Casts, Care, and Compartment Syndrome Nursing Best Practices
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In Diagnosis Must Be Based on Clinical Signs and Symptoms. in This Paper
242 POST-GRADUATE MEDICAL JOURNAL August, 1938 Postgrad Med J: first published as 10.1136/pgmj.14.154.242 on 1 August 1938. Downloaded from SOME REMARKS ON DIFFERENTIAL DIAGNOSIS OF BLOOD DISEASES. By A. PINEY, M.D., M.R.C.P. (Assistant Physician, St. Mary's Hospital for Women and Children.) Differential diagnosis of blood diseases has been discussed time and again, but, as a rule, blood-pictures, rather than clinical features, have been taken into account, so that the impression has become widespread that the whole problem is one for the laboratory, rather than for the bed-side. It is obvious, however, that the first steps in diagnosis must be based on clinical signs and symptoms. In this paper, there- fore, certain outstanding clinical features of blood diseases, and various rather puzzling syndromes will be described. The outstanding external sign that leads the practitioner to consider the possi- bility of a blood disease is pallor, which is not quite so simple a state as is often supposed. It is, of course, well known that cutaneous pallor is not an infallible sign of anaemia, but it is often presumed that well-coloured mucous membranes are fairly good evidence that anaemia is not present. This is not necessarily true. The conjunctive may be bright pink in spite of anaemia, because mild inflammationProtected by copyright. may be present, masking the pallor. This is quite frequently due to irritation by eyelash dyes. Similarly, the finger-nails, which used to serve as a reliable index of pallor, are now found disguised with coloured varnish. -
Review Cutaneous Patterns Are Often the Only Clue to a a R T I C L E Complex Underlying Vascular Pathology
pp11 - 46 ABstract Review Cutaneous patterns are often the only clue to a A R T I C L E complex underlying vascular pathology. Reticulate pattern is probably one of the most important DERMATOLOGICAL dermatological signs of venous or arterial pathology involving the cutaneous microvasculature and its MANIFESTATIONS OF VENOUS presence may be the only sign of an important underlying pathology. Vascular malformations such DISEASE. PART II: Reticulate as cutis marmorata congenita telangiectasia, benign forms of livedo reticularis, and sinister conditions eruptions such as Sneddon’s syndrome can all present with a reticulate eruption. The literature dealing with this KUROSH PARSI MBBS, MSc (Med), FACP, FACD subject is confusing and full of inaccuracies. Terms Departments of Dermatology, St. Vincent’s Hospital & such as livedo reticularis, livedo racemosa, cutis Sydney Children’s Hospital, Sydney, Australia marmorata and retiform purpura have all been used to describe the same or entirely different conditions. To our knowledge, there are no published systematic reviews of reticulate eruptions in the medical Introduction literature. he reticulate pattern is probably one of the most This article is the second in a series of papers important dermatological signs that signifies the describing the dermatological manifestations of involvement of the underlying vascular networks venous disease. Given the wide scope of phlebology T and its overlap with many other specialties, this review and the cutaneous vasculature. It is seen in benign forms was divided into multiple instalments. We dedicated of livedo reticularis and in more sinister conditions such this instalment to demystifying the reticulate as Sneddon’s syndrome. There is considerable confusion pattern. -
Anemia in Children with Palmar Pallor Aged 02 Months to 05 Years
