Rare Coexistence of Sialolithiasis and Actinomycosis in the Submandibular Gland

Total Page:16

File Type:pdf, Size:1020Kb

Rare Coexistence of Sialolithiasis and Actinomycosis in the Submandibular Gland Case Report ENT Updates 2016;6(3):148–151 doi:10.2399/jmu.2016003010 Rare coexistence of sialolithiasis and actinomycosis in the submandibular gland O¤uzhan Dikici1, Nuray Bayar Muluk2 1Department of Otorhinolaryngology, fievket Y›lmaz Training and Research Hospital, Bursa, Turkey 2Department of Otorhinolaryngology, Faculty of Medicine, K›r›kkale University, K›r›kkale, Turkey Abstract Özet: Submandibüler bezde siyalolityaz ve aktinomikozun seyrek görülen birlikteli¤i Sialolithiasis is a condition characterized by the obstruction of salivary Siyalolityaz, tükürük bezi veya boflalt›m kanal›n›n bir tafl veya siyalolit gland or its excretory duct by a calculus or sialolith. This condition pro- ile t›kanmas› ile karakterizedir. Bu durum etkilenmifl bezin fliflmesi, a¤- vokes swelling, pain, and infection of affected gland leading to salivary ecta- r›mas› ve enfeksiyonunu teflvik ederek tükürük bezi ektazisine, hatta da- sia and even causing the subsequent dilatation of the salivary gland. The ha sonra tükürük bezinin dilatasyonuna neden olmaktad›r. Bu olgu ra- aim of this case report is to present a rare condition of sialolithiasis of the porunun amac› submandibüler bezde siyalolitle birlikte aktinomikozun submandibular gland with actinomycosis. In this report, we presented a 35- görüldü¤ü nadir bir siyalolityaz olgusunu sunmakt›r. Bu raporda sub- year-old male patient having coexistence of submandibular sialolithiasis mandibüler siyalolit ve aktinomikozu olan 35 yafl›nda erkek hasta litera- and actinomycosis with a literature review. Patient underwent excision of tür taramas› eflli¤inde raporlanm›flt›r. Siyalolityaz nedeniyle sa¤ sub- the right submandibular gland due to siaololithiasis. Pathologic examina- mandibüler bez eksize edilmifltir. Patolojik incelemede gözlemlenen tion revealed chronic sialadenitis, sialolithiasis, actinomyces which all kronik siyaladenit, siyalolityaz ve aktinomiçes nedeniyle çap› 1.5 cm tafl- necessitate the excision of right submandibular gland with stones with 1.5 larla dolu sa¤ submandibüler bezin eksizyonunun gerekti¤i görülmüfl- cm in diameter. It should be keep in mind that sialolithiasis may be a pre- tür. Siyalolityaz›n submandibüler aktinomikozun predispozan faktörü disposing factor for submandibular actinomycosis and removal of the olabildi¤i, siyalolit veya bezin tümüyle ç›kart›lmas›n›n son derece öne- sialolith or the entire gland is of particular importance. mi oldu¤u ak›ldan ç›kart›lmamal›d›r. Keywords: Submandibular, sialolithiasis, actinomycosis. Anahtar sözcükler: Submandibüler, siyalolityaz, aktinomikoz. Actinomycosis is a chronic suppurative infection. This invasive when a mucosal lesion gains access to the subcu- infection is characterized by formation of multiple taneous tissue.[2] abscesses, draining sinuses, and granulation tissue. The Sialolithiasis usually appears between the age of 30 and three major clinical presentations of actinomycosis include [3] 60 years. It most commonly involves the submandibular the cervicofacial region, pulmothoracic region, and [1] gland (80% to 95%) and less frequently the parotid (5% to abdominopelvic regions. In cervicofacial actinomycosis, 20%). The sublingual gland and the minor salivary glands the submandibular area, parotid gland and buccal space [4] are affected. Primary involvement of the submandibular rarely (1% to 2%) have sialolithiasis. The stones may reach [5] gland is not frequent. Actinomycosis with sialolithiasis of from 0.1 to 30 mm. While 85% of submandibular gland the submandibular gland is a rare situation.