Ten Reasons Not to Use Pesticides
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Restricted Use Product Summary Report
Page 1 of 17 Restricted Use Product Summary Report (January 19, 2016) Percent Active Registration # Name Company # Company Name Active Ingredient(s) Ingredient 4‐152 BONIDE ORCHARD MOUSE BAIT 4 BONIDE PRODUCTS, INC. 2 Zinc phosphide (Zn3P2) 70‐223 RIGO EXOTHERM TERMIL 70 VALUE GARDENS SUPPLY, LLC 20 Chlorothalonil 100‐497 AATREX 4L HERBICIDE 100 SYNGENTA CROP PROTECTION, LLC 42.6 Atrazine 100‐585 AATREX NINE‐O HERBICIDE 100 SYNGENTA CROP PROTECTION, LLC 88.2 Atrazine 100‐669 CURACRON 8E INSECTICIDE‐MITICIDE 100 SYNGENTA CROP PROTECTION, LLC 73 Profenofos 100‐817 BICEP II MAGNUM HERBICIDE 100 SYNGENTA CROP PROTECTION, LLC 33; 26.1 Atrazine; S‐Metolachlor 100‐827 BICEP LITE II MAGNUM HERBICIDE 100 SYNGENTA CROP PROTECTION, LLC 28.1; 35.8 Atrazine; S‐Metolachlor 100‐886 BICEP MAGNUM 100 SYNGENTA CROP PROTECTION, LLC 33.7; 26.1 Atrazine; S‐Metolachlor 100‐898 AGRI‐MEK 0.15 EC MITICIDE/INSECTICIDE 100 SYNGENTA CROP PROTECTION, LLC 2 Abamectin 100‐903 DENIM INSECTICIDE 100 SYNGENTA CROP PROTECTION, LLC 2.15 Emamectin benzoate 100‐904 PROCLAIM INSECTICIDE 100 SYNGENTA CROP PROTECTION, LLC 5 Emamectin benzoate 100‐998 KARATE 1EC 100 SYNGENTA CROP PROTECTION, LLC 13.1 lambda‐Cyhalothrin 100‐1075 FORCE 3G INSECTICIDE 100 SYNGENTA CROP PROTECTION, LLC 3 Tefluthrin Acetochlor; Carbamothioic acid, dipropyl‐ 100‐1083 DOUBLEPLAY SELECTIVE HERBICIDE 100 SYNGENTA CROP PROTECTION, LLC 16.9; 67.8 , S‐ethyl ester 100‐1086 KARATE EC‐W INSECTICIDE 100 SYNGENTA CROP PROTECTION, LLC 13.1 lambda‐Cyhalothrin 100‐1088 SCIMITAR GC INSECTICIDE 100 SYNGENTA CROP PROTECTION, -
2,4-Dichlorophenoxyacetic Acid
2,4-Dichlorophenoxyacetic acid 2,4-Dichlorophenoxyacetic acid IUPAC (2,4-dichlorophenoxy)acetic acid name 2,4-D Other hedonal names trinoxol Identifiers CAS [94-75-7] number SMILES OC(COC1=CC=C(Cl)C=C1Cl)=O ChemSpider 1441 ID Properties Molecular C H Cl O formula 8 6 2 3 Molar mass 221.04 g mol−1 Appearance white to yellow powder Melting point 140.5 °C (413.5 K) Boiling 160 °C (0.4 mm Hg) point Solubility in 900 mg/L (25 °C) water Related compounds Related 2,4,5-T, Dichlorprop compounds Except where noted otherwise, data are given for materials in their standard state (at 25 °C, 100 kPa) 2,4-Dichlorophenoxyacetic acid (2,4-D) is a common systemic herbicide used in the control of broadleaf weeds. It is the most widely used herbicide in the world, and the third most commonly used in North America.[1] 2,4-D is also an important synthetic auxin, often used in laboratories for plant research and as a supplement in plant cell culture media such as MS medium. History 2,4-D was developed during World War II by a British team at Rothamsted Experimental Station, under the leadership of Judah Hirsch Quastel, aiming to increase crop yields for a nation at war.[citation needed] When it was commercially released in 1946, it became the first successful selective herbicide and allowed for greatly enhanced weed control in wheat, maize (corn), rice, and similar cereal grass crop, because it only kills dicots, leaving behind monocots. Mechanism of herbicide action 2,4-D is a synthetic auxin, which is a class of plant growth regulators. -
Growth Regulation and Other Secondary Effects of Herbicides Edivaldo D
