterium acnes overgrowth within follicles, & inflammatory response • Hormonal activation of pilosebaceous glands which may cause cyclic flares that coincide with menstruation Clinical Manifestations: • In areas with increased sebaceous glands (face, back, chest, upper arms) • Stage I: Comedones: small, inflammatory bumps from clogged pores - Open comedones (blackheads): incomplete blockage - Closed comedones (whiteheads): complete blockage • Stage II: Inflammatory: papules or pustules surrounded by inflammation • Stage III: Nodular or cystic acne: heals with scarring Differential Diagnosis: • Differentiate from rosacea which has no comedones** • Perioral dermatitis based on perioral and periorbital location • CS-induced acne lacks comedones and pustules are in same stage of development Diagnosis: • Mild: comedones, small amounts of papules &/or pustules • Moderate: comedones, larger amounts of papules &/or pustules • Severe: nodular (>5mm) or cystic Management: • Mild: topical – azelaic acid, salicylic acid, benzoyl peroxide, retinoids, Tretinoin topical (Retin A) or topical antibiotics [Clindamycin or Erythromycin with Benzoyl peroxide] • Moderate: above + oral antibiotics [Minocycline 50mg PO qd or Doxycycline 100 mg PO qd], spironolactone • Severe (refractory nodular acne): oral -
Mantke, Peitz, Surgical Ultrasound -- Index
419 Index A esophageal 218 Anorchidism 376 gallbladder 165 Aorta 364–366 A-mode imaging 97 gastric 220 abdominal aneurysm (AAA) AAA (abdominal aortic aneurysm) metastasis 142 20–21, 364, 366 20–21, 364, 366 pancreatic 149, 225 dissection 364, 366 Abdominal wall Adenofibroma, breast 263 perforation 366 abscess 300–301 Adenoma pseudoaneurysm 364 diagnostic evaluation 297 adrenal 214 Aortic rupture 20 hematoma 73, 300, 305 colorectal 231, 232 Aplasia, muscular 272 rectus sheath 297–300 duodenal papilla 229, 231 Appendicitis 1–4 hernia 300, 302–304 gallbladder 165 consequences for surgical indications for sonography 297 hepatic 54, 58, 141 treatment 2 seroma 298, 300, 305 multiple 141 sonographic criteria 1 trauma 297–300 parathyroid 213 Archiving 418 Abortion, tubal 30 renal 241 Arteriosclerosis 346, 348 Abscess thyroid 202–203 carotid artery 335, 337, 338 abdominal wall 300–301 Adenomyomatosis 8, 164, 165 plaque 337, 338, 345, 367, 370 causes 301 Adrenal glands 214–216 Arteriovenous (AV) malformation amebic 138 adenoma 214 139, 293, 326–329 breast 264 carcinoma 214 Artery chest wall 173, 178 cyst 214 carotid 334–339 diverticular 120, 123 hematoma 214 aneurysm 338 drainage 85–88, 93 hemorrhage 214 arteriosclerosis 335 hepatic 6, 138, 398 hyperplasia 214 plaque characteristics inflammatory bowel disease limpoma/myelipoma 214 337, 338, 345 116, 119 metastases 214 bifurcation 334, 337 intramural 5 sonographic criteria 214 bulb 339 lung 183, 186, 190 tuberculosis 214 dissection 338, 339, 346 pancreatic 11 Advanced dynamic flow (ADF) sonographic -
İstanbul University Cerrahpaşa Medical Faculty 2015-2016 Academic Year Synopsis of Curricula
İstanbul University Cerrahpaşa Medical Faculty 2015-2016 Academic Year Synopsis of Curricula For details visit: http://www.ctf.edu.tr/egitim_ogretim/indexen.htm Istanbul University, Cerrahpaşa Medical Faculty 2015-2016 Academic Year Synopsis of Curricula; Version 14-Jul-15; Page 1/125 Table of Contents 1st Year Courses .................................................................................................................................. 3 Course 1.1: Introduction to Medical Sciences ..................................................................................... 3 Course 1.2: Cell Tissue and Organ Systems I ...................................................................................... 8 Course 1.3: Cell Tissue and Organ Systems II .................................................................................. 12 Course 1.4: Introduction to Clinical Medicine ................................................................................... 19 2nd Year Courses ............................................................................................................................... 25 Course 2.1: Locomotor System .......................................................................................................... 25 Course 2.2: Cardiovascular System ................................................................................................... 28 Course 2.3: Respiratory System ......................................................................................................... 32 Course 2.4: -