[1] Actinomyces stones are located in Wharton’s duct, the remaining 15% are found as commensal organisms in the human oral cav- are in gland parenchyma.[6] However, giant sialoliths (>15 ity, respiratory and digestive tracts. The bacteria become mm) in the submandibular duct have rarely been reported.[7] Correspondence: Nuray Bayar Muluk, MD. Department of Otorhinolaryngology, Faculty of Medicine, Online available at: K›r›kkale University, K›r›kkale, Turkey. www.entupdates.org e-mail: [email protected] doi:10.2399/jmu.2016003010 QR code: Received: March 21, 2016; Accepted: April 14, 2016 ©2016 Continuous Education and Scientific Research Association (CESRA) Rare coexistence of sialolithiasis and actinomycosis in the submandibular gland Different aetiological hypotheses have been put about salivary gland calculi. These are mechanical, inflammatory, chemical, neurogenic, infectious, strange bodies, etc. Submandibular gland is more susceptible to the develop- ment of the stone compared to parotid gland. Wharton’s duct is longer and wider than the Stensen’s duct. Submandibular salivary flow is against gravity; and salivary pH is more alkaline; and mucin proteins, calcium and phos- phates are contained in greater amount than serous secre- tion of parotid saliva.[8] In this report, we presented a 35-year-old male patient having coexistence of submandibular sialolithiasis and actin- omycosis with a literature survey. Case Report Thirty-five-year-old male patient admitted to fiehit Kamil Government Hospital complaining of swelling in the right Fig. 1. Computed tomography scan of the right submandibular gland submandibular region. He had a history of episodes of showing calculus. right submandibular swelling occurring during meals and gradually decreased by resting. Physical examination showed increased sensation and pain on the right sub- Computerized Tomography examination showed bone mandibular gland. By intraoral examination, saliva flow density structure which was 1.5 cm of diameter in the right from the right Wharton’s duct was not observed. The ear, submandibular gland parenchyma (Fig. 1). Magnetic reso- nose and throat examination findings were normal. nance imaging (MRI) of the neck also demonstrated 1.5 cm Submandibular neck ultrasonography was reported as structure in the parenchyma (Figs. 2 and 3). “The right submandibular gland diameter increased; ducts Patient underwent excision of the right submandibular dilated in gland; and right sialoadenitis was present”. Neck gland. Cephalosporin antibiotic was administered and the Fig. 2. Axial MRI of the right submandibular gland. Fig. 3. Coronal MRI of the right submandibular gland. Volume 6 | Issue 3 | December 2016 149 Dikici O, Bayar Muluk N patient was discharged without problems. On the third day should be keep in mind that sialolithiasis may be a predis- of the patient’s discharge, he admitted to the hospital with posing factor for submandibular actinomycosis, and complaints of pain, swelling and discharge at the operation removal of the sialolith or the entire gland is of particular region. He was hospitalized, and pathology result of the importance. In our case, we thought that actinomyces infec- operation was reported simultaneously as “Chronic tion was present with the submandibular stone, and to be sialadenitis, sialolithiasis and actinomyces necessitate the induced by surgery. right submandibular gland excision with stones with 1.5 cm Actinomycosis in the head and neck has a better progno- in diameter”. Postoperative infection was thought to be sis than other sites since this region is more responsible to due to infection with Actinomyces. The patient was admin- surgery and antibiotic therapy. Surgery plays an important istered amoxicillin and clavulanic acid 1 g i.v (twice a day) role in diagnosis and treatment. High-dose intravenous for 10 days; and later, 1 g p.o (twice a day) for 21 days. The antibiotics for 2–4 weeks are a fundamental part of treat- patient's symptoms improved without the need for anoth- ment and 3–6 months of treatment with oral antibiotics is er surgical intervention. The patient gave informed con- recommended. Tetracycline and erythromycin are used in sent for using his data for scientific purposes. patients with penicillin allergy.