What Are Panic Attacks?
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No Limits Freediving
1 No Limits Freediving "The challenges to the respiratory function of the breath-hold diver' are formidable. One has to marvel at the ability of the human body to cope with stresses that far exceed what normal terrestrial life requires." Claes Lundgren, Director, Center for Research and Education in Special Environments A woman in a deeply relaxed state floats in the water next to a diving buoy. She is clad in a figure-hugging wetsuit, a dive computer strapped to her right wrist, and another to her calf. She wears strange form-hugging silicone goggles that distort her eyes, giving her a strange bug-eyed appearance. A couple of meters away, five support divers tread water near a diving platform, watching her perform an elaborate breathing ritual while she hangs onto a metal tube fitted with two crossbars. A few meters below the buoy, we see that the metal tube is in fact a weighted sled attached to a cable descending into the dark-blue water. Her eyes are still closed as she begins performing a series of final inhalations, breathing faster and faster. Photographers on the media boats snap pictures as she performs her final few deep and long hyperventilations, eliminating carbon dioxide from her body. Then, a thumbs-up to her surface crew, a pinch of the nose clip, one final lungful of air, and the woman closes her eyes, wraps her knees around the bottom bar of the sled, releases a brake device, and disappears gracefully beneath the waves. The harsh sounds of the wind and waves suddenly cease and are replaced by the effervescent bubbling of air being released from the regulators of scuba-divers. -
Respiratory Mechanics and Breathing Pattern T Jacopo P
Respiratory Physiology & Neurobiology 261 (2019) 48–54 Contents lists available at ScienceDirect Respiratory Physiology & Neurobiology journal homepage: www.elsevier.com/locate/resphysiol How to breathe? Respiratory mechanics and breathing pattern T Jacopo P. Mortola Department of Physiology, McGill University, 3655 Promenade Sir William Osler, room 1121, Montreal, Quebec, H3G 1Y6, Canada ARTICLE INFO ABSTRACT Keywords: On theoretical grounds any given level of pulmonary or alveolar ventilation can be obtained at various absolute Allometry lung volumes and through many combinations of tidal volume, breathing frequency and inspiratory and ex- Breathing frequency piratory timing. However, inspection of specific cases of newborn and adult mammals at rest indicates thatthe Human infant breathing pattern reflects a principle of economy oriented toward minimal respiratory work. The mechanisms Neonatal respiration that permit optimization of respiratory cost are poorly understood; yet, it is their efficiency and coordination that Work of breathing permits pulmonary ventilation at rest to require only a minimal fraction of resting metabolism. The sensitivity of the breathing pattern to the mechanical properties implies that tidal volume, breathing rate, mean inspiratory flow or other ventilatory parameters cannot be necessarily considered indicators proportional to thecentral neural respiratory ‘drive’. The broad conclusion is that the breathing pattern adopted by newborn and adult mammals is the one that produces the adequate alveolar ventilation -
Sleep Apnea Sleep Apnea
Health and Safety Guidelines 1 Sleep Apnea Sleep Apnea Normally while sleeping, air is moved at a regular rhythm through the throat and in and out the lungs. When someone has sleep apnea, air movement becomes decreased or stops altogether. Sleep apnea can affect long term health. Types of sleep apnea: 1. Obstructive sleep apnea (narrowing or closure of the throat during sleep) which is seen most commonly, and, 2. Central sleep apnea (the brain is causing a change in breathing control and rhythm) Obstructive sleep apnea (OSA) About 25% of all adults are at risk for sleep apnea of some degree. Men are more commonly affected than women. Other risk factors include: 1. Middle and older age 2. Being overweight 3. Having a small mouth and throat Down syndrome Because of soft tissue and skeletal alterations that lead to upper airway obstruction, people with Down syndrome have an increased risk of obstructive sleep apnea. Statistics show that obstructive sleep apnea occurs in at least 30 to 75% of people with Down syndrome, including those who are not obese. In over half of person’s with Down syndrome whose parents reported no sleep problems, sleep studies showed abnormal results. Sleep apnea causing lowered oxygen levels often contributes to mental impairment. How does obstructive sleep apnea occur? The throat is surrounded by muscles that are active controlling the airway during talking, swallowing and breathing. During sleep, these muscles are much less active. They can fall back into the throat, causing narrowing. In most people this doesn’t affect breathing. However in some the narrowing can cause snoring. -
