Acute Traumatic Spinal Cord Injury Guideline
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Internal Bleeding
Internal bleeding What is internal bleeding? It is a leakage of blood from the blood vessels of the surrounding tissues because of an injury affect the vessels and lead to rupture. Internal bleeding occurs inside the body cavities such as the head, chest, abdomen, or eye, and it is difficult to detect, because the leaked blood cannot be seen, and the person may not feel its occurrence till the symptoms associated with that bleeding start to appear. Note: It should be noted that people who take anticoagulant drugs are more likely to have this bleeding than others. What are symptoms of abdominal internal bleeding? There are many symptoms developed by the patients of internal bleeding in the abdomen or chest as below: • Feeling of pain in the abdomen. • Shortness of breath. • Feeling of chest pain. • Dizziness upon standing. • Bruises around the navel or on both sides of the abdomen. • Nausea, Vomiting. • Blood in urine. • Dark color stool. What are the symptoms of abdominal internal bleeding? Sometimes, internal bleeding may lead to loss of large amounts of blood, and in this case, the patient will have many symptoms, as below: • Accelerated heart beats • Low blood pressure • Skin sweating • General weakness • Feeling lethargic or feeling sleepy When should I go to seek medical care? Internal bleeding is very dangerous and life threatening and you should visit the doctor when experience one of the following cases:: ✓ After exposure to a severe injury, to ensure that there is no internal bleeding. ✓ Feeling severe pain in the abdomen ✓ Feeling acute shortness of breath ✓ feeling dizzy ✓ Seeing a change in vision Note: When these symptoms are noticed, you should go immediately to medical care or you must call the emergency services to avoid death. -
The European Guideline on Management Of
Rossaint et al. Critical Care (2016) 20:100 DOI 10.1186/s13054-016-1265-x RESEARCH Open Access The European guideline on management of major bleeding and coagulopathy following trauma: fourth edition Rolf Rossaint1, Bertil Bouillon2, Vladimir Cerny3,4,5,6, Timothy J. Coats7, Jacques Duranteau8, Enrique Fernández-Mondéjar9, Daniela Filipescu10, Beverley J. Hunt11, Radko Komadina12, Giuseppe Nardi13, Edmund A. M. Neugebauer14, Yves Ozier15, Louis Riddez16, Arthur Schultz17, Jean-Louis Vincent18 and Donat R. Spahn19* Abstract Background: Severe trauma continues to represent a global public health issue and mortality and morbidity in trauma patients remains substantial. A number of initiatives have aimed to provide guidance on the management of trauma patients. This document focuses on the management of major bleeding and coagulopathy following trauma and encourages adaptation of the guiding principles to each local situation and implementation within each institution. Methods: The pan-European, multidisciplinary Task Force for Advanced Bleeding Care in Trauma was founded in 2004 and included representatives of six relevant European professional societies. The group used a structured, evidence-based consensus approach to address scientific queries that served as the basis for each recommendation and supporting rationale. Expert opinion and current clinical practice were also considered, particularly in areas in which randomised clinical trials have not or cannot be performed. Existing recommendations were reconsidered and revised based on new scientific evidence and observed shifts in clinical practice; new recommendations were formulated to reflect current clinical concerns and areas in which new research data have been generated. This guideline represents the fourth edition of a document first published in 2007 and updated in 2010 and 2013. -
Title: ED Trauma: Trauma Nurse Clinical Resuscitation
