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Guidelines on the Diagnosis and Management of Pericardial
European Heart Journal (2004) Ã, 1–28 ESC Guidelines Guidelines on the Diagnosis and Management of Pericardial Diseases Full Text The Task Force on the Diagnosis and Management of Pericardial Diseases of the European Society of Cardiology Task Force members, Bernhard Maisch, Chairperson* (Germany), Petar M. Seferovic (Serbia and Montenegro), Arsen D. Ristic (Serbia and Montenegro), Raimund Erbel (Germany), Reiner Rienmuller€ (Austria), Yehuda Adler (Israel), Witold Z. Tomkowski (Poland), Gaetano Thiene (Italy), Magdi H. Yacoub (UK) ESC Committee for Practice Guidelines (CPG), Silvia G. Priori (Chairperson) (Italy), Maria Angeles Alonso Garcia (Spain), Jean-Jacques Blanc (France), Andrzej Budaj (Poland), Martin Cowie (UK), Veronica Dean (France), Jaap Deckers (The Netherlands), Enrique Fernandez Burgos (Spain), John Lekakis (Greece), Bertil Lindahl (Sweden), Gianfranco Mazzotta (Italy), Joa~o Morais (Portugal), Ali Oto (Turkey), Otto A. Smiseth (Norway) Document Reviewers, Gianfranco Mazzotta, CPG Review Coordinator (Italy), Jean Acar (France), Eloisa Arbustini (Italy), Anton E. Becker (The Netherlands), Giacomo Chiaranda (Italy), Yonathan Hasin (Israel), Rolf Jenni (Switzerland), Werner Klein (Austria), Irene Lang (Austria), Thomas F. Luscher€ (Switzerland), Fausto J. Pinto (Portugal), Ralph Shabetai (USA), Maarten L. Simoons (The Netherlands), Jordi Soler Soler (Spain), David H. Spodick (USA) Table of contents Constrictive pericarditis . 9 Pericardial cysts . 13 Preamble . 2 Specific forms of pericarditis . 13 Introduction. 2 Viral pericarditis . 13 Aetiology and classification of pericardial disease. 2 Bacterial pericarditis . 14 Pericardial syndromes . ..................... 2 Tuberculous pericarditis . 14 Congenital defects of the pericardium . 2 Pericarditis in renal failure . 16 Acute pericarditis . 2 Autoreactive pericarditis and pericardial Chronic pericarditis . 6 involvement in systemic autoimmune Recurrent pericarditis . 6 diseases . 16 Pericardial effusion and cardiac tamponade . -
NLRP3 Inflammasome Cutting Edge
Cutting Edge: Nitric Oxide Inhibits the NLRP3 Inflammasome Eduardo Hernandez-Cuellar, Kohsuke Tsuchiya, Hideki Hara, Rendong Fang, Shunsuke Sakai, Ikuo Kawamura, This information is current as Shizuo Akira and Masao Mitsuyama of September 25, 2021. J Immunol published online 24 October 2012 http://www.jimmunol.org/content/early/2012/10/24/jimmun ol.1202479 Downloaded from Supplementary http://www.jimmunol.org/content/suppl/2012/10/24/jimmunol.120247 Material 9.DC1 http://www.jimmunol.org/ Why The JI? Submit online. • Rapid Reviews! 30 days* from submission to initial decision • No Triage! Every submission reviewed by practicing scientists • Fast Publication! 4 weeks from acceptance to publication by guest on September 25, 2021 *average Subscription Information about subscribing to The Journal of Immunology is online at: http://jimmunol.org/subscription Permissions Submit copyright permission requests at: http://www.aai.org/About/Publications/JI/copyright.html Email Alerts Receive free email-alerts when new articles cite this article. Sign up at: http://jimmunol.org/alerts The Journal of Immunology is published twice each month by The American Association of Immunologists, Inc., 1451 Rockville Pike, Suite 650, Rockville, MD 20852 Copyright © 2012 by The American Association of Immunologists, Inc. All rights reserved. Print ISSN: 0022-1767 Online ISSN: 1550-6606. Published October 24, 2012, doi:10.4049/jimmunol.1202479 Cutting Edge: Nitric Oxide Inhibits the NLRP3 Inflammasome Eduardo Hernandez-Cuellar,*,† Kohsuke Tsuchiya,* Hideki Hara,* Rendong Fang,* Shunsuke Sakai,* Ikuo Kawamura,* Shizuo Akira,† and Masao Mitsuyama* Although the NLRP3 inflammasome plays a pivotal role in that NLRP3 inflammasome contributes to host defense against host defense, its uncontrolled activation is associated with microbialpathogens,excessiveactivationduetomutationsinthe inflammatory disorders, suggesting that regulation of the NLRP3 gene has been associated with a spectrum of autoin- inflammasome is important to prevent detrimental effects. -