[10] In our case, infection has been treated with proper therapy after the diagnosis with- Discussion out invasive surgery. Actinomycosis is an Actinomyces israeli infection. This com- The instrumental diagnosis of sialolithiasis is based on mensal organism exists at high amounts in tonsil tissue and several imaging techniques. Ultrasonography represents an carious teeth. Poor oral hygiene, diabetes, immune suppres- excellent first-level diagnostic technique because it reveals ductal and highly mineralized stones with a diameter of at sion, malnutrition and local tissue damage are predisposing [11] factors. These conditions may lead to infection and subse- least 1.5 mm with an accuracy of 99%. In this case, USG quent invasion of subcutaneous tissues.[8] Actinomycosis examination did not give definitive diagnosis but CT estab- infection usually spreads into nearby soft tissues without lished a definitive diagnosis. regard for tissue planes or lymphatic drainage.[2] The ultimate aim of giant sialolith treatment is to restore a normal salivary flow. When the stone can be pal- Cervicofacial actinomycosis is frequently the result of pated intraorally, the best option is to remove it through an oro-maxillofacial trauma, dental manipulation or dental intraoral approach. The choice of a surgical approach to caries. Cervicofacial actinomycosis mostly presents as a firm, access the sialolith with submandibular gland preservation painless, slow-growing swelling and a multiloculated abscess requires careful imaging evaluation, minimal invasive which frequently progresses to multiple discharging sinus- removal and transoral sialolithotomy. After surgical calculi es.[3] The infection, most commonly, presents as a chronic, removal,
Recommended publications
  • 29360-Oral Cavity Dr. Alexandra Borges.Pdf
    ORAL CAVITY: ANATOMY AND PATHOLOGIES Alexandra Borges, MD COI Disclosure Instituto Português de Oncologia de Lisboa I have nothing to disclose Champalimaud Foundation Lisbon, Portugal ECHNNR 2021 ECHNNR 2021 MR ANATOMY LEARNING OBJECTIVES • Become familiar with OC anatomy and the importance of using adequate terminology when reporting OC studies NASOPHARYNX NASAL CAVITY • Learn how to tailor imaging studies • Understand the different patterns of malignant tumor spread according to the different tumor subsites OROPHARYNX ORAL CAVITY HYPOPHARYNX BOUNDARIES CONTENTS: ORAL TONGUE Superior: • hard palate • superior alveolar ridge Inferior: • floor of the mouth • inferior alveolar ridge ITM Laterally: • cheeks and buccal mucosaosa Anterior: • Lips Posterior: • oropharynx ORAL TONGUE: Extrinsic tongue muscles EM: Styloglossus tongue retraction and elevation Styloglossus Palatoglossus Hyoglossus SG Geniglossus HG GG CN XII CN X EM: Palatoglossus elevation of the tongue EM: Hyoglossus tongue depression and retraction EM: Genioglossus tongue protrusion ORAL TONGUE: Extrinsic muscles GG GH TONGUE INNERVATION ORAL CAVITY IX sensitive and Spatial subdivision taste CN X Motor • Mucosal area • Tongue root • Sublingual space CN XII Motor • Submandibular space Lingual nerve: Sensitive (branch of V3) • Buccomasseteric region Taste (chorda tympani) ORAL MUCOSAL SPACE ROOT OF THE TONGUE 1. Lips 2. Gengiva (sup. alveolar ridge) 3. Gengiva (inf. alveolar ridge) 4. Buccal 5. Palatal 6. Sublingual/FOM 7. Retromolar trigone GG 8. Tongue ROT BOT Vestibule GH Mucosa
