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6/20/2017

Tami McDaniel, RRT, AE-C

#1 Cause of pediatric hospital admission

#1 Cause of death during the first year of life, except for congenital abnormalities

Almost all pediatric cardiac arrest begins as or respiratory arrest

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Pediatric

Pediatric Respiratory System

Large head, small mandible, small Large, posteriorly-placed High glottic opening Small airways Presence of , adenoids

Pediatric Respiratory System

Poor accessory muscle development Less rigid thoracic cage Horizontal ribs, primarily diaphragm breathers Increased metabolic rate, increased O2 consumption

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Respiratory Assessment

Airway Look, listen and feel

Look for chest and abdomen movement Listen for breath sounds Feel for movement at nose or mouth

Breathing Assess : Rate Effort Tidal volume Airway and lung sounds

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Normal respiratory rates by age

<1year 30-60

1-3 years 24-40

4-5 years 22-34

6-12 years 18-30

13-18 years 12-16

Any respiratory rate > 60 is abnormal

Signs of increased respiratory effort Nasal flaring Retractions Head bobbing Seesaw respirations Prolonged inspiratory or expiratory times

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Mouth Gasping Accessory muscle use Grunting

Grunting can be an ominous sign of impending respiratory failure

Pediatric Respiratory Diseases

Epiglottitis  Foreign body aspiration

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CROUP

Laryngotracheitis

 Laryngotracheobronchitis

Croup

PATHOPHYSIOLOGY • Viral (parainfluenza) • Affects , • Subglottic edema; Air flow obstruction

Croup

Incidence Affects mostly 6 months – 4 year olds Affects males more than females- 1.4/1 Mostly in fall and early winter

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Croup Low grade Hoarseness “Barking”cough Worsening  at night Respiratory distress

Severe Croup

Signs and Symptoms Suprasternal, intercostal, and subcostal retractions. Biphasic stridor Lethargy Agitation

Croup MILD CROUP-MANAGEMENT Reassurance Cool mist humidifier

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Croup SEVERE CROUP – MANAGEMENT

High concentration humidified oxygen Monitor oxygen saturations and ecg Racemic epi nebulizer treatments

BE PREPARED TO INTUBATE

EPIGLOTTITIS

Epiglottitis Pathophysiology Bacterial infection – H-influlenza B, Streptococcus of the and/or the supraglottictic tissue surrounding the epiglottis

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Epiglottitis Incidence 0.3 per 100,000 -Reduced incidence with HIB vaccine More males than females – 60%/40% No seasonal variation Most cases ages 2-7

Can occur at any age

Epiglottitis Signs & Symptoms Fever Sore throat  Respiratory distress Drooling Anxiety

Epiglottitis Signs & Symptoms Stridor –early on suprasternal, subcostal, and intercostal retractions Differentiate from croup by absence of “barking” cough

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Epiglottitis Management – Do’s Keep child calm Make parents informed of seriousness of situation Take child to OR to secure airway as soon as possible Anyone with a diagnosis of epiglottitis is intubated

Epiglottitis Management – Don’ts Agitate the child No direct visualization of the airway – except in the OR No IV’s until airway is secure No lab work until airway is secure No suctioning until airway is secure

Epiglottitis Steroids are not indicated for the management of epiglottitis Racemic epinephrine has no role in the treatment of acute epiglottitis

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ASTHMA

Asthma Pathophysiology  Mast cells, eosinophils, lymphocytes infiltrate airway after exposure to allergen or irritant  Airway hyperresponsiveness results in  Bronchial wall edema  Smooth muscle contraction  Excess mucous production

Asthma

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Asthma

Incidence 6.3 million children Prevalence highest in children <5 years 3rd ranked cause of pediatric hospitalizations 0.3 per 100,000 deaths in children

Asthma Signs & Symptoms Cough SOA Wheezing Chest tightness Retractions Air hunger Progressive fatigue

