615.9Barref.Pdf

Total Page:16

File Type:pdf, Size:1020Kb

615.9Barref.Pdf INDEX Abortifacient, abortifacients bees, wasps, and ants ginkgo, 492 aconite, 737 epinephrine, 963 ginseng, 500 barbados nut, 829 blister beetles goldenseal blister beetles, 972 cantharidin, 974 berberine, 506 blue cohosh, 395 buckeye hawthorn, 512 camphor, 407, 408 ~-escin, 884 hypericum extract, 602-603 cantharides, 974 calamus inky cap and coprine toxicity cantharidin, 974 ~-asarone, 405 coprine, 295 colocynth, 443 camphor, 409-411 ethanol, 296 common oleander, 847, 850 cascara, 416-417 isoxazole-containing mushrooms dogbane, 849-850 catechols, 682 and pantherina syndrome, mistletoe, 794 castor bean 298-302 nutmeg, 67 ricin, 719, 721 jequirity bean and abrin, oduvan, 755 colchicine, 694-896, 698 730-731 pennyroyal, 563-565 clostridium perfringens, 115 jellyfish, 1088 pine thistle, 515 comfrey and other pyrrolizidine­ Jimsonweed and other belladonna rue, 579 containing plants alkaloids, 779, 781 slangkop, Burke's, red, Transvaal, pyrrolizidine alkaloids, 453 jin bu huan and 857 cyanogenic foods tetrahydropalmatine, 519 tansy, 614 amygdalin, 48 kaffir lily turpentine, 667 cyanogenic glycosides, 45 lycorine,711 yarrow, 624-625 prunasin, 48 kava, 528 yellow bird-of-paradise, 749 daffodils and other emetic bulbs Laetrile", 763 yellow oleander, 854 galanthamine, 704 lavender, 534 yew, 899 dogbane family and cardenolides licorice Abrin,729-731 common oleander, 849 glycyrrhetinic acid, 540 camphor yellow oleander, 855-856 limonene, 639 cinnamomin, 409 domoic acid, 214 rna huang ricin, 409, 723, 730 ephedra alkaloids, 547 ephedra alkaloids, 548 Absorption, xvii erythrosine, 29 ephedrine, 547, 549 aloe vera, 380 garlic mayapple amatoxin-containing mushrooms S-allyl cysteine, 473 podophyllotoxin, 789 amatoxin poisoning, 273-275, gastrointestinal viruses milk thistle 279 viral gastroenteritis, 205 silibinin, 555 aspartame, 24 ginger, 485 mistletoe, 793 Medical Toxicology ofNatural Substances, by Donald G. Barceloux, MD Copyright © 2008 John Wiley & Sons, Inc. 1135 INDEX Absorption (cont'd) Acorus calamus. See Calamus Carolina jessamine, 841-842 muscarine-containing mushrooms Acquired immunodeficiency syndrome eat's claw, 422-424 and muscarine toxicity, 306 (AIDS) death camas, 707-709 mycotoxins Cryptosporidium parvum, 194 ergot and related compounds, aflatoxin, 329 Cyclospora cayetanensis, 195-196 332-340 ergot alkaloids, 336, 337 cyclosporiasis, 195 kaffi r lily, 710-711 fumonisin, 342 gastrointestinal viruses, 206 mescal beans, 733-734 ochratoxin, 345 human immunodeficiency virus zephyr lily, 716-717 trichothecene compounds, 353 (HIV), 78, 128, 144, 187, 191, Allergic phytodermatitis, 678-679 neem oil, 651 194-197,206,571,602,793, Allergic reactions nicotine, 811 910, 911, 981 ant envenomation, 956, 958-959, peppermint oil non-nucleoside reverse 964,1024 menthol, 655 transcriptase inhibitors, 603 bee envenomation, 959,961-962, protozoa and intestinal parasites protease inhibitors, 473, 603 964,1024 Cryptosporidium parvum, 194 suppression, 603 begonias, 748 rhubarb Salmonella, 146 black locust oxalate, 86 scabies mite, 910 pollen, 881 salmonella, 143 Acromelalgia, 269 blister beetles scombroid fish poisoning, 257 Paxillus syndrome, 313-315 cantharidin, 974 scorpions, 916 Acromelic acid A-E compounds, bugs Shiga