Weed Science 2010 58:351–354 Growth Regulation and Other Secondary Effects of Herbicides Edivaldo D. Velini, Maria L. B. Trindade, Luis Rodrigo M. Barberis, and Stephen O. Duke* As all herbicides act on pathways or processes crucial to plants, in an inhibitory or stimulatory way, low doses of any herbicide might be used to beneficially modulate plant growth, development, or composition. Glyphosate, the most used herbicide in the world, is widely applied at low rates to ripen sugarcane. Low rates of glyphosate also can stimulate plant growth (this effect is called hormesis). When applied at recommended rates for weed control, glyphosate can inhibit rust diseases in glyphosate-resistant wheat and soybean. Fluridone blocks carotenoid biosynthesis by inhibition of phytoene desaturase and is effective in reducing the production of abscisic acid in drought-stressed plants. Among the acetolactate synthase inhibitors, sulfometuron-methyl is widely used to ripen sugarcane and imidazolinones can be used to suppress turf species growth. The application of protoporphyrinogen oxidase inhibitors can trigger plant defenses against pathogens. Glufosinate, a glutamine synthetase inhibitor, is also known to improve the control of plant diseases. Auxin agonists (i.e., dicamba and 2,4-D) are effective, low-cost plant growth regulators. Currently, auxin agonists are still used in tissue cultures to induce somatic embryogenesis and to control fruit ripening, to reduce drop of fruits, to enlarge fruit size, or to extend the harvest period in citrus orchards. At low doses, triazine herbicides stimulate growth through beneficial effects on nitrogen metabolism and through auxin-like effects. Thus, sublethal doses of several herbicides have applications other than weed control. -
A Novel Role of Ethephon in Controlling the Noxious Weed Ipomoea Cairica
www.nature.com/scientificreports OPEN A novel role of ethephon in controlling the noxious weed Ipomoea cairica (Linn.) Sweet Received: 09 April 2015 1,* 1,* 1,3,* 2 1 Accepted: 22 May 2015 Zhong-Yu Sun , Tai-Jie Zhang , Jin-Quan Su , Wah Soon Chow , Jia-Qin Liu , 1 1 3 1 Published: 18 June 2015 Li-Ling Chen , Wei-Hua Li , Shao-Lin Peng & Chang-Lian Peng Several auxin herbicides, such as 2, 4-D and dicamba, have been used to eradicate an exotic invasive weed Ipomoea cairica in subtropical China, but restraining the re-explosion of this weed is still a challenge. Since ethylene is one of the major intermediate functioning products during the eradication process, we explored the possibility, mechanism and efficiency of using ethephon which can release ethylene to control Ipomoea cairica. The results of the pot experiment showed that 7.2 g /L ethephon could totally kill Ipomoea cairica including the stems and roots. The water culture experiment indicated that ethephon released an abundance of ethylene directly in leaves and caused increases in electrolyte leakage, 1-aminocyclopropane-1-carboxylic acid (ACC), abscisic acid (ABA) and H2O2 and decreases in chlorophyll content and photosynthetic activity, finally leading to the death of Ipomoea cairica. The field experiment showed that the theoretical effective concentration of ethephon for controlling Ipomoea cairica (weed control efficacy, WCE = 98%) was 4.06 g/L and the half inhibitory concentration (I50) was 0.56 g/L. More than 50% of the accompanying species were insensitive to the phytotoxicity of ethephon. Therefore, ethephon is an excellent alternative herbicide for controlling Ipomoea cairica. -
INDEX to PESTICIDE TYPES and FAMILIES and PART 180 TOLERANCE INFORMATION of PESTICIDE CHEMICALS in FOOD and FEED COMMODITIES
