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STIM1
Association of the IP3R to STIM1 Provides a Reduced Intraluminal
Na+ Influx Via Orai1 Inhibits Intracellular ATP-Induced Mtorc2 Signaling to Disrupt CD4 T Cell Gene Expression and Differentiation." Elife.6
KRAP Tethers IP3 Receptors to Actin and Licenses Them to Evoke Cytosolic Ca2+ Signals ✉ ✉ Nagendra Babu Thillaiappan 1,2 , Holly A
Supplementary Table S4. FGA Co-Expressed Gene List in LUAD
Alternative Splicing Switches STIM1 Targeting to Specialized Membrane Contact Sites and Modifies SOCE
STIM1 Phosphorylation at Y361 Recruits Orai1 to STIM1 Puncta And
Store-Operated Ca2+ Entry Regulates Ca2+-Activated Chloride Channels and Eccrine Sweat Gland Function
A New Family of Start Domain Proteins at Membrane Contact Sites Has a Role in ER-PM 2 Sterol Transport
Enhanced Stim1 Expression Is Associated with Acquired Chemo- Resistance of Cisplatin in Osteosarcoma Cells
IP3 Receptors – Lessons from Analyses Ex Cellula Ana M
Na Influx Via Orai1 Inhibits Intracellular ATP-Induced Mtorc2 Signaling
SUPPLEMENTARY APPENDIX Exome Sequencing Reveals Heterogeneous Clonal Dynamics in Donor Cell Myeloid Neoplasms After Stem Cell Transplantation
A Novel STIM1-Orai1 Gating Interface Essential for CRAC Channel Activation
STIM1L Traps and Gates Orai1 Channels Without Remodeling the Cortical ER
Coexpression Networks Based on Natural Variation in Human Gene Expression at Baseline and Under Stress
Inbred Mouse Strains Expression in Primary Immunocytes Across
Natural Genetic Variation in Stim1 Creates Stroke in the Spontaneously Hypertensive Rat
A Report of Novel STIM1 Deficiency and 6 Year Follow up of Two Previous Cases Associated with Mild Immunological Phenotype
Top View
STIM1 Gene Stromal Interaction Molecule 1
Integrin-Linked Kinase Controls Renal Branching Morphogenesis Via Dual Specificity Phosphatase 8
STIM1-Mediated Bidirectional Regulation of Ca2+ Entry Through Voltage-Gated Calcium Channels (VGCC) and Calcium-Release Activated Channels (CRAC)
Understanding the Regulation of STIM1-ORAI1 Interaction Using a Reaction-Diffusion Model
STIM Proteins: an Ever-Expanding Family
Structure and Function of IP3 Receptors
Calcium Flux and Endothelial Dysfunction During Acute Lung Injury: a Stimulating Target for Therapy
Gene Symbol Cancer Associated Expression Levelgene Name Size
Channels Induced by Local Ca2+ Entry in HEK293
Molecular Genetic Investigation Into Inherited Thrombocytopenia
P.E152K-STIM1 Mutation Deregulates Ca2+ Signaling Contributing to Chronic Pancreatitis
GRAM Domain Proteins Specialize Functionally Distinct ER-PM Contact
Calcium Channels in Lymphocytes Franz Fenninger and Wilfred A
Investigating the Role of BEST1 Protein in Best Vitelliform Macular
STIM2 Targets Orai1/STIM1 to the AKAP79 Signaling Complex and Confers Coupling of Ca2+ Entry with NFAT1 Activation
Table S1 All Prioritized Candidate Genes in L4 Drgs from Toppgene
Dynamic Interaction of Htrpc6 with the Orai1/STIM1 Complex Or Htrpc3 Mediates Its Role in Capacitative Or Non-Capacitative Ca2+ Entry Pathways I
STIM2 Targets Orai1/STIM1 to the AKAP79 Signaling Complex and Confers Coupling of Ca2+ Entry with NFAT1 Activation
The Regulation of STIM1 Translocation to the Plasma Membrane
Type 3 Inositol 1,4,5-Trisphosphate Receptor Is a Crucial Regulator of Calcium Dynamics Mediated by Endoplasmic Reticulum in HEK Cells
Effect of STIM1/2 and of Ceritinib on Platelet Function
ORAI1 Establishes Resistance to SARS-Cov-2 Infection by Regulating Tonic Type I Interferon Signaling
Structural and Functional Differences Between L-Type Calcium Channels: Crucial Issues for Future Selective Targeting
Structural Elements of Stromal Interaction Molecule Function T ⁎ ⁎ Matthew J
The Short Isoform of Extended Synaptotagmin-2 Controls Ca2+
Table of Contents List of Investigators
Regulation of Endothelial Barrier Integrity Via Store-Operated Ca2+ Entry
ER-Localized Bestrophin 1 Activates Ca2+-Dependent Ion Channels TMEM16A and SK4 Possibly by Acting As a Counterion Channel
Stable Expression and Function of the Inositol 1,4,5-Triphosphate Receptor Requires Palmitoylation by a DHHC6/Selenoprotein K Complex
Store-Operated Ca2+ Entry Plays a Role in HMGB1-Induced Vascular Endothelial Cell Hyperpermeability
Orai–Vascular Endothelial-Cadherin Signaling Complex Regulates High
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Remodeling of ER–Plasma Membrane Contact Sites but Not STIM1 Phosphorylation Inhibits Ca2+ Influx in Mitosis
1 Na+ Influx Via Orai1 Inhibits Intracellular ATP Induced Mtorc2 Signaling To
Activating Mutations in STIM1 and ORAI1 Cause Overlapping Syndromes of Tubular Myopathy and Congenital Miosis
Endoplasmic Reticulum‐Plasma Membrane Contact Sites As An
Than Just Simple Interaction Between STIM and Orai Proteins: CRAC Channel Function Enabled by a Network of Interactions with Regulatory Proteins