2010 CVMA Convention: Scientific Presentations
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University of Bradford Ethesis
University of Bradford eThesis This thesis is hosted in Bradford Scholars – The University of Bradford Open Access repository. Visit the repository for full metadata or to contact the repository team © University of Bradford. This work is licenced for reuse under a Creative Commons Licence. THE IDENTIFICATION OF BOVINE TUBERCULOSIS IN ZOOARCHAEOLOGICAL ASSEMBLAGES VOLUME 1 (1 OF 2) J. E. WOODING PhD UNIVERSITY OF BRADFORD 2010 THE IDENTIFICATION OF BOVINE TUBERCULOSIS IN ZOOARCHAEOLOGICAL ASSEMBLAGES Working towards differential diagnostic criteria Volume 1 (1 of 2) Jeanette Eve WOODING Submitted for the degree of Doctor of Philosophy School of Life Sciences Division of Archaeological, Geographical and Environmental Sciences University of Bradford 2010 Jeanette Eve WOODING The identification of bovine tuberculosis in zooarchaeological assemblages. Working towards differential diagnostic criteria. Keywords: Palaeopathology, zooarchaeology, human osteoarchaeology, zoonosis, Iron Age, Viking Age, Iceland, Orkney, England ABSTRACT The study of human palaeopathology has developed considerably in the last three decades resulting in a structured and standardised framework of practice, based upon skeletal lesion patterning and differential diagnosis. By comparison, disarticulated zooarchaeological assemblages have precluded the observation of lesion distributions, resulting in a dearth of information regarding differential diagnosis and a lack of standard palaeopathological recording methods. Therefore, zoopalaeopathology has been restricted to the analysis of localised pathologies and ‘interesting specimens’. Under present circumstances, researchers can draw little confidence that the routine recording of palaeopathological lesions, their description or differential diagnosis will ever form a standard part of zooarchaeological analysis. This has impeded the understanding of animal disease in past society and, in particular, has restricted the study of systemic disease. -
The Yellow Cat: Diagnostic & Therapeutic Strategies
Peer Reviewed THE YELLOW CAT: DIAGNOSTIC & THERAPEUTIC STRATEGIES The Yellow Cat: Diagnostic & Therapeutic Strategies Craig B. Webb, PhD, DVM, Diplomate ACVIM (Small Animal Internal Medicine) Colorado State University There is no mystery when it comes to a “yellow” cat. Icterus and jaundice—both of which describe a yellowish pigmentation of the skin—indicate hyperbilirubinemia, a 5- to 10-fold elevation in serum bilirubin concentration. However, this is where the certainty ends and the diagnostic challenge begins. The icteric cat presentation is not a sensitive or specific marker of disease, despite the visually obvious and impressive clinical sign (Figure 1).1 The objective of this article is to briefly review differentials for hyperbilirubinemia in the cat, and present a diagnostic and therapeutic strategy that will help practitioners approach this problem in an efficient and effective manner. FIGURE 1. Icteric pinna of a cat in the critical HYPERBILIRUBINEMIA: ORGANIZATION care isolation unit; prehepatic hemolysis BY LOCATION and anemia are a result of Cytauxzoon felis infection. Hyperbilirubinemia The differentials for hyperbilirubinemia should be results when organized by location: prehepatic, hepatic, and serum bilirubin posthepatic. While, in cats, it is common to find Hepatic Disease concentrations concurrent disease processes, starting from this A significant decrease, or loss, of hepatocellular reach 2 to 3 mg/dL foundation is the first step toward an effective and function effects bilirubin metabolism, and (35–50 mcmol/L). efficient diagnostic workup of icteric cats. frequently results in intrahepatic cholestasis (Table 1, page 40). Unconjugated bilirubin from damaged Prehepatic Disease hepatocytes is present, although the majority of Hemolysis releases hemoglobin, which is then bilirubin that appears in the cat’s circulation is metabolized through biliverdin to bilirubin in the conjugated, having completed the metabolic step liver. -
Musculoskeletal Radiology
