DOCSLIB.ORG

Multiple Medications

Total Page：16

File Type：pdf, Size：1020Kb

Download full-text PDF Read full-text
Multiple Medications Unless specifically noted, Medications need to be discontinued for 48 hours prior to exam and 24 hours post exam ALL OVER THE COUNTER SUPPLEMENTS OFF FOR 5 DAYS (examples: papaya enzymes, turmeric, cinnamon, vit. E, etc.) Abilify Asprin- 24 hours ConZip Abciximab- 24 hours Atomoxetine Coumadin- 5 days Acetophenazine Aventyl Cyclobenzaprine Actremra- 5 days Azilect Cycloserine Actron- 24 hours Bayer Asprin- 24 hours Cyclosporine Adapin Benzphentamine Cylert Adderall Bextra-24 hours Cymbalta Adipenx Bontril Dabigatran Advil- 24 hours Brilinta- 5 days Dalteparin- 5 days Aflaxin- 24 hours Brufen- 24 hours Daypro- 24 hours Aggrenox- 7 days Brupropion Dayrun- 24 hours Aleve- 24 hours Busiprone Daytrana Algix – 24 hours BuSpar Demerol Alka Seltzer- 24 hours Cambia-24 hours Desipramine Amantadine Cataflam- 24 hours Desoxyn Amitriptyline Ceeoxx- 24 hours Desvenlafaxine Amitriptyline + Chlordiazepoxide Celebrex- 24 hours Desyrel Amitriptyline + Perphenazine Celecoxib- 24 hours Dexedrine Amoxapine Celexa Dexketoprofen- 24 hours Amphetamine Ceoxx- 24 hours Dexmethylphenidate Amphetamine mixture Chlorpromazine Dextroamphetamine Amrix Chlorprothixene Diclofenac- 24 hours Anafranil Cilostazol- 5 days Didrex Anaprox- 24 hours Citalopram Diethylprophm Ansaid- 24 hours Clinoril- 24 hours Diflunisal- 24 hours Apixaban- Clomipramine Disalcid- 24 hours Aripiprazole Clopidogrel- 5 days Dividose Arixtra- 5 days Clopidogrel Bisulfate- 5 days Dolobid- 24 hours Armodafinil Clotam Rapid- 24 hours Dopram Arthrotec-24 hours Clozapine Doxapram Asaid-24 hours Clozaril Doxepin Asendin Compazine Droperidol Duexis-24 hours Fluphenazine Latuda Duloxetine Flurbiprofen- 24 hours Levoprome Duragesic Flurwood- 24 hours Lexapro Duraprox- 24 hours Fluvoxamine Licofel- 24 hours Dynastat- 24 hours Focalin XR Licofelone- 24 hours Effexor Fondaparinux- 5 days Limbitrol DS Effient- 5 days Fragmin- 5 days Lithium Elavil Froben – 24 hours Lithobid Eldepryl Furazolidone Lithotabs Eliquis Furoxone Lodine (Lodine XL)- 24 hours Enoxaparin-15 hours Geodon Lonamin Ephedrine Haldol Lornoxicam- 24 hours Escitalopram Haloperidol Lovenox- 15 hours Eskalith Heparin- Radiologist Preference Loxapine Ethopropazine Ibuprofen- 24 hours Loxitane Etodolac- 24 hours Imipenem-cilastatin Loxomac-24 hours Etoricoxib- 24 hours Imipramine Loxonin- 24 hours Etrafon Indocin (Indocin SR/IV)- 24 hours Loxoprofen- 24 hours Eutonyl Indomethacin- 24 hours Ludiomil Excedrin Migraine- 24 hours Indomethegan- 24 hours Lumiracoxib- 24 hours Famotidine- 24 hours Invega Luvox Feldene- 24 hours Isocarboxazid Maprotiline Fenoprofen- 24 hours Isoniazid Marplan Fenopron- 24 hours Jantoven- 1-3 days (check INR prior) Matulane Fentanyl