Title: ED Trauma: Trauma Nurse Clinical Resuscitation Document Category: Clinical Document Type: Policy Department/Committee Owner: Practice Council Original Date: Approved By (last review): Director of Emergency Services, Approval Date: 07/28/2014 Trauma Medical Director, Medical Director Emergency (Complete history at end of document.) Services POLICY: To provide immediate, effective and efficient patient care to the trauma patient, designated nursing staff will respond to the trauma room when a trauma page is received. TRAUMA CONTROL NURSE: 1) Role: a) The trauma control nurse (TCN) is a registered nurse (RN) with specialized training in the care of the traumatized patient, and who will function as the trauma team’s lead nurse. b) The TCN shall have successfully completed the Trauma Nurse Core Course (TNCC), Advanced Cardiac Life Support (ACLS), Emergency Nurse Pediatric Course (ENPC) or Pediatric Advanced Life Support (PALS), and role orientation with trauma services. c) Full-time employee or regularly scheduled part-time Emergency Department (ED) nurse. d) RN must have 6 months of LMH ED experience. 2) Trauma Control Duties: a) Inspects and stocks trauma room at beginning of each shift and after each trauma patient is discharged from the ED. b) Attempts to maintain trauma room temperature at 80-82 degrees Fahrenheit. c) Communicates with pre-hospital personnel to obtain patient information and prior field treatment and response. d) Makes determination that a patient meets Type I or Type II criteria and immediately notifies LMH’s Call System to initiate the Trauma Activation System. e) Assists physician with orders as directed. f) Acts as liaison with patient’s family/law enforcement/emergency medical services (EMS)/flight crews. -
Symptomatic Intracranial Hemorrhage (Sich) and Activase® (Alteplase) Treatment: Data from Pivotal Clinical Trials and Real-World Analyses
Symptomatic intracranial hemorrhage (sICH) and Activase® (alteplase) treatment: Data from pivotal clinical trials and real-world analyses Indication Activase (alteplase) is indicated for the treatment of acute ischemic stroke. Exclude intracranial hemorrhage as the primary cause of stroke signs and symptoms prior to initiation of treatment. Initiate treatment as soon as possible but within 3 hours after symptom onset. Important Safety Information Contraindications Do not administer Activase to treat acute ischemic stroke in the following situations in which the risk of bleeding is greater than the potential benefit: current intracranial hemorrhage (ICH); subarachnoid hemorrhage; active internal bleeding; recent (within 3 months) intracranial or intraspinal surgery or serious head trauma; presence of intracranial conditions that may increase the risk of bleeding (e.g., some neoplasms, arteriovenous malformations, or aneurysms); bleeding diathesis; and current severe uncontrolled hypertension. Please see select Important Safety Information throughout and the attached full Prescribing Information. Data from parts 1 and 2 of the pivotal NINDS trial NINDS was a 2-part randomized trial of Activase® (alteplase) vs placebo for the treatment of acute ischemic stroke. Part 1 (n=291) assessed changes in neurological deficits 24 hours after the onset of stroke. Part 2 (n=333) assessed if treatment with Activase resulted in clinical benefit at 3 months, defined as minimal or no disability using 4 stroke assessments.1 In part 1, median baseline NIHSS score was 14 (min: 1; max: 37) for Activase- and 14 (min: 1; max: 32) for placebo-treated patients. In part 2, median baseline NIHSS score was 14 (min: 2; max: 37) for Activase- and 15 (min: 2; max: 33) for placebo-treated patients. -
Guidelines for Trauma Team Activation (TTA)
Guidelines for Trauma Team Activation (TTA) ONE of the following criteria must be present with associated traumatic mechanism L e v e Measure Vital Signs and level of consciousness l Trauma Team Activation ALL TTA 1 & 2's MUST BE TRANSPORTED TO RGH Rural Travel time greater than 1 hour, failed airway · Glasgow Coma Scale less than 13 or immediate life threat divert to local facility and · Systolic Blood Pressure less than 90mmHg arrange STAT transport to RGH Trauma Center · Respiratory Rate less than 10 or greater then 29 breaths Prehospital per minute (less than 20 in infant), or advanced airway · Assess patient and determine TTA Level 1 support required · Early activation to receiving facility with: TTA Level, MIVT Report, ETA · STARS Activation or ALS (ACP) intercept NO · Update facility as needed Yes · Transport to Trauma Center Assess anatomy of injury Triage Nurse · Alert TTL Physician with TTA level, MIVT Report, ETA · TTL has a 20min response time · All penetrating injuries to head, neck, torso, and · Alert switchboard to overhead page: extremities proximal to elbow or knee Trauma Level ‘#’ ETA · Chest wall instability or deformity (e.g. flail chest) Trauma Team Lead · Two or more proximal long-bone fractures · Update ER on incoming Rural Trauma patients · Crushed, degloved, mangled, pulseless or amputation · Assume lead role and MRP status of an extremity proximal to wrist or ankle · Prepare resuscitation team · Pelvic fractures (high impact) · Assess, Treat and Stabilize patient 2 · Major facial or head trauma including depressed/open -
