Inflammation 1. Serous fluid: +a) is characterized by moderate protein content; +b) is characterized by a low relative density; c) is characterized by an increased content of cell elements; +d) typical for inflammation of the serous membranes (chest, heart, joints, etc.); +e) the most frequently observed during the burn, immune inflammation. 2. Purulent exudate: +a) is characterized by the presence of large amounts of purulent corpuscles; b) is characterized by a low content of polymorphonuclear leukocytes, mainly killed and destroyed; +c) has a high proteolytic activity; +d) is characteristic of inflammation caused by coccal infection; +e) is more common during acute inflammation. 3. Fibrinous exudate: +a) has a high content of fibrin; +b) is observed in inflammation with a significant increase in vascular permeability; +c) is characteristic of inflammation of the oral mucosa, bronchi, stomach, intestine; d) occurs only in chronic inflammatory processes; +e) may be observed in diphtheria, dysentery, tuberculosis of the lungs. 4. Type of exudate during inflammation caused by staphylococcus and streptococcus a) hemorrhagic; +b) purulent; c) fibrinous; d) serous; e) mixed. 5. The main source of hydrolytic enzymes in inflammation are a) products of vital activity of microbes; +b) destroyed white blood cells; c) mast cells; d) microbial cells; e) platelets. 6. At an inflammation caused by pyogenic microorganisms is dominated in the composition of exudate +a) neutrophils; b) eosinophils; c) basophils, d) monocytes; e) lymphocytes. 7. When purulent acute inflammation is observed +a) neutrophilic leukocytosis; b) lymphocytosis; c) monocytosis. 8. The sequence of emigration of leukocytes in acute inflammation: a) neutrophils, eosinophils, monocytes; +b) neutrophils, monocytes, lymphocytes; c) monocytes, lymphocytes, neutrophils; d) lymphocytes, monocytes, neutrophils; e) macrophages, neutrophils, monocytes. 9. Emigrate in inflammatory focus primarily +a) neutrophils; b) lymphocytes; c) eosinophils. 10. Contributes to "rolling" of leukocytes in inflammatory focus: a) acceleration of blood flow; b) expression of integrins on the surface of leukocytes; c) reducing the amount of receptors of the adhesion on endothelium; d) expression of immunoglobulin-like molecules on the endothelium; +e) activation of L- and E-selectins. 11. Provides the strong bond of leukocytes with the endothelium in inflammatory focus: a) L-selectins; +b) integrins; c) immunoglobulins; d) E-selectins; e) prostaglandins. 12. Macrophages are +a) monocytes; b) lymphocytes; c) neutrophils; d) mast cells; e) fibroblasts. 13. The local manifestations of inflammation are: a) fever, leukocytosis, accelerated erythrocyte sedimentation rate; +b) pain, redness, fever, organ dysfunction; c) headache, sleep disturbance, loss of appetite; d) myalgia, ossalgiya; e) hypoalbuminemia, hypergammaglobulinemia. 14. Latin name of "redness" as local signs of inflammation is a) calor; b) dolor; +c) rubor; d) tumor; e) funcio laesa. 15. Latin name of "pain" as the local signs of inflammation is a) calor; +b) dolor; c) rubor; d) tumor; e) funcio laesa. 16. Latin name of "swelling" as local signs of inflammation is a) calor; b) dolor; c) rubor; +d) tumor; e) funcio laesa. 17. Latin name of "dysfunction" as the local signs of inflammation is a) calor; b) dolor; c) rubor; d) tumor; +e) funcio laesa. 18. Latin name of "fever" as local signs of inflammation is +a) calor; b) dolor; c) rubor; d) tumor; e) funcio laesa. 19. Pathogenic factor of the local temperature rise in inflammation is a) venous hyperemia; b) ischemia; +c) arterial hyperemia; d) stasis; e) embolism. 20. Pathogenesis of local temperature increase in inflammation is associated with: a) violation of the outflow of blood through the veins; +b) development of arterial hyperemia and "fire" of metabolism; c) compression of the wall of venules by exudate; d) emigration of leukocytes; e) effect of catecholamines on the vascular wall. 21. Redness in the inflammatory focus is associated with +a) arterial hyperemia; b) ischemia; c) increasing metabolism; d) physical and chemical changes; e) venous hyperemia. 22. Indicates to presence of inflammatory process in the body +a) ESR acceleration; b) leukopenia; c) anemia. 23. Сommon manifestations of inflammation are: a) pain, redness, organ dysfunction; +b) symptoms of intoxication; +c) ESR acceleration; +d) fever, leukocytosis; e) swelling due to edema. 24. The process of proliferation during inflammation is inhibited by: a) fibroblast growth factor; b) growth hormone; +c) chalones; d) IL-I; e) somatomedin. 25. Inflammation is regarded as adaptive response of the body because: +a) limits the area of damage, prevents the spread of flogogenic factor and products of alteration in the body; +b) inactivates flogogenic agent and products of alteration of tissue; c) prevents an allergization of the organism; +d) mobilizes the body's protective factors. 26. How is changed the content of K, Na, Ca in the inflamed tissue? +a) increases; b) decreases. 27. What are the signs of inflammation associated with hyperosmia? +a) swelling; b) redness; с) fever; +d) pain; +e) disturbance of function. 28. What causes to acidosis in inflammatory focus? +a) release and accumulation of large amounts of acids; b) accumulation of chloride ions; c) increase in the content of the polypeptide; d) accumulation of sodium ions; e) accumulation of potassium ions. 29. What hormones induce amplification of the inflammatory response? a) glucocorticoids; +b) mineralocorticoids; c) thyroxine; d) STH; e) insulin. 30. What hormones inhibit the inflammatory response? +a) glucocorticoids; b) mineralocorticoids; c) thyroxine; d) STH; e) insulin.