Desquamative Gingivitis Desquamative Gingivitis
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Long-Term Uncontrolled Hereditary Gingival Fibromatosis: a Case Report
Long-term Uncontrolled Hereditary Gingival Fibromatosis: A Case Report Abstract Hereditary gingival fibromatosis (HGF) is a rare condition characterized by varying degrees of gingival hyperplasia. Gingival fibromatosis usually occurs as an isolated disorder or can be associated with a variety of other syndromes. A 33-year-old male patient who had a generalized severe gingival overgrowth covering two thirds of almost all maxillary and mandibular teeth is reported. A mucoperiosteal flap was performed using interdental and crevicular incisions to remove excess gingival tissues and an internal bevel incision to reflect flaps. The patient was treated 15 years ago in the same clinical facility using the same treatment strategy. There was no recurrence one year following the most recent surgery. Keywords: Gingival hyperplasia, hereditary gingival hyperplasia, HGF, hereditary disease, therapy, mucoperiostal flap Citation: S¸engün D, Hatipog˘lu H, Hatipog˘lu MG. Long-term Uncontrolled Hereditary Gingival Fibromatosis: A Case Report. J Contemp Dent Pract 2007 January;(8)1:090-096. © Seer Publishing 1 The Journal of Contemporary Dental Practice, Volume 8, No. 1, January 1, 2007 Introduction Hereditary gingival fibromatosis (HGF), also Ankara, Turkey with a complaint of recurrent known as elephantiasis gingiva, hereditary generalized gingival overgrowth. The patient gingival hyperplasia, idiopathic fibromatosis, had presented himself for examination at the and hypertrophied gingival, is a rare condition same clinic with the same complaint 15 years (1:750000)1 which can present as an isolated ago. At that time, he was treated with full-mouth disorder or more rarely as a syndrome periodontal surgery after the diagnosis of HGF component.2,3 This condition is characterized by had been made following clinical and histological a slow and progressive enlargement of both the examination (Figures 1 A-B). -
ABC of Oral Health Periodontal Disease John Coventry, Gareth Griffiths, Crispian Scully, Maurizio Tonetti
Clinical review ABC of oral health Periodontal disease John Coventry, Gareth Griffiths, Crispian Scully, Maurizio Tonetti Most periodontal disease arises from, or is aggravated by, accumulation of plaque, and periodontitis is associated particularly with anaerobes such as Porphyromonas gingivalis, Bacteroides forsythus, and Actinobacillus actinomycetemcomitans. Calculus (tartar) may form from calcification of plaque above or below the gum line, and the plaque that collects on calculus exacerbates the inflammation. The inflammatory reaction is associated with progressive loss of periodontal ligament and alveolar bone and, eventually, with mobility and loss of teeth. Periodontal diseases are ecogenetic in the sense that, in subjects rendered susceptible by genetic or environmental factors (such as polymorphisms in the gene for interleukin 1, cigarette smoking, immune depression, and diabetes), the infection leads to more rapidly progressive disease. Osteoporosis also seems to have some effect on periodontal bone loss. The possible effects of periodontal disease on systemic Chronic marginal gingivitis showing erythematous oedematous appearance health, via pro-inflammatory cytokines, have been the focus of much attention. Studies to test the strength of associations with atherosclerosis, hypertension, coronary heart disease, cerebrovascular disease, and low birth weight, and any effects on diabetic control, are ongoing. Gingivitis Chronic gingivitis to some degree affects over 90% of the population. If treated, the prognosis is good, but otherwise it may progress to periodontitis and tooth mobility and loss. Marginal gingivitis is painless but may manifest with bleeding from the gingival crevice, particularly when brushing the teeth. The gingival margins are slightly red and swollen, eventually with mild gingival hyperplasia. Management—Unless plaque is assiduously removed and Gingivitis with hyperplasia kept under control by tooth brushing and flossing and, where necessary, by removal of calculus by scaling and polishing by dental staff, the condition will recur. -
Dentinal Hypersensitivity: a Review
