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SF3B1
Mutated SF3B1 Is Associated with Transcript Isoform Changes of The
Anti-Phospho-SF3B1 (Sap155) (Ser129) Pab
Monoclonal B-Cell Lymphocytosis Is Characterized by Mutations in CLL Putative Driver Genes and Clonal Heterogeneity Many Years Before Disease Progression
SF3B2-Mediated RNA Splicing Drives Human Prostate Cancer Progression
Flexible, Unbiased Analysis of Biological Characteristics Associated with Genomic Regions
Full-Length Transcript Characterization of SF3B1 Mutation in Chronic Lymphocytic Leukemia Reveals Downregulation of Retained Introns
Report Summary 4 Clinically Significant Variants & Combinations 0 Relevant Therapies 0 Other Biomarkers
Cryptic Splicing Events in the Iron Transporter ABCB7 and Other Key Target Genes in SF3B1-Mutant Myelodysplastic Syndromes
Roles of Splicing Factors in Hormone-Related Cancer Progression
The Biological Function and Clinical Significance of SF3B1 Mutations In
Titration of SF3B1 Activity Reveals Distinct Effects on the Transcriptome and Cell Physiology
Clinical Characterization and Prognostic Impact of SF3B1 and TP53 Gene Mutations in Myelodysplastic Syndromes
Expression Levels of SF3B3 Correlate with Prognosis and This Is the Author's Manuscript of Article Published in Final Edited Form As
SF3B1-Mutated Chronic Lymphocytic Leukemia Shows Evidence Of
In Silico Analysis of a Highly Mutated Gene in Cancer Provides Insight Into Abnormal Mrna Splicing: Splicing Factor 3B Subunit 1K700E Mutant
SF3B2-Mediated RNA Splicing Drives Human Prostate
Disruption of SF3B1 Results in Deregulated Expression And
Cross-Talk Between PRMT1-Mediated Methylation and Ubiquitylation on RBM15 Controls RNA Splicing
Top View
Somatic SF3B1 Mutations in Myelodysplastic Syndrome with Ring Sideroblasts and Chronic Lymphocytic Leukaemia
Enhanced DNA-Repair Capacity and Resistance to Chemically Induced
Alternative Splicing of EZH2 Pre-Mrna by SF3B3 Contributes To
The Cryo-EM Structure of the Sf3b Spliceosome Complex Bound to a Splicing Modulator Reveals a Pre-Mrna Substrate Competitive Mechanism of Action
Age, JAK2V617F and SF3B1 Mutations Are the Main Predicting Factors for Survival in Refractory Anaemia with Ring Sideroblasts and Marked Thrombocytosis
Altered RNA Processing in Cancer Pathogenesis and Therapy
Interactome Analyses Revealed That the U1 Snrnp Machinery Overlaps
Selective Inhibition of CDK7 Reveals High-Confidence Targets and New Models for TFIIH Function in Transcription
Related Mutations in the Sf3b1 Protein on Pre-Mrna Splicing in Vitro
Rapalog-Mediated Repression of Tribbles Pseudokinase 3 Regulates Pre- Mrna Splicing
1 in Silico Analyses Reveal New Putative Breast Cancer RNA-Binding Proteins
1 CDK9 and PP2A Regulate the Link Between RNA Polymerase II
Emerging Roles of the Spliceosomal Machinery in Myelodysplastic Syndromes and Other Hematological Disorders
RNA Splicing Factors As Oncoproteins and Tumour Suppressors
Splicing Modulators Act at the Branch Point Adenosine Binding Pocket Defined by the PHF5A–Sf3b Complex
Aberrant Splicing Regulation and Therapeutic Targets in Cancer
Hotspot SF3B1 Mutations Induce Metabolic Reprogramming and Vulnerability to Serine Deprivation
Expression Levels of SF3B3 Correlate with Prognosis And