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IMPACT FACTOR 5.923 an Open Access Journal by MDPI Neurobiology of Aging and Aging-Related Disorders Guest Editors: Message from the Guest Editors Dr. Livia D’Angelo Brain ageing is a complex process of all vertebrate Department of Veterinary organisms and is influenced by systemic, genetic, and Medicine and Animal environmental factors. In course of ageing, the brain Productions, University of Naples Federico II, Naples, Italy undergoes general morpho-physiological changes that [email protected] include shrinkage of brain mass and myelin, decline of synaptic connections, and decreased neurotransmitter Dr. Paolo de Girolamo release. These modifications are associated with cognitive Department of Veterinary decline in information processing speed, memory, Medicine, University of Naples reasoning, and executive function. Furthermore, ageing is Federico II, 80137 Naples, Italy one of the major contributing factors for [email protected] neurodegenerations and other brain diseases. Much of Prof. Dr. María Trinidad what we know about the impact of ageing on brain comes Herrero from model organisms, which have been instrumental to Faculty of Medicine, Universidad identify factors that cause and modulate the ageing de Murcia, Murcia, Spain process. In this collection, we aim to address various [email protected] aspects of the neurobiology of ageing and ageing related disorders, including a) new experimental approaches to modulate the ageing process with consequences on brain Deadline for manuscript morpho-physiology; b) the identification of genetic and submissions: environmental factors related to brain ageing; and c) new 28 February 2022 imaging techniques and bioinformatics approaches to detect changes in the brain during ageing. mdpi.com/si/73796 SpeciaIslsue IMPACT FACTOR 5.923 an Open Access Journal by MDPI Editor-in-Chief Message from the Editor-in-Chief Prof. Dr. Maurizio Battino The International Journal of Molecular Sciences (IJMS, 1. Department of ISSN 1422-0067) is an open access journal, which was Odontostomatologic and established in 2000. The journal aims to provide a forum Specialized Clinical Sciences, Sez-Biochimica, Faculty of for scholarly research on a range of topics, including Medicine, Università Politecnica biochemistry, molecular and cell biology, molecular delle Marche, Via Ranieri 65, biophysics, molecular medicine, and all aspects of 60100 Ancona, Italy molecular research in chemistry. IJMS publishes both 2. International Research Center for Food Nutrition and Safety, original research and review articles, and regularly Jiangsu University, Zhenjiang publishes special issues to highlight advances at the 212013, China cutting edge of research. We invite you to read recent articles published in IJMS and consider publishing your next paper with us. Author Benefits Open Access:— free for readers, with article processing charges (APC) paid by authors or their institutions. High Visibility: indexed within Scopus, SCIE (Web of Science), PubMed, PMC, MEDLINE, Embase, CAPlus / SciFinder, and many other databases. Journal Rank: JCR - Q1 (Biochemistry & Molecular Biology) / CiteScore - Q1 (Inorganic Chemistry) Contact Us International Journal of Molecular Tel: +41 61 683 77 34 mdpi.com/journal/ijms Sciences Fax: +41 61 302 89 18 [email protected] MDPI, St. Alban-Anlage 66 www.mdpi.com @IJMS_MDPI 4052 Basel, Switzerland.

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REWARD LIST ‐ RANK C JCR 2018 ‐CiteScore 2018 WOS Edition/ Scopus Main Rank Subject Category Journal Title ISSN Subject Area code IEEE TRANSACTIONS ON ULTRASONICS SCIE ACOUSTICS 08853010 C FERROELECTRICS AND FREQUENCY SCIE ACOUSTICS JOURNAL OF VIBRATION AND CONTROL 10775463 C SCIE AGRICULTURAL ECONOMICS & POLICY JOURNAL OF AGRICULTURAL ECONOMICS 0021857X C SCIE AGRICULTURAL ENGINEERING BIOMASS & BIOENERGY 09619534 C SCIE AGRICULTURE, DAIRY & ANIMAL SCIENCE ANIMAL REPRODUCTION SCIENCE 03784320 C SCIE AGRICULTURE, DAIRY & ANIMAL SCIENCE APPLIED ANIMAL BEHAVIOUR SCIENCE 01681591 C SCIE AGRICULTURE, DAIRY & ANIMAL SCIENCE ARCHIVES OF ANIMAL NUTRITION 1745039X C JOURNAL OF ANIMAL PHYSIOLOGY AND SCIE AGRICULTURE, DAIRY & ANIMAL SCIENCE 09312439 C ANIMAL NUTRITION SCIE AGRICULTURE, DAIRY & ANIMAL SCIENCE Animals 20762615 C SCIE AGRICULTURE, MULTIDISCIPLINARY Journal of Land Use Science 1747423X C RENEWABLE AGRICULTURE AND FOOD SCIE AGRICULTURE, MULTIDISCIPLINARY 17421705 C SYSTEMS SCIE AGRICULTURE, MULTIDISCIPLINARY ANNALS OF APPLIED BIOLOGY 00034746 C SCIE AGRICULTURE, MULTIDISCIPLINARY CALIFORNIA AGRICULTURE 00080845 C SCIE AGRONOMY PLANT PATHOLOGY 00320862 C SCIE AGRONOMY IRRIGATION SCIENCE 03427188 C SCIE AGRONOMY Rice Science 16726308 C SCIE AGRONOMY Agronomy‐Basel 20734395 C SCIE AGRONOMY CROP PROTECTION 02612194 C SCIE AGRONOMY EXPERIMENTAL AGRICULTURE 00144797 C JOURNAL OF PLANT NUTRITION AND SOIL SCIE AGRONOMY 14368730 C SCIENCE SCIE ALLERGY CONTACT DERMATITIS 01051873 C SCIE ALLERGY Allergy Asthma & Immunology Research 20927355 C Advances