Clinical Excellence Series Volume VI an Evidence-Based Approach to Infectious Disease
Clinical Excellence Series n Volume VI An Evidence-Based Approach To Infectious Disease Inside The Young Febrile Child: Evidence-Based Diagnostic And Therapeutic Strategies Pharyngitis In The ED: Diagnostic Challenges And Management Dilemmas HIV-Related Illnesses: The Challenge Of Emergency Department Management Antibiotics In The ED: How To Avoid The Common Mistake Of Treating Not Wisely, But Too Well Brought to you exclusively by the publisher of: An Evidence-Based Approach To Infectious Disease CEO: Robert Williford President & Publisher: Stephanie Ivy Associate Editor & CME Director: Jennifer Pai • Associate Editor: Dorothy Whisenhunt Director of Member Services: Liz Alvarez • Marketing & Customer Service Coordinator: Robin Williford Direct all questions to EB Medicine: 1-800-249-5770 • Fax: 1-770-500-1316 • Non-U.S. subscribers, call: 1-678-366-7933 EB Medicine • 5550 Triangle Pkwy Ste 150 • Norcross, GA 30092 E-mail: [email protected] • Web Site: www.ebmedicine.net The Emergency Medicine Practice Clinical Excellence Series, Volume Volume VI: An Evidence-Based Approach To Infectious Disease is published by EB Practice, LLC, d.b.a. EB Medicine, 5550 Triangle Pkwy Ste 150, Norcross, GA 30092. Opinions expressed are not necessarily those of this publication. Mention of products or services does not constitute endorsement. This publication is intended as a general guide and is intended to supplement, rather than substitute, professional judgment. It covers a highly technical and complex subject and should not be used for making specific medical decisions. The materials contained herein are not intended to establish policy, procedure, or standard of care. Emergency Medicine Practice, The Emergency Medicine Practice Clinical Excellence Series, and An Evidence-Based Approach To Infectious Disease are trademarks of EB Practice, LLC, d.b.a. -
CHEST RADIOLOGY: Goals and Objectives
Harlem Hospital Center Department of Radiology Residency Training Program CHEST RADIOLOGY: Goals and Objectives ROTATION 1 (Radiology Years 1): Resident responsibilities: • ED chest CTs • Inpatient and outpatient plain films including the portable intensive care unit radiographs • Consultations with referring clinicians MEDICAL KNOWLEDGE: • Residents must demonstrate knowledge about established and evolving biomedical, clinical, and cognitive sciences and the application of this knowledge to patient care. At the end of the rotation, the resident should be able to: • Identify normal radiographic and CT anatomy of the chest • Identify and describe common variants of normal, including aging changes. • Demonstrate a basic knowledge of radiographic interpretation of atelectasis, pulmonary infection, congestive heart failure, pleural effusion and common neoplastic diseases of the chest • Identify the common radiologic manifestation of thoracic trauma, including widened mediastinum, signs of aortic laceration, pulmonary contusion/laceration, esophageal and diaphragmatic rupture. • Know the expected postoperative appearance in patients s/p thoracic surgery and the expected location of the life support and monitoring devices on chest radiographs of critically ill patients (intensive care radiology); be able to recognize malpositioned devices. • Identify cardiac enlargement and know the radiographic appearance of the dilated right vs. left atria and right vs. left ventricles, and pulmonary vascular congestion • Recognize common life-threatening -
CT Signs in the Lungs Girish S