Dentinal Hypersensitivity: A Review Abstract Dentinal hypersensitivity is generally reported by the patient after experiencing a sharp pain caused by one of several different stimuli. The pain response varies substantially from one person to another. The condition generally involves the facial surfaces of teeth near the cervical aspect and is very common in premolars and canines. The most widely accepted theory of how the pain occurs is Brannstrom’s hydrodynamic theory, fluid movement within the dentinal tubules. The dental professional, using a variety of diagnostic techniques, will discern the condition from other conditions that may cause sensitive teeth. Treatment of the condition can be invasive or non-invasive in nature. The most inexpensive and efficacious first line of treatment for most patients is a dentifrice containing a desensitizing active ingredient such as potassium nitrate and/or stannous fluoride. This review will address the prevalence, diagnosis, and treatment of dentinal hypersensitivity. In addition the home care recommendations will focus on desensitizing dentifrices. Keywords: Dentinal hypersensitivity, hydrodynamic theory, stannous fluoride, potassium nitrate Citation: Walters PA. Dentinal Hypersensitivity: A Review. J Contemp Dent Pract 2005 May;(6)2:107-117. © Seer Publishing 1 The Journal of Contemporary Dental Practice, Volume 6, No. 2, May 15, 2005 Introduction The prevalence of dentinal hypersensitivity Dentifrices and mouth rinses are routinely used has been reported over the years in a variety as a delivery system for therapeutic agents of ways: as greater than 40 million people such as antimicrobials and anti-sensitivity in the U.S. annually1, 14.3% of all dental agents. Therapeutic oral care products are patients2, between 8% and 57% of adult dentate available to assist the patient in the control of population3, and up to 30% of adults at some time dental caries, calculus formation, and dentinal during their lifetime.4 hypersensitivity to name a few. -
Generalized Aggressive Periodontitis Associated with Plasma Cell Gingivitis Lesion: a Case Report and Non-Surgical Treatment
Clinical Advances in Periodontics; Copyright 2013 DOI: 10.1902/cap.2013.130050 Generalized Aggressive Periodontitis Associated With Plasma Cell Gingivitis Lesion: A Case Report and Non-Surgical Treatment * Andreas O. Parashis, Emmanouil Vardas, † Konstantinos Tosios, ‡ * Private practice limited to Periodontics, Athens, Greece; and, Department of Periodontology, School of Dental Medicine, Tufts University, Boston, MA, United States of America. †Clinic of Hospital Dentistry, Dental Oncology Unit, University of Athens, Greece. ‡ Private practice limited to Oral Pathology, Athens, Greece. Introduction: Plasma cell gingivitis (PCG) is an unusual inflammatory condition characterized by dense, band-like polyclonal plasmacytic infiltration of the lamina propria. Clinically, appears as gingival enlargement with erythema and swelling of the attached and free gingiva, and is not associated with any loss of attachment. The aim of this report is to present a rare case of severe generalized aggressive periodontitis (GAP) associated with a PCG lesion that was successfully treated and maintained non-surgically. Case presentation: A 32-year-old white male with a non-contributory medical history presented with gingival enlargement with diffuse erythema and edematous swelling, predominantly around teeth #5-8. Clinical and radiographic examination revealed generalized severe periodontal destruction. A complete blood count and biochemical tests were within normal limits. Histological and immunohistochemical examination were consistent with PCG. A diagnosis of severe GAP associated with a PCG lesion was assigned. Treatment included elimination of possible allergens and non- surgical periodontal treatment in combination with azithromycin. Clinical examination at re-evaluation revealed complete resolution of gingival enlargement, erythema and edema and localized residual probing depths 5 mm. One year post-treatment the clinical condition was stable. -
Periodontal Health, Gingival Diseases and Conditions 99 Section 1 Periodontal Health
CHAPTER Periodontal Health, Gingival Diseases 6 and Conditions Section 1 Periodontal Health 99 Section 2 Dental Plaque-Induced Gingival Conditions 101 Classification of Plaque-Induced Gingivitis and Modifying Factors Plaque-Induced Gingivitis Modifying Factors of Plaque-Induced Gingivitis Drug-Influenced Gingival Enlargements Section 3 Non–Plaque-Induced Gingival Diseases 111 Description of Selected Disease Disorders Description of Selected Inflammatory and Immune Conditions and Lesions Section 4 Focus on Patients 117 Clinical Patient Care Ethical Dilemma Clinical Application. Examination of the gingiva is part of every patient visit. In this context, a thorough clinical and radiographic assessment of the patient’s gingival tissues provides