Bermudagrass Control in Southern Lawns
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2,4-Dichlorophenoxyacetic Acid
2,4-Dichlorophenoxyacetic acid 2,4-Dichlorophenoxyacetic acid IUPAC (2,4-dichlorophenoxy)acetic acid name 2,4-D Other hedonal names trinoxol Identifiers CAS [94-75-7] number SMILES OC(COC1=CC=C(Cl)C=C1Cl)=O ChemSpider 1441 ID Properties Molecular C H Cl O formula 8 6 2 3 Molar mass 221.04 g mol−1 Appearance white to yellow powder Melting point 140.5 °C (413.5 K) Boiling 160 °C (0.4 mm Hg) point Solubility in 900 mg/L (25 °C) water Related compounds Related 2,4,5-T, Dichlorprop compounds Except where noted otherwise, data are given for materials in their standard state (at 25 °C, 100 kPa) 2,4-Dichlorophenoxyacetic acid (2,4-D) is a common systemic herbicide used in the control of broadleaf weeds. It is the most widely used herbicide in the world, and the third most commonly used in North America.[1] 2,4-D is also an important synthetic auxin, often used in laboratories for plant research and as a supplement in plant cell culture media such as MS medium. History 2,4-D was developed during World War II by a British team at Rothamsted Experimental Station, under the leadership of Judah Hirsch Quastel, aiming to increase crop yields for a nation at war.[citation needed] When it was commercially released in 1946, it became the first successful selective herbicide and allowed for greatly enhanced weed control in wheat, maize (corn), rice, and similar cereal grass crop, because it only kills dicots, leaving behind monocots. Mechanism of herbicide action 2,4-D is a synthetic auxin, which is a class of plant growth regulators. -
Exposure to Herbicides in House Dust and Risk of Childhood Acute Lymphoblastic Leukemia
Journal of Exposure Science and Environmental Epidemiology (2013) 23, 363–370 & 2013 Nature America, Inc. All rights reserved 1559-0631/13 www.nature.com/jes ORIGINAL ARTICLE Exposure to herbicides in house dust and risk of childhood acute lymphoblastic leukemia Catherine Metayer1, Joanne S. Colt2, Patricia A. Buffler1, Helen D. Reed3, Steve Selvin1, Vonda Crouse4 and Mary H. Ward2 We examine the association between exposure to herbicides and childhood acute lymphoblastic leukemia (ALL). Dust samples were collected from homes of 269 ALL cases and 333 healthy controls (o8 years of age at diagnosis/reference date and residing in same home since diagnosis/reference date) in California, using a high-volume surface sampler or household vacuum bags. Amounts of agricultural or professional herbicides (alachlor, metolachlor, bromoxynil, bromoxynil octanoate, pebulate, butylate, prometryn, simazine, ethalfluralin, and pendimethalin) and residential herbicides (cyanazine, trifluralin, 2-methyl-4- chlorophenoxyacetic acid (MCPA), mecoprop, 2,4-dichlorophenoxyacetic acid (2,4-D), chlorthal, and dicamba) were measured. Odds ratios (OR) and 95% confidence intervals (CI) were estimated by logistic regression. Models included the herbicide of interest, age, sex, race/ethnicity, household income, year and season of dust sampling, neighborhood type, and residence type. The risk of childhood ALL was associated with dust levels of chlorthal; compared to homes with no detections, ORs for the first, second, and third tertiles were 1.49 (95% CI: 0.82–2.72), 1.49 (95% CI: 0.83–2.67), and 1.57 (95% CI: 0.90–2.73), respectively (P-value for linear trend ¼ 0.05). The magnitude of this association appeared to be higher in the presence of alachlor. -
Weed Control in Direct-Seeded Field Pea Gregory J
Weed Control in Direct-seeded Field Pea Gregory J. Endres and Blaine G. Schatz Weed control and field pea response to selected soil- and POST-applied herbicides were evaluated in a randomized complete-block design with three replicates. The experiment was conducted on a Heimdahl loam soil with 6.7 pH and 2.9% organic matter at the NDSU Carrington Research Extension Center. Herbicide treatments were applied to 5- by 25-ft plots with a pressurized hand-held plot sprayer at 17 gal/A and 30 psi through 8002 flat-fan nozzles. Fall sulfentrazone treatments were applied October 25, 2004 to a moist soil surface with 47 F, 71% RH, 15% clear sky, and 11 mph wind. On April 28, 2005, inoculated 'Integra' field pea was seeded into standing wheat stubble in 7-inch rows at a rate of 300,000 pure live seeds/A. PRE treatments were applied to a dry soil surface on April 30 with 31 F, 64% RH, 30% clear sky, and 10 mph wind. Rainfall totaled 1.22 inches 8 d following PRE application. The trial area was treated on May 6 with a PRE burn-down application of glyphosate at 0.75 lb ae/A plus ammonium sulfate at 1% v/v. The early POST (EPOST) treatment was applied on May 23 with 73 F, 35% RH, 100% cloudy sky, and 6 mph wind to 2-inch tall field pea, 1- to 2-leaf green and yellow foxtail, 0.5-inch tall common lambsquarters, 0.5-inch tall prostrate and redroot pigweed, and 0.5-inch tall wild buckwheat. -