eCommons@AKU Department of Paediatrics and Child Health Division of Woman and Child Health 2-28-2017 Anemia in children with palmar pallor aged 02 months to 05 years Saroop Chand Farzana Shaikh Chetan Das Yasmeen Memon Mohammad Akbar Nizamani See next page for additional authors Follow this and additional works at: https://ecommons.aku.edu/ pakistan_fhs_mc_women_childhealth_paediatr Part of the Pediatrics Commons Authors Saroop Chand, Farzana Shaikh, Chetan Das, Yasmeen Memon, Mohammad Akbar Nizamani, and Zulfiqar Ali Qutrio Baloch IAJPS 2017, 4 (02), 290-295 Zulfiqar Ali Qutrio Baloch et al ISSN 2349-7750 CODEN (USA): IAJPBB ISSN: 2349-7750 INDO AMERICAN JOURNAL OF PHARMACEUTICAL SCIENCES http://doi.org/10.5281/zenodo.345648 Available online at: http://www.iajps.com Research Article ANEMIA IN CHILDREN WITH PALMAR PALLOR AGED 02 MONTHS TO 05 YEARS Dr. Saroop Chand1, Dr. Farzana Shaikh1, Dr. Chetan Das1, Dr. Yasmeen Memon1, Dr. Mohammad Akbar Nizamani1 and *Dr. Zulfiqar Ali Qutrio Baloch2 1Department of pediatrics Liaquat University of Medical and Health Sciences (LUMHS). 2Brandon Regional Hospital, Brandon, Florida. Received: 10 February 2016 Accepted: 25 February 2017 Published: 28 February 2017 Absract: Objective: To determine the frequency of anemia in children with palmar pallor aged 02 months to 05 years Patients and Methods: This cross sectional descriptive study of six months (01-12-2012 to 31-05-2013) was conducted in the department of paediatrics at Liaquat University Hospital Hyderabad. All the children, from 02 months to 05 years, of either gender had palmar pallor on examination were recruited and evaluated for anemia by assessing the level of haemoglobin and categorized anemia as mild, moderate and severe. -
Livedoid Vasculopathy Associated with Peripheral Neuropathy: a Report of Two Cases* Vasculopatia Livedoide Associada a Neuropatia Periférica: Relato De Dois Casos
CASE REPORT 227 s Livedoid vasculopathy associated with peripheral neuropathy: a report of two cases* Vasculopatia livedoide associada a neuropatia periférica: relato de dois casos Mariana Quirino Tubone1 Gabriela Fortes Escobar1 Juliano Peruzzo1 Pedro Schestatsky2 Gabriela Maldonado3 DOI: http://dx.doi.org/10.1590/abd1806-4841.20132363 Abstract: Livedoid vasculopathy (LV) is a chronic and recurrent disease consisting of livedo reticularis and sym- metric ulcerations, primarily located on the lower extremities, which heal slowly and leave an atrophic white scar ("atrophie blanche"). Neurological involvment is rare and presumed to be secondary to the ischemia from vascu- lar thrombosis of the vasa nervorum. Laboratory evaluation is needed to exclude secondary causes such as hyper- coagulable states, autoimmune disorders and neoplasms. We present two patients with a rare association of peripheral neuropathy and LV, thereby highlighting the importance of a multidisciplinary approach to reach the correct diagnosis. Keywords: Livedo reticularis; Mononeuropathies; Polyneuropathies; Skin diseases, vascular Resumo: Vasculopatia livedoide é uma doença crônica e recorrente caracterizada por livedo reticular e úlceras simétricas nos membros inferiores, que cicatrizam e deixam uma cicatriz branca atrófica ("atrophie blanche"). Envolvimento neurológico é raro e está provavelmente associado a isquemia pela trombose dos vasa nervorum. Avaliação laboratorial é indicada com o intuito de excluir causas secundárias como estados de hipercoagulabili- dade, doenças autoimunes e neoplasias. Apresentamos dois pacientes com uma rara associação de vasculopatia livedoide com neuropatia periférica, enfatizando a importância de uma abordagem multidisciplinar na busca do diagnóstico correto. Palavras-chave: Dermatopatias vasculares; Livedo reticular; Mononeuropatias; Polineuropatias INTRODUCTION Livedoid vasculopathy (LV) is a chronic and resentation of the dermo-hypodermic junction, was recurrent disease, usually restricted to the skin, and compatible with LV. -