the dental practitioner with invaluable diagnostic information that is critical to determining the health status of the gingiva. The dental hygienist is often the first member of the dental team to be able to detect the early signs of periodontal disease. In 2017, the American Academy of Periodontology (AAP) and the European Federation of Periodontology (EFP) developed a new worldwide classification scheme for periodontal and peri-implant diseases and conditions. Included in the new classification scheme is the category called “periodontal health, gingival diseases/conditions.” Therefore, this chapter will first review the parameters that define periodontal health. Appreciating what constitutes as periodontal health serves as the basis for the dental provider to have a stronger understanding of the different categories of gingival diseases and conditions that are commonly encountered in clinical practice. Learning Objectives • Define periodontal health and be able to describe the clinical features that are consistent with signs of periodontal health. • List the two major subdivisions of gingival disease as established by the American Academy of Periodontology and the European Federation of Periodontology. -
Third Molar (Wisdom) Teeth
Third molar (wisdom) teeth This information leaflet is for patients who may need to have their third molar (wisdom) teeth removed. It explains why they may need to be removed, what is involved and any risks or complications that there may be. Please take the opportunity to read this leaflet before seeing the surgeon for consultation. The surgeon will explain what treatment is required for you and how these issues may affect you. They will also answer any of your questions. What are wisdom teeth? Third molar (wisdom) teeth are the last teeth to erupt into the mouth. People will normally develop four wisdom teeth: two on each side of the mouth, one on the bottom jaw and one on the top jaw. These would normally erupt between the ages of 18-24 years. Some people can develop less than four wisdom teeth and, occasionally, others can develop more than four. A wisdom tooth can fail to erupt properly into the mouth and can become stuck, either under the gum, or as it pushes through the gum – this is referred to as an impacted wisdom tooth. Sometimes the wisdom tooth will not become impacted and will erupt and function normally. Both impacted and non-impacted wisdom teeth can cause problems for people. Some of these problems can cause symptoms such as pain & swelling, however other wisdom teeth may have no symptoms at all but will still cause problems in the mouth. People often develop problems soon after their wisdom teeth erupt but others may not cause problems until later on in life. -
A Concise Guide to Treating Painful Oral Lesions
Drugs Used to Treat Osteoporosis and Bone Cancer Perio & Implant Centers The Team for of the Monterey Bay (831) 648-8800 Jochen P. Pechak, DDS, MSD in Silicon Valley (408) 738-3423 Which May Cause Osteonecrosis of the Jaws mobile: www.DrPechakapp.com he many bisphosphonates and monoclonal antibodies which are used to treat osteoporosis and bone cancer often web: GumsRus.com causeDrugsDrugs osteonecrosis Used Used of the to jaws.to Treat AsTreat dental clinicians,Osteoporosis Osteoporosis it is important that and andwe are Bone awareBone of this Cancers Cancers side effect before Ttreating our patients who are taking these drugs. The tables below summarize these drugs, the route these drugs are administered, andWhich Whichtheir likelihood May May of causing Cause Cause osteonecrosis Osteonecrosis Osteonecrosis of the jaws as reported byof of Dr. the theRobert Jaws JawsMarx at the University of Miami Division of Oral and Maxillofacial Surgery. PDL tm Osteoporosis Drugs Drugs Osteoporosis Used to Treat Drugs Osteoporosis PerioDontaLetter Jochen P. Pechak, DDS, MSD, Periodontics and Implant Dentistry Spring DrugDrug ClassificationClassification ActionAction DoseDose RouteRoute %% of of ReportedReported CasesCases of of OsteonecrosisOsteonecrosis AlendronateAlendronate BisphosphonateBisphosphonate OsteoclastOsteoclast 7070 mg/wk mg/wk OralOral 8282%% From Our Office A Concise Guide to Treating (Fosamax(Fosamax ToxicityToxicity to Yours... Generic)Generic) Painful Oral Lesions ResidronateResidronate BisphosphonateBisphosphonate OsteoclastOsteoclast 3535 mg/wk mg/wk OralOral 1%1% As dentists specializing in treat- (Actonel Toxicity (Actonel Toxicity ment of diseases of the oral cavity atients present frequently with Treating Cold Sores Atelvia)Atelvia) and associated structures, we are painful oral lesions. They are often also called upon to treat pain- IbandronateIbandronate BisphosphonateBisphosphonate OsteoclastOsteoclast 150150 mg/mos mg/mos OralOral 1%1% usually not serious, but patients And Canker Sores (Boniva) Toxicity IV ful oral lesions in the mouth. -