Keral- 24 hours Meclofenamic Acid- 24 hours Fexmid Ketoflam- 24 hours Meclomen- 24 hours Firocoxib- 24 hours Ketoprofen- 24 hours Medipren- 5 days Fish Oil- 5 days Ketorolac- 24 hours Mefenamic Acid- 24 hours Flexeril Khedezla Mellaril Fluoxetine Larasidone Melox- 24 hours Fluoxetine Symbol Largon Meloxicam- 24 hours Meperidine Nimesulide- 24 hours Piroxicam- 24 hours Meridia Norpramin Placil Mesoridazine Nortriptyline Plavix- 5 days Mesulid- 24 hours Nuprin- 24 hours Pletal- 5 days Metadate Nurofen- 24 hours Ponstel- 24 hours Methamphetamine Nuvigil Prasugrel- 5 days Methdilazine Ocuvite- 5 days (vitamin) Prevacid-24 hours Methotraxate- 5 days Olanzapine Previcox- 24 hours Methylphenidate Orap Prexige- 24 hours Midol 24 hours Orudis- 24 hours Primaxin Milnacipran Oruvail- 24 hours Pristiq Mirtazapine Oxaporozin- 24 hours Procarbazine Moban Oxeno- 24 hours Prochlorperazine Mobic- 24 hours Paliperidone Proclorperazine Mobiflex-5 days Pamelor Prolixin Modafinil Paradaxa- 1-2 days if CrCl >50, 3-5 Promazine days if CrCl <50 Molindone Promethazine Parecoxib- 24 hours Mono-Gesic- 5 days Protriptyline Pargyline Motrin- 5 days Provigil Parnate Movalis-5 days Prozac Paroxetine Nabumetone-24 hours Pseudoephedrine Parsidol Nalfron- 24 hours Quetiapine Paxil NapraPac-24 hours Rasagiline Pemoline Naprelan- 24 hours Recoxa- 24 hours Permitil Naprosyn- 24 hours Reopro-24 hours Perphenazine Naproxen- 24 hours Relafen- 24 hours Phendimetrazine Nardil Remeron Phenelzine Navane Rheumatrex-5 days Phenergan Nefazodone Risperdal Phentermine Neoral Risperidone Pimozide Nimalox- 24 hours Ritalin Rivaroxaban Tenoxicam- 24 hours Ultram Rofecoxib- 24 hours Theochron Urbifen- 24 hours Rybix ODT Theo-Dur Valdecoxib- 24 hours Salflex- 24 hours Theophylline VanSpar Salsalate- 24 hours Thiethylperazine Venlafaxine Salsltab- 24 hours Thioidazine Vesprin Sandimmune Thiothixine Viibryd Sarafem Thorazine Vilazodone HCL Savella Ticagrelor- 5 days Vimovo Sawpalmetto- 2 weeks Ticlid- 7 days Vioxx- 24 hours Selegiline Ticlopidine- 7 days Vivactil Serentil Tindal Voltaren (Voltaren- XR)- 24 hours Seromycin Tizanidine Warfarin- 5 days Seroquel Tocilizumab- 5 days Wellbutrin Sertraline Tofranil Xarelto Serzone Tolectin- 24 hours Xatmep – 5 days Sibutramine Tolfenamic Acid- 24 hours Xefo- 24 hours Sinequan Tolmetin- 24 hours Zanaflex Slo-bid Toradol (Toradol IV/IM)- 24 hours Zelapar Slo-Phyllin Torecan Ziprasidone Sparine Tramadol Zipsor-24 hours Sprix-24 hours Tranylcypromine Zoloft Stelazine Trazodone Zyban Strattera Trexall – 5 days Zyprexa Sulide- 24 hours Trifluoperazine Sulindac- 24 hours Triflupromazine Surmontil Trilafon Symmetrel Trimipramine Tacaryl Tufnll- 24 hours Taractan Tumeric- 5 days Tenaute Ultracet .
Load more

Recommended publications
  • What Are the Acute Treatments for Migraine and How Are They Used?