MUSCULOSKELETAL RADIOLOGY Developed by The Education Committee of the American Society of Musculoskeletal Radiology 1997-1998 Charles S. Resnik, M.D. (Co-chair) Arthur A. De Smet, M.D. (Co-chair) Felix S. Chew, M.D., Ed.M. Mary Kathol, M.D. Mark Kransdorf, M.D., Lynne S. Steinbach, M.D. INTRODUCTION The following curriculum guide comprises a list of subjects which are important to a thorough understanding of disorders that affect the musculoskeletal system. It does not include every musculoskeletal condition, yet it is comprehensive enough to fulfill three basic requirements: 1.to provide practicing radiologists with the fundamentals needed to be valuable consultants to orthopedic surgeons, rheumatologists, and other referring physicians, 2.to provide radiology residency program directors with a guide to subjects that should be covered in a four year teaching curriculum, and 3.to serve as a “study guide” for diagnostic radiology residents. To that end, much of the material has been divided into “basic” and “advanced” categories. Basic material includes fundamental information that radiology residents should be able to learn, while advanced material includes information that musculoskeletal radiologists might expect to master. It is acknowledged that this division is somewhat arbitrary. It is the authors’ hope that each user of this guide will gain an appreciation for the information that is needed for the successful practice of musculoskeletal radiology. I. Aspects of Basic Science Related to Bone A. Histogenesis of developing bone 1. Intramembranous ossification 2. Endochondral ossification 3. Remodeling B. Bone anatomy 1. Cellular constituents a. Osteoblasts b. Osteoclasts 2. Non cellular constituents a. -
Feline Obesity: Food Toys and Owner-Perceived Quality of Life During a Prescribed Weight Loss Plan
Feline Obesity: Food Toys and Owner-Perceived Quality of Life During a Prescribed Weight Loss Plan Lauren Elizabeth Dodd Thesis submitted to the faculty of the Virginia Polytechnic Institute and State University in partial fulfillment of the requirements for the degree of Master of Science In Biomedical Veterinary Sciences Megan Shepherd, Chair Sherrie Clark Nick Dervisis Kathy Hosig May 16, 2019 Blacksburg, VA Keywords: Feline, Obesity, Weight loss Copyright © Lauren Dodd Use or inclusion of any portion of this document in another work intended for commercial use will require permission from the copyright owner. ACADEMIC ABSTRACT The prevalence of overweight and obesity in the feline population is estimated to be 25.7% and 33.8%, respectively. Feline obesity is associated with comorbidities such as insulin resistance and hepatic lipidosis. Several risk factors are associated with obesity including middle age, neuter status, decreased activity, and diet. Obesity management is multifaceted and includes client education, diet modification, and consistent monitoring. Successful obesity management may be dependent on owner perception of their cat’s quality of life during a prescribed weight loss plan. Low perceived quality of life may result in failure to complete the weight loss process. Food toys may be used to enhance environmental enrichment, allow cats to express their natural predatory behavior and overall improve owner-perceived quality of life. Therefore, we set out to investigate the role of food toys in owner-perceived quality of life of obese cats during a prescribed weight loss plan. Fifty-five cats with a BCS > 7 were enrolled in a double-blinded weight loss study and randomized into one of two groups: food toy (n=26) or food bowl (n=29). -
Chapter 12: Imaging Techniques
ith advancing technology, diagnostic im- aging techniques available for avian pa- CHAPTER tients now include ultrasound, fluoros- W copy, computed tomography (CT) and nu- clear scintigraphy; however, routine radiography re- mains the most frequently performed imaging mo- dality in birds and frequently is diagnostic without the need for more sophisticated procedures. Informa- tion obtained from radiographs will frequently com- plement results from other testing methods, provid- 12 ing for a more thorough evaluation of a disease process. Both risk and benefit to the patient should be consid- ered when radiography is used as a screening proce- dure in an apparently normal companion bird. In general, radiography should be performed only when IMAGING indicated by historical information, physical exami- nation findings and laboratory data. Indiscriminate TECHNIQUES radiographic studies create an unnecessary risk to the patient and technical staff. Radiographic findings should always be correlated with surgical, endoscopic or necropsy findings. These comparisons will refine a clinician’s ability to detect subtle radiographic changes, and improve diagnostic capabilities and therapeutic results. Marjorie C. McMillan 247 CHAPTER 12 IMAGING TECHNIQUES graphic contrast is controlled by subject contrast, scatter, and film contrast and fog. Detail is improved Technical Considerations by using a small focal spot, the shortest possible exposure time (usually 0.015 seconds), adequate fo- cus-film distance (40 inches), a collimated beam, sin- gle emulsion film and a rare earth, high-detail The size (mainly thickness), composition (air, soft screen. The contact between the radiographic cas- tissue and bone) and ability to arrest motion are the sette and the patient should be even, and the area of primary factors that influence radiographic tech- interest should be as close as possible to the film. -