CT Signs in the Lungs Girish S. Shroff, MD,* Edith M. Marom, MD,† Myrna C.B. Godoy, MD, PhD,* Mylene T. Truong, MD,* and Caroline Chiles, MDz Radiologic signs are often based on items or patterns that are encountered in everyday life. They are especially useful because their observation allows the differential diagnosis to be narrowed, and in some cases, enables a diagnosis to be made. In this review, several clas- sic and newer computed tomography signs in the lungs are discussed. Semin Ultrasound CT MRI 40:265-274 © 2018 Elsevier Inc. All rights reserved. Introduction should be considered when the pipe cleaner sign is seen (Fig. 2). Nodule distribution varies slightly among the con- adiologic signs are often based on items or patterns that ditions—in sarcoidosis, nodules tend to predominate along R are encountered in everyday life. They are especially use- larger bronchovascular bundles and in the subpleural ful because their observation allows the differential diagnosis regions whereas in silicosis and coal worker’s pneumoconi- to be narrowed, and in some cases, enables a diagnosis to be osis, nodules tend to predominate in the centrilobular and made. Furthermore, early recognition of signs associated subpleural regions.2 Smooth or nodular interlobular septal with aggressive infections may be life-saving. In this review, thickening is usually the dominant feature in lymphangitic the following computed tomography (CT) signs in the lungs carcinomatosis. Interlobular septal thickening is typically will be discussed: pipe cleaner, halo, reversed halo, air cres- absent in granulomatous diseases such as sarcoidosis and cent, Monod, Cheerio, straight edge, air bronchogram, tree- silicosis. -
Pneumonia (CAP)
肺實質化病變與肺塌陷 胸腔內科周百謙醫師 Dr. Pai-chien Chou MD PhD Department of Thoracic Medicine Taipei Medical University Hospital Chest X-ray • P-A view • Lateral view • Oblique view • Lordotic view • Expiratory film • Decubitus view • Overpenetrated grid film The Elements of a chest x-ray (CXR) • The Broncho-vascular markings in the lung • The borders of the heart • The contours of the mediastinum and pleural space • The ribs and spine Segmental anatomy Segmental Anatomy Cardiomediastinal outlines on Chest X-ray Density of image ◆ Gas ◆ Water ◆ Fat ◆ Metal and bone ◆ Thinking of pathogenesis Basic thinking of a lesion on Chest X-ray ◆ Size ◆ Location (Silhouette sign) – Anterior, posterior – Which lobe is involved ◆ Intrapulmonary (Air bronchogram sign) ◆ Extrapulmonary (Incomplete border sign) Infiltrate in the lungs • Fluid accumulates in lung, predominate in the alveolar (airspace) compartment or the interstitial compartment. interstitial compartment Lymphatic compartment Alveolar unit Vascular unit Air space opacification The opacification is caused by fluid or solid material within the airways that causes a difference in the relative attenuation of the lung: • transudate, e.g. pulmonary edema secondary to heart failure • pus, e.g. bacterial pneumonia • blood, e.g. pulmonary hemorrhage • cells, e.g. bronchoalveolar carcinoma • protein, e.g. alveolar proteinosis • fat, e.g. lipoid pneumonia • gastric contents, e.g. aspiration pneumonia • water, e.g. drowning When considering the likely causes of airspace opacification, it is useful to determine chronicity -
Signs in Chest Imaging
Diagn Interv Radiol 2011; 17:18–29 CHEST IMAGING © Turkish Society of Radiology 2011 PICTORIAL ESSAY Signs in chest imaging Oktay Algın, Gökhan Gökalp, Uğur Topal ABSTRACT adiological practice includes classification of illnesses with similar A radiological sign can sometimes resemble a particular object characteristics through recognizable signs. Knowledge of and abil- or pattern and is often highly suggestive of a group of similar pathologies. Awareness of such similarities can shorten the dif- R ity to recognize these signs can aid the physician in shortening ferential diagnosis list. Many such signs have been described the differential diagnosis list and deciding on the ultimate diagnosis for for X-ray and computed tomography (CT) images. In this ar- ticle, we present the most frequently encountered plain film a patient. In this report, 23 important and frequently seen radiological and CT signs in chest imaging. These signs include for plain signs are presented and described