    2. Acute Treatment CQ II-2-1 What are the acute treatments for migraine and how are they used? Recommendation The mainstay of acute treatment for migraine is pharmacotherapy. The drugs used include (1) acetaminophen, (2) non-steroidal anti-inflammatory drugs (NSAIDs), (3) ergotamines, (4) triptans and (5) antiemetics. Stratified treatment according to the severity of migraine is recommended: use NSAIDs such as aspirin and naproxen for mild to moderate headache, and use triptans for moderate to severe headache, or even mild to moderate headache when NSAIDs were ineffective in the past. It is necessary to give guidance and cautions to patients having acute attacks, and explain the methods of using medications (timing, dose, frequency of use) and medication use during pregnancy and breast-feeding. Grade A Background and Objective The objective of acute treatment is to resolve the migraine attack completely and rapidly and restore the patient’s normal functions. An ideal treatment should have the following characteristics: (1) resolves pain and associated symptoms rapidly; (2) is consistently effective; (3) no recurrence; (4) no need for additional use of medication; (5) no adverse effects; (6) can be administered by the patients themselves; and (7) low cost. Literature was searched to identify acute treatments that satisfy the above conditions. Comments and Evidence The acute treatment drugs for migraine generally include (1) acetaminophens, (2) non-steroidal anti-inflammatory drugs (NSAIDs), (3) ergotamines, (4) triptans, and (5) antiemetics. For severe migraines including status migrainosus and migraine attacks refractory to treatment, (6) anesthetics, and (7) corticosteroids (dexamethasone) are used (Tables 1 and 2).1)-9) There are two approaches to the selection and sequencing of these medications: “step care” and “stratified care”.
    [Show full text]
  • AMANTADIP\Dle: on Nemolebtic-II[WDUCED CATALEPSY UV the RAT
    J. Fac. Plmnz. Istanbul lstanbul Ecz. Fak. Mec. 29, 1 (1993) 29, 1 (1993) THE EFFECT OF CYPROHEPT~~AWEb AMANTADIP\dlE: ON NEmOLEBTIC-II[WDUCED CATALEPSY UV THE RAT SUMMARY Catalepsy is not a unitary phenomenon. With respect to biochemical mechanisms and neurotransmitter sytems, the origin of this behavioural response varies according to different substances which cause catalepsy. For example, the catdeptogenic effect of ne- uroleptics has been related to the blockage of striatal doparnine receptors. Neurophysio- logical and biochemical data have shown that a mechanism caused by gama-aminobuty- ric acid and serotonin have been effective in the proper functioning of the dopaminergic nigrosbriatal tract. We have studied the effects of cyproheptadine, a serotonin antagonist, and of amantadin which is used in the treatment of Parkinson syndrome on the catalepsy indu- ced by trifluoperazine, a phenothiazin compound, and pimozid, a drug used as a neuro- leptic. It was observed that when rats were pretreated with cyproheptadine as well as amantadiie, the catalepsy induced by the above neuroleptic drugs was attenuated. The results of our study show that substances which have an effect on the seroto- nergic and dopaminergic systems also play a role on cases of catalepsy induced by the neuroleptic drugs mentioned above. (*) Faculty of Pharmacy, Depament of Pharmacology, University of Istanbul, 31152, Istanbul, Turkey Katalepsi bir tek nedene bagh OWortaya qllian bit durum degildir. Biyokimya- sal ve norotransmitter sistemler agsmdan bu davran~glnsebebi, katalepsi olughYan qe- gitli maddeler iqin fad&&. Omegin noroleptiklerin kataleptojenik etkisi striatal dopa- min reseptorlerinin blokaj~nabajjlanmqtx. Niirofizyolojik ve biyokimyasal veriler do- pamine jik nigro-striatal sistemin duzenli $&$masmda gma-aminobutirik asid ve sero- tonin'e bagh olan bir mekanizmmn etkili oldugunu gostermigtir.