The Carnivore Connection to Nutrition in Cats
1202ttn.qxd 11/6/2002 11:13 AM Page 1559 Timely Topics in Nutrition The carnivore connection to nutrition in cats Debra L. Zoran, DVM, PhD, DACVIM The JAVMA welcomes contributions to this feature. nutritional biochemistry of cats. In addition, informa- Articles submitted for publication will be fully reviewed tion is included on possible roles of nutrition in the with the American College of Veterinary Nutrition (ACVN) development of obesity, idiopathic hepatic lipidosis acting in an advisory capacity to the editors. Inquiries (IHL), inflammatory bowel disease, and diabetes melli- should be sent to Dr. John E. Bauer, Department of Small tus in cats. Animal Medicine and Surgery, College of Veterinary Medicine, Texas A&M University, College Station, TX Protein 77843-4474. The natural diet of cats in the wild is a meat-based regimen (eg, rodents, birds) that contains little CHO; n another time long ago, Leonardo da Vinci said, thus, cats are metabolically adapted to preferentially I“The smallest feline is a masterpiece.”1 And for those use protein and fat as energy sources (Appendix 1). of us who marvel at the wonder that is a cat, there is no This evolutionary difference in energy metabolism doubt that his statement was remarkable for its sim- mandates cats to use protein for maintenance of blood glucose concentrations even when sources of protein plicity as well as its truth. Cats are amazing creatures, 2 unique and interesting in almost every way imaginable. in the diet are limiting. The substantial difference in Despite this, it has been common for veterinarians to protein requirements between cats and omnivores, consider cats and dogs as similar beings for anesthesia such as dogs, serves to illustrate this important meta- protocols, clinical diseases, and treatments. -
Subbarao K Skeletal Dysplasia (Sclerosing Dysplasias – Part I)
REVIEW ARTICLE Skeletal Dysplasia (Sclerosing dysplasias – Part I) Subbarao K Padmasri Awardee Prof. Dr. Kakarla Subbarao, Hyderabad, India Introduction Dysplasia is a disturbance in the structure of Monitoring germ cell Mutations using bone and disturbance in growth intrinsic to skeletal dysplasias incidence of dysplasia is bone and / or cartilage. Several terms have 1500 for 9.5 million births (15 per 1,00,000) been used to describe Skeletal Dysplasia. Skeletal dysplasias constitute a complex SCLEROSING DYSPLASIAS group of bone and cartilage disorders. Three main groups have been reported. The first Osteopetrosis one is thought to be x-linked disorder Pyknodysostosis genetically inherited either as dominant or Osteopoikilosis recessive trait. The second group is Osteopathia striata spontaneous mutation. Third includes Dysosteosclerosis exposure to toxic or infective agent Worth’s sclerosteosis disrupting normal skeletal development. Van buchem’s dysplasia Camurati Engelman’s dysplasia The term skeletal dysplasia is sometimes Ribbing’s dysplasia used to include conditions which are not Pyle’s metaphyseal dysplasia actually skeletal dysplasia. According to Melorheosteosis revised classification of the constitutional Osteoectasia with hyperphosphatasia disorders of bone these conditions are Pachydermoperiosteosis (Touraine- divided into two broad groups: the Solente-Gole Syndrome) osteochondrodysplasias and the dysostoses. There are more than 450 well documented Evaluation by skeletal survey including long skeletal dysplasias. Many of the skeletal bones thoracic cage, hands, feet, cranium and dysplasias can be diagnosed in utero. This pelvis is adequate. Sclerosing dysplasias paper deals with post natal skeletal are described according to site of affliction dysplasias, particularly Sclerosing epiphyseal, metaphyseal, diaphyseal and dysplasias. generalized. Dysplasias with increased bone density Osteopetrosis (Marble Bones): Three forms are reported. -