US Environmental Protection Agency Office of Pesticide Programs INDEX to PESTICIDE TYPES and FAMILIES and PART 180 TOLERANCE INFORMATION of PESTICIDE CHEMICALS in FOOD and FEED COMMODITIES Note: Pesticide tolerance information is updated in the Code of Federal Regulations on a weekly basis. EPA plans to update these indexes biannually. These indexes are current as of the date indicated in the pdf file. For the latest information on pesticide tolerances, please check the electronic Code of Federal Regulations (eCFR) at http://www.access.gpo.gov/nara/cfr/waisidx_07/40cfrv23_07.html 1 40 CFR Type Family Common name CAS Number PC code 180.163 Acaricide bridged diphenyl Dicofol (1,1-Bis(chlorophenyl)-2,2,2-trichloroethanol) 115-32-2 10501 180.198 Acaricide phosphonate Trichlorfon 52-68-6 57901 180.259 Acaricide sulfite ester Propargite 2312-35-8 97601 180.446 Acaricide tetrazine Clofentezine 74115-24-5 125501 180.448 Acaricide thiazolidine Hexythiazox 78587-05-0 128849 180.517 Acaricide phenylpyrazole Fipronil 120068-37-3 129121 180.566 Acaricide pyrazole Fenpyroximate 134098-61-6 129131 180.572 Acaricide carbazate Bifenazate 149877-41-8 586 180.593 Acaricide unclassified Etoxazole 153233-91-1 107091 180.599 Acaricide unclassified Acequinocyl 57960-19-7 6329 180.341 Acaricide, fungicide dinitrophenol Dinocap (2, 4-Dinitro-6-octylphenyl crotonate and 2,6-dinitro-4- 39300-45-3 36001 octylphenyl crotonate} 180.111 Acaricide, insecticide organophosphorus Malathion 121-75-5 57701 180.182 Acaricide, insecticide cyclodiene Endosulfan 115-29-7 79401 -
California Restricted Materials Requirements (English)
CALIFORNIA RESTRICTED MATERIALS REQUIREMENTS FEDERAL RESTRICTED USE PESTICIDES RESTRICTED USE PESTICIDE A (Included by reference as California Restricted Materials) DUE TO (reason for restricted use classification) Pesticides display the RESTRICTED USE PESTICIDE (RUP) statement on For retail sale to and use only by Certified Applicators or the pesticide container similar to the statement shown here. RUPs require an persons under their direct supervision and only for those RUP statement enclosed in a box, at the top of the front panel of the label. uses covered by the Certified Applicator's certification. Some product labels require a Certified Applicator be “physically present” at the use site. B CALIFORNIA RESTRICTED MATERIALS This section is written in a quick reference format; refer to Title 3, California Code of Regulations (3 CCR) section 6400 for complete text. Acrolein, labeled for use as an aquatic Chlorpyrifos, labeled for the Metam sodium, labeled for the Potassium n-methyldithiocarbamate herbicide production of an agricultural production of agricultural plant (metam-potassium), labeled for the Aldicarb – unregistered commodity commodities production of agricultural plant All dust (except products containing Dazomet, labeled for the production Methamidophos – unregistered commodities only exempt pesticides)** of agricultural plant commodities Methidathion Propanil (3,4-dichloropropionanilide) Aluminum phosphide Dicamba* Methomyl†† Sodium cyanide Any pesticide containing active 2,4-dichlorophenoxyacetic acid Methyl bromide Sodium -
AP-42, CH 9.2.2: Pesticide Application