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Jennifer I. Luebke 1 Curriculum Vitae Jennifer I. Luebke, Ph.D. Associate
Jennifer I. Luebke Curriculum Vitae Jennifer I. Luebke, Ph.D. Associate Professor of Anatomy & Neurobiology Associate Professor of Psychiatry Director, Laboratory of Cellular Neurobiology Boston University School of Medicine 85 East Newton Street, M949 Boston, Massachusetts 02118 Voice: 617-638-4930 Email: [email protected] Education and Employment History: 1980-1984: B.S. (Biology) Randolph-Macon College, Ashland, Virginia 1984-1986: Laboratory Technician (Laboratory of Cell Biology and Genetics, NIDDK) National Institutes of Health, Bethesda, Maryland 1986-1990: Ph.D. Student (Anatomy & Neurobiology), Boston University School of Medicine, Boston, Massachusetts (Linda L. Wright, mentor) 1990-1992: Postdoctoral Fellow, Department of Psychiatry, Harvard Medical School, Boston, Massachusetts (Robert W. McCarley and Robert W. Greene mentors) 1992-1995: Postdoctoral Fellow, Department of Physiology, Tufts University Medical School, Boston, Massachusetts (Kathleen Dunlap, mentor) 1995-2003: Assistant Professor, Department of Anatomy & Neurobiology and Department of Psychiatry, Boston University School of Medicine, Boston, Massachusetts 2004-Present: Associate Professor, Department of Anatomy & Neurobiology and Department of Psychiatry, Boston University School of Medicine, Boston, Massachusetts 2010-Present: Adjunct Associate Professor, Department of Neuroscience, Mount Sinai School of Medicine, New York, New York Research Summary: Research is directed toward understanding alterations in the structure and function of individual cortical pyramidal cells in a rhesus monkey model of normal aging and in transgenic mouse models of neurodegenerative disease. Using whole-cell patch-clamp methods and ultra-high resolution confocal microscopy, Dr. Luebke has demonstrated marked alterations in action potential firing patterns (and underlying ionic currents), glutamatergic and GABAergic synaptic response properties and detailed dendritic and spine architecture in cortical pyramidal cells both in normal aging and in mouse models of neurodegenerative disease.
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Predicting Cognitive Decline: Genetic, Environmental and Lifestyle Risk Factors
Predicting Cognitive Decline: Genetic, Environmental and Lifestyle Risk Factors Shea J. Andrews April 2017 A thesis by compilation submitted for the degree of Doctor of Philosophy of The Australian National University © Copyright by Shea John Frederick Andrews 2017 All Rights Reserved i Declaration This work was conducted from February 2013 to August 2016 at the Genome Diversity and Health Group, John Curtin School of Medical Research, The Australian National University, Canberra, ACT. This thesis by compilation consists of five original publications describing the analyses I have performed during my candidature investigating the role of genetic, environmental and lifestyle risk factors in normal cognitive aging. All five publications have been published in Q1 ranked journals according to the SCImago Journal & Country Rankings in the fields of neurology, genetics, psychiatry and mental health, and geriatric and gerontology. My specific contribution to each manuscript is detailed in the subsequent pages in the form of a statement signed by the senior author of each publication. This document has not been submitted for qualifications at any other academic institution. Shea Andrews Canberra, Australia April 2017 ii Published Papers Andrews, SJ, Das, D., Anstey, KJ., Easteal, S. (2015). Interactive effect of APOE genotype and blood pressure on cognitive decline: The PATH through life project. Journal of Alzheimer's Disease. 44(4): 1087-98. DOI: 10.3233/JAD- 140630 For this publication, I designed and performed all statistical analyses and wrote the manuscript. A slightly modified version of this paper is presented in Chapter 3. ____________________ Simon Easteal Senior Author April 2017 Andrews SJ, Das D, Cherbuin N, Anstey KJ, Easteal S.
