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MGME1
Mice Lacking the Mitochondrial Exonuclease MGME1 Accumulate Mtdna Deletions Without Developing Progeria
Loss-Of-Function Mutations in MGME1 Impair Mtdna Replication and Cause Multisystemic Mitochondrial Disease
Roles of the Mitochondrial Replisome in Mitochondrial DNA Deletion Formation
Oxygen Tension Modulates the Mitochondrial Genetic Bottleneck and Influences the Segregation of a Heteroplasmic Mtdna Variant in Vitro
DNA Repair and Mutagenesis in Vertebrate Mitochondria: Evidence for the Asymmetric DNA Strand Inheritance
2014-Platform-Abstracts.Pdf
Dominant Mutations in Mtdna Maintenance Gene SSBP1 Cause Optic Atrophy and Foveopathy
Transposon Mutagenesis Reveals Fludarabine Resistance Mechanisms in Chronic Lymphocytic Leukemia
Effects of Mitochondrial Nucleases on Mt DNA Degradation
The Role of the Aryl Hydrocarbon Receptor in 2,3,7,8- Tetrachlorodibenzo-Ρ-Dioxin-Induced Mitochondrial Dysfunction in Mouse Hepatoma Cells
Clinical Implementation of RNA Sequencing for Mendelian Disease Diagnostics
Current Progress with Mammalian Models of Mitochondrial DNA Disease
Selective Mitochondrial DNA Degradation Following Double-Strand Breaks
Human Mitochondrial DNA Replication Machinery and Disease. Matthew J Ouny G Southern Illinois University School of Medicine
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Generation of CRISPR Engineered Prostate Cancer Cell Line Models to Study Androgen Receptor Signalling in Advanced Prostate Cancer
Anti-MGME1 Mouse Mab
Mitochondrial Transcription Factor a Promotes DNA Strand Cleavage at Abasic Sites
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Combining Genome Wide Association Studies and Differential Gene
Identification and Functional Validation of New Mtdna and Nuclear Gene Variants Responsible for Mitochondrial Disorders
Autosomal Recessive Variants in Intellectual Disability and Autism Spectrum Disorder
Adult-Onset Mendelian PEO Associated with Mitochondrial Disease
Pathological Ribonuclease H1 Causes R-Loop Depletion and Aberrant DNA Segregation in Mitochondria
Mitochondrial Disease Heterogeneity: a Prognostic Challenge
The 2020 Version of the Gene Table of Neuromuscular Disorders (Nuclear Genome) Louise Benarroch, Gisèle Bonne, Francois Rivier, Dalil Hamroun
Dedicated Surveillance Mechanism Controls G-Quadruplex Forming Non-Coding Rnas in Human Mitochondria
The 2021 Version of the Gene Table of Neuromuscular Disorders (Nuclear Genome) Louise Benarroch, Gisèle Bonne, Francois Rivier, Dalil Hamroun
A Two-Nuclease Pathway Involving Rnase H1 Is Required for Primer Removal at Human Mitochondrial Oril
Elucidating the Constitutional Genetic Basis of Multiple Primary Tumours
Clonal Expansion of Secondary Mitochondrial DNA Deletions Associated with Spinocerebellar Ataxia Type 28
The Exonuclease Activity of DNA Polymerase Γ Is Required for Ligation During Mitochondrial DNA Replication
Linear Mitochondrial DNA Is Rapidly Degraded by Components of the Replication Machinery