9.2.2PesticideApplication 9.2.2.1General1-2 Pesticidesaresubstancesormixturesusedtocontrolplantandanimallifeforthepurposesof increasingandimprovingagriculturalproduction,protectingpublichealthfrompest-bornediseaseand discomfort,reducingpropertydamagecausedbypests,andimprovingtheaestheticqualityofoutdoor orindoorsurroundings.Pesticidesareusedwidelyinagriculture,byhomeowners,byindustry,andby governmentagencies.Thelargestusageofchemicalswithpesticidalactivity,byweightof"active ingredient"(AI),isinagriculture.Agriculturalpesticidesareusedforcost-effectivecontrolofweeds, insects,mites,fungi,nematodes,andotherthreatstotheyield,quality,orsafetyoffood.Theannual U.S.usageofpesticideAIs(i.e.,insecticides,herbicides,andfungicides)isover800millionpounds. AiremissionsfrompesticideusearisebecauseofthevolatilenatureofmanyAIs,solvents, andotheradditivesusedinformulations,andofthedustynatureofsomeformulations.Mostmodern pesticidesareorganiccompounds.EmissionscanresultdirectlyduringapplicationorastheAIor solventvolatilizesovertimefromsoilandvegetation.Thisdiscussionwillfocusonemissionfactors forvolatilization.Thereareinsufficientdataavailableonparticulateemissionstopermitemission factordevelopment. 9.2.2.2ProcessDescription3-6 ApplicationMethods- Pesticideapplicationmethodsvaryaccordingtothetargetpestandtothecroporothervalue tobeprotected.Insomecases,thepesticideisapplieddirectlytothepest,andinotherstothehost plant.Instillothers,itisusedonthesoilorinanenclosedairspace.Pesticidemanufacturershave developedvariousformulationsofAIstomeetboththepestcontrolneedsandthepreferred -
Advances in Enzyme-Based Biosensors for Pesticide Detection
biosensors Review Advances in Enzyme-Based Biosensors for Pesticide Detection Bogdan Bucur 1 ID , Florentina-Daniela Munteanu 2 ID , Jean-Louis Marty 3,* and Alina Vasilescu 4 ID 1 National Institute of Research and Development for Biological Sciences, Centre of Bioanalysis, 296 Splaiul Independentei, 060031 Bucharest, Romania; [email protected] 2 Faculty of Food Engineering, Tourism and Environmental Protection, “Aurel Vlaicu” University of Arad, Elena Dragoi, No. 2, 310330 Arad, Romania; fl[email protected] 3 BAE Laboratory, Université de Perpignan via Domitia, 52 Avenue Paul Alduy, 66860 Perpignan, France 4 International Centre of Biodynamics, 1B Intrarea Portocalelor, 060101 Bucharest, Romania; [email protected] * Correspondence: [email protected]; Tel.: +33-468-66-1756 Received: 28 February 2018; Accepted: 20 March 2018; Published: 22 March 2018 Abstract: The intensive use of toxic and remanent pesticides in agriculture has prompted research into novel performant, yet cost-effective and fast analytical tools to control the pesticide residue levels in the environment and food. In this context, biosensors based on enzyme inhibition have been proposed as adequate analytical devices with the added advantage of using the toxicity of pesticides for detection purposes, being more “biologically relevant” than standard chromatographic methods. This review proposes an overview of recent advances in the development of biosensors exploiting the inhibition of cholinesterases, photosynthetic system II, alkaline phosphatase, cytochrome P450A1, peroxidase, tyrosinase, laccase, urease, and aldehyde dehydrogenase. While various strategies have been employed to detect pesticides from different classes (organophosphates, carbamates, dithiocarbamates, triazines, phenylureas, diazines, or phenols), the number of practical applications and the variety of environmental and food samples tested remains limited. -
Developmental Ramifications of Dithiocarbamate Pesticide Exposure in Zebrafish