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Brain Ageing in the New Millennium
Brain ageing in the new millennium Julian N. Trollor, Michael J. Valenzuela Objective: This paper examines the current literature pertaining to brain ageing. The objective of this review is to provide an overview of the effects of ageing on brain structure and function and to examine possible mediators of these changes. Methods: A MEDLINE search was conducted for each area of interest. A selective review was undertaken of relevant articles. Results: Although fundamental changes in fluid intellectual abilities occur with age, global cognitive decline is not a hallmark of the ageing process. Decline in fluid intellectual ability is paralleled by regionally specific age related changes apparent from both structural and functional neuroimaging studies. The histopathological mediators of these changes do not appear to be reduction in neuronal number, which, with the exception of selected hippo- campal regions, remain relatively stable across age. At the molecular level, several mech- anisms of age related change have been postulated. Such theoretical models await refinement and may eventually provide a basis for therapy designed to reduce effects of the ageing process. The role of possible protective factors such as ‘brain reserve’, neuro- protective agents and hormonal factors in modifying individual vulnerability to the ageing process has been the focus of a limited number of studies. Conclusion: Our understanding of the functional and structural changes associated with both healthy and pathological ageing is rapidly gaining in sophistication and complexity. An awareness of the fundamental biological substrates underpinning the ageing process will allow improved insights into vulnerability to neuropsychiatric disease associated with advancing age. Key words: brain ageing, cognition, dementia, neuroimaging.
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The Effects of Normal Aging on Myelin and Nerve Fibers: a Review∗
Journal of Neurocytology 31, 581–593 (2002) The effects of normal aging on myelin and nerve fibers: A review∗ ALAN PETERS Department of Anatomy and Neurobiology, Boston University School of Medicine, 715, Albany Street, Boston, MA 02118, USA [email protected] Received 7 January 2003; revised 4 March 2003; accepted 5 March 2003 Abstract It was believed that the cause of the cognitive decline exhibited by human and non-human primates during normal aging was a loss of cortical neurons. It is now known that significant numbers of cortical neurons are not lost and other bases for the cognitive decline have been sought. One contributing factor may be changes in nerve fibers. With age some myelin sheaths exhibit degenerative changes, such as the formation of splits containing electron dense cytoplasm, and the formation on myelin balloons. It is suggested that such degenerative changes lead to cognitive decline because they cause changes in conduction velocity, resulting in a disruption of the normal timing in neuronal circuits. Yet as degeneration occurs, other changes, such as the formation of redundant myelin and increasing thickness suggest of sheaths, suggest some myelin formation is continuing during aging. Another indication of this is that oligodendrocytes increase in number with age. In addition to the myelin changes, stereological studies have shown a loss of nerve fibers from the white matter of the cerebral hemispheres of humans, while other studies have shown a loss of nerve fibers from the optic nerves and anterior commissure in monkeys. It is likely that such nerve fiber loss also contributes to cognitive decline, because of the consequent decrease in connections between neurons.
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Technology and Aging in America (Part 14 Of
Appendix B The Cell Biology of Aging Introduction spans that seem to be “programmed” into them accord- ing to species. Mechanisms underlying this programmed The distinction between the effects of aging as a aging could involve an intrinsic genetic component of chronological process and diseases whose prevalence the tissues (24), and/or an extrinsic “neuroendocrine increase with advancing age remains blurred. Condi- pacemaker” (16,17). tions once thought to be inevitable companions of old The genetic version of the biological clock is bol- age are now known to in fact be pathological in origin stered by the observation that the survival time of and thus possibly treatable and preventable. One ex- tissues grafted (or transferred) from an older animal ample is senile dementia, no longer the mysterious fate into a younger one of the same species seems to de- of the very old, but often a clinical manifestation of pend on the age of the donor and is not influenced arteriosclerotic or biochemical brain disease. by the “younger” environment into which it has been Further research may elucidate the mechanisms introduced (24). Similarly, when nuclei from younger underlying aging and their possible connection to age- cells are transplanted into the cytoplasm of older cells, related diseases, leading to the development of medi- the recipients then live longer and regain the ability cal techniques that delay the onset and progression to replicate. These observations all suggest that a of symptoms. While it may not lead to actual exten- “clock” controlling aging does exist and may be located sion of the human life span, such knowledge could re- in the nucleus, possibly in the genetic material (24).