enterotoxins, 152 Paxillus syndrome, 313-315 venom, 971 snow drop Actaea racemosa. See Black cohosh burdock root, 401 galant amine, 714 Adult respiratory distress syndrome buttercup family, 691 solanine, 80 (ARDS) castor bean Vibrio species, 168 ants, 961 dust, 722 sassafras bees, 961 pulp, 719-720 safrole, 584 colchicine-containing plants, 695­ ricin saw palmetto 696,699 inhaled, 722 oral absorption, 588 hornets, 961 eat's claw sea snakes, 1076 Hymenoptera order, 961 allergic interstitial nephritis, senna marine animals 424 rhein anthrone, 593 palytoxin, 1098 centipede envenomation, 952 sennosides, 592, 593 mycotoxins, 356 chamomile tea, 427 skullcap, 597-598 peppermint oil, 655 chaparral, 431-432 St. John's wort, 602 pine oil, 669 chase tree, 436 terrestrial snakes, 1035 toxic oil syndrome, 11 Clostridium botulinum tree tobacco and other piperidine­ turpentine oil, 669 horse-derived antitoxin, 109 containing plants wasps, 961 clove anabasine, 812 rattlesnake envenomation, 1031, leaf oil, 440 nicotine, 811 1034 cinnamon oil, 42 ultraviolet, 731, 823 Aflatoxins,327-332 daffodils, 704 water hemlock and water dropwort, cancer, 187,331 dust mites, 355 823 black pepper seed contaminant, 72 echinacea, 571-572 yarrow, 624 food contaminant, 5 eugenol, 440 yohimbe bark and yohimbine, 629 microvesicular steatosis, 92 feverfew, 467-468 Acceptable daily intake (ADI) African blue lily, chemical fly bites, 981 food additives and sensitivities, composition and toxicity, fruit skin, 50 23-25 743-744 garlic, 474 limonene, 639 Airborne contact phytodermatitis, 685 ginkgo, 493-494 Acetaldehyde, Akee fruit, 34-38 grass pea, 64 coprine-containing mushrooms, 295 Alfimeprase, copperhead snakes, 1042 guar gum-containing products, 28 false morel, 287 Alimentary toxic aleukia (ATA), hornet food additives, 25 trichothecene mycotoxins, vespid venom, 958 peppermint, 654 354-355 horse chestnut purple coneflower, 570 Alkaloids. See also specific alkaloids pollen, 884 Achelase I and II, caterpillar venom, aconitum, 737-739 jequirity bean, 731 991 black cohosh, 388-399 jellyfish envenomation, 1089 Aconite, 736 blue cohosh, 394-396 juniper, 523 1136 INDEX lavender, 534 Allopathic medicine, senna, 591-592 fly and mosquito bites, 981 limonene, 639 S-Allyl cysteine, garlic, 473 Latrodectus antivenom, 931 Listeria monocytogenes Almonds rattlesnake venom, 1032 penicillin, 137 botanical description, 761 Anatoxin-a, cyanobacteria, 184 lupine Laetrile" extracts, 761-762 Anemonin, physiochemical properties seeds, 786 poisonous parts, 762 and toxicity, 691-692 milk thistle Aloe vera, 370-380, 414 Anethole, chemical structure, 608-609 extract, 556 Aloin, 378-380 Angelica sinensis, 461-463 mosquito bites, 981 Alternaria species Animals and animal products. See mushrooms, 268 in air, 320 also Insects; Reptiles penicillin G, 279 classification and identification, aflatoxins, 327 mycotoxins 320-324 Bacillus cereus, 89 airborne molds, 318, 320, 323 Amanita mushrooms Campylobacter jejuni, 94 antigen-specific IgA mycological identification and Clostridium botulinum, 104 concentrations, 358 classification, 265-268 Clostridium perfringens, 114-115 fungi, 324 Pantherina syndrome, 298-302 dog laurel toxicity and, 845-846 damp environments, 355-356 Paxillus syndrome, 312-315 Escherichia coli, 121 mold, 324, 326 poisonous parts, 273-275 food additives