Anzcvs Science Week 2018
ANZCVS SCIENCE WEEK 2018 Small Animal Medicine proceedings in collaboration with Feline Medicine, Radiology and Pharmacology Chapters With thanks to Hills Pet Nutrition for their ongoing support of the Small Animal Medicine Chapter Science Week Program 2018 ANZCVS Science Week Contents Stem cell therapy in cats: what’s the evidence? Keshuan Chow ………………………..…...4 Treatment guidelines for respiratory tract infections in the cat. Jane Sykes……………….....7 Diagnostic approach to fever in cats. Jane Sykes………………………………………….....9 Funny feline syndromes: the oddities of the cat. Katherine Briscoe………………………...11 Feline nutrition: a clinician’s perspective. Sue Foster………………………………………16 Hepatic CT including portosystemic shunt assessment. Chris Ober………………………..25 Thoracic CT imaging. Chris Ober………………………………………………………….28 Imaging in Oncology. Chris Ober…………………………………………………………...31 Personal infection control practices. Angela Willemsen…………………………………….35 Brucella Suis seroprevalence. Cathy Kneipp………………………………………………..35 Feline listeriosis. .Tommy Fluen……………………………………………………………..36 2 2018 ANZCVS Science Week Macronutrient intake and behaviour in cats. Sophia Little………………………………………36 Body condition and morbidity, survival and lifespan in cats. Kendy Teng………………….37 DGGR lipase concentrations and hyperadrenocorticsm. Amy Collings……………………..37 Canine mast cell tumours. Benjamin Reynolds ……………………………………………..38 Effect of melatonin on cyclicity and lactation in queens. Mark Vardanega………………...38 Lower motor neuron paresis in dogs. Melissa Robinson…………………………………….39 Management -
Feline Lower Urinary Tract Disease from Wikipedia, the Free Encyclopedia
Log in / create account article discussion edit this page history Feline lower urinary tract disease From Wikipedia, the free encyclopedia Feline lower urinary tract disease (FLUTD) is a term that is used to cover many problems of the feline urinary tract, including stones and cystitis. The term feline urologic syndrome (FUS) is an older term which is still sometimes used for this condition. The condition can lead to plugged navigation penis syndrome also known as blocked cat syndrome. It is a common disease in adult cats, though it can strike in young cats too. It may Main page present as any of a variety of urinary tract problems, and can lead to a complete blockage of the urinary system, which if left untreated is fatal. Contents FLUTD is not a specific diagnosis in and of itself, rather, it represents an array of problems within one body system. Featured content Current events FLUTD affects cats of both sexes, but tends to be more dangerous in males because they are more susceptible to blockages due to their longer, Random article narrower urethrae. Urinary tract disorders have a high rate of recurrence, and some cats seem to be more susceptible to urinary problems than others. search Contents 1 Symptoms Go Search 2 Causes interaction 3 Treatment About Wikipedia 4 Further reading Community portal 5 External links Recent changes Contact Wikipedia Symptoms [edit] Donate to Wikipedia Help Symptoms of the disease include prolonged squatting and straining during attempts to urinate, frequent trips to the litterbox or a reluctance to leave toolbox the area, small amounts of urine voided in each attempt, blood in the urine, howling, crying, or other vocalizations. -
A Surgeon's Perspective on the Current Trends in Managing
A Surgeon’s Perspective on the Current Trends in Managing Osteoarthritis David Dycus, DVM, MS, DACVS, CCRP Veterinary Orthopedic and Sports Medicine Group Annapolis Junction, MD Osteoarthritis (OA) is a chronic, progressive disease that affects both dogs and cats. It has been noted that up to 20% of adult dogs and 60% of adult cats have radiographic evidence of OA.1,2 Owners, themselves are becoming increasingly aware that bone and joint problems are and issue with their pet. Much of this increased awareness has come through the use of the Internet and social media. The overall outcome of osteoarthritis is centered on destruction of the articular cartilage and breakdown of the joint. Because of this OA must be thought of as a global disease process rather than an isolated disease entity. There is considerable cross talk among the tissues that make up a joint. For this reason the joint must be thought of as an organ and the final pathway of OA is organ failure of the joint. OA primarily affects diarthrodial joints. A diarthrodial joint is composed of the joint capsule, synovial lining, articular cartilage, and the surrounding muscles, ligaments, tendons, and bone. The joint capsule is composed of two layers: the outer fibrous layer and the inner subsynovial layer. Both layers have a rich blood and nerve supply. One explanation of pain associated with OA is distention of the joint capsule due to joint effusion. The synovial lining covers ever structure in the joint except for the cartilage/menisci. It provides a low friction lining and is responsible for the production of synovial fluid. -