Complications from COVID-19 – Not for the Faint of Heart Jeannette Guerrasio, MD
Complications from COVID-19 – Not for the faint of heart Jeannette Guerrasio, MD Anytime a person suffers a health event there is always the risk for complications. For example, if you are walking through the living room and bump your leg into the coffee table, you will get a superficial bruise. You may not have any complications at all. But, if you also get a tiny cut on your shin from the corner of the coffee table and bacteria that normally live on the skin get into the cut, you will develop a complication in the form of a skin infection (cellulitis). If you are on blood thinners, you may bleed more than the average person and develop the complication of a deeper, lumpy, more painful bruise called a hematoma. The same is true for COVID-19. When an individual gets the virus, they may or may not go on to develop complications. Some of the complications are more likely than others and some of them are temporary while others may become permanent. The most common complications of COVID-19 are the development of blood clots and low oxygen levels. Patients can form blood clots in the veins or arteries anywhere in the body. The most common locations are in their legs (deep vein thrombosis or DVT) and lungs (pulmonary embolism or PE.) Some patients who get blood clots need to be on blood thinners, like Warfarin, Xarelto or Eliquis, for months while others need them for the rest of their lives. COVID-19 disrupts the iron in patient’s red blood cells, making it harder for their blood to carry oxygen from their lungs to the organs of their body, resulting in low oxygen levels. -
The Internal Treatment of Traumatic Injury
THE INTERNAL TREATMENT OF TRAUMATIC INJURY The focus of this paper is the treatment of traumatic injury with internally ingested Chinese herbal formulas. Whereas the strategy for external treatment of traumatic injury is governed by clinical manifestation, internal treatment strategies are governed by proper identification of progressive stages. GENERAL SIGNS/SYMPTOMS OF ACUTE INJURY There are three distinct stages of traumatic injury, which are expressed by a limited number of clinical manifest- ations. The three primary manifestations of the early stages of trauma are heat, swelling, and pain. Western medicine, since the time of the great Roman physician, Galen, has specified five signs, but the differences, from our point of view, is negligible. The five signs discussed by Western medicine are: pain, swelling, redness, heat, and loss of function. Oriental medicine combines heat and redness into one sign, since both a sensation of warmth and the visible sign of redness are classified as heat. The “loss of function” sign is seen by Oriental medicine as a mechanical consequence of significant qi and blood stasis, and cannot be addressed separately from qi and blood stasis by internal treatments. Thus, both East and West are in basic agreement about the signs of early stage injury. If acute injury develops into a chronic issue, other signs can come into play, such as numbness/tingling, localized weakness, and aggravation by external evils such as cold. A WORD ABOUT BLEEDING Bleeding is a special manifestation of traumatic injury, and is a pattern unto itself. In most injuries where there is bleeding, it must be stopped before further assessment is made. -
Myocardial Infarction (Heart Attack)