Respiratory Issues in Rett Syndrome Dr. Marianna Sockrider, Pediatric
RettEd Q&A: Respiratory Issues in Rett Syndrome Dr. Marianna Sockrider, Pediatric Pulmonologist, Texas Children's Hospital Webcast 02/13/2018 Facilitator: Paige Nues, Rettsyndrome.org Recording link: https://register.gotowebinar.com/recording/1810253924903738120 Attendee Questions Response Breathing Irregularities Why is breathing so funny when our girls/ A behavioral arousal can trigger breathing abnormalities in Rett boys wake up, almost as if startled? syndrome. A person goes through stages of sleep and particularly if aroused from a deep sleep state (REM) may be more disoriented or startled. This can trigger the irregular breathing. Could she address the stereotypical You may observe breathing abnormalities like breath holding breathing abnormalities such as gasping and hyperventilation more with the stress of an acute and breath holding and how they play a respiratory illness. Breath holding and hyperventilation do not part in respiratory illness? directly cause respiratory illness. If a person has difficulty with swallowing and has these breathing episodes while trying to eat or drink, aspiration could occur which can cause respiratory symptoms. If one has very shallow breathing, especially when there is more mucus from acute infection, it may be more likely to build up in the lower lungs causing airway obstruction and atelectasis (collapse of some air sacs). Is there any evidence (even anecdotal) Frequent breath-holding and hyperventilation has been Reference 1 that breathing patterns change in Rett reported to become less evident with increasing age though it is patients over time? not certain whether this could be that families are used to the irregular breathing and don’t report it as much or that it is the people who live longer who are less symptomatic. -
Dyspnoea and Its Measurement
REVIEW Key points Dyspnoea is the sensation of breathing discom- fort that can be described with different terms according to different pathophysiological mechanisms that vary in intensity. The mechanisms of dyspnoea are complex. In COPD, whilst the intensity and quality of dys- pnoea during activity correlates with the magni- tude of lung hyperinflation and inspiratory events, it correlates poorly with FEV1. Valid, reliable and responsive instruments are available to measure the severity of dyspnoea in patients with respiratory disease. 100 Breathe | December 2004 | Volume 1 | No 2 REVIEW N. Ambrosino1 Dyspnoea and its G. Scano2 measurement 1 Pulmonary Unit, Cardio-Thoracic Dept, University-Hospital Pisa, Pisa, and 2Clinica Medica, University-Hospital Careggi, CME article: educational aims Firenze, Italy. To introduce dyspnoea and explain its mechanisms. Correspondence: To present dyspnoea descriptors, which may help in the understanding of the language of N. Ambrosino dyspnoea, and to relate these to specific diseases. Pulmonary Unit To describe some of the methods available for the measurement of dyspnoea. Cardio-Thoracic Dept Azienda Ospedaliera-Universitaria Pisana Via Paradisa 2, Cisanello Summary 56124 Pisa Italy Fax: 39 50996786 Dyspnoea, a term used to characterise a subjective experience of breathing discomfort, is E-mail: perhaps the most important symptom in cardiorespiratory disease. Receptors in the air- [email protected] ways, lung parenchyma, respiratory muscles and chemoreceptors provide sensory feed- back via vagal, phrenic and intercostal nerves to the spinal cord, medulla and higher cen- tres. Knowledge of dyspnoea descriptors can help in understanding the language of dys- pnoea and these are presented here. It is important to appreciate that differences in lan- guage, race, culture, sex and previous experience can all change the perception of and the manner in which the feeling of being dyspnoeic is expressed to others. -