MC(' Potential Exposure of Humans to 2
(MC( FUNDAMENTAL AND APPLIED TOXICOLOGY 1:3 3 9-3 4 6 (1981) Potential Exposure of Humans to 2,4,5-T and TCDD in the Oregon Coast Ranges MICHAEL NEWTON" and LOGAN A. NORRISB "Professor of Forest Ecology, Oregon State University, Corvallis; BChief Research Chemist, USDA Forest Service, Corvallis, Oregon ABSTRACT Potential Exposure of Humans to 2,4,5-T and TCDD in Humans may be exposed to herbicides through drift; inges- the Oregon Coast Ranges. Newton, M. and Norris, L.A. tion of wild and domestic meat, vegetables, and fruit; con- (1981). F.undam. AppL Toxicol. 1:339-346. Research on the sumption of water; and dermal contact while handling the use of 2,4,5-trichlorophenoxyacetic acid (2,4,5-T) contami- chemicals, equipment, and treated vegetation. The range of -8 nated with 2.5 X 10 parts 2,3,7,8-tetrachlorodibenzo-p- potential exposure extends from zero, if there is no encounter dioxin (TCDD) in forests of the Oregon Coast Ranges per- with the herbicide, to the worst situation where the person has mits estimates of human exposures for both compounds. encountered the highest levels of water contamination, drift Estimated total exposure of nearby ( ^ 1/8 mile distant) resi- exposure, meat contamination, and dermal exposure simul- dents during the first week after application is 0.0039 mg/kg taneously. We have brought estimates of all sources together of 2,4,5-T for a 70-kg adult. Exposure to TCDD in the same to determine the possible range of total exposure from episode would be 1.9 X 10 b ° mg/kg. -
Appendix C - Wildlife 9/8/2008
INVASIVE PLANT BIOLOGICAL ASSESSMENT Umatilla and Wallowa-Whitman National Forests Appendix C - Wildlife 9/8/2008 APPENDIX C - WILDLIFE C-1 INVASIVE PLANT BIOLOGICAL ASSESSMENT Umatilla and Wallowa-Whitman National Forests Appendix C - Wildlife 9/8/2008 C-2 INVASIVE PLANT BIOLOGICAL ASSESSMENT Umatilla and Wallowa-Whitman National Forests Appendix C - Wildlife 9/8/2008 Appendix C ......................................................................................................................... 1 Wildlife ............................................................................................................................... 1 Exposure Groups for Forest Service Sensitive Wildlife ................................................. 6 Effects of the Alternatives on Sensitive Wildlife ........................................................... 7 Tables C-5 – C-13 Herbicides ................................................................................... 11 Umatilla and Wallowa-Whitman National Forest Herbicide Spray Buffers ................ 12 Aquatics............................................................................................................. 15 Wildlife .............................................................................................................. 15 Worker Health: Based on backpack spray applications. ................................. 15 Public Health: ................................................................................................... 16 Summary of Herbicide Effects to Wildlife -
INDEX to PESTICIDE TYPES and FAMILIES and PART 180 TOLERANCE INFORMATION of PESTICIDE CHEMICALS in FOOD and FEED COMMODITIES
US Environmental Protection Agency Office of Pesticide Programs INDEX to PESTICIDE TYPES and FAMILIES and PART 180 TOLERANCE INFORMATION of PESTICIDE CHEMICALS in FOOD and FEED COMMODITIES Note: Pesticide tolerance information is updated in the Code of Federal Regulations on a weekly basis. EPA plans to update these indexes biannually. These indexes are current as of the date indicated in the pdf file. For the latest information on pesticide tolerances, please check the electronic Code of Federal Regulations (eCFR) at http://www.access.gpo.gov/nara/cfr/waisidx_07/40cfrv23_07.html 1 40 CFR Type Family Common name CAS Number PC code 180.163 Acaricide bridged diphenyl Dicofol (1,1-Bis(chlorophenyl)-2,2,2-trichloroethanol) 115-32-2 10501 180.198 Acaricide phosphonate Trichlorfon 52-68-6 57901 180.259 Acaricide sulfite ester Propargite 2312-35-8 97601 180.446 Acaricide tetrazine Clofentezine 74115-24-5 125501 180.448 Acaricide thiazolidine Hexythiazox 78587-05-0 128849 180.517 Acaricide phenylpyrazole Fipronil 120068-37-3 129121 180.566 Acaricide pyrazole Fenpyroximate 134098-61-6 129131 180.572 Acaricide carbazate Bifenazate 149877-41-8 586 180.593 Acaricide unclassified Etoxazole 153233-91-1 107091 180.599 Acaricide unclassified Acequinocyl 57960-19-7 6329 180.341 Acaricide, fungicide dinitrophenol Dinocap (2, 4-Dinitro-6-octylphenyl crotonate and 2,6-dinitro-4- 39300-45-3 36001 octylphenyl crotonate} 180.111 Acaricide, insecticide organophosphorus Malathion 121-75-5 57701 180.182 Acaricide, insecticide cyclodiene Endosulfan 115-29-7 79401 -
