DOCSLIB.ORG
Explore
Sign Up
Log In
Upload
Search
Home
» Tags
» NPR2
NPR2
NPR1 Paralogs of Arabidopsis and Their Role in Salicylic Acid Perception
A Novel Variant of FGFR3 Causes Proportionate Short Stature
Mutations in C-Natriuretic Peptide (NPPC): a Novel Cause of Autosomal Dominant Short Stature
Cldn19 Clic2 Clmp Cln3
Regulation of the Natriuretic Peptide Receptor 2 (Npr2) by Phosphorylation of Juxtamembrane Serine and Threonine Residues Is
Dephosphorylation of the NPR2 Guanylyl Cyclase Contributes to Inhibition of Bone Growth by Fibroblast Growth Factor
Regulation of the Natriuretic Peptide Receptor 2 (Npr2) By
(12) United States Patent (10) Patent No.: US 8,741,861 B2 Mann (45) Date of Patent: Jun
(12) Patent Application Publication (10) Pub. No.: US 2003/0198970 A1 Roberts (43) Pub
C-Type Natriuretic Peptide (CNP) Is a Bifurcation Factor for Sensory Neurons
Dephosphorylation of the NPR2 Guanylyl Cyclase Contributes To
Dephosphorylation of the NPR2 Guanylyl Cyclase Contributes to Inhibition of Bone Growth by Fibroblast Growth Factor
Peripheral Nerve Single-Cell Analysis Identifies Mesenchymal Ligands That Promote Axonal Growth
Dephosphorylation of Juxtamembrane Serines and Threonines of the NPR2
Current Modulation of Guanylate Cyclase Pathway Activity—Mechanism and Clinical Implications
Luteinizing Hormone Reduces the Activity of the NPR2 Guanylyl
Study of Cellular Heterogeneity and Differential Dynamics of Autophagy
Supplementary Figures Figure S1
Top View
A. Supplementary Figure 1
Kin-2787, Is Active in Class Ii and Class Iii Braf Mutant Models
Dephosphorylation of the NPR2 Guanylyl Cyclase
C-Type Natriuretic Peptide/Natriuretic Peptide Receptor 2 Is Involved in Cell Proliferation and Testosterone Production in Mouse Leydig Cells
Dephosphorylation and Inactivation of NPR2 Guanylyl Cyclase In
Newborndxtm Advanced Sequencing Evaluation Disorders List
Ncounter® Mouse Metabolic Pathways Panel - Gene and Probe Details
Baena 2020.Pdf
The Receptor Guanylyl Cyclase Npr2 Is Essential for Sensory Axon
Combined Genetic and Chemical Screens Indicate Protective Potential
Genes Implicated
The Receptor Guanylyl Cyclase Npr2 Regulates the Bifurcation of Cranial Sensory Axons
In FLT3-ITD+ Acute Myeloid Leukemia (AML) with Concurrent Gene Mutations in NPM1, DNMT3A Or TET2
Appendix 3. Non-HK Human TM Expressed Genes Grouped Based on Function*
Genetic Investigation of Patients with Tall Stature
Neuronal, Stromal, and T-Regulatory Cell Crosstalk in Murine Skeletal Muscle
Natriuretic Peptides Appeared After Their Receptors in Vertebrates Anna Grandchamp†, Shifa Tahir† and Philippe Monget*
Supplementary Material