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TRADD
Accumulation of P53 and Reductions in XIAP Abundance Promote the Apoptosis of Prostate Cancer Cells
Life, Death, and the Pursuit of Apoptosis
Expression of the Tumor Necrosis Factor Receptor-Associated Factors
Supplementary Table 2 Supplementary Table 1
Beyond Tumor Necrosis Factor Receptor: TRADD Signaling in Toll-Like Receptors
The Molecular Links Between Cell Death and Inflammasome
Bcl-2 Inhibition to Overcome Resistance to Chemo- and Immunotherapy
Caspase Activation & Apoptosis
Regulation of Caspase-8 Activity at the Crossroads of Pro-Inflammation
Death Signaling Paper Discussion • Apaf-1, a Human Protein
Characterizing the Sphingomyelinase Pathway Triggered by PRIMA-1 Derivatives in Lung Cancer Cells with Differing P53 Status
A Novel Mechanism of TRAF Signaling Revealed by Structural and Functional Analyses of the TRADD–TRAF2 Interaction
Apo2l/TRAIL-Dependent Recruitment of Endogenous FADD and Caspase-8 to Death Receptors 4 and 5
REVIEW the IAP Family: Endogenous Caspase Inhibitors with Mul- Tiple Biological Activities
Signaling TNF-Like Ligand 1A/Death Receptor 3 the Adaptor Protein
Introduction Cytotoxic Granule Contents Inhibitors of Apoptosis
REVIEW Death and Decoy Receptors and P53-Mediated Apoptosis
N Domains Help Define Novel Apoptosis and Immunity
Top View
Therapeutic Small Molecules Target Inhibitor of Apoptosis Proteins In
Apoptosis of Tumor-Infiltrating T Lymphocytes: a New Immune
Growth of Human Prostate Cancer Cells Is Significantly Suppressed in Vitro with Sodium Butyrate Through Apoptosis
Survivin – an Anti-Apoptosis Protein: Its Biological Roles and Implications
CD30-Dependent Degradation of TRAF2: Implications for Negative Regulation of TRAF Signaling and the Control of Cell Survival
The Role of Bcl-2 and Its Pro-Survival Relatives in Tumourigenesis and Cancer Therapy
TRADD Mediates the Tumor Necrosis Factor-Induced Apoptosis of L929
The Tumor Necrosis Factor Receptor 2 Signal Transducers TRAF2
Genetic Events Inhibiting Apoptosis in Diffuse Large B Cell Lymphoma
Expression of Antiapoptotic and Proapoptotic Molecules in Diabetic
Evidence Against the Hypothesis That BCL-2 Inhibits Apoptosis Through an Anti-Oxidant Effect
Activation of the NF-Kb Pathway by Caspase 8 and Its Homologs
Future Therapeutic Directions for Smac-Mimetics
FADD) Sensitizes Osteosarcoma to Tnfα-Induced Cell Death Mario G
The Inhibitor-Of-Apoptosis Protein Survivin Increases P34cdc2
EVER2 Protein Binds TRADD to Promote TNF–Induced Apoptosis
Of Caspase-3 Activation Synthesis of Bcl-2 and Mediated by Inhibition
(TWEAK) Enhances Poly(I:C)-Induced RIPK1-Mediated Necroptosis