Weed Management—Major Crops
Weed Technology 2010 24:1–5 Weed Management—Major Crops Annual Grass Control in Strip-Tillage Peanut Production with Delayed Applications of Pendimethalin W. Carroll Johnson, III, Eric P. Prostko, and Benjamin G. Mullinix, Jr.* In strip-tillage peanut production, situations occur when dinitroaniline herbicides are not applied in a timely manner. In these cases, dinitroaniline herbicides would be applied days or weeks after seeding. However, there is no information that documents the effects of delayed applications on weed control. Trials were conducted in 2004, 2005, and 2007 in Georgia to determine the weed control efficacy of delayed applications of pendimethalin in strip-tillage peanut production. Treatments included seven timings of pendimethalin application and three pendimethalin-containing herbicide combinations. Timings of application were immediately after seeding (PRE), vegetative emergence of peanut (VE), 1 wk after VE (VE+1wk), VE+2wk, VE+3wk, VE+4wk, and a nontreated control. Pendimethalin containing herbicide programs included pendimethalin plus paraquat, pendimethalin plus imazapic, and pendimethalin alone. Among the possible treatment combinations was a current producer standard timing for nonpendimethalin weed control programs in peanut, which was either imazapic or paraquat alone applied VE+3wk. Pendimethalin alone did not effectively control Texas millet regardless of time of application (69 to 77%), whereas southern crabgrass was controlled by pendimethalin alone PRE (87%). Delayed applications of pendimethalin controlled Texas millet and southern crabgrass when combined with either paraquat or imazapic, with imazapic being the preferred combination due to better efficacy on southern crabgrass than paraquat at most delayed applications. Peanut yield was improved when any of the herbicide combinations were applied PRE compared to later applications. -
Reregistration Eligibility Decision (RED) for Glyphosate
GLYPHOSATE RED September 1993 GLYPHOSATE REREGISTRATION ELIGIBILITY TEAM Office of Pesticide Programs: Special Review and Reregistration Division Eric Feris .................................................. Reregistration Branch Health Effects Division Jane Smith ........................................ Chemical Coordination Branch Krystyna Locke .............................................. Toxicology Branch I Jeff Evans ........................... Occupational and Residential Exposure Branch Randolph Perfetti ......................... Chemistry Branch - Reregistration Support Biological and Economic Analysis Division James G. Saulmon .....................................Biological Analysis Branch Eric Maurer ........................................... Economic Analysis Branch Environmental Fate and Effects Division Candace Brassard ..................................... Ecological Effects Branch Kevin Poff ............................. Environmental Fate and Groundwater Branch Bernice Slutsky ............................ Science Analysis and Coordination Staff Registration Division Mark Perry .......................................... Registration Support Branch Karen P. Hicks ....................................... Fungicide-Herbicide Branch Policy and Special Projects Staff Jean Frane ............................. Food Safety & Regulation Tracking Section Office of General Counsel: Pesticides and Toxic Substances Division Debra Burton ................................................. Pesticides Branch Office of Compliance Monitoring: -
Toxicological Profile for Glyphosate Were