Eagle's Syndrome, Elongated Styloid Process and New
Musculoskeletal Science and Practice 50 (2020) 102219 Contents lists available at ScienceDirect Musculoskeletal Science and Practice journal homepage: www.elsevier.com/locate/msksp Masterclass Eagle’s syndrome, elongated styloid process and new evidence for pre-manipulative precautions for potential cervical arterial dysfunction Andrea M. Westbrook a,*, Vincent J. Kabbaz b, Christopher R. Showalter c a Method Manual Physical Therapy & Wellness, Raleigh, NC, 27617, USA b HEAL Physical Therapy, Monona, WI, USA c Program Director, MAPS Accredited Fellowship in Orthopedic Manual Therapy, Cutchogue, NY, 11935, USA ARTICLE INFO ABSTRACT Keywords: Introduction: Safety with upper cervical interventions is a frequently discussed and updated concern for physical Eagle’s syndrome therapists, chiropractors and osteopaths. IFOMPT developed the framework for safety assessment of the cervical Styloid spine, and this topic has been discussed in-depth with past masterclasses characterizing carotid artery dissection CAD and cervical arterial dysfunction. Our masterclass will expand on this information with knowledge of specific Carotid anatomical anomalies found to produce Eagle’s syndrome, and cause carotid artery dissection, stroke and even Autonomic Manipulation death. Eagle’s syndrome is an underdiagnosed, multi-mechanism symptom assortment produced by provocation of the sensitive carotid space structures by styloid process anomalies. As the styloid traverses between the internal and external carotid arteries, provocation of the vessels and periarterial sympathetic nerve fiberscan lead to various neural, vascular and autonomic symptoms. Eagle’s syndrome commonly presents as neck, facial and jaw pain, headache and arm paresthesias; problems physical therapists frequently evaluate and treat. Purpose: This masterclass aims to outline the safety concerns, assessment and management of patients with Eagle’s syndrome and styloid anomalies. -
Pdfs/ Ommended That Initial Cultures Focus on Common Pathogens, Pscmanual/9Pscssicurrent.Pdf)
Clinical Infectious Diseases IDSA GUIDELINE A Guide to Utilization of the Microbiology Laboratory for Diagnosis of Infectious Diseases: 2018 Update by the Infectious Diseases Society of America and the American Society for Microbiologya J. Michael Miller,1 Matthew J. Binnicker,2 Sheldon Campbell,3 Karen C. Carroll,4 Kimberle C. Chapin,5 Peter H. Gilligan,6 Mark D. Gonzalez,7 Robert C. Jerris,7 Sue C. Kehl,8 Robin Patel,2 Bobbi S. Pritt,2 Sandra S. Richter,9 Barbara Robinson-Dunn,10 Joseph D. Schwartzman,11 James W. Snyder,12 Sam Telford III,13 Elitza S. Theel,2 Richard B. Thomson Jr,14 Melvin P. Weinstein,15 and Joseph D. Yao2 1Microbiology Technical Services, LLC, Dunwoody, Georgia; 2Division of Clinical Microbiology, Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, Minnesota; 3Yale University School of Medicine, New Haven, Connecticut; 4Department of Pathology, Johns Hopkins Medical Institutions, Baltimore, Maryland; 5Department of Pathology, Rhode Island Hospital, Providence; 6Department of Pathology and Laboratory Medicine, University of North Carolina, Chapel Hill; 7Department of Pathology, Children’s Healthcare of Atlanta, Georgia; 8Medical College of Wisconsin, Milwaukee; 9Department of Laboratory