    [Show full text]
  • The Oromaxillofacial Rehabilitation in Orthodontic-Surgical Protocols
    DOI: 10.1051/odfen/2015044 J Dentofacial Anom Orthod 2016;19:203 © The authors The oromaxillofacial rehabilitation in orthodontic-surgical protocols Th. Gouzland1,2, M. Fournier1 1 Kinesiologist, specializing in oromaxillofacial rehabilitation 2 Educator SUMMARY Oro-maxillo-facial rehabilitation is an ancient practice that has developed over recent years through research and integration with physiotherapists in multidisciplinary teams, as is the case with orthodontic- surgical procedures. At the same time, the progress made in orthognathic and orthodontic surgery over the last 20 years encourages more and more patients to undergo surgery. Preoperative treatment is based on early assessment and preparation for optimal surgical conditions to come up with a functional plan. A short stay in a hospital, focusing on rehabilitation, is recommended. During the postoperative phase, the key objectives are to ensure the muscles and arteries all function perfectly, acceptance of the new face, and the immediate correction of any orofacial dyspraxia that has occurred during myofunc- tional therapy. The various specialists in this multidisciplinary team must constantly be in communication. The importance of postoperative physiotherapy will be illustrated by a study consisting of 35 cases of maxillomandibular osteotomy with orthodontic preparation and monitoring. The purpose of this study is to show occurrence of suboccipital and cervical muscle tensions as well as masticatory muscles. Then we will be able to see the importance of these practices, the impact on recovery, the impact on posture and how best to treat. KEYWORDS Physiotherapy, orthognathic surgery, orthodontic, myofunctional therapy, muscles, posture INTRODUCTION The progress made in the techniques and comfortable recovery. This fits into and the results obtained by coupling the current social view where one’s orthognathic surgery with orthodontics image is given greater importance.
    [Show full text]
  • ODONTOGENTIC INFECTIONS Infection Spread Determinants
    ODONTOGENTIC INFECTIONS The Host The Organism The Environment In a state of homeostasis, there is Peter A. Vellis, D.D.S. a balance between the three. PROGRESSION OF ODONTOGENIC Infection Spread Determinants INFECTIONS • Location, location , location 1. Source 2. Bone density 3. Muscle attachment 4. Fascial planes “The Path of Least Resistance” Odontogentic Infections Progression of Odontogenic Infections • Common occurrences • Periapical due primarily to caries • Periodontal and periodontal • Soft tissue involvement disease. – Determined by perforation of the cortical bone in relation to the muscle attachments • Odontogentic infections • Cellulitis‐ acute, painful, diffuse borders can extend to potential • fascial spaces. Abscess‐ chronic, localized pain, fluctuant, well circumscribed. INFECTIONS Severity of the Infection Classic signs and symptoms: • Dolor- Pain Complete Tumor- Swelling History Calor- Warmth – Chief Complaint Rubor- Redness – Onset Loss of function – Duration Trismus – Symptoms Difficulty in breathing, swallowing, chewing Severity of the Infection Physical Examination • Vital Signs • How the patient – Temperature‐ feels‐ Malaise systemic involvement >101 F • Previous treatment – Blood Pressure‐ mild • Self treatment elevation • Past Medical – Pulse‐ >100 History – Increased Respiratory • Review of Systems Rate‐ normal 14‐16 – Lymphadenopathy Fascial Planes/Spaces Fascial Planes/Spaces • Potential spaces for • Primary spaces infectious spread – Canine between loose – Buccal connective tissue – Submandibular – Submental