Case Report Forearm Compartment Syndrome Following Thrombolytic Therapy for Massive Pulmonary Embolism: a Case Report and Review of Literature
Hindawi Publishing Corporation Case Reports in Orthopedics Volume 2011, Article ID 678525, 4 pages doi:10.1155/2011/678525 Case Report Forearm Compartment Syndrome following Thrombolytic Therapy for Massive Pulmonary Embolism: A Case Report and Review of Literature Ravi Badge and Mukesh Hemmady Department of Trauma and Orthopaedics Surgery, Wrightington, Wigan and Leigh NHS Trust, Wigan WN1 2NN, UK Correspondence should be addressed to Ravi Badge, [email protected] Received 2 November 2011; Accepted 6 December 2011 Academic Editor: M. K. Lyons Copyright © 2011 R. Badge and M. Hemmady. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Use of thrombolytic therapy in pulmonary embolism is restricted in cases of massive embolism. It achieves faster lysis of the thrombus than the conventional heparin therapy thus reducing the morbidity and mortality associated with PE. The compartment syndrome is a well-documented, potentially lethal complication of thrombolytic therapy and known to occur in the limbs involved for vascular lines or venepunctures. The compartment syndrome in a conscious and well-oriented patient is mainly diagnosed on clinical ground with its classical signs and symptoms like disproportionate pain, tense swollen limb and pain on passive stretch. However these findings may not be appropriately assessed in an unconscious patient and therefore the clinicians should have high index of suspicion in a patient with an acutely swollen tense limb. In such scenarios a prompt orthopaedic opinion should be considered. In this report, we present a case of acute compartment syndrome of the right forearm in a 78 years old male patient following repeated attempts to secure an arterial line for initiating the thrombolytic therapy for the management of massive pulmonary embolism. -
Cerebral Venous Thrombosis and Livedo Reticularis in a Case with MTHFR 677TT Homozygote
Journal of Clinical Neurology / Volume 2 / June, 2006 Case Report Cerebral Venous Thrombosis and Livedo Reticularis in a Case with MTHFR 677TT Homozygote Jee-Young Lee, M.D., Manho Kim, M.D., Ph.D. Department of Neurology, College of Medicine, Seoul National University, Seoul, Korea Hyperhomocysteinemia associated with methylene terahydrofolate reductase (MTHFR) mutation can be a risk factor for idiopathic cerebral venous thrombosis. We describe the first case of MTHFR 677TT homozygote with cerebral venous thrombosis and livedo reticularis. A 45-year-old man presented with seizures and mottled-like skin lesions, that were aggravated by cold temperature. Hemorrhagic infarct in the right frontoparietal area with superior sagittal sinus thrombosis was observed. He had hyperhomocysteinemia, low plasma folate level, and MTHFR 677TT homozygote genotype, which might be associated with livedo reticularis and increase the risk for cerebral venous thrombosis. J Clin Neurol 2(2):137-140, 2006 Key Words : Livedo reticularis, Methylene tetrahydrofolate reductase, Cerebral venous thrombosis Hyperhomocysteinemia causes vascular endothelial venous infarct due to cerebral venous thrombosis. damage that result in atherosclerosis and ischemic strokes.1 It is also associated with prothrombotic state or venous thromboembolism2 including cerebral venous CASE REPORT thrombosis.3 Among the thrombophilic factors with hyperhomocysteinemia, methylene tetrahydrofolate reduc- A 45 year-old man was brought to the emergency tase (MTHFR) mutant (C677 → T, homozygote) with room with uncontrolled seizures. Two days ago, sudden low plasma folate concentration increases the risk for paresthesia in left arm developed, which progressed to cerebral venous thrombosis.4 MTHFR 677TT is thermo- tonic posturing and leftward head version, followed by labile and sensitive to temperature alteration.5 a generalized tonic clonic seizure. -