and sensitivities, 23 nutmeg, 69 silibinin as antidote, 554 fumonisins, 342-343 peppermint Amanitins gastrointestinal viruses, 204 oil, 655 amanita mushroom, 266-274 Giardia lamblia, 197-198 philodendron, 771 Amatoxin grayanotoxins in, 871 plant dermatitis, 677-679, 682, 684­ arnatoxin-containing mushrooms, lantana toxicity in, 868 685,687 265-284 Listeria monocytogenes, 133 rosemary, 576 amatoxin poisoning, 268-280 ochratoxins, 344-347 rue Ambient air oleander poisoning in, 849 extract, 580 fungal spore concentrations, Salmonella in, 142 saw palmetto 321-324 Shiga enterotoxins, 151 extract, 589 mycotoxins in, 319-320 Staphylococcus aureus, 156-157 Scheff/era species, 771 Amebiasis, 191-192 Streptococcus species, 162 scombroid fish, 258 American short-tailed shrew, 1133 tannins, 894-895 scorpion envenomation, 922 American trypanosomiasis, kissing tetrodotoxins in, 247-248 sea urchin bug, 971 Vibrio species, 167-168 spines, 1108 Amines, bee venom, 957-958 Yersinia enterocolitica, 175 senna, 594 a-Amino-5-methyl-3­ Anisatin, chemical structure, 608-609 snake envenomation, 1059 hydroxyisoxazolone-a­ Anthozoa, classification and antivenom,1034-1038 propionate (AMPA), domoic characteristics, 1096-1098 spider acid toxicity, 215 Anthranoid laxatives, cascara, antivenom, 930 1-Aminocyclopropanol, coprine toxins, 415-419 bites, 941 295 Anthraquinone derivatives sponges ~-Aminopropionitrile, osteolathyrism, cascara, 415-419 Dogger Bank itch, 1103 807-808 mushrooms, 290-293 spurge family, 756 Amnesic shellfish poisoning and rhubarb poisoning, 86 dog's mercury domoic acid, 212-217 Antibiotics, food additives and pollen, 757 Amphetamine derivatives, myristicin sensitivities, 27-28 St. John's wort metabolites and, 68 Anticholinergic signs, hypericum extract, 604 Amphibians. See Frogs; Toads; specific botulism, 106-107 tansy, 615-616 species bulbs, 705 tea tree Amygdalin burdock root, 401 oil, 661 cyanogenic foods, 45-50 Datura species, 777-781 ticks, 912 fruit kernels, 45 Escherichia coli, 127 turpentine Laetrile" derivatives, 762 European bittersweet, 804 oil, 669 Anabaena species, neurotoxins, 184 isoxazole-containing mushrooms, wandering Jew, 819 Anabasine, 810-813 301 wasp envenomation, 958, 1024 Analgesics, rattlesnake venom, lupine, 786 yellow jackets, 955 1039-1040 nutmeg, 68-69 yarrow, 624 Anaphylactoid reactions puffer fish, 249 yew, 901 ants, wasps, and bees, 961-963 snowdrop, 712-714 1137 INDEX Anticholinergic signs (cont 'd) garlic, 472 Assassin bug valerian, 620 gingko tree, 490 behavior, 970-971 water hemlock, 824 hawthorn, 511 clinical response, 971 Antidotes methyl apigenin geographic distribution, 970 belladonna alkaloid toxicity, 781 valerian, 618, 620 identifying characteristics, 969 ciguatoxins, 244 passionflower, 559 venom composition and apparatus, colchicine toxicity, 699 rosemary, 575-576 971 eugenol toxicity, 440-441 skullcap, 597 Asteraceae family nicotine toxicity, 813 yarrow, 624 airborne contact dermatitis, 685
Recommended publications
  • Ergot Alkaloids Mycotoxins in Cereals and Cereal-Derived Food Products: Characteristics, Toxicity, Prevalence, and Control Strategies