Sacramento Heart & Vascular Medical Associates February 19, 2012 500 University Ave. Sacramento, CA 95825 Page 1 916-830-2000 Fax: 916-830-2001 Patient Information For: Only A Test Myocardial Infarction (Heart Attack) What is a myocardial infarction (MI)? Myocardial infarction (MI) is a heart attack. It happens when blood flow to a part of the heart is suddenly blocked. How does it occur? Myocardial infarction may occur at any time and often occurs without warning. As we grow older, our coronary arteries may become narrowed by the buildup of cholesterol plaque. When the arteries narrow, less blood can go through them, and less oxygen gets to the heart muscle. The process of narrowing is called atherosclerosis. The narrower the artery becomes, the more likely it is that a blood clot may form and block the artery completely, causing a heart attack. Sometimes sudden blockages can occur even in places where the artery was not narrow before. A heart attack may also occur when the heart muscle needs more oxygen than the blood vessels can provide. This might happen, for example, during hard exercise such as shoveling snow, or with a sudden increase in blood pressure. Less commonly, a heart attack can occur due to coronary spasm. Coronary spasm is a sudden and temporary narrowing of a small part of an artery that supplies blood to the heart. It may be caused by smoking or drugs such as cocaine. Risk factors for heart disease include: - cigarette smoking - a family history of heart attack - diabetes - overweight - high blood pressure - high blood cholesterol - low HDL cholesterol (that is, too little "good" cholesterol) - stress - a lifestyle that does not include much physical activity. -
Management of Hypovolaemic Shock in the Trauma Patient HYPOVOLAEMIC SHOCK GUIDELINE
HypovaolaemicShock_FullRCvR.qxd 3/2/07 3:03 PM Page 1 ADULT TRAUMA CLINICAL PRACTICE GUIDELINES :: Management of Hypovolaemic Shock in the Trauma Patient HYPOVOLAEMIC SHOCK GUIDELINE blood O-neg HypovaolaemicShock_FullRCvR.qxd 3/2/07 3:03 PM Page 2 Suggested citation: Ms Sharene Pascoe, Ms Joan Lynch 2007, Adult Trauma Clinical Practice Guidelines, Management of Hypovolaemic Shock in the Trauma Patient, NSW Institute of Trauma and Injury Management. Authors Ms Sharene Pascoe (RN), Rural Critical Care Clinical Nurse Consultant Ms Joan Lynch (RN), Project Manager, Trauma Service, Liverpool Hospital Editorial team NSW ITIM Clinical Practice Guidelines Committee Mr Glenn Sisson (RN), Trauma Clinical Education Manager, NSW ITIM Dr Michael Parr, Intensivist, Liverpool Hospital Assoc. Prof. Michael Sugrue, Trauma Director, Trauma Service, Liverpool Hospital This work is copyright. It may be reproduced in whole or in part for study training purposes subject to the inclusion of an acknowledgement of the source. It may not be reproduced for commercial usage or sale. Reproduction for purposes other than those indicated above requires written permission from the NSW Insititute of Trauma and Injury Management. © NSW Institute of Trauma and Injury Management SHPN (TI) 070024 ISBN 978-1-74187-102-9 For further copies contact: NSW Institute of Trauma and Injury Management PO Box 6314, North Ryde, NSW 2113 Ph: (02) 9887 5726 or can be downloaded from the NSW ITIM website http://www.itim.nsw.gov.au or the NSW Health website http://www.health.nsw.gov.au January 2007 HypovolaemicShock_FullRep.qxd 3/2/07 12:36 PM Page i blood O-neg Important notice! 'Management of Hypovolaemic Shock in the Trauma Patient’ clinical practice guidelines are aimed at assisting clinicians in informed medical decision-making. -
Management of Hypovolaemic Shock in the Trauma Patient (Full Guideline)
HypovaolaemicShock_FullRCvR.qxd 3/2/07 3:03 PM Page 1 ADULT TRAUMA CLINICAL PRACTICE GUIDELINES :: Management of Hypovolaemic Shock in the Trauma Patient HYPOVOLAEMIC SHOCK GUIDELINE blood O-neg HypovaolaemicShock_FullRCvR.qxd 3/2/07 3:03 PM Page 2 Suggested citation: Ms Sharene Pascoe, Ms Joan Lynch 2007, Adult Trauma Clinical Practice Guidelines, Management of Hypovolaemic Shock in the Trauma Patient, NSW Institute of Trauma and Injury Management. Authors Ms Sharene Pascoe (RN), Rural Critical Care Clinical Nurse Consultant Ms Joan Lynch (RN), Project Manager, Trauma Service, Liverpool Hospital Editorial team NSW ITIM Clinical Practice Guidelines Committee Mr Glenn Sisson (RN), Trauma Clinical Education Manager, NSW ITIM Dr Michael Parr, Intensivist, Liverpool Hospital Assoc. Prof. Michael Sugrue, Trauma Director, Trauma Service, Liverpool Hospital This work is copyright. It may be reproduced in whole or in part for study training purposes subject to