AN ABSTRACT OF THE DISSERTATION OF Fred Tilton for the degree of Doctor of Philosophy in Toxicology presented on May 24, 2006. Title:Developmental Ramifications of Dithiocarbamate Pesticide Exposure in Zebrafish. Redacted for Privacy Abstract approved Robert Dithiocarbamates are widely used agricultural pesticides, industrial chemicals and effluent additives. DTCs and their related compounds have historical and current relevance in clinical and experimental medicine. DTC developmental toxicity is well established, but poorly understood. Dithiocarbamates according to the U.S. EPA have a mechanism of action involving, "the inhibition of metal-dependent and sulfhydryl enzyme systems in fungi, bacteria, plants, as well as mammals." We hypothesized that by using the zebrafish development model we could better define the mechanism of action of DTCs and for the first time establish a molecular understanding of DTC developmental toxicity in vertebrates. We have established that all types of dithiocarbamate pesticides and some degradation products have the potential to elicit a common toxic effect on development resulting in a distorted notochord and a significant impact to the body axis. We provide evidence to support the hypothesis that metal chelation is not the primary mechanism of action by which DTCs impact the developing vertebrate. By manipulating the exposure window of zebrafish we hypothesized that somitogenesis was the targeted developmental process. We tested this by using the Affymetrix microarray to observe gene expression induced by the N-methyl dithiocarbamate, metam sodium (NaM). Throughout this process it is clear that genes related to muscle development are perturbed. These gene signatures are consistent with the morphological changes observed in larval and adult animals and that somitogenesis is the developmental target. -
AP-42, Vol. 1, Final Background Document for Pesticide Application
Emission Factor Documentation for AP-42 Section 9.2.2 Pesticide Application Final Report For U.S. Environmental Protection Agency Office of Air Quality Planning and Standards Emission Inventory Branch EPA Contract No. 68-D2-0159 Work Assignment No. I-08 MRI Project No. 4601-08 September 1994 Emission Factor Documentation for AP-42 Section 9.2.2 Pesticide Application Final Report For U.S. Environmental Protection Agency Office of Air Quality Planning and Standards Emission Inventory Branch Research Triangle Park, NC 27711 Attn: Mr. Dallas Safriet (MD-14) Emission Factor and Methodology EPA Contract No. 68-D2-0159 Work Assignment No. I-08 MRI Project No. 4601-08 September 1994 NOTICE The information in this document has been funded wholly or in part by the United States Environmental Protection Agency under Contract No. 68-D2-0159 to Midwest Research Institute. It has been subjected to the Agency's peer and administrative review, and it has been approved for publication as an EPA document. Mention of trade names or commercial products does not constitute endorsement or recommendation for use. iii iv PREFACE This report was prepared by Midwest Research Institute (MRI) for the Office of Air Quality Planning and Standards (OAQPS), U.S. Environmental Protection Agency (EPA), under Contract No. 68-D2-0159, Assignment No. 005 and I-08. Mr. Dallas Safriet was the EPA work assignment manager for this project. Approved for: MIDWEST RESEARCH INSTITUTE Roy M. Neulicht Program Manager Environmental Engineering Department Jeff Shular Director, Environmental Engineering Department September 29, 1994 v vi CONTENTS LIST OF FIGURES ................................................ viii LIST OF TABLES ................................................ -
2019 Minnesota Chemicals of High Concern List