    agronomy Review Ergot Alkaloids Mycotoxins in Cereals and Cereal-Derived Food Products: Characteristics, Toxicity, Prevalence, and Control Strategies Sofia Agriopoulou Department of Food Science and Technology, University of the Peloponnese, Antikalamos, 24100 Kalamata, Greece; [email protected]; Tel.: +30-27210-45271 Abstract: Ergot alkaloids (EAs) are a group of mycotoxins that are mainly produced from the plant pathogen Claviceps. Claviceps purpurea is one of the most important species, being a major producer of EAs that infect more than 400 species of monocotyledonous plants. Rye, barley, wheat, millet, oats, and triticale are the main crops affected by EAs, with rye having the highest rates of fungal infection. The 12 major EAs are ergometrine (Em), ergotamine (Et), ergocristine (Ecr), ergokryptine (Ekr), ergosine (Es), and ergocornine (Eco) and their epimers ergotaminine (Etn), egometrinine (Emn), egocristinine (Ecrn), ergokryptinine (Ekrn), ergocroninine (Econ), and ergosinine (Esn). Given that many food products are based on cereals (such as bread, pasta, cookies, baby food, and confectionery), the surveillance of these toxic substances is imperative. Although acute mycotoxicosis by EAs is rare, EAs remain a source of concern for human and animal health as food contamination by EAs has recently increased. Environmental conditions, such as low temperatures and humid weather before and during flowering, influence contamination agricultural products by EAs, contributing to the Citation: Agriopoulou, S. Ergot Alkaloids Mycotoxins in Cereals and appearance of outbreak after the consumption of contaminated products. The present work aims to Cereal-Derived Food Products: present the recent advances in the occurrence of EAs in some food products with emphasis mainly Characteristics, Toxicity, Prevalence, on grains and grain-based products, as well as their toxicity and control strategies.
    [Show full text]
  • Feed Safety 2016
    Annual Report The surveillance programme for feed materials, complete and complementary feed in Norway 2016 - Mycotoxins, fungi and bacteria NORWEGIAN VETERINARY INSTITUTE The surveillance programme for feed materials, complete and complementary feed in Norway 2016 – Mycotoxins, fungi and bacteria Content Summary ...................................................................................................................... 3 Introduction .................................................................................................................. 4 Aims ........................................................................................................................... 5 Materials and methods ..................................................................................................... 5 Quantitative determination of total mould, Fusarium and storage fungi ........................................ 6 Chemical analysis .......................................................................................................... 6 Bacterial analysis .......................................................................................................... 7 Statistical analysis ......................................................................................................... 7 Results and discussion ...................................................................................................... 7 Cereals .....................................................................................................................
