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C3a receptor
The 'C3ar Antagonist' SB290157 Is a Partial C5ar2 Agonist
BD Biosciences New RUO Reagents - November 2020
An Overview on G Protein-Coupled Receptor-Induced Signal Transduction in Acute Myeloid Leukemia
G Protein-Coupled Receptors
Novel Therapies for Eosinophilic Disorders
International Union of Basic and Clinical Pharmacology. LXXXVII
Techniques for Immune Function Analysis Application Handbook 1St Edition
Initial, Transient, and Specific Interaction Between G Protein
The C3ar Promotes Macrophage Infiltration and Regulates ANCA Production but Does Not Affect Glomerular Injury in Experimental Anti- Myeloperoxidase Glomerulonephritis
Derivation of Ligands for the Complement C3a Receptor from the C-Terminus of C5a
Induction of Complement C3a Receptor Responses by Kallikrein-Related Peptidase 14
Myeloid Innate Immunity Mouse Vapril2018
Multifaceted Role of Β-Arrestins in Inflammation and Disease
Multifaceted Roles of Neutrophils in Autoimmune Diseases
Complement-Activation Fragment C4a Mediates Effector Functions by Binding As Untethered Agonist to Protease-Activated Receptors 1 and 4
Exploring the Role of C5a-C5ar1 Signalling in Development Through Pluripotent Stem Cell Modelling
Complement and Cancer—A Dysfunctional Relationship?
The Role of Complement C3a Receptor in Stroke
Top View
Cause Prolonged ERK1/2 Phosphorylation Human
Expression of the Anaphylatoxin Receptors C3ar and C5ar Is Increased in Fatal Asthma
Wo2018/232195
The Effect of Exercise on Gene Expression and Signaling in Mouse
Dimerization of Complement C5a Receptors in Inflammatory Responses
C5ar1 Regulates T Follicular Helper Differentiation and Chronic Graft-Versus-Host Disease Bronchiolitis Obliterans
Protease Activated Receptor 4 As a Novel Modulator of Regulatory T Cell Function
Of C3a Receptor Marrow Hematopoietic Cells Independently
Journal of Prosthodontic Research 65 ( 2021) 393–399
Contribution of Complement Anaphylatoxin Receptors to Melanoma Growth: Potential Therapeutic Targets
14 Responses by Kallikrein-Related Peptidase Induction of Complement
Complement C3a Receptor
Targeting the PGD2/CRTH2/DP1 Signaling Pathway in Asthma and Allergic Disease: Current Status and Future Perspectives
Mediate and Regulate Leukocyte Functions Protein-Coupled
New Insights Into the Immune Functions of Complement
Antagonism of the Prostaglandin D 2 Receptor CRTH2 Attenuates Asthma
Regulation of C3a Receptor Signaling in Human Mast Cells by G Protein Coupled Receptor Kinases
Complement As a Regulator of Adaptive Immunity
The Contribution of Mast Cells to Antiviral Immune Responses: Characterization and Functional Definition of Receptor Transporter Protein 4 (RTP4)
The Open Pain Journal, 2016, 9, 26-37 the Open Pain Journal
(12) United States Patent (10) Patent No.: US 9,512,236 B2 Adams Et Al
Endotoxin-Shock Protective Anti-Inflammatory Role for C3a In
Conserved Second Residue of Helix 8 of GPCR May Confer Subclass-Characteristic and Distinct Roles Through a Rapid Initial
Chemoattractant G Protein-Coupled Receptors in Human Mast Cells
G Protein-Coupled Receptors
G Protein-Coupled Receptors
And C5-Binding Proteins in Renal Injury
Differential Involvement of ACKR3 C-Tail in Β-Arrestin Recruitment, Trafficking and Internalization
G Protein-Coupled Receptor GPCR
Leukocyte Trafficking to the Lungs and Beyond: Lessons from Influenza For
Identification and Interaction Analysis of Molecular Markers in Myocardial Infarction by Integrated Bioinformatics Analysis
The Role of Coagulation and Complement Factors for Mast Cell Activation in the Pathogenesis of Chronic Spontaneous Urticaria
C3a and C5a Promote Renal Ischemia-Reperfusion Injury
Complement C3a and C5a Receptor Blockade Modulates Regulatory T Cell Conversion in Head and Neck Cancer
Endothelial C3a Receptor Mediates Vascular Inflammation and Blood-Brain Barrier Permeability During Aging
More Choice BD Multicolor Antibody Reagents Catalog 18Th Edition
Bioinformatics Analysis of Key Genes and Pathways for Obesity
MACS® Marker Screen Plate Allocations
Functional C3a Receptor Activated Human T Lymphocytes Express A
Pharmacological Targeting of Anaphylatoxin Receptors During the Effector Phase of Allergic Asthma Suppresses Airway Hyperresponsiveness and Airway Inflammation