Periodontal Disease)
Patient Fact Sheet Gum Disease (Periodontal Disease) It can often go unnoticed – until it’s too late. While you may not think periodontal (gum) disease affects you, 75 percent of adults over the age of 35 show signs and symptoms. In fact, periodontal disease is the leading cause of tooth loss in adults. Why? Because it occurs at an age when cavities are usually a thing of the past and the initial symptoms often go unnoticed. Recent studies have also shown a possible link between periodontal disease and heart disease. One theory in support of this is that the bacteria that cause periodontal disease enter the bloodstream and promote blood clots and narrowing of the arteries that cause heart attacks. It has also been shown that if a woman develops severe periodontal disease during pregnancy, she is more likely to give birth to a low birth weight infant. Research also shows periodontal disease is linked to many other health problems, as well. What is gum (periodontal) If I have no symptoms, how do I How can I prevent periodontal disease? What causes it? know if I have gum disease? disease? Periodontal disease, or gum disease, is a bacterial Periodontal disease can be easily detected by your • Brush your teeth twice a day with a soft-bristled infection of the gums, ligaments and bone that support general dentist or a periodontist (a specialist in toothbrush. Hold the brush at a 45-degree angle to the teeth and anchor them in the jaw. The bacteria, periodontal diseases) during regular dental the gum line and gently clean where the gums meet which act mainly on certain carbohydrates in our diets, examinations.Therefore, regular checkups, ideally every your teeth. -
Hereditary Gingival Fibromatosis, Inherited Disease, Gingivectomy
Clinical Practice 2014, 3(1): 7-10 DOI: 10.5923/j.cp.20140301.03 Hereditary Gingival Fibromatosis - A Case Report Anand Kishore1,*, Vivek Srivastava2, Ajeeta Meenawat2, Ambrish Kaushal3 1King George Medical College, Lucknow 2BBD College of dental sciences 3Chandra Dental College & Hospital Abstract Hereditary gingival fibromatosis is characterized by a slow benign enlargement of gingival tissue. It causes teeth being partially or totally covered by enlarged gingiva, causing esthetic and functional problems. It is usually transmitted both as autosomal dominant trait and autosomal recessive inheritance although sporadic cases are commonly reported. This paper reports three cases of gingival fibromatosis out of which one was in a 15 year old girl treated with convectional gingivectomy. Keywords Hereditary gingival fibromatosis, Inherited disease, Gingivectomy having the gingival enlargement before the patient’s birth 1. Introduction and she got operated in the village government hospital. No further relevant medical history was present. Hereditary gingival fibromatosis (HGF) or Idiopathic gingival fibromatosis is a rare, benign, asymptomatic, non-hemorrhagic and non-exudative proliferative fibrous lesion of gingival tissue occurring equally among men and women, in both arches with varying intensity in individuals within the same family [1]. It occurs as an autosomal dominant condition although recessive form also does occur. Consanguinity seems to increase the risk of autosomal dominant inheritance. It affects the marginal gingival, attached gingival and interdental papilla presenting as pink, non-hemorrhagic and have a firm, fibrotic consistency [2]. It also shows a generalized firm nodular enlargement with smooth to stippled surfaces and minimal tendency to bleed. Figure 1. Gingival enlargement However, in some cases the enlargement can be so firm and dense that it feels like bone on palpation [3]. -
Oral Diseases Associated with Human Herpes Viruses: Aetiology, Clinical Features, Diagnosis and Management