    [Show full text]
  • National Resource Material Green and Black Poison Frog (Dendrobates Auratus)
    Indicative 10 Project National Resource Material Green and Black Poison frog (Dendrobates auratus) Michelle T. Christy and Win Kirkpatrick 2017 Department of Primary Industries and Regional Development 3 Baron-Hay Court, South Perth, WA 6151 An Invasive Animals CRC Project Contents Summary ............................................................................. 2 Key Messages ................................................................... 2 Classification ................................................................... 2 Common names ................................................................ 3 Biology and Ecology ................................................................ 3 Identification ................................................................... 3 Behaviours and Traits ......................................................... 4 Food and Foraging ............................................................. 4 Reproduction and Lifecycle ................................................. 5 Habitat ......................................................................... 5 Global Range ........................................................................ 5 Potential for Introduction ........................................................ 6 Potential for Eradication.......................................................... 7 Impacts ............................................................................... 7 Economic ........................................................................ 7 Environmental
    [Show full text]
  • Clostridium Perfringens
    CLOSTRIDIUM PERFRINGENS: SPORES & CELLS MEDIA & MODELING Promotor: prof. dr. ir. Frans M. Rombouts Hoogleraar in de levensmiddelenhygiëne en –microbiologie Co-promotor: dr. Rijkelt R. Beumer Universitair docent Leerstoelgroep levensmiddelenmicrobiologie Promotiecommissie: prof. dr. ir. Johan M. Debevere (Universiteit Gent, België) dr. ir. Servé H.W. Notermans (TNO Voeding, Zeist) prof. dr. Michael W. Peck (Institute of Food Research, Norwich, UK) prof. dr. ir. Marcel H. Zwietering (Wageningen Universiteit) CLOSTRIDIUM PERFRINGENS: SPORES & CELLS MEDIA & MODELING Aarieke Eva Irene de Jong Proefschrift ter verkrijging van de graad van doctor op gezag van de rector magnificus van Wageningen Universiteit, prof. dr. ir. L. Speelman, in het openbaar te verdedigen op dinsdag 21 oktober 2003 des namiddags te vier uur in de Aula A.E.I. de Jong – Clostridium perfringens: spores & cells, media & modeling – 2003 Thesis Wageningen University, Wageningen, The Netherlands – With summary in Dutch ISBN 90-5808-931-2 ABSTRACT Clostridium perfringens is one of the five major food borne pathogens in the western world (expressed in cases per year). Symptoms are caused by an enterotoxin, for which 6% of type A strains carry the structural gene. This enterotoxin is released when ingested cells sporulate in the small intestine. Research on C. perfringens has been limited to a couple of strains that sporulate well in Duncan and Strong (DS) medium. These abundantly sporulating strains in vitro are not necessarily a representation of the most dangerous strains in vivo. Therefore, sporulation was optimized for C. perfringens strains in general. None of the tested media and methods performed well for all strains, but Peptone-Bile- Theophylline medium (with and without starch) yielded highest spore numbers.
    [Show full text]
  • Advances in Anticancer Antibody-Drug Conjugates and Immunotoxins
    Send Orders for Reprints to [email protected] Recent Patents on Anti-Cancer Drug Discovery, 2014, 9, 35-65 35 Advances in Anticancer Antibody-Drug Conjugates and Immunotoxins Franco Dosio1,*, Barbara Stella1, Sofia Cerioni1, Daniela Gastaldi2 and Silvia Arpicco1 1Dipartimento di Scienza e Tecnologia del Farmaco, University of Torino, Torino, I-10125, Italy; 2Dipartimento di Bio- tecnologie Molecolari e Scienze per la Salute, University of Torino, Torino, I-10125, Italy Received: December 13, 2012; Accepted: February 21, 2013; Revised: March 7, 2013 Abstract: Antibody-delivered drugs and toxins are poised to become important classes of cancer therapeutics. These bio- pharmaceuticals have potential in this field, as they can selectively direct highly potent cytotoxic agents to cancer cells that present tumor-associated surface markers, thereby minimizing systemic toxicity. The activity of some conjugates is of particular interest receiving increasing attention, thanks to very promising clinical trial results in hematologic cancers. Over twenty antibody-drug conjugates and eight immunotoxins in clinical trials as well as some recently approved drugs, support the maturity of this approach. This review focuses on recent advances in the development of these two classes of biopharmaceuticals: conventional toxins and anticancer drugs, together with their mechanisms of action. The processes of conjugation and purification, as reported in the literature and in several patents, are discussed and the most relevant results in clinical trials are listed. Innovative technologies and preliminary results on novel drugs and toxins, as reported in the literature and in recently-published patents (up to February 2013) are lastly examined. Keywords: Antibody drug conjugate, anticancer agents, auristatins immunotoxin, calicheamicins, cross-linkers, duocarmycins, maytansinoids.