using chest X-rays, computed tomog- films the air bronchogram sign, silhouette sign, deep sulcus raphy (CT) images, illustrations and photographs. sign, Continuous diaphragm sign, air crescent (“meniscus”) sign, Golden S sign, cervicothoracic sign, Luftsichel sign, scim- itar sign, doughnut sign, Hampton hump sign, Westermark Plain films sign, and juxtaphrenic peak sign, and for CT the gloved finger Air bronchogram sign sign, CT halo sign, signet ring sign, comet tail sign, CT an- giogram sign, crazy paving pattern, tree-in-bud sign, feeding Bronchi, which are not normally seen, become visible as a result of vessel sign, split pleura sign, and reversed halo sign. opacification of the lung parenchyma. -
Common Pediatric Pulmonary Issues
Common Pediatric Pulmonary Issues Chris Woleben MD, FAAP Associate Dean, Student Affairs VCU School of Medicine Assistant Professor, Emergency Medicine and Pediatrics Objectives • Learn common causes of upper and lower airway disease in the pediatric population • Learn basic management skills for common pediatric pulmonary problems Upper Airway Disease • Extrathoracic structures • Pharynx, larynx, trachea • Stridor • Externally audible sound produced by turbulent flow through narrowed airway • Signifies partial airway obstruction • May be acute or chronic Remember Physics? Poiseuille’s Law Acute Stridor • Febrile • Laryngotracheitis (croup) • Retropharyngeal abscess • Epiglottitis • Bacterial tracheitis • Afebrile • Foreign body • Caustic or thermal airway injury • Angioedema Croup - Epidemiology • Usually 6 to 36 months old • Males > Females (3:2) • Fall / Winter predilection • Common causes: • Parainfluenza • RSV • Adenovirus • Influenza Croup - Pathophysiology • Begins with URI symptoms and fever • Infection spreads from nasopharynx to larynx and trachea • Subglottic mucosal swelling and secretions lead to narrowed airway • Development of barky, “seal-like” cough with inspiratory stridor • Symptoms worse at night Croup - Management • Keep child as calm as possible, usually sitting in parent’s lap • Humidified saline via nebulizer • Steroids (Dexamethasone 0.6 mg/kg) • Oral and IM route both acceptable • Racemic Epinephrine • <10kg: 0.25 mg via nebulizer • >10kg: 0.5 mg via nebulizer Croup – Management • Must observe for 4 hours after -
Chronic Cough- Whoop It
3/3/2016 Chronic Cough- Whoop it Cassaundra Hefner PULMONARY ANATOMY DNP, FNP-BC FryeCare Lung Center Upper Airway Nasopharynx Oropharynx Laryngopharynx Lower Larynx Trachea Bronchi Bronchopulmonary segments Terminal bronchioles Acinus (alveolar regions) Upper and Lower Airway are lined with cilia which propel mucus and trapped bacteria toward the oropharynx Cough COUGH ACTION Protective reflex that keeps throat clear allowing for mucocilliary clearance of airway secretion Intrathoracic process of air from a vigorous cough through nearly closed vocal cords can approach 300mmHG, the velocities tear off mucus from the airway walls. The velocity can be up to 500mph 4 Cough/Sputum Defense mechanism to prevent aspiration- cough center stimulated- cough begins with deep inspiration to 50 % vital capacity- maximum expiratory flow increases coil - decreasing airway resistance- glottis opens wide and takes in large amount of air - glottis then rapidly closes - abdominal and intercostal muscles contract- increases intrapleural pressure - the glottis reopens- explosive release of air the tracheobronchial tree narrows rips the mucous off the walls = sputum 1 3/3/2016 Chronic Cough Defined (AACP, 2016) Effects of cough that prompts visit Talierco & Umur, 2014 Acute Sub-acute Chronic Fatigue 57% Cough Cough 3-8 Unexplained chronic less than weeks cough(UCC) Insomnia 45% 3 weeks Excessive perspiration 42% Cough lasting greater Incontinence 39% than 8 weeks in 15 yo or older MSK pain 45% Cough lasting greater Inguinal herniation than 4 weeks in Dysrhythmias those under the Headaches age of 15 Quality of life questionnaires are recommended for adolescents and children (CQLQ) Work loss Data Institute (NCG) (2016) Cough Referral to Pulmonology 80%-90% chronic cough Most common symptom for PCP visits in the U.S.