DBF4
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- Checkpoint Inhibition of Origin Firing Prevents Inappropriate Replication
- Mechanisms Governing DDK Regulation of the Initiation of DNA Replication
- Identification of Novel Anti-Tumor Therapeutic Target Via Proteomic
- Amplification of a Broad Transcriptional Program by a Common Factor Triggers the Meiotic Cell Cycle in Mice
- CDC7 Kinase Antibody
- Structural and Mechanistic Insights Into Mcm2-7 Double-Hexamer Assembly
- Analysis of the Crystal Structure of an Active MCM Hexamer Justin M Miller†, Buenafe T Arachea†, Leslie B Epling, Eric J Enemark*
- Cryo-EM Structure of Mcm2-7 Double Hexamer on DNA Suggests A
- Schwann Cell Reprogramming and Lung Cancer Progression: a Meta-Analysis of Transcriptome Data
- FARE2015 WINNERS Sorted by Study Section
- Incorporation Into the Prereplicative Complex Activates the Mcm2–7 Helicase for Cdc7–Dbf4 Phosphorylation
- Distinct Surfaces on Cdc5/PLK Polo-Box Domain Orchestrate Combinatorial Substrate Recognition During Cell Division Ahmad W
- The Role of Dbf4-Dependent Kinase in Maintaining Genome Stability
- Dbf4 Recruitment by Forkhead Transcription Factors Defines an Upstream Rate-Limiting Step in Determining Origin Firing Timing
- In Silico Study for Decoding the Correlated Role of MCM7 Gene in Progression of Breast Cancer and Alzheimer’S Disorder
- Macrophage Activation JUNB Is a Key Transcriptional Modulator Of
- Characterization of the Association of Dbf4 and Cdc7 with Mcm2-7 And
- 1 Global Rnaseq of Ocular Cells Reveals Gene Dysregulation in Both