Orthopedic Trauma Postoperative Care and Rehab
Orthopedic Trauma Postoperative Care and Rehab Serge Charles Kaska, MD Name that Beach 100$ Still 100$ 75$ 50$ 1$ Omaha • June 6th 1941 • !st Infantry Division • 2000 KIA LIFE OR LIMB THREAT 1. Compartment Syndrome 2. Fat Emboli Syndrome 3. Pulmonary Embolism 4. Shock Compartment syndrome case A 16 year old male was retrieving a tire from his truck bed on the side of the highway in the pouring rain when a car careens off of the road and sandwiches the patients legs between the bumpers at freeway speed. Acute compartment syndrome Compartment syndrome DEFINED Definition: Elevated tissue pressure within a closed fascial space • Pathogenesis – Too much in-flow: results in edema or hemorrhage – Decreased outflow: results in venous obstruction caused by tight dressing and/or cast. • Reduces tissue perfusion • Results in cell death Compartment syndrome tissue survival • Muscle – 3-4 hours: reversible changes – 6 hours: variable damage – 8 hours: irreversible changes • Nerve – 2 hours: looses nerve conduction – 4 hours: neuropraxia – 8 hours: irreversible changes Physical exam 1. Pain 2. Pain 3. Pain Physical exam • Inspection – Swelling, skin is tight and shiny • Motion – Active motion will be refused or unable. Must see dorsiflexion • Palpation – Severe pain with palpation • Alarming pain with passive stretch Physical exam • Dorsiflexion Physical Exam • Palpation – Severe pain with palpation • Alarming pain with passive stretch Physical exam • Evaluations from nurses, therapists, and orthotech’s are CRITICAL • If you call a doctor and say that -
Mechanical Ventilation Guide
MAYO CLINIC MECHANICAL VENTILATION GUIDE RESP GOALS INITIAL MONITORING TARGETS FAILURE SETTINGS 6 P’s BASIC HEMODYNAMIC 1 BLOOD PRESSURE SBP > 90mmHg STABILITY PEAK INSPIRATORY 2 < 35cmH O PRESSURE (PIP) 2 BAROTRAUMA PLATEAU PRESSURE (P ) < 30cmH O PREVENTION PLAT 2 SAFETY SAFETY 3 AutoPEEP None VOLUTRAUMA Start Here TIDAL VOLUME (V ) ~ 6-8cc/kg IBW PREVENTION T Loss of AIRWAY Female ETT 7.0-7.5 AIRWAY / ETT / TRACH Patent Airway MAINTENANCE Male ETT 8.0-8.5 AIRWAY AIRWAY FiO2 21 - 100% PULSE OXIMETRY (SpO2) > 90% Hypoxia OXYGENATION 4 PEEP 5 [5-15] pO2 > 60mmHg 5’5” = 350cc [max 600] pCO2 40mmHg TIDAL 6’0” = 450cc [max 750] 5 VOLUME 6’5” = 500cc [max 850] ETCO2 45 Hypercapnia VENTILATION pH 7.4 GAS GAS EXCHANGE BPM (RR) 14 [10-30] GAS EXCHANGE MINUTE VENTILATION (VMIN) > 5L/min SYNCHRONY WORK OF BREATHING Decreased High Work ASSIST CONTROL MODE VOLUME or PRESSURE of Breathing PATIENT-VENTILATOR AC (V) / AC (P) 6 Comfortable Breaths (WOB) SUPPORT SYNCHRONY COMFORT COMFORT 2⁰ ASSESSMENT PATIENT CIRCUIT VENT Mental Status PIP RR, WOB Pulse, HR, Rhythm ETT/Trach Position Tidal Volume (V ) Trachea T Blood Pressure Secretions Minute Ventilation (V ) SpO MIN Skin Temp/Color 2 Connections Synchrony ETCO Cap Refill 2 Air-Trapping 1. Recognize Signs of Shock Work-up and Manage 2. Assess 6Ps If single problem Troubleshoot Cause 3. If Multiple Problems QUICK FIX Troubleshoot Cause(s) PROBLEMS ©2017 Mayo Clinic Foundation for Medical Education and Research CAUSES QUICK FIX MANAGEMENT Bleeding Hemostasis, Transfuse, Treat cause, Temperature control HYPOVOLEMIA Dehydration Fluid Resuscitation (End points = hypoxia, ↑StO2, ↓PVI) 3rd Spacing Treat cause, Beware of hypoxia (3rd spacing in lungs) Pneumothorax Needle D, Chest tube Abdominal Compartment Syndrome FLUID Treat Cause, Paralyze, Surgery (Open Abdomen) OBSTRUCTED BLOOD RETURN Air-Trapping (AutoPEEP) (if not hypoxic) Pop off vent & SEE SEPARATE CHART PEEP Reduce PEEP Cardiac Tamponade Pericardiocentesis, Drain. -