    [Show full text]
  • Aetio-Pathogenesis and Clinical Pattern of Orofacial Infections
    2 Aetio-Pathogenesis and Clinical Pattern of Orofacial Infections Babatunde O. Akinbami Department of Oral and Maxillofacial Surgery, University of Port Harcourt Teaching Hospital, Rivers State, Nigeria 1. Introduction Microbial induced inflammatory disease in the orofacial/head and neck region which commonly arise from odontogenic tissues, should be handled with every sense of urgency, otherwise within a short period of time, they will result in acute emergency situations.1,2 The outcome of the management of the conditions are greatly affected by the duration of the disease and extent of spread before presentation in the hospital, severity(virulence of causative organisms) of these infections as well as the presence and control of local and systemic diseases. Odontogenic tissues include 1. Hard tooth tissue 2. Periodontium 2. Predisposing factors of orofacial infections Local factors and systemic conditions that are associated with orofacial infections are listed below. Local factors Systemic factors 1. Caries, impaction, pericoronitis Human immunodeficiency virus 2. Poor oral hygiene, periodontitis Alcoholism 3. Trauma Measles, chronic malaria, tuberculosis Diabetis mellitus, hypo- and 4. Foreign body, calculi hyperthyroidism 5. Local fungal and viral infections Liver disease, renal failure, heart failure 6. Post extraction/surgery Blood dyscrasias 7. Irradiation Steroid therapy 8. Failed root canal therapy Cytotoxic drugs 9. Needle injections Excessive antibiotics, 10. Secondary infection of tumors, cyst, Malnutrition fractures 11.
    [Show full text]
  • How to Manage a Buccal Space Mass – a Case Series
    Open Access Austin Head & Neck Oncology Case Report How to Manage a Buccal Space Mass – A Case Series Franzen A, Glitzki S and Coordes A* Department of Otorhinolaryngology, Head and Neck Abstract Surgery, Brandenburg Medical School, Campus Ruppiner Introduction: Patients presenting with masses in the cheek are common Kliniken, Neuruppin, Germany for head and neck specialists and present a diagnostic challenge against the *Corresponding author: Coordes Annekatrin, backdrop of a wide variety of etiologies. Based on a case series the specific Department of Otorhinolaryngology, Head and Neck problems of differential diagnosis and management are discussed. Surgery, Charité Universitätsmedizin Berlin, Germany Case series: Six patients of our series presenting with a buccal mass Received: September 12, 2018; Accepted: October 04, suffered from a pleomorphic adenoma of an accessory parotid gland, an 2018; Published: October 11, 2018 epidermoid cyst, a carcinoma of the Stensen`s duct, a carcinoma from the maxillary sinus, a secondary metastasis from oropharyngeal cancer and a distant metastasis of pulmonary cancer. Discussion: Our case series underlines the vast origins of buccal masses. Important hints for malignancy are rapid and painful tumor development and a medical history of malignant disease. Clinical examination, sonography and CT/MRI scans are performed for diagnostic evaluation. Histologic examination is required if the proper diagnosis cannot be achieved and the tumor growth is not in spontaneous remission. The surgical management may be challenging depending on the location and tumor size. Keywords: Cheek; Differential Diagnosis; Accessory Parotid Gland; Salivary Duct; Neoplasm; Metastasis Introduction 2.5cm in diameter that was anechoic and polygonally limited – an organ relation, especially to the parotid gland, cannot be described.