A f Toxicological Profile for Glyphosate August 2020 GLYPHOSATE II DISCLAIMER Use of trade names is for identification only and does not imply endorsement by the Agency for Toxic Substances and Disease Registry, the Public Health Service, or the U.S. Department of Health and Human Services. GLYPHOSATE III FOREWORD This toxicological profile is prepared in accordance with guidelines developed by the Agency for Toxic Substances and Disease Registry (ATSDR) and the Environmental Protection Agency (EPA). The original guidelines were published in the Federal Register on April 17, 1987. Each profile will be revised and republished as necessary. The ATSDR toxicological profile succinctly characterizes the toxicologic and adverse health effects information for these toxic substances described therein. Each peer-reviewed profile identifies and reviews the key literature that describes a substance's toxicologic properties. Other pertinent literature is also presented, but is described in less detail than the key studies. The profile is not intended to be an exhaustive document; however, more comprehensive sources of specialty information are referenced. The focus of the profiles is on health and toxicologic information; therefore, each toxicological profile begins with a relevance to public health discussion which would allow a public health professional to make a real-time determination of whether the presence of a particular substance in the environment poses a potential threat to human health. The adequacy of information to determine a substance's -
US EPA, Pesticide Product Label, SHARDA GLYPHOSATE 20.5 % +
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Paspalum Vaginatum) Turf
BERMUDAGRASS (CYNODON DACTYLON) AND GOOSEGRASS (ELEUSINE INDICA) MANAGEMENT IN SEASHORE PASPALUM (PASPALUM VAGINATUM) TURF A THESIS SUBMITTED TO THE GRADUATE DIVISION OF THE UNIVERSITY OF HAWAII AT MĀNOA IN PARTIAL FULFILLMENT OF THE REQUIREMENTS FOR THE DEGREE OF MASTER OF SCIENCE IN TROPICAL PLANT AND SOIL SCIENCES MAY 2018 By Alex J. Lindsey Thesis Committee: Joseph DeFrank, Chairperson Orville Baldos Zhiqiang Cheng ACKNOWLEDGEMENTS I would like to thank Dr. Zhiqiang Cheng and Dr. Joseph DeFrank for providing funding for my thesis through CTAHR’s competitive Supplemental Funding Program. I would like to thank my advisor, Dr. Joseph DeFrank, for his continual support and guidance throughout the completion of my thesis. I appreciate the skills and knowledge he has taught me that will help me with my future endeavors. I would like to express my gratitude and appreciation to my committee members, Dr. Zhiqiang Cheng (co-advisor) and Dr. Orville Baldos, who were always there to help and provide valuable inputs throughout this process. I would also like to thank Craig Okazaki, Magoon Research Station supervisor, for providing research material and assisting as a graduate student and Rey Ito, The Green Doctor, for providing knowledge and valuable inputs for my thesis research. Thanks to Sean Fong, Hawaiian Turfgrass, for providing research materials; the Pali Golf Course, the Hoakalei Country Club, and the West Loch Golf Course for your cooperation and providing space for field trials; and to BASF, Bayer, and Syngenta for providing the herbicides used in this study. Lastly, I would like to thank my friends and family for all their love and support throughout this process. -
Multiple Resistance to Glyphosate, Paraquat and Accase
Research Article Received: 5 September 2017 Revised: 16 October 2017 Accepted article published: 26 October 2017 Published online in Wiley Online Library: 8 December 2017 (wileyonlinelibrary.com) DOI 10.1002/ps.4774 Multiple resistance to glyphosate, paraquat and ACCase-inhibiting herbicides in Italian ryegrass populations from California: confirmation and mechanisms of resistance Parsa Tehranchian,a* Vijay Nandula,b Mithila Jugulam,c Karthik Puttac and Marie Jasieniuka Abstract BACKGROUND: Glyphosate, paraquat and acetyl CoA carboxylase (ACCase)-inhibiting herbicides are widely used in California annual and perennial cropping systems. Recently, glyphosate, paraquat, and ACCase- and acetolactate synthase (ALS)-inhibitor resistance was confirmed in several Italian ryegrass populations from the Central Valley of California. This research characterized the possible mechanisms of resistance. RESULTS: Multiple-resistant populations (MR1, MR2) are resistant to several herbicides from at least three modes of action. Dose–response experiments revealed that the MR1 population was 45.9-, 122.7- and 20.5-fold, and the MR2 population was 24.8-, 93.9- and 4.0-fold less susceptible to glyphosate, sethoxydim and paraquat, respectively, than the susceptible (Sus) population. Accumulation of shikimate in Sus plants was significantly greater than in MR plants 32 h after light pretreatments. Glyphosate resistance in MR plants was at least partially due to Pro106-to-Ala and Pro106-to-Thr substitutions at site 106 of 5-enolpyruvylshikimate-3-phosphate synthase (EPSPS). EPSPS gene copy number and expression level were similar in plants from the Sus and MR populations. An Ile1781-to-Leu substitution in ACCase gene of MR plants conferred a high level of resistance to sethoxydim and cross-resistance to other ACCase-inhibitors.