Medicine, Cleveland Clinic, Ohio; 10Department of Pathology and Laboratory Medicine, Beaumont Health, Royal Oak, Michigan; 11Dartmouth- Hitchcock Medical Center, Lebanon, New Hampshire; 12Department of Pathology and Laboratory Medicine, University of Louisville, Kentucky; 13Department of Infectious Disease and Global Health, Tufts University, North Grafton, Massachusetts; 14Department of Pathology and Laboratory Medicine, NorthShore University HealthSystem, Evanston, Illinois; and 15Departments of Medicine and Pathology & Laboratory Medicine, Rutgers Robert Wood Johnson Medical School, New Brunswick, New Jersey Contents Introduction and Executive Summary I. -
Chronic Orofacial Pain: Burning Mouth Syndrome and Other Neuropathic
anagem n M e ai n t P & f o M l e Journal of a d n i c r i u n o e J Pain Management & Medicine Tait et al., J Pain Manage Med 2017, 3:1 Review Article Open Access Chronic Orofacial Pain: Burning Mouth Syndrome and Other Neuropathic Disorders Raymond C Tait1, McKenzie Ferguson2 and Christopher M Herndon2 1Saint Louis University School of Medicine, St. Louis, USA 2Southern Illinois University Edwardsville School of Pharmacy, Edwardsville, USA *Corresponding author: RC Tait, Department of Psychiatry, Saint Louis University School of Medicine,1438 SouthGrand, Boulevard, St Louis, MO-63104, USA, Tel: 3149774817; Fax: 3149774879; E-mail: [email protected] Recevied date: October 4, 2016; Accepted date: January 17, 2017, Published date: January 30, 2017 Copyright: © 2017 Raymond C Tait, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Abstract Chronic orofacial pain is a symptom associated with a wide range of neuropathic, neurovascular, idiopathic, and myofascial conditions that affect a significant proportion of the population. While the collective impact of the subset of the orofacial pain disorders involving neurogenic and idiopathic mechanisms is substantial, some of these are relatively uncommon. Hence, patients with these disorders can be vulnerable to misdiagnosis, sometimes for years, increasing the symptom burden and delaying effective treatment. This manuscript first reviews the decision tree to be followed in diagnosing any neuropathic pain condition, as well as the levels of evidence needed to make a diagnosis with each of several levels of confidence: definite, probable, or possible. -
Influences of Estrogen and Progesterone on Periodontium 26 Deepa D
CODS Journal of Dentistry Ocial Publication of College of Dental Sciences Alumni Association, Davanagere Volume 6, Issue 1, 2014 CONTENTS Director’s Message 1 V.V. Subba Reddy President’s Message 2 Vasundhara Shivanna Secretary’s Message 3 Praveen S. Basandi Editorial 4 Nandini D.B Original Articles Effect of alcohol containing and alcohol free mouth rinses on microhardness of three 5 esthetic restorative materials Vasundhara Shivanna, Rucha Nilegaonkar Prevalence and distribution of dental anomalies and fluorosis in a small cohort of 9 Indian school children and teenagers Selvamani. M , Praveen S Basandi, Madhushankari G.S Review Articles Paperless dentistry - The future 13 Mala Ram Manohar, Gajendra Bhansali Photo activated disinfection in restorative dentistry - A technical review 16 Deepak B.S, Mallikarjun Goud K, Nishanth P An overview of occupational hazards in dental practice and preventive measures. 19 Poorya Naik .D.S, Chetan .S, Gopal Krishna.B.R, Naveen Shamnur An overview on influences of estrogen and progesterone on periodontium 26 Deepa D CODS Journal of Dentistry 2014, Volume 6, Issue 1 CODS Journal of Dentistry Ocial Publication of College of Dental Sciences Alumni Association, Davanagere Volume 6, Issue 1, 2014 CONTENTS Review Articles Dental home - A new approach for child oral health care 30 Poornima P, Meghna Bajaj, Nagaveni N.B, Roopa K.B, V.V. Subba Reddy Variants of inferior alveolar nerve block: A review 35 Anuradha M, Yashavanth Kumar D.S, Harsha .V. Babji, Rahul Seth Case Reports Ellis-van Creveld syndrome affecting siblings: A case report and review 40 Mamatha G.P, Manisha Jadhav , Rajeshwari G Annigeri, Poornima .P, V.V Subba Reddy Integrated approach of ceramic and composite veneers in tetracycline stained teeth: A case report. -
