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MEN1
TERT Promoter Mutations Occur Frequently in Gliomas and a Subset of Tumors Derived from Cells with Low Rates of Self-Renewal
SUPPLEMENTARY NOTE Co-Activation of GR and NFKB
Multiple Endocrine Neoplasia Type 1 (MEN1)
Reversal of Preexisting Hyperglycemia in Diabetic Mice by Acute Deletion of the Men1 Gene
Twenty Years of Menin: Emerging Opportunities for Restoration of Transcriptional Regulation in MEN1
International Journal of Infection Prevention Issn No: 2690-4837
Cdx4 and Menin Co-Regulate Hoxa9 Expression in Hematopoietic Cells Jizhou Yan, Ya-Xiong Chen, Angela Desmond, Albert Silva, Yuqing Yang, Haoren Wang, Xianxin Hua*
Integrative Genomic Characterization Identifies Molecular Subtypes of Lung Carcinoids
Dynamic Epigenetic Regulation by Menin During Pancreatic Islet Tumor Formation Wenchu Lin1,2,3,4, Hideo Watanabe1,2,3, Shouyong Peng1,2,3, Joshua M
Full Text (PDF)
Global Phosphoproteomic Profiling Reveals Perturbed Signaling in a Mouse Model of Dilated Cardiomyopathy
Multiple Endocrine Neoplasia Type 1 (MEN1)
Epigenetic Silencing of Tumor Suppressor Genes During in Vitro
Computational Simulations to Predict Creatine Kinase-Associated Factors: Protein-Protein Interaction Studies of Brain and Muscle Types of Creatine Kinases
Tissue Selectivity in Multiple Endocrine Neoplasia Type 1-Associated Tumorigenesis Ana Gracanin,1 Koen M
Association of Somatotrophinomas with Loss of Alleles on Chromosome 11 and with Gsp Mutations
MEN1 Gene Menin 1
Menin and Menin-Associated Proteins Coregulate Cancer Energy Metabolism
Top View
Gene Expression Profiling in Insulinomas of Men1 B-Cell Mutant Mice Reveals Early Genetic and Epigenetic Events Involved in Panc
Neuroendocrine Neoplasms of the Pancreas: the Pathological Viewpoint
Analysis of MEN1 C.482G>A (P.Gly161asp) Mutation in A
Kmt2a Cooperates with Menin to Suppress Tumorigenesis in Mouse Pancreatic Islets
Clinical Testing for Multiple Endocrine Neoplasia Type 1 in a DNA Diagnostic Laboratory Roger D
MEN1 C.825‑1G>A Mutation in a Family
Relative Telomere Lengths in Tumor and Normal Mucosa Are Related to Disease Progression and Chromosome Instability Profiles in Colorectal Cancer
Nets: Organ-Related Epigenetic Derangements and Potential Clinical Applications
Epigenetic Regulation in the Tumorigenesis of MEN1-Associated Endocrine Cell Types
A Rapid and Easy Method for Multiple Endocrine Neoplasia Type 1 Mutation Detection Using Conformation-Sensitive Gel Electrophoresis
The Tumor Suppressor Protein Menin Interacts with NF-Kb Proteins and Inhibits NF-Kb-Mediated Transactivation
NUP98 and KMT2A
Identification of a Novel MEN1 Gene Mutation in Saudi Arabian Patient with Multiple Endocrine Tumors
Rabbit Anti-Phospho-NFKB1-SL20075R-FITC
Mutational and Gross Deletion Study of the MEN1 Gene and Correlation With
Genetics of Endocrinology
Allelic Loss of 11Q13 As Detected by MEN1-FISH Is Not Associated with Mutation of the MEN1 Gene in Lymphoid Neoplasms
A MEN1 Syndrome with a Paraganglioma
Differentially Expressed Genes in Men1 Knockout and Wild‑Type Embryoid Bodies for Pancreatic Islet Development
Identification of Significant Pathways in Gastric Cancer Based on Protein-Protein Interaction Networks and Cluster Analysis
Ncomms4518.Pdf
Multiple Tumor Suppressor Pathways Negatively Regulate Telomerase
Mutation Analysis of the MEN1 Gene in Multiple Endocrine Neoplasia Type 1, Familial Acromegaly and Familial Isolated Hyperparathyroidism*
The Tumor Suppressor Menin Regulates Hematopoiesis and Myeloid Transformation by Influencing Hox Gene Expression
The Trithorax Protein Partner Menin Acts in Tandem with EZH2 to Suppress C/EBP Α and Differentiation in MLL-AF9 Leukemia Austin T
Telomerase Reverse Transcriptase Promoter Region Mutations and the Clinical Characteristics of Pulmonary Neuroendocrine Tumors
Recapitulation of Pancreatic Neuroendocrine Tumors in Human Multiple Endocrine Neoplasia Type I Syndrome Via Pdx1-Directed Inactivation of Men1
MEN1 Tumorigenesis in the Pituitary and Pancreatic Islet Requires Cdk4 but Not Cdk2
Men1 Maintains Exocrine Pancreas Homeostasis in Response to Inflammation and Oncogenic Stress
MEN1 Silencing Aggravates Tumorigenic Potential of AR-Independent Prostate Cancer Cells Through Nuclear Translocation and Activa
Germline Mutation Landscape of Multiple Endocrine Neoplasia Type 1
Wild-Type Menin Is Rapidly Degraded Via the Ubiquitin-Proteasome Pathway in a Rat Insulinoma Cell Line
Involvement of the MEN1 Gene in Hormone-Related Cancers: Clues from Molecular Studies, Mouse Models, and Patient Investigations
Genes Involved in Angiogenesis and Mtor Pathways Are Frequently
Telomere Dysfunction Suppresses Multiple Endocrine Neoplasia in Mice
Menin Is Necessary for Long Term Maintenance of Meningioma-1 Driven Leukemia
Happy 20Th Anniversary MEN1: from Positional Cloning to Gene Function Restoration
Thoracic and Duodenopancreatic Neuroendocrine Tumors in Multiple Endocrine Neoplasia Type 1: Natural History and Function of Menin in Tumorigenesis
An Unknown Phenotype of Multiple
Comprehensive Analysis of MEN1 Mutations and Their Role in Cancer
Prognostic Factors for the Outcome of Nonfunctioning Pancreatic Neuroendocrine Tumors in MEN1: a Systematic Review of Literature
Parathyroid Tumor Development Involves Deregulation of Homeobox Genes
Molecular Genetic Studies of Sporadic and MEN1-Associated Endocrine Pancreatic Tumors
Predictive Testing for Multiple Endocrine Neoplasia Type 1 Using DNA Polymorphisms
Long-Term Outcome of Multiple Endocrine Neoplasia Type 1 Related Manifestations
A Rare Case of Multiple Endocrine Neoplasia Type 1 Initially Presenting
HOXA9 Promotes MYC-Mediated Leukemogenesis by Maintaining
Telomere Length Abnormalities and Telomerase RNA Component Expression in Gastroenteropancreatic Neuroendocrine Tumors
Role of Menin in the Regulation of Telomerase Activity in Normal and Cancer Cells