    [Show full text]
  • Menstrually Related and Nonmenstrual Migraines in A
    MENSTRUALLY RELATED AND NONMENSTRUAL MIGRAINES IN A FREQUENT MIGRAINE POPULATION: FEATURES, CORRELATES, AND ACUTE TREATMENT DIFFERENCES A dissertation presented to the faculty of the College of Arts and Sciences of Ohio University In partial fulfillment of the requirements for the degree Doctor of Philosophy Brenda F. Pinkerman March 2006 This dissertation entitled MENSTRUALLY RELATED AND NONMENSTRUAL MIGRAINES IN A FREQUENT MIGRAINE POPULATION: FEATURES, CORRELATES, AND ACUTE TREATMENT DIFFERENCES by BRENDA F. PINKERMAN has been approved for the Department of Psychology and the College of Arts and Sciences by Kenneth A. Holroyd Distinguished Professor of Psychology Benjamin M. Ogles Interim Dean, College of Arts and Sciences PINKERMAN, BRENDA F. Ph.D. March 2006. Clinical Psychology Menstrually Related and Nonmenstrual Migraines in a Frequent Migraine Population: Features, Correlates, and Acute Treatment Differences (307 pp.) Director of Dissertation: Kenneth A. Holroyd This research describes and compares menstrually related migraines as defined by recent proposed guidelines of the International Headache Society (IHS, 2004) to nonmenstrual migraines in a population of female migraineurs with frequent, disabling migraines. Migraines are compared by frequency per day of the menstrual cycle, headache features, use of abortive and rescue medications, and acute migraine treatment outcomes. In addition, this study explores predictors of acute treatment response and headache recurrence within 24 hours following acute migraine treatment for menstrually related migraines. Participants are 107 menstruating female migaineurs who met IHS (2004) proposed criteria for menstrually related migraines and completed headache diaries using hand-held computers. Diary data are analyzed using repeated measures logistic regression. The frequency of migraines is significantly increased during the perimenstrual period, and menstrually related migraines are of longer duration and greater frequency with longer lasting disability than nonmenstrual migraines.
    [Show full text]
  • Use of Analgesics in Exotic Felids Edward C. Ramsay, DVM Professor, Department of Small Animal Clinical Sciences, University Of
    Use of Analgesics in Exotic Felids Formatted: Centered Edward C. Ramsay, DVM Professor, Department of Small Animal Clinical Sciences, University of Tennessee, and Consulting Veterinarian, Knoxville Zoological Gardens, Knoxville, Tennessee. Corresponding address: Ed Ramsay Dept. of Sm Animal Clin Sci C247, Veterinary Teaching Hospital University of Tennessee Knoxville, TN 37996-4544 Phone: 865-755-8219 FAX: 865-974-5554 Email: [email protected] 2 Treatment of pain in domestic and non-domestic cats has been a challenge for the clinician. Many cat species are stoic and show few or very subtle external signs of pain. Additionally, the adverse effects of nonsteroidal antiinflammatory drugs (NSAIDs) in domestic cats are well documented and have discouraged many practitioners from trying novel NSAID’s in exotic felids. As in other animals, each cat’s response to pain and analgesics will vary, necessitating an individualized treatment plan. As a rule, always treat painful felids to effect, and not by rote reliance on published dosages. It is frequently necessary to try different agents and combinations to find which produces the optimal analgesic effect in exotic felids. In order to minimize adverse effects, it is desirable to work toward treatment with the lowest effective dose when treating chronic pain. Non-steroidal Antiinflammatory Drugs NSAIDs are antiinflammatory drugs which act both centrally and peripherally. The primary effects are believed to be caused by their ability to inhibit cyclooxygenase (COX) enzymes in the arachidonic acid metabolism cascade. The COX-1 isoform is regarded as constitutive (continuously expressed) and is responsible for many homeostatic processes, such as maintenance of gastric mucosal integrity, platelet function, and renal autoregulation.
    [Show full text]
  • Delusional Parasitosis Mimicking Cutaneous Infestation in Elderly Patients
    LESSONS FROM PRACTICE LESSONS FROM PRACTICE Delusional parasitosis mimicking cutaneous infestation in elderly patients Clinical records Patient 1 Patient 3 An 88-year-old man gave a 12-month history of seeing insects attacking An 81-year-old woman was referred with a persistent belief that his legs and crawling along the floor of his house. He described these she had scabies and lice infestation of her eyes, nose, arms and insects as 4 cm long, black and white bugs with beaks, which pecked anus. This resulted in her persistently washing her clothes and at hisThe legs, Medical causing Journal wounds. of He Australia often felt ISSN:a sharp 0025-729X stinging sensation 18 herself and reporting the retirement village where she lived to the heraldingAugust their 2003 presence. 179 4 209-210 He also had burning pain in both legs below Health Department. She had received anti-scabies treatment the knees. He had had his house fumigated twice in the previous year empirically. ©The Medical Journal of Australia 2003 www.mja.com.au andLessons put various from chemicals practice across his doorways and bed to ward off the She had a long history of severe depression after the death of her bugs. He described no other hallucinations or delusions. husband, for which she took doxepin. She had paranoid ideation He was not using any regular medications and had never been a about her neighbours and saw things crawling down the walls, consumer of alcohol. He lived alone and managed all activities of and had moved residences several times to avoid these problems.