Instant Notes: Immunology, Second Edition
Immunology Second Edition The INSTANT NOTES series Series Editor: B.D. Hames School of Biochemistry and Molecular Biology, University of Leeds, Leeds, UK Animal Biology 2nd edition Biochemistry 2nd edition Bioinformatics Chemistry for Biologists 2nd edition Developmental Biology Ecology 2nd edition Immunology 2nd edition Genetics 2nd edition Microbiology 2nd edition Molecular Biology 2nd edition Neuroscience Plant Biology Chemistry series Consulting Editor: Howard Stanbury Analytical Chemistry Inorganic Chemistry 2nd edition Medicinal Chemistry Organic Chemistry 2nd edition Physical Chemistry Psychology series Sub-series Editor: Hugh Wagner Dept of Psychology, University of Central Lancashire, Preston, UK Psychology Cognitive Psychology Forthcoming title Physiological Psychology Immunology Second Edition P.M. Lydyard Department of Immunology and Molecular Pathology, Royal Free and University College Medical School, University College London, London, UK A. Whelan Department of Immunology, Trinity College and St James’ Hospital, Dublin, Ireland and M.W. Fanger Department of Microbiology and Immunology, Dartmouth Medical School, Lebanon, New Hampshire, USA © Garland Science/BIOS Scientific Publishers Limited, 2004 First published 2000 This edition published in the Taylor & Francis e-Library, 2005. “To purchase your own copy of this or any of Taylor & Francis or Routledge’s collection of thousands of eBooks please go to www.eBookstore.tandf.co.uk.” Second edition published 2004 All rights reserved. No part of this book may be reproduced or -
Principle of Infection
23/09/56 Principle of Infection La-or Chompuk, M.D. Department of pathology Faculty of Medicine Infection • Definition: Invasion and multiplication of microorganisms in body tissues • No symptom, local cellular injury, localized symptom, dissemination • Mechanism; competitive metabolism, toxins, intracellular replication, immune response 1 23/09/56 Classification of infectious agents: - classification according to structure - classification according to pathogenesis - classification according to site of multiplication Classification according to structure - Prion - Fungi - Viruses - Protozoa, metazoa - Bacteria - Ectoparasite - Rickettsia, chlamydia, mycoplasma 2 23/09/56 Classification according to pathogenesis • Pathogenic agents; - Virulence: the degree of pathogenicity of a microorganism - Indicated by the severity of disease, the ability to invade tissue - high virulence - low virulence • Opportunistic infection Classification according to site of multiplication - obligate intracellular organisms; Prions, viruses, rickettsiae, chlamydia, some protozoa - facultative intracellular organism; Mycobacteria, Actinomyces, Pseudomonas spp. - extracellular organisms; mycoplasma, fungi, bacteria, metazoa 3 23/09/56 Pathogenesis of Infectious Disease -Host - Pathogen; organism or parasite that cause disease Host factors: 1. General factors; socioeconomic status, behavior pattern, occupational, and internal factors 2. Natural defense mechanism; skin and normal flora, respiratory tract and mucociliary mechanism, Hcl production in stomach, or -
Herpes Simplex Virus-Associated Dermatitis with Either High Or Normal Ige Responded Well to Antiviral Therapy: a Study of 787 Quick-Tzanck-Test-Positive Patients