    [Show full text]
  • Description Concept ID Synonyms Definition
    Description Concept ID Synonyms Definition Category ABNORMALITIES OF TEETH 426390 Subcategory Cementum Defect 399115 Cementum aplasia 346218 Absence or paucity of cellular cementum (seen in hypophosphatasia) Cementum hypoplasia 180000 Hypocementosis Disturbance in structure of cementum, often seen in Juvenile periodontitis Florid cemento-osseous dysplasia 958771 Familial multiple cementoma; Florid osseous dysplasia Diffuse, multifocal cementosseous dysplasia Hypercementosis (Cementation 901056 Cementation hyperplasia; Cementosis; Cementum An idiopathic, non-neoplastic condition characterized by the excessive hyperplasia) hyperplasia buildup of normal cementum (calcified tissue) on the roots of one or more teeth Hypophosphatasia 976620 Hypophosphatasia mild; Phosphoethanol-aminuria Cementum defect; Autosomal recessive hereditary disease characterized by deficiency of alkaline phosphatase Odontohypophosphatasia 976622 Hypophosphatasia in which dental findings are the predominant manifestations of the disease Pulp sclerosis 179199 Dentin sclerosis Dentinal reaction to aging OR mild irritation Subcategory Dentin Defect 515523 Dentinogenesis imperfecta (Shell Teeth) 856459 Dentin, Hereditary Opalescent; Shell Teeth Dentin Defect; Autosomal dominant genetic disorder of tooth development Dentinogenesis Imperfecta - Shield I 977473 Dentin, Hereditary Opalescent; Shell Teeth Dentin Defect; Autosomal dominant genetic disorder of tooth development Dentinogenesis Imperfecta - Shield II 976722 Dentin, Hereditary Opalescent; Shell Teeth Dentin Defect;
    [Show full text]
  • Complex Odontogenic Infections
    Complex Odontogenic Infections Larry ). Peterson CHAPTEROUTLINE FASCIAL SPACE INFECTIONS Maxillary Spaces MANDIBULAR SPACES Secondary Fascial Spaces Cervical Fascial Spaces Management of Fascial Space Infections dontogenic infections are usually mild and easily and causes infection in the adjacent tissue. Whether or treated by antibiotic administration and local sur- not this becomes a vestibular or fascial space abscess is 0 gical treatment. Abscess formation in the bucco- determined primarily by the relationship of the muscle lingual vestibule is managed by simple intraoral incision attachment to the point at which the infection perfo- and drainage (I&D) procedures, occasionally including rates. Most odontogenic infections penetrate the bone dental extraction. (The principles of management of rou- in such a way that they become vestibular abscesses. tine odontogenic infections are discussed in Chapter 15.) On occasion they erode into fascial spaces directly, Some odontogenic infections are very serious and require which causes a fascial space infection (Fig. 16-1). Fascial management by clinicians who have extensive training spaces are fascia-lined areas that can be eroded or dis- and experience. Even after the advent of antibiotics and tended by purulent exudate. These areas are potential improved dental health, serious odontogenic infections spaces that do not exist in healthy people but become still sometimes result in death. These deaths occur when filled during infections. Some contain named neurovas- the infection reaches areas distant from the alveolar cular structures and are known as coinpnrtments; others, process. The purpose of this chapter is to present which are filled with loose areolar connective tissue, are overviews of fascial space infections of the head and neck known as clefts.
    [Show full text]
  • Anitha Subbappa Ashwini Tukuru Shivakumar* Vishal Shrishial
    ORIGINAL RESEARCH PAPER Volume-9 | Issue-1 | January-2020 | PRINT ISSN No. 2277 - 8179 | DOI : 10.36106/ijsr INTERNATIONAL JOURNAL OF SCIENTIFIC RESEARCH REAL TIME PCR ANALYSIS & ANTIBIOTIC SENSITIVITY OF BUCCAL SPACE INFECTION OF PERIODONTAL ORIGIN: A CASE REPORT Dental Science Reader, Department of Periodontology, JSSDCH, Dental-In-charge, JSSMCH, Anitha Subbappa JSSAHER *-Assisstant Professor, Department of OMFS, JSSDCH, JSSAHER Ashwini Tukuru Reader, Department of Conservative and Endodontics, JSSDCH, JSSAHER, Shivakumar* *Corresponding Author Vishal Shrishial Assisstant Professor, Department of Orthodontics, JSSDCH,, JSSAHER Kudagi ABSTRACT Background: Microbiology of odontogenic infections was inconsistent. It was difficult to compare various bacteriological