Minnesota Department of Health, Chemicals of High Concern List, 2019 Persistent, Bioaccumulative, Toxic (PBT) or very Persistent, very High Production CAS Bioaccumulative Use Example(s) and/or Volume (HPV) Number Chemical Name Health Endpoint(s) (vPvB) Source(s) Chemical Class Chemical1 Maine (CA Prop 65; IARC; IRIS; NTP Wood and textiles finishes, Cancer, Respiratory 11th ROC); WA Appen1; WA CHCC; disinfection, tissue 50-00-0 Formaldehyde x system, Eye irritant Minnesota HRV; Minnesota RAA preservative Gastrointestinal Minnesota HRL Contaminant 50-00-0 Formaldehyde (in water) system EU Category 1 Endocrine disruptor pesticide 50-29-3 DDT, technical, p,p'DDT Endocrine system Maine (CA Prop 65; IARC; IRIS; NTP PAH (chem-class) 11th ROC; OSPAR Chemicals of Concern; EuC Endocrine Disruptor Cancer, Endocrine Priority List; EPA Final PBT Rule for 50-32-8 Benzo(a)pyrene x x system TRI; EPA Priority PBT); Oregon P3 List; WA Appen1; Minnesota HRV WA Appen1; Minnesota HRL Dyes and diaminophenol mfg, wood preservation, 51-28-5 2,4-Dinitrophenol Eyes pesticide, pharmaceutical Maine (CA Prop 65; IARC; NTP 11th Preparation of amino resins, 51-79-6 Urethane (Ethyl carbamate) Cancer, Development ROC); WA Appen1 solubilizer, chemical intermediate Maine (CA Prop 65; IARC; IRIS; NTP Research; PAH (chem-class) 11th ROC; EPA Final PBT Rule for 53-70-3 Dibenzo(a,h)anthracene Cancer x TRI; WA PBT List; OSPAR Chemicals of Concern); WA Appen1; Oregon P3 List Maine (CA Prop 65; NTP 11th ROC); Research 53-96-3 2-Acetylaminofluorene Cancer WA Appen1 Maine (CA Prop 65; IARC; IRIS; NTP Lubricant, antioxidant, 55-18-5 N-Nitrosodiethylamine Cancer 11th ROC); WA Appen1 plastics stabilizer Maine (CA Prop 65; IRIS; NTP 11th Pesticide (EPA reg. -
INSECT, WEED, Anddisease CONTROL in TURFGRASS
SC-039 5/17 WEED,INSECT, and DISEASE CONTROL in TURFGRASS 2017–18 WEED, INSECT, and DISEASE CONTROL in TURFGRASS Editor Casey Reynolds, Assistant Professor and Extension Turfgrass Specialist Authors Casey Reynolds, Assistant Professor and Extension Turfgrass Specialist Matt Elmore, Assistant Professor and Extension Turfgrass Specialist Young-Ki Jo, Associate Professor and Extension Turfgrass Specialist Diane Silcox Reynolds, Post-doctoral Research Associate, Entomology AggieTurf: http://aggieturf.tamu.edu Contents Introduction . 1 Herbicide Mode of Action (MOA) classification . 3 Herbicides for general control of grassy and broadleaf weeds . 4 Preemergence herbicides for grassy and broadleaf weeds . 4 Selective postemergence herbicides . 9 Synthetic auxin postemergence herbicides for broadleaf weeds . 19 Product formulations containing synthetic auxin herbicides . 21 Nonsynthetic auxin postemergence herbicides for broadleaf weeds . 23 Nonselective herbicides for general weed control . 24 Herbicides for commonly occurring weeds . 25 Crabgrass (Digitaria spp ). 25 Goosegrass (Eleusine indica) . 27 Sandbur (Cenchrus spp ). 30 Annual bluegrass (Poa annua L ). 33 Dallisgrass (Paspalum dilatatum Poir ). 39 WEEDS Bermudagrass (Cynodon spp ). 41 Nutsedge (Cyperus spp ). and kyllinga (Kyllinga spp ). 43 Khakiweed and mat chafflower (Alternanthera spp ). 46 Herbicides containing sulfentrazone . 47 Herbicides containing quinclorac . 48 Turfgrass tolerance to postemergence herbicides . 49 Plant growth regulators . 51 Insect pests in turfgrasses . 53 Insecticide Mode of Action (MOA) classification . 55 Insecticides registered for use in turfgrasses . 56 Ants . 56 Armyworms . 58 Billbugs . 61 Black turfgrass ataenius . 63 Chinch bugs . 66 Cutworms . 69 Green June beetles . 72 Mealybugs . 74 Mites . 75 INSECTS Mole crickets . 76 Red imported fire ants . 79 Sod webworms . 81 White grubs . 84 Diseases in Texas turfgrasses . 86 Fungicide Mode of Action (MOA) classification .