the inclusion of an acknowledgement of the source. It may not be reproduced for commercial usage or sale. Reproduction for purposes other than those indicated above requires written permission from the NSW Insititute of Trauma and Injury Management. © NSW Institute of Trauma and Injury Management SHPN (TI) 070024 ISBN 978-1-74187-102-9 For further copies contact: NSW Institute of Trauma and Injury Management PO Box 6314, North Ryde, NSW 2113 Ph: (02) 9887 5726 or can be downloaded from the NSW ITIM website http://www.itim.nsw.gov.au or the NSW Health website http://www.health.nsw.gov.au January 2007 HypovolaemicShock_FullRep.qxd 3/2/07 12:36 PM Page i blood O-neg Important notice! 'Management of Hypovolaemic Shock in the Trauma Patient’ clinical practice guidelines are aimed at assisting clinicians in informed medical decision-making. -
Chapter 24 Abdomen Injuries
44093_CH024_0001_0021.qxd 1/18/07 4:35 PM Page 1 SECTION 4TRAUMA Chapter 24 Abdomen Injuries Objectives Cognitive 4-8.17 Describe the epidemiology, including the morbidity/mortality and prevention strategies 4-8.1 Describe the epidemiology, including the for hollow organ injuries. (p 24.11) morbidity/mortality and prevention strategies for a patient with abdominal trauma. 4-8.18 Explain the pathophysiology of hollow organ (p 24.6, 24.7) injuries. (p 24.11) 4-8.2 Describe the anatomy and physiology of organs 4-8.19 Describe the assessment findings associated and structures related to abdominal injuries. with hollow organ injuries. (p 24.11) (p 24.6) 4-8.20 Describe the treatment plan and management of 4-8.3 Predict abdominal injuries based on blunt and hollow organ injuries. (p 24.15) penetrating mechanisms of injury. 4-8.21 Describe the epidemiology, including the (p 24.5, 24.8) morbidity/mortality and prevention strategies 4-8.4 Describe open and closed abdominal injuries. for abdominal vascular injuries. (p 24.12) (p 24.7, 24.8) 4-8.22 Explain the pathophysiology of abdominal 4-8.5 Explain the pathophysiology of abdominal vascular injuries. (p 24.12) injuries. (p 24.10) 4-8.23 Describe the assessment findings associated 4-8.6 Describe the assessment findings associated with abdominal vascular injuries. (p 24.12) with abdominal injuries. (p 24.10) 4-8.24 Describe the treatment plan and management of 4-8.7 Identify the need for rapid intervention and abdominal vascular injuries. (p 24.15) transport of the patient with abdominal injuries 4-8.25 Describe the epidemiology, including the based on assessment findings. -
Anytown Trauma Center Trauma Protocols
ANYTOWN TRAUMA CENTER TRAUMA PROTOCOLS TITLE: TRAUMA TEAM ACTIVATION PROTOCOL PURPOSE: The purpose of the protocol is to establish guidelines for trauma team activation and define the members of the responding trauma team to facilitate the resuscitation and management of critical or seriously injured patients who require rapid, organized resuscitation, evaluation and stabilization to promote optimal outcomes. It also serves to provide triage guidelines for adult and pediatric patients. PROCESS: 1. TRAUMA TEAM ACTIVATION PROTCOL A. The criteria for activation of the trauma team is clearly defined and posted at the Emergency Department triage desk, by the EMS communication station and in the resuscitation rooms. B. The trauma team may be activated prior to arrival based on the EMS communication and their assessment. C. The trauma surgeon, emergency medicine physician, emergency department charge nurse/ house supervisor, emergency department nurses and the Trauma Program Manager may activate the trauma team. D. The person calling the trauma activation will initiate the trauma page to group page the trauma team and will specify the MOI, BP, HR, ETA and level of activation required and age if available. E. If the trauma team members are present in the emergency department and alert is still communicated to ensure everyone is notified. F. Trauma team member notification and arrival times will be documented on the trauma flow sheet (paper or electronic). G. Trauma team members will sign-in when they arrive. H. Trauma team members will be activated for all patients who meet the following criteria: 1. Level 1 trauma activation (major): life threatening injuries and/or unstable vital signs, limb-threating or disability threatening injury 2.