investigations because of different methods and materials used. Streptococci which can be seen in the composition of microbial dental plaque may cause inflammatory periodontal disease and dental caries. Microbiological isolation helped in identifying the organisms and advocating antibiotics. Case report: We present a case of buccal space infection in a 51-year-old patient presented with a swelling in the upper left cheek region. He was on antibiotics as prescribed by a dentist for toothache for a week. It was diagnosed as a buccal space infection due to periodontal origin. Intra orally there was chronic generalized periodontitis with 9mm probing pocket depth, clinical attachment loss of 6 mm, mobility & exudation in upper left first and second molar. Pus sample was collected and
    [Show full text]
  • Cross-Sectional Imaging of Third Molar–Related Abnormalities
    Published September 10, 2020 as 10.3174/ajnr.A6747 REVIEW ARTICLE Cross-Sectional Imaging of Third Molar–Related Abnormalities R.M. Loureiro, D.V. Sumi, H.L.V.C. Tames, S.P.P. Ribeiro, C.R. Soares, R.L.E. Gomes, and M.M. Daniel ABSTRACT SUMMARY: Third molars may be associated with a wide range of pathologic conditions, including mechanical, inflammatory, infectious, cystic, neoplastic, and iatrogenic. Diagnosis of third molar–related conditions can be challenging for radiologists who lack experience in dental imaging. Appropriate imaging evaluation can help practicing radiologists arrive at correct diagnoses, thus improving patient care. This review discusses the imaging findings of various conditions related to third molars, highlighting relevant anatomy and cross-sectional imaging techniques. In addition, key imaging findings of complications of third molar extraction are presented. ABBREVIATIONS: CBCT ¼ cone-beam CT; MDCT ¼ multidetector-row CT hird molars, or wisdom teeth, are a more common source of molars usually erupt between 18 and 25 years of age.4 Every tooth Tpathologic conditions than other teeth. They are the last teeth is anatomically divided into a crown and a root by the cementoe- to develop and usually fail to erupt correctly. Impac- namel junction. The crown is the outer portion exposed in the ted third molars have been associated with inflammatory and oral cavity, and the root is the portion covered by the alveolar infectious conditions as well as development of cysts and tumors.1 ridge (Fig 1).3 Each crown has 5 free
    [Show full text]
  • EMERGENCY MEDICINE PRACTICE EMPRACTICE.NET an EVIDENCE-BASED APPROACH to EMERGENCY MEDICINE May 2003 Acute Dental Emergencies Volume 5, Number 5
    EMERGENCY MEDICINE PRACTICE EMPRACTICE.NET AN EVIDENCE-BASED APPROACH TO EMERGENCY MEDICINE May 2003 Acute Dental Emergencies Volume 5, Number 5 In Emergency Medicine Author “What do you mean, you don’t have a dentist in the emergency department?” Kip Benko, MD, FACEP Clinical Instructor in Emergency Medicine, “Go ahead, Doc—just pull it.” University of Pittsburgh School of Medicine; Attending Physician, Mercy Hospital of Pittsburgh, “I called my dentist’s office but got no answer. He pulled my tooth this morning Pittsburgh, PA. and now it’s bleeding like crazy. Oh, yeah—I’m on Coumadin!” Peer Reviewers OUND familiar? Complaints pertaining to teeth are common, and Marianne C. Burke, MD Spatients frequently present to the ED for initial care. Many patients Emergency Medicine Consultants, Los Angeles, CA. realize that definitive care must be provided by a dentist or oral surgeon, but either pain, trauma, inability to contact their dentist, or the lack of Charles Stewart, MD, FACEP Colorado Springs, CO. financial resources leads patients to our EDs.1 While treating dental emergencies in the ED can be challenging and frustrating, it can also be CME Objectives immensely satisfying. There is no more appreciative patient than one relieved of severe dental pain. Many emergency physicians are unable to Upon completing this article, you should be able to: recognize and treat acute dental problems because of a lack of specific 1. describe the structure, classification, training, yet proper initial care will limit morbidity such as tooth loss, pain, and identification of primary and infection, and, potentially, craniofacial abnormality. Moreoever, while permanent teeth; “dental patients” are often triaged as non-emergent, some of these patients 2.