Asthma Management Quick history and physical exam Auscultation Use of accessory muscles Heart rate Respiratory rate O2 sats Peak Flow (PEFR)

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Asthma Management –mild to moderate exacerbation ( PEFR 50-80%) Nebulized beta agonist- up to 3 doses in 1 hour O2 for SpO2 <90% Oral steroids Reevaluate Continue neb tx’s 1-3 hours provided there is improvement

Asthma Management – Severe exacerbation (PEFR<50%) Nebulized high dose beta agonist every 20 minutes or continuous for 1 hour O2 for SpO2 <90% IV corticosteroids Reevaluate

Asthma Management – Severe exacerbation (PEFR<50%) Continue nebulized high dose beta agonist hourly or continuously

Be prepared to intubate

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Asthma Emergency interventions should NOT include

CPT Mucolytics Sedation

BRONCHIOLITIS

Bronchiolitis Pathophysiology Viral infection RSV most common cause (85%) Affects small airways Thick mucous – increased production Air trapping Tissue inflammation in airways

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Bronchiolitis Incidence Affects children less than 2 years of age Affects 4% of children <1year old Affects 6% of children 1-2 years old Mostly from November – April Peaks in January & February

Bronchiolitis Incidence More males than females (1.25 X) 90,000 hospitalization/year 4500 deaths /year

Bronchiolitis Signs & Symptoms Viral upper respiratory infection Cough Low grade fever SOA Expiratory wheezes Poor feeding

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Bronchiolitis Signs & Symptoms  Retractions Grunting Apnea

Bronchiolitis Management Humidified oxygen Nasal & oral suctioning ( with saline drops) IV’s as needed for hydration

Bronchiolitis Management Trial of bronchodilator therapy to assess effectiveness Discontinue if not effective and agitation occurs Use bronchodilator therapy for children with history of asthma

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Bronchiolitis Despite the prominent role that inflammation plays in the pathogenesis of , have not clearly been shown to be of significant benefit in improving the clinical status of patients with bronchiolitis.

FOREIGN BODY AIRWAY OBSTRUCTION

FBAO Incidence High risk in children <7 years 90% of deaths are in children <5

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FBAO Signs & Symptoms Choking Coughing Stridor Respiratory distress Wheezing Inability to ventilate during CPR

FBAO Management Minimal intervention if child is conscious maintaining own airway O2

FBAO Management – totally obstructed Infant – 5 back blows followed by 5 chest thrusts

Child – 5 abdominal thrusts

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FBAO Management – Totally obstructed

 Continue CPR until object dislodged

Pulse Oximetry  Two light emitting diodes – one at the red spectrum and one at the infared spectrum Must be positioned so that the emitter and the detector are exactly opposite each other

Pulse Oximetry Sensor can be placed on finger, toe, top of ear and lobe of ear – for infants and newborns palm of hand or sole of foot Needs to be 5- 10 mm of tissue between the two diodes

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Pulse Oximetry- Limitations Motion artifact is the most common  Recognized by a distorted wave form Poor Perfusion  Inadequate arterial pulsation at the measurement site  Ambient light interference causes “flooding” effect  Can be avoided by covering sensor

CASE STUDIES

 2 year old presents with cough, stridor, temp 99.5,child has a hoarse cry and a barking cough. In your assessment you note retractions and inspiratory and expiratory stridor.  What do you think is wrong with this child?  What would your treatment be?

 3 year old with a history of fever, sore throat that has progressively worsened over the last 3 hours. You find the child sitting up leaning forward with hands on knees. The mother says the child has not had a cough. Your assessment notes drooling, retractions and anxiety.  What do you think is wrong with this child?  What would your treatment be?

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 2 year old brought to the ER by EMS. Mom says she went in to wake the child up for day care and the child took one breath then stopped breathing. You notice that the chest is not rising with ventilations via bag/mask.  What do you think is wrong with this child?  What would your treatment be?

QUESTIONS?

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