    [Show full text]
  • AMATOXIN MUSHROOM POISONING in NORTH AMERICA 2015-2016 by Michael W
    VOLUME 57: 4 JULY-AUGUST 2017 www.namyco.org AMATOXIN MUSHROOM POISONING IN NORTH AMERICA 2015-2016 By Michael W. Beug: Chair, NAMA Toxicology Committee Assessing the degree of amatoxin mushroom poisoning in North America is very challenging. Understanding the potential for various treatment practices is even more daunting. Although I have been studying mushroom poisoning for 45 years now, my own views on potential best treatment practices are still evolving. While my training in enzyme kinetics helps me understand the literature about amatoxin poisoning treatments, my lack of medical training limits me. Fortunately, critical comments from six different medical doctors have been incorporated in this article. All six, each concerned about different aspects in early drafts, returned me to the peer reviewed scientific literature for additional reading. There remains no known specific antidote for amatoxin poisoning. There have not been any gold standard double-blind placebo controlled studies. There never can be. When dealing with a potentially deadly poisoning (where in many non-western countries the amatoxin fatality rate exceeds 50%) treating of half of all poisoning patients with a placebo would be unethical. Using amatoxins on large animals to test new treatments (theoretically a great alternative) has ethical constraints on the experimental design that would most likely obscure the answers researchers sought. We must thus make our best judgement based on analysis of past cases. Although that number is now large enough that we can make some good assumptions, differences of interpretation will continue. Nonetheless, we may be on the cusp of reaching some agreement. Towards that end, I have contacted several Poison Centers and NAMA will be working with the Centers for Disease Control (CDC).
    [Show full text]
  • The Notification of Injuries from Hazardous Substances
    The Notification of Injuries from Hazardous Substances Draft Guidelines and Reference Material for Public Health Services To be used to assist in a pilot study from July – December 2001 April 2001 Jeff Fowles, Ph.D. Prepared by the Institute of Environmental Science and Research, Limited Under contract by the New Zealand Ministry of Health DISCLAIMER This report or document ("the Report") is given by the Institute of Environmental Science and Research Limited ("ESR") solely for the benefit of the Ministry of Health, Public Health Service Providers and other Third Party Beneficiaries as defined in the Contract between ESR and the Ministry of Health, and is strictly subject to the conditions laid out in that Contract. Neither ESR nor any of its employees makes any warranty, express or implied, or assumes any legal liability or responsibility for use of the Report or its contents by any other person or organisation. Guidelines for the Notification of Injuries from Hazardous Substances April 2001 Acknowledgements The author wishes to thank Dr Michael Bates and Melissa Perks (ESR), Sally Gilbert and Helen Saba (Ministry of Health), Dr Donald Campbell (Midcentral Health), Dr Deborah Read (ERMANZ), and Dr Nerida Smith (National Poisons Centre) for useful contributions and helpful suggestions on the construction of these guidelines. Guidelines for the Notification of Injuries from Hazardous Substances April 2001 TABLE OF CONTENTS SUMMARY ........................................................................................................................1
    [Show full text]
  • Changing Pattern of Epidemic Dropsy in North India
    Epidemic Dropsy in North India N. Sharma et al. ORIGINAL ARTICLE Changing Pattern of Epidemic Dropsy in North India NAVNEET SHARMA1,*, NAINA MOHAN 2, ASHISH BHALLA1, AMAN SHARMA1, SURJIT SINGH 1 1 The Department of Internal Medicine, Postgraduate Institute of Medical Education and Research, Chandigarh, India 2 King's College London, Strand, London, United Kingdom Abstract Background: Epidemic dropsy occurs due to ingestion of mustard oil contaminated with oil from Argemone mexicana, leading to edema and tenderness of the abdomen, upper and lower limbs. In this study, clinical profiles of patients presented with epidemic dropsy in north India are described. Methods: This was a prospective study of patients presented with epidemic dropsy to the emergency department of Nehru Hospital, during the period from March 2004 to December 2011. Inclusion criteria were patients presenting with tender bilateral pitting leg edema and dermal telangiectasia. Clinical and laboratory data of patients were entered into case record forms at the time of presentation until discharge from the hospital. Results: Leg edema was the principal symptom in our series, and was in concurrence with current literature. Erythema has only been reported in 35-82% of published series, though it was present in all of our patients. Similarly, features such as diarrhea, hepatomegaly and anemia were more frequent in our cases compared to the literature. Furthermore, pancytopenia which was documented on peripheral blood counts in 54% of our cases has never been reported before. Conclusion: Epidemic dropsy should be considered in patients presenting with progressive erythema, edema, and tenderness of the limbs who had a history of consumption of mustard oil and confirmation of Argemone oil contamination according to laboratory tests.