Acute Compartment Syndrome Complicating Deep Venous Thrombosis
SMGr up Case Report SM Journal of Acute Compartment Syndrome Case Reports Complicating Deep Venous Thrombosis Senthil Dhayalan1*, David Jardine1*, Tony Goh2 and Nicholas Lash3 1Senior registrar and General Physician, Dept of General Medicine, NZ 2Consultant Radiologist, Dept of Radiology, NZ 3Orthopaediac Surgeon, Dept of Orthopaedics, NZ A 40-yr old, heavily-built man initially presented to his general practitioner 2 days before Article Information admission with recent onset of pain and swelling in his left calf. A duplex ultrasound scan Received date: Nov 21, 2018 demonstrated a popliteal and lower leg deep vein thrombosis [DVT], extending 15 cm above the Accepted date: Nov 26, 2018 knee into the femoral vein. He was started appropriately on enoxaparin 130 mg bd. Despite the Published date: Nov 27, 2018 treatment, the pain got worse, particularly when standing, and he was admitted for symptom control. Five weeks earlier he had undergone anterior cruciate ligament repair of the left knee, *Corresponding author [without heparin prophylaxis] and made a satisfactory recovery (Figure 1). Jardine D, General Medicine On examination he was mildly distressed with a low-normal blood pressure [110/70 mmHg] Department, Christchurch hospital, and a persistent low-grade fever [temperature ranged from 37.0-37.8 oC]. The left leg was moderately Private Bag 4710, Christchurch 8140, diffusely swollen, slightly warm and darker in colour [figure]. The pain was localised to the posterior New Zealand, compartments and was exacerbated by dorsi-flexing the ankle and squeezing the gastrocnemius. Email: [email protected] There were no signs of joint effusion, thrombophlebitis, lymphadenopathy or cellulitis. -
Bilateral Atraumatic Compartment Syndrome of the Legs Leading to Rhabdomyolysis and Acute Renal Failure Following Prolonged Kneeling in a Heroin Addict
PAJT 10.5005/jp-journals-10030-1075 CASE REPORTBilateral Atraumatic Compartment Syndrome of the Legs Leading to Rhabdomyolysis and Acute Renal Failure Bilateral Atraumatic Compartment Syndrome of the Legs Leading to Rhabdomyolysis and Acute Renal Failure Following Prolonged Kneeling in a Heroin Addict. A Case Report and Review of Relevant Literature Saptarshi Biswas, Ramya S Rao, April Duckworth, Ravi Kothuru, Lucio Flores, Sunil Abrol ABSTRACT cerrado, que interfiere con la circulación de los componentes mioneurales del compartimento. Síndrome compartimental Introduction: Compartment syndrome is defined as a symptom bilateral de las piernas es una presentación raro que requiere complex caused by increased pressure of tissue fluid in a closed una intervención quirúrgica urgente. En un reporte reciente osseofascial compartment which interferes with circulation (Khan et al 2012), ha habido reportados solo 8 casos de to the myoneural components of the compartment. Bilateral síndrome compartimental bilateral. compartment syndrome of the legs is a rare presentation Se sabe que el abuso de heroína puede causar el síndrome requiring emergent surgical intervention. In a recent case report compartimental y rabdomiólisis traumática y atraumática. El (Khan et al 2012) there have been only eight reported cases hipotiroidismo también puede presentarse independiente con cited with bilateral compartment syndrome. rabdomiólisis. Heroin abuse is known to cause compartment syndrome, traumatic and atraumatic rhabdomyolysis. Hypothyroidism can Presentación del caso: Presentamos un caso de una mujer also independently present with rhabdomyolysis. de 22 años quien presentó con tumefacción bilateral de las piernas asociado con la perdida de la sensación, después Case presentation: We present a case of a 22 years old female de pasar dos días arrodillado contra una pared después de who presented with bilateral swelling of the legs with associated usar heroína intravenosa. -