www.sada.co.za / SADJ Vol 71 No. 6 CLINICAL REVIEW < 253 Oral diseases associated with human herpes viruses: aetiology, clinical features, diagnosis and management SADJ July 2016, Vol 71 no 6 p253 - p259 R Ballyram1, NH Wood2, RAG Khammissa3, J Lemmer4, L Feller5 ABSTRACT Human herpesviruses (HHVs) are very prevalent DNA ACRONYMS viruses that can cause a variety of orofacial diseases. EM: erythema multiforme Typically they are highly infectious, are contracted early in HHV: human herpes virus life, and following primary infection, usually persist in a latent form. Primary oral infections are often subclinical, but may PCR: polymerase chain reaction be symptomatic as in the case of herpes simplex virus- HSV, HHV-1: herpes simplex virus induced primary herpetic gingivostomatitis. Reactivation VZV, HHV-3: varicella-zoster virus of the latent forms may result in various conditions: herpes EBV, HHV-4: Epstein-Barr virus simplex virus (HSV) can cause recurrent herpetic orolabial CMV, HHV-5: cytomegalovirus lesions; varicella zoster virus (VZV) can cause herpes zoster; Epstein-Barr virus (EBV) can cause oral hairy Key words: herpes simplex virus, human herpes virus-8, leukoplakia; and reactivation of HHV-8 can cause Kaposi varicella zoster virus, Epstein-Barr virus, recurrent herpes sarcoma. In immunocompromised subjects, infections labialis, recurrent intraoral herpetic ulcers, treatment, val- with human herpesviruses are more extensive and aciclovir, aciclovir, famcicylovir. severe than in immunocompetent subjects. HSV and VZV infections are treated with nucleoside analogues aciclovir, valaciclovir, famciclovir and penciclovir. These agents INTRODucTION have few side effects and are effective when started The human herpesvirus (HHV) family comprises a diverse early in the course of the disease. -
Denture Technology Curriculum Objectives
Health Licensing Agency 700 Summer St. NE, Suite 320 Salem, Oregon 97301-1287 Telephone (503) 378-8667 FAX (503) 585-9114 E-Mail: [email protected] Web Site: www.Oregon.gov/OHLA As of July 1, 2013 the Board of Denture Technology in collaboration with Oregon Students Assistance Commission and Department of Education has determined that 103 quarter hours or the equivalent semester or trimester hours is equivalent to an Associate’s Degree. A minimum number of credits must be obtained in the following course of study or educational areas: • Orofacial Anatomy a minimum of 2 credits; • Dental Histology and Embryology a minimum of 2 credits; • Pharmacology a minimum of 3 credits; • Emergency Care or Medical Emergencies a minimum of 1 credit; • Oral Pathology a minimum of 3 credits; • Pathology emphasizing in Periodontology a minimum of 2 credits; • Dental Materials a minimum of 5 credits; • Professional Ethics and Jurisprudence a minimum of 1 credit; • Geriatrics a minimum of 2 credits; • Microbiology and Infection Control a minimum of 4 credits; • Clinical Denture Technology a minimum of 16 credits which may be counted towards 1,000 hours supervised clinical practice in denture technology defined under OAR 331-405-0020(9); • Laboratory Denture Technology a minimum of 37 credits which may be counted towards 1,000 hours supervised clinical practice in denture technology defined under OAR 331-405-0020(9); • Nutrition a minimum of 4 credits; • General Anatomy and Physiology minimum of 8 credits; and • General education and electives a minimum of 13 credits. Curriculum objectives which correspond with the required course of study are listed below. -
Idiopathic Gingival Fibromatosis Associated with Generalized Aggressive Periodontitis: a Case Report
Clinical P RACTIC E Idiopathic Gingival Fibromatosis Associated with Generalized Aggressive Periodontitis: A Case Report Contact Author Rashi Chaturvedi, MDS, DNB Dr. Chaturvedi Email: rashichaturvedi@ yahoo.co.in ABSTRACT Idiopathic gingival fibromatosis, a benign, slow-growing proliferation of the gingival tissues, is genetically heterogeneous. This condition is usually part of a syndrome or, rarely, an isolated disorder. Aggressive periodontitis, another genetically transmitted disorder of the periodontium, typically results in severe, rapid destruction of the tooth- supporting apparatus. The increased susceptibility of the host population with aggressive periodontitis may be caused by the combined effects of multiple genes and their inter- action with various environmental factors. Functional abnormalities of neutrophils have also been implicated in the etiopathogenesis of aggressive periodontitis. We present a rare case of a nonsyndromic idiopathic gingival fibromatosis associated with general- ized aggressive periodontitis. We established the patient’s diagnosis through clinical and radiologic assessment, histopathologic findings and immunologic analysis of neutrophil function with a nitro-blue-tetrazolium reduction test. We describe an interdisciplinary approach to the treatment of the patient. For citation purposes, the electronic version is the definitive version of this article: www.cda-adc.ca/jcda/vol-75/issue-4/291.html diopathic gingival fibromatosis is a rare syndromic, have been genetically linked to hereditary condition