    [Show full text]
  • Schizophrenia Care Guide
    August 2015 CCHCS/DHCS Care Guide: Schizophrenia SUMMARY DECISION SUPPORT PATIENT EDUCATION/SELF MANAGEMENT GOALS ALERTS Minimize frequency and severity of psychotic episodes Suicidal ideation or gestures Encourage medication adherence Abnormal movements Manage medication side effects Delusions Monitor as clinically appropriate Neuroleptic Malignant Syndrome Danger to self or others DIAGNOSTIC CRITERIA/EVALUATION (PER DSM V) 1. Rule out delirium or other medical illnesses mimicking schizophrenia (see page 5), medications or drugs of abuse causing psychosis (see page 6), other mental illness causes of psychosis, e.g., Bipolar Mania or Depression, Major Depression, PTSD, borderline personality disorder (see page 4). Ideas in patients (even odd ideas) that we disagree with can be learned and are therefore not necessarily signs of schizophrenia. Schizophrenia is a world-wide phenomenon that can occur in cultures with widely differing ideas. 2. Diagnosis is made based on the following: (Criteria A and B must be met) A. Two of the following symptoms/signs must be present over much of at least one month (unless treated), with a significant impact on social or occupational functioning, over at least a 6-month period of time: Delusions, Hallucinations, Disorganized Speech, Negative symptoms (social withdrawal, poverty of thought, etc.), severely disorganized or catatonic behavior. B. At least one of the symptoms/signs should be Delusions, Hallucinations, or Disorganized Speech. TREATMENT OPTIONS MEDICATIONS Informed consent for psychotropic
    [Show full text]
  • Acute Migraine Treatment
    Acute Migraine Treatment Morris Levin, MD Professor of Neurology Director, Headache Center UCSF Department of Neurology San Francisco, CA Mo Levin Disclosures Consulting Royalties Allergan Oxford University Press Supernus Anadem Press Amgen Castle Connolly Med. Publishing Lilly Wiley Blackwell Mo Levin Disclosures Off label uses of medication DHE Antiemetics Zolmitriptan Learning Objectives At the end of the program attendees will be able to 1. List all important options in the acute treatment of migraine 2. Discuss the evidence and guidelines supporting the major migraine acute treatment options 3. Describe potential adverse effects and medication- medication interactions in acute migraine pharmacological treatment Case 27 y/o woman has suffered ever since she can remember from “sick headaches” . Pain is frontal, increases over time and is generally accompanied by nausea and vomiting. She feels depressed. The headache lasts the rest of the day but after sleeping through the night she awakens asymptomatic 1. Diagnosis 2. Severe Headache relief Diagnosis: What do we need to beware of? • Misdiagnosis of primary headache • Secondary causes of headache Red Flags in HA New (recent onset or change in pattern) Effort or Positional Later onset than usual (middle age or later) Meningismus, Febrile AIDS, Cancer or other known Systemic illness - Neurological or psych symptoms or signs Basic principles of Acute Therapy of Headaches • Diagnose properly, including comorbid conditions • Stratify therapy rather than treat in steps • Treat early
    [Show full text]
  • Guideline for the Treatment of Breakthrough and the Prevention of Refractory Chemotherapy-Induced Nausea and Vomiting in Children with Cancer