    [Show full text]
  • Epidemic Dropsy in India
    Postgrad Med J 1999;75:657–661 © The Fellowship of Postgraduate Medicine, 1999 Postgrad Med J: first published as 10.1136/pgmj.75.889.657 on 1 November 1999. Downloaded from Epidemic dropsy in India B D Sharma, Sanjay Malhotra, Vikram Bhatia, Mandeep Rathee Summary Epidemic dropsy results from ingestion of edible oil adulterated with Argemone Epidemic dropsy is a clinical state mexicana (Mexican Poppy) oil. The outbreak of epidemic dropsy in the Indian resulting from use of edible oils capital, New Delhi, during the rainy season of 1998 was of one of the most severe adulterated with Argemone mexi- forms and had repercussions in both health and political circles. Some 2552 cana oil. Sanguinarine and dehyd- cases were reported and 65 deaths occurred between 5 August and 12 October, rosanguinarine are two major causing untold misery and economic loss to the aVected families. The actual fig- toxic alkaloids of Argemone oil, ures are likely to be much higher due to nonreporting of milder cases to the hos- which cause widespread capillary pitals. The aim of this article is to consolidate and update the available dilatation, proliferation and in- information on clinical aspects of epidemic dropsy. creased capillary permeability. The condition was first reported by Lyon in 1877 from Calcutta1 and has since Leakage of the protein-rich occurred in other countries including the Fiji Islands, Mauritius, Madagascar, plasma component into the extra- South Africa and Burma (Myanmar).2 In India, it has been reported from time cellular compartment leads to the to time from the States of West Bengal, Bihar, Orissa, Madhya Pradesh, Uttar formation of oedema.
    [Show full text]
  • Hallucinogens: a Cause of Convulsive Ergot Psychoses
    Loma Linda University TheScholarsRepository@LLU: Digital Archive of Research, Scholarship & Creative Works Loma Linda University Electronic Theses, Dissertations & Projects 6-1976 Hallucinogens: a Cause of Convulsive Ergot Psychoses Sylvia Dahl Winters Follow this and additional works at: https://scholarsrepository.llu.edu/etd Part of the Psychiatry Commons Recommended Citation Winters, Sylvia Dahl, "Hallucinogens: a Cause of Convulsive Ergot Psychoses" (1976). Loma Linda University Electronic Theses, Dissertations & Projects. 976. https://scholarsrepository.llu.edu/etd/976 This Thesis is brought to you for free and open access by TheScholarsRepository@LLU: Digital Archive of Research, Scholarship & Creative Works. It has been accepted for inclusion in Loma Linda University Electronic Theses, Dissertations & Projects by an authorized administrator of TheScholarsRepository@LLU: Digital Archive of Research, Scholarship & Creative Works. For more information, please contact [email protected]. ABSTRACT HALLUCINOGENS: A CAUSE OF CONVULSIVE ERGOT PSYCHOSES By Sylvia Dahl Winters Ergotism with vasoconstriction and gangrene has been reported through the centuries. Less well publicized are the cases of psychoses associated with convulsive ergotism. Lysergic acid amide a powerful hallucinogen having one.-tenth the hallucinogenic activity of LSD-25 is produced by natural sources. This article attempts to show that convulsive ergot psychoses are mixed psychoses caused by lysergic acid amide or similar hallucinogens combined with nervous system
    [Show full text]
  • Cassava Cyanide Diseases & Neurolathyrism Network Issue Number 20, December 2012