Assessment, Management and Decision Making in the Treatment of Polytrauma Patients with Head Injuries
Compartment Syndrome Andrew H. Schmidt, M.D. Professor, Dept. of Orthopedic Surgery, Univ. of Minnesota Chief, Department of Orthopaedic Surgery Hennepin County Medical Center April 2016 Disclosure Information Andrew H. Schmidt, M.D. Conflicts of Commitment/ Effort Board of Directors: OTA Critical Issues Committee: AOA Editorial Board: J Knee Surgery, J Orthopaedic Trauma Medical Director, Director Clinical Research: Hennepin County Med Ctr. Disclosure of Financial Relationships Royalties: Thieme, Inc.; Smith & Nephew, Inc. Consultant: Medtronic, Inc.; DGIMed; Acumed; St. Jude Medical (spouse) Stock: Conventus Orthopaedics; Twin Star Medical; Twin Star ECS; Epien; International Spine & Orthopedic Institute, Epix Disclosure of Off-Label and/or investigative Uses I will not discuss off label use and/or investigational use in my presentation. Objectives • Review Pathophysiology of Acute Compartment Syndrome • Review Current Diagnosis and Treatment – Risk Factors – Clinical Findings – Discuss role and technique of compartment pressure monitoring. Pathophysiology of Compartment Syndrome Pressure Inflexible Fascia Injured Muscle Vascular Consequences of Elevated Intracompartment Pressure: A-V Gradient Theory Pa (High) Pv (Low) artery arteriole capillary venule vein Local Blood Pa - Pv Flow = R Matsen, 1980 Increased interstitial pressure Pa (High) Tissue ischemia artery arteriole capillary venule vein Lysis of cell walls Release of osmotically active cellular contents into interstitial fluid Increased interstitial pressure More cellular -
Cerebellar Disease in the Dog and Cat
CEREBELLAR DISEASE IN THE DOG AND CAT: A LITERATURE REVIEW AND CLINICAL CASE STUDY (1996-1998) b y Diane Dali-An Lu BVetMed A thesis submitted for the degree of Master of Veterinary Medicine (M.V.M.) In the Faculty of Veterinary Medicine University of Glasgow Department of Veterinary Clinical Studies Division of Small Animal Clinical Studies University of Glasgow Veterinary School A p ril 1 9 9 9 © Diane Dali-An Lu 1999 ProQuest Number: 13815577 All rights reserved INFORMATION TO ALL USERS The quality of this reproduction is dependent upon the quality of the copy submitted. In the unlikely event that the author did not send a com plete manuscript and there are missing pages, these will be noted. Also, if material had to be removed, a note will indicate the deletion. uest ProQuest 13815577 Published by ProQuest LLC(2018). Copyright of the Dissertation is held by the Author. All rights reserved. This work is protected against unauthorized copying under Title 17, United States C ode Microform Edition © ProQuest LLC. ProQuest LLC. 789 East Eisenhower Parkway P.O. Box 1346 Ann Arbor, Ml 48106- 1346 GLASGOW UNIVERSITY lib ra ry ll5X C C ^ Summary SUMMARY________________________________ The aim of this thesis is to detail the history, clinical findings, ancillary investigations and, in some cases, pathological findings in 25 cases of cerebellar disease in dogs and cats which were presented to Glasgow University Veterinary School and Hospital during the period October 1996 to June 1998. Clinical findings were usually characteristic, although the signs could range from mild tremor and ataxia to severe generalised ataxia causing frequent falling over and difficulty in locomotion.