Article ID: WMC004846 ISSN 2046-1690 Herpes Simplex Virus-Associated Dermatitis with Either High or Normal IgE Responded Well to Antiviral Therapy: A Study of 787 Quick-Tzanck-Test-Positive Patients Peer review status: No Corresponding Author: Dr. Lily Hsiao, Vice president, Moriya Eye and Skin Clinic, 5-7-1, Mizukino, 302-0121 - Japan Submitting Author: Dr. Lily Hsiao, Vice president, Moriya Eye and Skin Clinic, 5-7-1, Mizukino, 302-0121 - Japan Article ID: WMC004846 Article Type: Original Articles Submitted on:20-Mar-2015, 07:33:49 AM GMT Published on: 20-Mar-2015, 07:34:27 AM GMT Article URL: http://www.webmedcentral.com/article_view/4846 Subject Categories:DERMATOLOGY Keywords:herpes simplex virus, quick Tzanck test, erythema multiforme, atopic dermatitis, intrinsic atopic dermatitis How to cite the article:Hsiao L. Herpes Simplex Virus-Associated Dermatitis with Either High or Normal IgE Responded Well to Antiviral Therapy: A Study of 787 Quick-Tzanck-Test-Positive Patients. WebmedCentral DERMATOLOGY 2015;6(3):WMC004846 Copyright: This is an open-access article distributed under the terms of the Creative Commons Attribution License(CC-BY), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Source(s) of Funding: None Competing Interests: None Additional Files: HSVADWMC15 WebmedCentral > Original Articles Page 1 of 33 WMC004846 Downloaded from http://www.webmedcentral.com on 20-Mar-2015, 07:38:29 AM Herpes Simplex Virus-Associated Dermatitis with Either High or Normal IgE Responded Well to Antiviral Therapy: A Study of 787 Quick-Tzanck-Test-Positive Patients Author(s): Hsiao L Abstract Abbreviations Background: The overall age-adjusted 1. -
Pattern of Inflammatory Salivary Gland Diseases Among Sudanese Patients Dr
DOI: 10.21276/sjams Scholars Journal of Applied Medical Sciences (SJAMS) ISSN 2320-6691 (Online) Sch. J. App. Med. Sci., 2017; 5(4F):1668-1673 ISSN 2347-954X (Print) ©Scholars Academic and Scientific Publisher (An International Publisher for Academic and Scientific Resources) www.saspublisher.com Original Research Article Pattern of inflammatory salivary gland diseases among Sudanese patients Dr. Manahil Abuzeid1, Dr. Sharfi Ahmed2, Dr. Yousif O.Yousif3 1MBBS, faculty of Medicine, Bahr El Ghazal University 2Associated Professor, Faculty of Medicine, Omdurman Islamic University, Sudan, DOHNS London UK 3Assisstant Professor, faculty of Dentist, Khartoum University Consultant oral and Maxillofacial surgeon, Sudan *Corresponding author Dr. Sharfi Abdelgadir Omer Ahmed Email: [email protected] Abstract: Inflammatory conditions are the most common pathology to affect the salivary glands. Typical features of a comprehensive range of pathology including obstructive and sialadenitis, Sjogrens syndrome, sarcoidosis and HIV sialopathy. This study aims to know the pattern of inflammatory conditions of the salivary glands among 105 Sudanese patients in Khartoum state. This is a retrospective, cross- sectional, analytic and hospital based study from January 2014 to May 2016. Conducted in Otorhinolaryngological, Head and neck and Oromaxillofacial hospitals. The commonest inflammatory disease is ranula in sublingual glands. The most common site of stones in salivary gland was within glandular tissue. Inflammatory conditions were most common in salivary glands. Keywords: Salivary disease, inflammatory conditions INTRODUCTION within the ductal system of the gland, 80% percent of Inflammatory conditions are the most common all salivary calculi occur in the submandibular gland, pathology to affect the salivary glands [1]. Acute with approximately 70% of these demonstrable as sialadenitis is a bacterial inflammation of the salivary radio-opacities on routine plain radiography consisting gland. -
