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ASCC3
Hypopituitarism May Be an Additional Feature of SIM1 and POU3F2 Containing 6Q16 Deletions in Children with Early Onset Obesity
S41467-020-18249-3.Pdf
The Interaction of DNA Repair Factors ASCC2 and ASCC3 Is Affected by Somatic Cancer Mutations
Supplementary Information
Multiomic Analysis of the UV-Induced DNA Damage Response
The Role of SMARCAD1 During Replication Stress Sarah Joseph
ALKBH3 Partner ASCC3 Mediates P-Body Formation and Selective
The Neurodegenerative Diseases ALS and SMA Are Linked at The
An Evaluation of Cancer Subtypes and Glioma Stem Cell Characterisation Unifying Tumour Transcriptomic Features with Cell Line Expression and Chromatin Accessibility
Whole-Exome Sequencing of Polycythemia Vera Revealed Novel Driver Genes and Somatic Mutation Shared by T Cells and Granulocytes
Kevetrin Induces Apoptosis in TP53 Wild‑Type and Mutant Acute Myeloid Leukemia Cells
ALKBH5 Is a Mammalian RNA Demethylase That Impacts RNA Metabolism and Mouse Fertility
Identification of a Core TP53 Transcriptional Program with Highly Distributed Tumor Suppressive Activity
Interactome Analyses Revealed That the U1 Snrnp Machinery Overlaps
Genome-Wide Transcriptional Effects of the Anti-Cancer Agent Camptothecin
Cellular Response to Small Molecules That Selectively Stall Protein
A Pan-Cancer Genome-Wide Analysis Reveals Tumour Dependencies by Induction of Nonsense-Mediated Decay
The Interaction of DNA Repair Factors ASCC2 and ASCC3 Is Affected By
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Network-Based Analysis of Oligodendrogliomas Predicts Novel
Gene-Based Association Analysis Identifies 190 Genes Affecting
Activation and Regulation of the ALKBH3-ASCC Alkylation Repair Pathway
RNA M6a Modification Orchestrates a LINE-1Â
Cellular Context-Dependent Consequences of Apc Mutations on Gene Regulation and Cellular Behavior
The Oxidative Demethylase ALKBH3 Marks Hyperactive Gene Promoters in Human Cancer Cells Robert Liefke2,3†, Indra M
Identification of a Novel Trigger Complex That Facilitates Ribosome
Deep Sequencing Reveals 50 Novel Genes for Recessive Cognitive Disorders
Characterization of a Dominant Cone Degeneration in A
Detailed Phenotype–Genotype Study in Five Patients with Chromosome 6Q16 Deletion: Narrowing the Critical Region for Prader–Willi-Like Phenotype