Livedoid Vasculopathy Associated with Peripheral Neuropathy: a Report of Two Cases* Vasculopatia Livedoide Associada a Neuropatia Periférica: Relato De Dois Casos
CASE REPORT 227 s Livedoid vasculopathy associated with peripheral neuropathy: a report of two cases* Vasculopatia livedoide associada a neuropatia periférica: relato de dois casos Mariana Quirino Tubone1 Gabriela Fortes Escobar1 Juliano Peruzzo1 Pedro Schestatsky2 Gabriela Maldonado3 DOI: http://dx.doi.org/10.1590/abd1806-4841.20132363 Abstract: Livedoid vasculopathy (LV) is a chronic and recurrent disease consisting of livedo reticularis and sym- metric ulcerations, primarily located on the lower extremities, which heal slowly and leave an atrophic white scar ("atrophie blanche"). Neurological involvment is rare and presumed to be secondary to the ischemia from vascu- lar thrombosis of the vasa nervorum. Laboratory evaluation is needed to exclude secondary causes such as hyper- coagulable states, autoimmune disorders and neoplasms. We present two patients with a rare association of peripheral neuropathy and LV, thereby highlighting the importance of a multidisciplinary approach to reach the correct diagnosis. Keywords: Livedo reticularis; Mononeuropathies; Polyneuropathies; Skin diseases, vascular Resumo: Vasculopatia livedoide é uma doença crônica e recorrente caracterizada por livedo reticular e úlceras simétricas nos membros inferiores, que cicatrizam e deixam uma cicatriz branca atrófica ("atrophie blanche"). Envolvimento neurológico é raro e está provavelmente associado a isquemia pela trombose dos vasa nervorum. Avaliação laboratorial é indicada com o intuito de excluir causas secundárias como estados de hipercoagulabili- dade, doenças autoimunes e neoplasias. Apresentamos dois pacientes com uma rara associação de vasculopatia livedoide com neuropatia periférica, enfatizando a importância de uma abordagem multidisciplinar na busca do diagnóstico correto. Palavras-chave: Dermatopatias vasculares; Livedo reticular; Mononeuropatias; Polineuropatias INTRODUCTION Livedoid vasculopathy (LV) is a chronic and resentation of the dermo-hypodermic junction, was recurrent disease, usually restricted to the skin, and compatible with LV. -
How to Help Someone Having a Panic Attack
How to Help Someone Having a Panic Attack 1. Understand what a panic attack is. A panic attack is a sudden attack of extreme anxiety. It can occur without warning and for no obvious reason. The symptoms are listed under the tips sections of this article. In extreme cases, the symptoms may be accompanied by an acute fear of dying. Although they are quite distressing, panic attacks are not usually life-threatening and can last from 5 - 20 minutes. It is important to note that the signs and symptoms of a panic attack can be similar to those of a heart attack. 2. If this is the first time the person has had something like this, seek emergency medical attention. When in doubt, it is always best to seek immediate medical attention. If the person has diabetes, asthma or other medical problems, seek medical help. 3. Find out the cause of the attack. Talk to the person and determine if he or she is having a panic attack and not another kind of medical emergency (such as a heart or asthma attack) which would require immediate medical attention. Check that the cause of poor breathing is not asthma, as asthma is an entirely different condition and requires different treatments. WARNINGS • Panic attacks, especially to someone who has never had one before, often seem like heart attacks. But heart attacks can be deadly, and if there's any question as to which one it is, it's best to call emergency services. • It should be noted that many asthma sufferers have panic attacks. -
Cerebral Venous Thrombosis and Livedo Reticularis in a Case with MTHFR 677TT Homozygote
