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MRAS
COVID-19—The Potential Beneficial Therapeutic Effects of Spironolactone During SARS-Cov-2 Infection
Deciphering the Nature of Trp73 Isoforms in Mouse
SHOC2–MRAS–PP1 Complex Positively Regulates RAF Activity and Contributes to Noonan Syndrome Pathogenesis
The Tumor Suppressor SCRIB Is a Negative Modulator of the Wnt/-Catenin Signaling Pathway
In Vivo Sensitivity of Human Melanoma to Tumor Necrosis Factor
Stool-Derived Eukaryotic RNA Biomarkers for Detection of High-Risk Adenomas
Hnag-1 Increases Lifespan by Regulating Energy Metabolism And
Supplemental Information Loss of Estrogen Regulated MIR135A1 At
The RAS–Effector Interaction As a Drug Target Adam B
Identifying Genetic Risk Variants for Coronary Heart Disease in Familial Hypercholesterolemia: an Extreme Genetics Approach
Download a Subgraph Composed of the Initially 32 Raso- of Proteins Underlying Rasopathies in Order to Iden- Pathy Proteins Selected in This Study
Identification of RRAS Gene Related to Nasopharyngeal Carcinoma Based on Pathway and Network-Based Analyses
R-Ras Gtpases Signaling Role in Myelin Neurodegenerative Diseases
Delineation of LZTR1 Mutation-Positive Patients with Noonan Syndrome and Identification of LZTR1 Binding to RAF1–PPP1CB Complexes
SHOC2 Complex-Driven RAF Dimerization Selectively Contributes to ERK Pathway Dynamics
Isoform-Specific Differences in the Effector Binding Regions
MRAS: a Close but Understudied Member of the RAS Family
Genome-Wide Gene Expression Profiling of Randall's Plaques In
Top View
A Truncated Form of the Polarity Protein SCRIB Induces Apoptosis in MDA-MB231 Cells by Regulating Components of Several Signalling Pathways
In Silico Analysis Predicting Effects of Deleterious Snps of Human
Mutations in the RAS-BRAF-MAPK-ERK Pathway
HDAC and MAPK/ERK Inhibitors Cooperate to Reduce
HDAC and MAPK/ERK Inhibitors Cooperate to Reduce Viability And
Dissecting the Involvement of Ras Gtpases in Kidney Fibrosis
LZTR1 Facilitates Polyubiquitination and Degradation of RAS-Gtpases
Herefore, Additional Strategies Are Needed to Target Even These Benign Rasopathy Manifestations
Small Gtpases* 1,2 3§ David J
TET1 Interacts Directly with NANOG Via Independent Regions Containing
Supplementary Figures and Tables 1, 2, 4-7
MRAS Mouse Monoclonal Antibody [Clone ID: OTI3C4] – TA502864
Homozygous Mutation in SAMHD1 Gene Causes Cerebral Vasculopathy and Early Onset Stroke
Genomic Variations in Patients with Myelodysplastic Syndrome and Karyotypes Without Numerical Or Structural Changes Cristiano Luiz Ribeiro 1,5*, Irene P
The Ins and Outs of RAS Effector Complexes
The Scribble Family in Cancer: Twentieth Anniversary
1 Classification Title Short Title Authors Author
Diagnostic Difficulties and Possibilities of NF1-Like Syndromes in Childhood
MRAS (NM 001085049) Human Recombinant Protein – TP318272
Structures of N-Terminally Processed KRAS Provide Insight Into the Role of N-Acetylation Received: 16 October 2018 Srisathiyanarayanan Dharmaiah1, Timothy H
In Vitro Protection of Vascular Function from Oxidative Stress and Inflammation by Pulsatility in Resistance Arteries
Elucidation of MRAS-Mediated Noonan Syndrome with Cardiac Hypertrophy