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Integrated Transcriptomic and Neuroimaging Brain Model Decodes
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NeuroRegulation The Official Journal of . Volume 8, Number 1, 2021 NeuroRegulation Editor-in-Chief Rex L. Cannon, PhD: SPESA Research Institute, Knoxville, TN, USA Executive Editor Nancy L. Wigton, PhD: 1) Grand Canyon University, Phoenix, AZ, USA; 2) Applied Neurotherapy Center, Tempe, AZ, USA Associate Editors Scott L. Decker, PhD: University of South Carolina, Department of Psychology, Columbia, SC, USA Jon A. Frederick, PhD: Lamar University, Beaumont, TX, USA Genomary Krigbaum, PsyD: University of Wyoming, Family Medicine Residency, Casper, WY, USA Randall Lyle, PhD: Mount Mercy University, Cedar Rapids, IA, USA Tanya Morosoli, MSc: 1) Clínica de Neuropsicología Diagnóstica y Terapéutica, Mexico City, Mexico; 2) ECPE, Harvard T. H. Chan School oF Public Health, Boston, MA, USA Sarah Prinsloo, PhD: MD Anderson Cancer Center, Houston, TX, USA Deborah Simkin, MD: 1) Emory University School of Medicine, Department of Psychiatry, Atlanta, GA, USA; 2) Attention, Memory, and Cognition Center, Destin, FL, USA Estate M. Sokhadze, PhD: University of South Carolina, School of Medicine Greenville, Greenville, SC, USA Larry C. Stevens, PhD: Northern Arizona University, Department of Psychological Services, Flagstaff, AZ, USA Tanju Surmeli, MD: Living Health Center for Research and Education, Sisli, Istanbul, Turkey Production Editor Jacqueline Luk Paredes, Phoenix, AZ, USA NeuroRegulation (ISSN: 2373-0587) is published quarterly by the International Society for Neurofeedback and Research (ISNR), 13876 SW 56th Street, PMB 311, Miami, FL 33175-6021, USA. Copyright NeuroRegulation is open access with no submission fees or APC (Author Processing Charges). This journal provides immediate open access to its content on the principle that making research freely available to the public supports a greater global exchange of knowledge.
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2020-21 Student Handbook for Pharmacology and Neuroscience
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Listado De Revistas Extranjeras Homologadas 2019
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Dr. F. Gonzalez-Lima CV Updated Jan. 10, 2017
(Updated January 10, 2017) CURRICULUM VITAE Francisco Gonzalez-Lima 1. POSITION George I. Sanchez Centennial Professorship in Liberal Arts and Sciences Professor, Behavioral Neuroscience Area, Department of Psychology, College of Liberal Arts Professor, Division of Pharmacology and Toxicology, College of Pharmacy Professor, Institute for Neuroscience, College of Natural Sciences, Graduate Studies Program The University of Texas at Austin 2. ADDRESS The University of Texas at Austin, 108 E. Dean Keeton Stop A8000, Austin, TX 78712-1043, USA. Home: 5739 Merrywing Circle, Austin, TX 78730-1411, USA. Phone: Cell (512) 202-7375, Office (512) 475-8497, Fax (512) 910-8247. Email: [email protected] Webpage: http://www.utexas.edu/cola/depts/psychology/faculty/fg 3. EDUCATION B.S., Biology, Tulane University, New Orleans, Louisiana, USA, 1976. B.A., Psychology, Tulane University, New Orleans, Louisiana, USA, 1977. Ph.D., Anatomy and Neurobiology, Biomedical Sciences Graduate Program, School of Medicine, University of Puerto Rico Medical Sciences Campus, San Juan, Puerto Rico, USA, 1980. Postdoctoral Research Fellow, Behavioral Neuroscience, Alexander von Humboldt Foundation, Technische Universität Darmstadt, Germany, 1983. 4. EXPERIENCE 4.1. Appointments Assistant Professor 1980-83, Associate Professor 1983-85, Department of Anatomy, Ponce School of Medicine, Ponce, Puerto Rico, USA. Assistant Professor 1986-90, Department of Anatomy and Neurobiology, College of Medicine, Texas A&M University, College Station, Texas, USA. Associate Professor 1991-96, Professor 1997-present, Head 1997-2001, Behavioral Neuroscience Area, George I. Sanchez Centennial Professorship in Liberal Arts and Sciences 2000-present, Director 2002-2011, Texas Consortium in Behavioral Neuroscience, Departments of Psychology, Pharmacology and Toxicology, Institute for Neuroscience, Imaging Research Center and Center for Behavioral Neuroendocrinology, The University of Texas at Austin, Texas, USA.
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