Journal of Clinical Neurology / Volume 2 / June, 2006 Case Report Cerebral Venous Thrombosis and Livedo Reticularis in a Case with MTHFR 677TT Homozygote Jee-Young Lee, M.D., Manho Kim, M.D., Ph.D. Department of Neurology, College of Medicine, Seoul National University, Seoul, Korea Hyperhomocysteinemia associated with methylene terahydrofolate reductase (MTHFR) mutation can be a risk factor for idiopathic cerebral venous thrombosis. We describe the first case of MTHFR 677TT homozygote with cerebral venous thrombosis and livedo reticularis. A 45-year-old man presented with seizures and mottled-like skin lesions, that were aggravated by cold temperature. Hemorrhagic infarct in the right frontoparietal area with superior sagittal sinus thrombosis was observed. He had hyperhomocysteinemia, low plasma folate level, and MTHFR 677TT homozygote genotype, which might be associated with livedo reticularis and increase the risk for cerebral venous thrombosis. J Clin Neurol 2(2):137-140, 2006 Key Words : Livedo reticularis, Methylene tetrahydrofolate reductase, Cerebral venous thrombosis Hyperhomocysteinemia causes vascular endothelial venous infarct due to cerebral venous thrombosis. damage that result in atherosclerosis and ischemic strokes.1 It is also associated with prothrombotic state or venous thromboembolism2 including cerebral venous CASE REPORT thrombosis.3 Among the thrombophilic factors with hyperhomocysteinemia, methylene tetrahydrofolate reduc- A 45 year-old man was brought to the emergency tase (MTHFR) mutant (C677 → T, homozygote) with room with uncontrolled seizures. Two days ago, sudden low plasma folate concentration increases the risk for paresthesia in left arm developed, which progressed to cerebral venous thrombosis.4 MTHFR 677TT is thermo- tonic posturing and leftward head version, followed by labile and sensitive to temperature alteration.5 a generalized tonic clonic seizure. -
Diver Medical | Participant Questionnaire Directions
Diver Medical | Participant Questionnaire Recreational scuba diving and freediving requires good physical and mental health. There are a few medical conditions which can be hazardous while diving, listed below. Those who have, or are predisposed to, any of these conditions, should be evaluated by a physician. This Diver Medical Participant Questionnaire provides a basis to determine if you should seek out that evaluation. If you have any concerns about your diving fitness not represented on this form, consult with your physician before diving. If you are feeling ill, avoid diving. If you think you may have a contagious disease, protect yourself and others by not participating in dive training and/or dive activities. References to “diving” on this form encompass both recreational scuba diving and freediving. This form is principally designed as an initial medical screen for new divers, but is also appropriate for divers taking continuing education. For your safety, and that of others who may dive with you, answer all questions honestly. Directions Complete this questionnaire as a prerequisite to a recreational scuba diving or freediving course. Note to women: If you are pregnant, or attempting to become pregnant, do not dive. Yes I have had problems with my lungs/breathing, heart, blood, or have been diagnosed with COVID-19. No 1 Go to Box A Yes I am over 45 years of age. No 2 Go to Box B I struggle to perform moderate exercise (for example, walk 1.6 kilometer/one mile in 14 minutes 3 or swim 200 meters/yards without resting), OR I have been unable to participate in a normal Yes No physical activity due to fitness or health reasons within the past 12 months. -
Influence of Exercise and C02 on Breathing Pattern in Patients with Chronic Obstructive Lung Disease (COLD)
Eur Respir J 1988, 1, 139-144 Influence of exercise and C02 on breathing pattern in patients with chronic obstructive lung disease (COLD) G. Scano*, F. Gigliotti*, A. van Meerhaeghe**, A. De Coster**, R. Sergysels** Influence of exercise and C02 on breathing pattern in patients with chronic • Clinica Medica III, Universita di Firenze, obstructive lung disease (COLD). G. Scano, F. Gigliotti, A. van Meerhaeghe. Italy. A. De Coster, R. Sergyse/s. •• Pulmonary Division, Hopital Universitaire ABSTRACT: In ten eucapnic patients with chronic obstructive lung disease St. Pierre (ULB), Brussels, Belgium. (COLD) we evaluated the breathing pattern during induced progressive Keywords: Breathing pattern; exercise; COPD; hypercapnia (C0 rebreathing) and progressive exercise on an ergometric 1 col rebreathing. bicycle (30 W /3 min). The time and volume components of the respiratory cycle were