    Working together to eliminate cyanide poisoning, konzo, tropical ataxic neuropathy (TAN) and neurolathyrism Cassava Cyanide Diseases & Neurolathyrism Network (ISSN 1838-8817 (Print): ISSN 1838-8825 (Online) Issue Number 20, December 2012 Contents Historical Awareness of Neurolathyrism, Historical Awareness of Neurolathyrism, and Cassava Toxicity and Cassava Toxicity ................................... 1 Most people studying lathyrism are likely to Acute cyanide poisoning from cassava: is have training in bio-scientific methods or in social it still common? ........................................... 4 science, yet there are considerable gaps of thinking International conference on “Recent and practice between different disciplines, e.g. from plant biologists to historians of food in human Trends in Lathyrus sativus Research” cultures. There are different approaches to what is (Hyderabad, India, November 8-9, 2012). ... 6 considered ‘well-founded knowledge’, and how it Neurolathyrism in Bidar and Medak may be established. This paper recognises such districts of South India ................................ 7 differences, and their relevance to knowledge- Residual Cyanide content In Cassava development in neurolathyrism, with brief Product of India ............................................ 7 comparison of cassava (manioc) and its toxicity. It also considers why a broader approach to knowledge is important, and whether historical CCDNN Coordinators: knowledge can be made useful to people who face Prof Fernand Lambein increased food scarcity, and are ‘below the radar’ of Ghent University, Institute for plant Biotechnology government attention. Outreach (IPBO) Review articles in lathyrism often begin with a Proeftuinstraat 86 N1, B-9000 Ghent, Belgium Phone: +32 484 417 5005 glimpse of history, citing ancient texts where E-mail: [email protected] or Lathyrus sativus seems to appear, or [email protected] archaeological reports of seeds found from antiquity.
    [Show full text]
  • A Case of Mushroom Poisoning with Russula Subnigricans: Development of Rhabdomyolysis, Acute Kidney Injury, Cardiogenic Shock, and Death
    CASE REPORT Nephrology http://dx.doi.org/10.3346/jkms.2016.31.7.1164 • J Korean Med Sci 2016; 31: 1164-1167 A Case of Mushroom Poisoning with Russula subnigricans: Development of Rhabdomyolysis, Acute Kidney Injury, Cardiogenic Shock, and Death Jong Tae Cho and Jin Hyung Han Mushroom exposures are increasing worldwide. The incidence and fatality of mushroom poisoning are reported to be increasing. Several new syndromes in mushroom poisoning Department of Internal Medicine, College of have been described. Rhabdomyolytic mushroom poisoning is one of new syndromes. Medicine, Dankook University, Cheonan, Korea Russula subnigricans mushroom can cause delayed-onset rhabdomyolysis with acute Received: 17 April 2015 kidney injury in the severely poisoned patient. There are few reports on the toxicity of R. Accepted: 6 June 2015 subnigricans. This report represents the first record of R. subnigricans poisoning with rhabdomyolysis in Korea, describing a 51-year-old man who suffered from rhabdomyolysis, Address for Correspondence: Jong Tae Cho, MD acute kidney injury, severe hypocalcemia, respiratory failure, ventricular tachycardia, Department of Internal Medicine, College of Medicine, cardiogenic shock, and death. Mushroom poisoning should be considered in the evaluation Dankook University, 201 Manghyang-ro, Dongnam-gu, Cheonan 31116, Korea of rhabdomyolysis of unknown cause. Furthermore, R. subnigricans should be considered E-mail: [email protected] in the mushroom poisoning with rhabdomyolysis. Keywords: Mushroom Poisoning; Rhabdomyolysis; Acute Kidney Injury; Respiratory Failure; Cardiogenic Shock INTRODUCTION in August, 2010 at the Jujak mountain located on the province of Jeollanam­do, the southern area of Korea. He was a bus driv­ More leisure time for hobbies, hiking, and trekking has led to er.
    [Show full text]