    [Show full text]
  • Clinical Evaluation of Class II and Class III Gingival Recession Defects of Maxillary Posterior Teeth Treated with Pedicled Buccal Fat Pad: a Pilot Study
    Dental Research Journal Original Article Clinical evaluation of Class II and Class III gingival recession defects of maxillary posterior teeth treated with pedicled buccal fat pad: A pilot study D. Deepa1, K. V. Arun Kumar2 Departments of 1Periodontology and 2Oral and Maxillofacial Surgery, Subharti Dental College and Hospital, Meerut, Uttar Pradesh, India ABSTRACT Background: Buccal fat pad (BFP) is a specialized vascular tissue adequately present in buccal space and is close to the maxillary posterior quadrant. The aim of this clinical study was to evaluate the utility of pedicled BFP (PBFP) in the treatment of Class II and III gingival recession. Materials and Methods: Ten systemically healthy patients with age ranging from 35 to 55 years with Class II and Class III gingival recession in the maxillary molars were selected. Before the surgical phase, patients were enrolled in a strict maintenance program including oral hygiene instructions and scaling and root planing. A horizontal incision of 1–1.5 cm was made in the buccal sulcus of the maxillary molar region; buccinator muscle was separated bluntly to expose the BFP. The fat was then teased out from its bed and spread to cover defects adequately. It was then secured and sutured without tension. Clinical parameters such as probing depth, recession width, recession length (RL), and width of keratinized gingiva were recorded at baseline and at 6 months postoperatively, and Received: June 2016 weekly assessment was done at 1 week, 2 weeks, 3 weeks, and after 4 weeks for observations Accepted: August 2017 during the postoperative healing. Results: Treated recession defects healed successfully without any significant postoperative Address for correspondence: Dr.
    [Show full text]
  • Buccal Fat Pad Augmentation for Facial Rejuvenation Steven R
    TITLE PAGE (AUTHORS, CORRESPONDING AUTHOR, AFFILIATION, FINANCIAL DISCLOSURE, Buccal Fat Pad Augmentation for Facial Rejuvenation 1,2 Steven R. Cohen MD, FACS 1 Emily Fireman MS4 2 Sierra Hewett 1,2 Ahmad Saad MD From the Division of Plastic Surgery, University of California, San Diego1 and FACES+ Plastic Surgery, Dermatology, Skin 2 and Laser Center, San Diego, California Corresponding Author: Steven R. Cohen MD 4510 Executive Drive Suite 200 San Diego, CA 92121; [email protected]; 858-453-7224 Disclosures: Dr. Cohen consults for Tulip Medical, Inc. and Millenium Medical Technologies, Inc. and is a paid investigator, consultant and minor shareholder for Cytori, Inc. MANUSCRIPT (TITLE, MANUSCRIPT TEXT, REFERENCES, LEGENDS) Buccal Fat Pad Augmentation for Facial Rejuvenation ABSTRACT Background: The buccal space and its fat pad (BFP) is a valuable, overlooked target in facial rejuvenation procedures. We identified a specific group of patients who have normal or prominent malar projection in the presence of atrophy of the buccal fat pad, with or without prominent gonial angles. Materials and Methods: Eight of 24 prospectively studied patients (BIOMED IRB), who had fat grafts and facelifts received an average of 2.7 ml of fat transferred into the buccal space. Immediate visual correction of the buccal depression was noted. No overcorrection was carried out. Results: None of the 8 patients suffered an adverse event from trans-oral buccal space fat grafting. Persistent facial volume in this area has been noted up to 24 months after treatment. Conclusions: In patients with buccal fat pad atrophy, fat grafting into the buccal space can be safely performed through an intraoral approach.
    [Show full text]