Sexually Transmitted Infections and Increased Risk of Co-Infection with Human Immunodeficiency Virus
REVIEW ARTICLE Sexually Transmitted Infections and Increased Risk of Co-infection with Human Immunodeficiency Virus Margaret R.H. Nusbaum, DO, MPH; Robin R. Wallace, MD; Lisa M. Slatt, MEd; Elin C. Kondrad, MD The incidence of trichomoniasis (Trichomonas vaginalis) Clinical Presentation in the United States is estimated at 5 million cases annu- Urethritis, Epididymitis, and Proctitis ally; chlamydia (Chlamydia trachomatis) at 3 million; gon- In men, STIs usually remain confined to the urethra. Symptoms orrhea (Neisseria gonorrhoeae), 650,000; and syphilis (Tre- of urethritis include urethral discharge, dysuria, or urethral ponema pallidum), 70,000. However, most sexually itching. The discharge of nongonococcal urethritis (NGU) is transmitted infections (STIs) are asymptomatic—con- often slight, and may not be apparent without massaging the tributing to underdiagnosis estimated at 50% or more. urethra. Discharge of NGU is usually minimal and gray, white, Diagnosis of an STI signals sexual health risk because an or mucoid rather than yellow. Discharge that is yellow and pre- STI facilitates the transmission and acquisition of other sent in greater volume most often signals infection with N STIs, including human immunodeficiency virus (HIV). gonorrhoeae. In fact, comorbid STIs increase patients’ susceptibility of Epididymitis presents as acute unilateral testicular pain acquiring and transmitting HIV by two- to fivefold. Sev- and swelling. Clinical findings include tenderness of the epi- eral studies have shown that aggressive STI prevention, didymis and ductus deferens, erythema and edema of the testing, and treatment reduces the transmission of HIV. overlying scrotal skin, urethral discharge, and dysuria. Swelling The authors discuss common clinical presentations, and tenderness may be localized or may extend to the entire screening, diagnosis, and treatment for trichomoniasis, epididymis and surrounding areas, making the epididymis less chlamydia, gonorrhea, syphilis, and herpes simplex virus. -
Eagle's Syndrome
PRACTICE case report Eagle’s syndrome: an unusual cause of a clicking jaw D R P Godden,1 S Adam,2 and R T M Woodwards,3 her jaw, although it could not be palpated. Calcification of the stylohyoid ligament is a well recognised There was mild ill-defined tenderness in radiographic finding in dental practice. Fortunately, affected the right retromandibular region. Exami- individuals seldom develop symptoms. We report a case of a nation of the TMJ was normal, with full patient whose main complaint was a loud click following jaw range of jaw movement, no muscle ten- derness, and no palpable click from the movement. This unusual presentation has not been described joint. Deep palpation of the right tonsillar before and should be considered in the differential diagnosis of fossa elicited tenderness. Examination of ‘clicking jaw’. the pharynx was otherwise normal. The panoral radiograph showed a thickened articulated stylohyoid process. Eagle’s syndrome was diagnosed and the patient Mineralisation of the stylohyoid ligament radiated to the ear. Her medical practi- underwent excision through an extra-oral is a well recognised radiographic finding tioner suspected internal derangement of approach. Through a skin crease incision, and an incidence of 18.2% has been the temporomandibular joint (TMJ) and the carotid artery, internal jugular vein reported on panoramic radiographs.1 advised her to consult her dental practi- and IX, X, XI and XII cranial nerves were The majority of patients are asympto- tioner. A panoral radiograph was taken dissected out and the stylohyoid ligament matic. However, in 1937, Eagle was the (fig.