measured breath by breath. When compared to hypercapnia, the increase Received: January 30, 1987; accepted after in ventilation (VE) during exercise was associated with a smaDer increase in revision: September 17, 1987. tidal volume (VT) and a greater increase in respiratory frequency (fl). Plots of tidal volume (VT) against both inspiratory time (Tt) and expiratory time (TE) showed a greater decrease in both TI and TE during exercise than with hypercapnia. Analysis of YE in terms of flow (VT/TI) and timing (Tt/'I'r) showed VE to increase by a similar increase to that in VT/TI during both exerci.se and hypercapnia, while Tt/TT did not change significantly. When the patients were matched for a given VE (28 I· min -J ), exercise induced a smaller increase in VT (p < 0.05), a greater increase in fl (p < 0.025); Tl (p < 0.025) and TE (p < 0.01) were found to be smaller during exercise than hypercapnia. -
Carbon Dioxide Exposure Effects – Fact Sheet
CARBON DIOXIDE EXPOSURE EFFECTS – FACT SHEET Studies by NIOSH in 1976 dispelled the myth that carbon dioxide is an asphyxiant gas and only causes adverse health effects when it displaces oxygen. Symptoms of overexposure by inhalation include dizziness, headache, nausea, rapid breathing, shortness of breath, deeper breathing, increased heart rate (tachycardia), eye and extremity twitching, cardiac arrhythmia, memory disturbances, lack of concentration, visual and hearing disturbances (including photophobia, blurred vision, transient blindness, hearing loss and ringing in the ears), sweating, restlessness, vomiting, shaking, confusion, flushed skin, panic, parathesis (a sensation of numbness in the extremities), disorientation, convulsions, unconsciousness, coma, and death. CO2 Duration Physiological Impact/Health Effect Concentration 1,000 ppm Less than Impairs judgment, decision-making ability, and thinking skills on a 2½ hrs. short-term basis, even for healthy individuals. 2,500 ppm Less than Many individuals are rendered cognitively marginal or 2½ hrs. dysfunctional. 5,000 ppm with Headache, lethargy, mental slowness, emotional irritation, and 20.9% Oxygen sleep disruption. 6% 1-2 mins. Hearing and visual disturbances 7% (70,000 ppm) 5 mins. death with 20.9% Oxygen 10% to 15% Dizziness, drowsiness, severe muscle twitching, unconsciousness and death within a few minutes. Within 1 Loss of controlled and purposeful activity, unconsciousness, coma, 17% to 30% min. convulsions, and death 30% carbon 30 secs. Unconsciousness, with some subjects having seizures that were dioxide, with 70% characterized as decerebrate (no cerebral functioning). oxygen Even though oxygen is necessary to carry out cell functions, it is not the lack of oxygen that stimulates breathing. Breathing is stimulated by an excess of CO2. -
Beneath Tropic Seas; a Record of Diving Among the Coral Reefs of Haiti
^'%^^ C Ij > iUiJs'fr) R/V ChTiiit-UBRARY BOOKS BY WILLIAM BEEBE TWO BIRD-LOVERS IN MEXICO Houghton, Mifflin Co.—ig05 THE BIRD Henry Holt and Co.—igo6 THE LOG OP THE SUN Henry Holt and Co.—igo6 OUR SEARCH FOR A WILDERNESS Henry Holt and Co.—ig/o TROPICAL WILD LIFE New York Zoological Society—igr^ JUNGLE PEACE Henry Holt and Co. —igiS EDGE OF THE JUNGLE Henry Holt and Co.—ig2l A MONOGRAPH OF THE PHEASANTS H. F. Witherby and Co.—igi8-ig22 GALAPAGOS: WORLD'S END G. P. Putnam's Sons—ig24 JUNGLE DAYS G. P. Putnam's Sons—ig2S THE ARCTURUS ADVENTURE G. P. Putnam's Sons—ig26 pheasants: their lives and homes Doubleday, Page ^ Co. —1926 pheasant JUNGLES G. P. Putnam's Sons—Jg2y BENEATH TROPIC BEAS G. P. Putnam's Sons—1928 ^1 No-Man's-Land Five Fathoms Down Painted by Zarh Pritchard many feet beneath the water on a coral reef in the Lagoon of Maraa, Tahiti ^' BENEATH TROPIC SEAS C" ^ A RECORD OF DIVING AMONG THE CORAL REEFS OF HAITI BY WILLIAM BEEBE, ScD. ^Director of the 'Department of 'tropical 'Research of the V^ew york 'Zoological Society TH SIXTY ILLUSTRATIONS G. P. PUTNAM'S SONS NEW YORK — LONDON 1928 R/V Cjri>*«W BENEATH TROPIC SEAS ex- Copyright, 1928 by William Beebe First printing, September, 1928 Second printing, September, 1928 ^^ Made in the United States of America To ELSWYTH THANE I PREFACE The Tenth Expedition of the Department of Tropical Research of the New York Zoological Society was made possible by generous contribu- tions from members of the Society.