    This is a repository copy of Guideline for the Treatment of Breakthrough and the Prevention of Refractory Chemotherapy-induced Nausea and Vomiting in Children with Cancer. White Rose Research Online URL for this paper: https://eprints.whiterose.ac.uk/97043/ Version: Accepted Version Article: Flank, Jaqueline, Robinson, Paula, Holdsworth, Mark et al. (7 more authors) (2016) Guideline for the Treatment of Breakthrough and the Prevention of Refractory Chemotherapy-induced Nausea and Vomiting in Children with Cancer. Pediatric blood & cancer. ISSN 1545-5009 https://doi.org/10.1002/pbc.25955 Reuse Items deposited in White Rose Research Online are protected by copyright, with all rights reserved unless indicated otherwise. They may be downloaded and/or printed for private study, or other acts as permitted by national copyright laws. The publisher or other rights holders may allow further reproduction and re-use of the full text version. This is indicated by the licence information on the White Rose Research Online record for the item. Takedown If you consider content in White Rose Research Online to be in breach of UK law, please notify us by emailing [email protected] including the URL of the record and the reason for the withdrawal request. [email protected] https://eprints.whiterose.ac.uk/ Pediatric Blood & Cancer Guideline for the Treatment of Breakthrough and the Prevention of Refractory Chemotherapy-induced Nausea and Vomiting in Children with Cancer Journal: Pediatric Blood & Cancer Manuscript ID PBC-15-1233.R1 Wiley - ManuscriptFor
    [Show full text]
  • (12) Patent Application Publication (10) Pub. No.: US 2006/0024365A1 Vaya Et Al
    US 2006.0024.365A1 (19) United States (12) Patent Application Publication (10) Pub. No.: US 2006/0024365A1 Vaya et al. (43) Pub. Date: Feb. 2, 2006 (54) NOVEL DOSAGE FORM (30) Foreign Application Priority Data (76) Inventors: Navin Vaya, Gujarat (IN); Rajesh Aug. 5, 2002 (IN)................................. 699/MUM/2002 Singh Karan, Gujarat (IN); Sunil Aug. 5, 2002 (IN). ... 697/MUM/2002 Sadanand, Gujarat (IN); Vinod Kumar Jan. 22, 2003 (IN)................................... 80/MUM/2003 Gupta, Gujarat (IN) Jan. 22, 2003 (IN)................................... 82/MUM/2003 Correspondence Address: Publication Classification HEDMAN & COSTIGAN P.C. (51) Int. Cl. 1185 AVENUE OF THE AMERICAS A6IK 9/22 (2006.01) NEW YORK, NY 10036 (US) (52) U.S. Cl. .............................................................. 424/468 (22) Filed: May 19, 2005 A dosage form comprising of a high dose, high Solubility active ingredient as modified release and a low dose active ingredient as immediate release where the weight ratio of Related U.S. Application Data immediate release active ingredient and modified release active ingredient is from 1:10 to 1:15000 and the weight of (63) Continuation-in-part of application No. 10/630,446, modified release active ingredient per unit is from 500 mg to filed on Jul. 29, 2003. 1500 mg, a process for preparing the dosage form. Patent Application Publication Feb. 2, 2006 Sheet 1 of 10 US 2006/0024.365A1 FIGURE 1 FIGURE 2 FIGURE 3 Patent Application Publication Feb. 2, 2006 Sheet 2 of 10 US 2006/0024.365A1 FIGURE 4 (a) 7 FIGURE 4 (b) Patent Application Publication Feb. 2, 2006 Sheet 3 of 10 US 2006/0024.365 A1 FIGURE 5 100 ov -- 60 40 20 C 2 4.