Cat Flea from Wikipedia, the Free Encyclopedia
Log in / create account article discussion edit this page history Cat flea From Wikipedia, the free encyclopedia The cat flea, Ctenocephalides felis, is one of the most abundant and widespread fleas in the world. Cat flea Contents navigation Main page 1 Overview Contents 2 Life cycle Featured content 3 Effects on the hosts Current events 4 Disease transmission Random article 5 References search 6 External links 6.1 Flea Treatment Scientific classification Go Search Overview [edit] Domain: Eukaryota interaction The cat flea's primary host is the domestic cat, but this is also the primary flea infesting dogs in most of the Kingdom: Animalia About Wikipedia world. The cat flea can also maintain its life cycle on other carnivores and on the Virginia opossum. Rabbits, Phylum: Arthropoda Community portal rodents, ruminants and humans can be infested or bitten, but a population of cat fleas cannot be sustained by Recent changes Class: Insecta [citation needed] Contact Wikipedia these aberrant hosts. Order: Siphonaptera Donate to Wikipedia Help Life cycle [edit] Family: Pulicidae Genus: Ctenocephalides toolbox The female cat flea lays her eggs on the host, but the eggs, once dry, What links here have evolved to filter out of the haircoat of the host into the resting and Species: C. felis Related changes sheltering area of the host. Upload file Binomial name Special pages Ctenocephalides felis Printable version (Bouché, 1835) Permanent link Cite this page The eggs hatch into larvae, which are negatively phototaxic, meaning that languages Photo showing some they hide from light in the substrate. Flea larvae feed on a variety of organic characteristics used to identify from Deutsch substances, but most importantly subsist on dried blood that is filtered out other fleas, including genal comb Français of the haircoat of the host after it is deposited there as adult flea fecal Nederlands material. -
Chapter 25: Oncology
eoplasia is an abnormal, uncontrolled, pro- gressive proliferation of cells in any tissue CHAPTER or organ. Classification of neoplasms is N based upon general tissue origin (epithelial vs. mesenchymal), specific cell lineage and whether the neoplasm is benign (-oma) or malignant (sarcoma or carcinoma). Classification of some neoplasms as benign or malignant may require knowledge of the biological behavior of the neoplasm. 25 The majority of the veterinary medical literature has reported the incidence, gross appearance and micro- scopic characteristics of neoplasms of domesticated birds, especially poultry.20,22,23,101,109 Furthermore, the study of retroviral-induced neoplasia in poultry has advanced medical knowledge of retroviral molecular biology, as well as that of neoplasm development, growth and metastasis.91,101 Similar information con- ONCOLOGY cerning neoplasms of captive and free-ranging birds is almost nonexistent. One ultrastructural survey of various budgerigar neoplasms failed to disclose retroviral particles, but sampling errors are a known complication of such studies.52 More recently, papil- lomaviruses have been demonstrated as the etiologic agents of cutaneous papillomas in African Grey Par- rots, Chaffinches and Bramblings.73,87,94,96 Kenneth S. Latimer Reports of neoplasia are extant for captive as op- posed to free-ranging birds,5,6,7,12,15,49,51,83,102,108 espe- cially budgerigars, where the overall incidence of neoplasia ranges from 16.8% to 24.2%.12,15,51 In a veterinary diagnostic laboratory with a diverse avian caseload, budgerigars accounted for 69.7% of all psit- tacine neoplasms and 41% of all avian neoplasms recorded. The overall incidence of neoplasia approxi- mated 3.8% in all avian submissions.108 Compared to free-ranging birds, neoplasia is re- ported more frequently in companion and aviary birds because such birds are observed closely for abnormalities, have a longer life span and may have a genetic predisposition to neoplasia through in- breeding.