    [Show full text]
  • Psychedelics in Psychiatry: Neuroplastic, Immunomodulatory, and Neurotransmitter Mechanismss
    Supplemental Material can be found at: /content/suppl/2020/12/18/73.1.202.DC1.html 1521-0081/73/1/202–277$35.00 https://doi.org/10.1124/pharmrev.120.000056 PHARMACOLOGICAL REVIEWS Pharmacol Rev 73:202–277, January 2021 Copyright © 2020 by The Author(s) This is an open access article distributed under the CC BY-NC Attribution 4.0 International license. ASSOCIATE EDITOR: MICHAEL NADER Psychedelics in Psychiatry: Neuroplastic, Immunomodulatory, and Neurotransmitter Mechanismss Antonio Inserra, Danilo De Gregorio, and Gabriella Gobbi Neurobiological Psychiatry Unit, Department of Psychiatry, McGill University, Montreal, Quebec, Canada Abstract ...................................................................................205 Significance Statement. ..................................................................205 I. Introduction . ..............................................................................205 A. Review Outline ........................................................................205 B. Psychiatric Disorders and the Need for Novel Pharmacotherapies .......................206 C. Psychedelic Compounds as Novel Therapeutics in Psychiatry: Overview and Comparison with Current Available Treatments . .....................................206 D. Classical or Serotonergic Psychedelics versus Nonclassical Psychedelics: Definition ......208 Downloaded from E. Dissociative Anesthetics................................................................209 F. Empathogens-Entactogens . ............................................................209
    [Show full text]
  • Blood Thinners These Medications Require up to 2 Weeks of Discontinued Use Prior to a Myelogram Procedure
    MEDICATIONS TO AVOID PRE & POST MYELOGRAMS Radiocontrast agents used in myelography have the ability to lower the seizure threshold and when used with other drugs that also have this ability, may lead to convulsions. Drugs that possess the ability to lower seizure threshold should be discontinued at least 48 hours prior to myelography if possible and should not be resumed until at least 24 hours post-procedure. Drugs are listed below. ***Always consult with your prescribing physician prior to stoppage of your medication. Phenothiazines Antidepressants MAO Inhibitors Acetophenazine (Tindal) Amitriptyline (Elavil) Furazolidone (Furoxone) Chlorpromazine (Thorazine) Amoxapine (Asendin) Isocarboxazid (Marplan) Promethazine (Phenergan) Bupropion *(Welbutrin, Zyban) Pargyline (Eutonyl) Ethopropazine (Parsidol) Clomipramine (Anafranil, Placil) Phenelzine (Nardil) Fluphenazine (Prolixin) Desipramine (Norapramin) Procarbazine (Matulane) Mesoridazine (Serentil) Doxepin (Sinequan) Tranlcypromine (Parnate) Methdilazine (Tacaryl) Imipramine (Tofranil) Perphenazine (Trilafon) Maprotiline (Ludiomil) CNS Stimulants Proclorperazine (Compazine) Nortriptyline (Pamelor) Amphetamines Promazine (Sparine) Protriptyline (Vivactil) Pemoline (Cylert) Promethazine (Phenergan) Trimipramine (Surmontil) Thiethylperazine (Torecan) Thioridazine (Mellaril) Combination Antidepressants Trifluoperazine (Stelazine) Amitriptyline + Chlordiazepoxide (Limbitrol DS) Triflupromazine (Vesprin) Amitriptyline + Perphenazine (Triavil) Largon Levoprome Miscellaneous Antipsychotics
    [Show full text]
  • Drug Repurposing Opportunities in Pancreatic Ductal Adenocarcinoma
    pharmaceuticals Review Drug Repurposing Opportunities in Pancreatic Ductal Adenocarcinoma Rita Rebelo 1,2,† ,Bárbara Polónia 1,2,†,Lúcio Lara Santos 3,4, M. Helena Vasconcelos 1,2,5,* and Cristina P. R. Xavier 1,2,5,* 1 Cancer Drug Resistance Group, IPATIMUP—Institute of Molecular Pathology and Immunology, University of Porto, 4200-135 Porto, Portugal; [email protected] (R.R.); [email protected] (B.P.) 2 i3S—Instituto de Investigação e Inovação em Saúde, Universidade do Porto, 4200-135 Porto, Portugal 3 Experimental Pathology and Therapeutics Group, IPO—Instituto Português de Oncologia, 4200-072 Porto, Portugal; [email protected] 4 ICBAS—Biomedical Sciences Institute Abel Salazar, Universidade do Porto, 4050-313 Porto, Portugal 5 Department of Biological Sciences, FFUP—Faculty of Pharmacy of the University of Porto, 4200-135 Porto, Portugal * Correspondence: [email protected] (M.H.V.); [email protected] (C.P.R.X.) † These authors equally contributed to this work. Abstract: Pancreatic ductal adenocarcinoma (PDAC) is considered one of the deadliest tumors worldwide. The diagnosis is often possible only in the latter stages of the disease, with patients already presenting an advanced or metastatic tumor. It is also one of the cancers with poorest prognosis, presenting a five-year survival rate of around 5%. Treatment of PDAC is still a major challenge, with cytotoxic chemotherapy remaining the basis of systemic therapy. However, no major advances have been made recently, and therapeutic options are limited and highly toxic. Thus, novel therapeutic options are urgently needed. Drug repurposing is a strategy for the development of novel treatments using approved or investigational drugs outside the scope of the original clinical indication.
    [Show full text]