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TP53INP1
Mir-155 Targets TP53INP1 to Regulate Liver Cancer Stem Cell Acquisition and Self-Renewal ⇑ Fengchao Liu, Xin Kong, Lin Lv, Jian Gao
Mir-504 Promotes Tumour Growth and Metastasis in Human Osteosarcoma by Targeting TP53INP1
TP53INP1 Down-Regulation Activates a P73-Dependent DUSP10/ERK Signaling Pathway To
Rna-Sequencing Applications: Gene Expression Quantification and Methylator Phenotype Identification
Oxidative Stress-Induced P53 Activity Is Enhanced by a Redox-Sensitive TP53INP1 Sumoylation
TP53INP1, a Tumor Suppressor, Interacts with LC3 and ATG8-Family Proteins Through the LC3-Interacting Region (LIR) and Promotes Autophagy-Dependent Cell Death
Original Article Microrna-155 Acts As an Oncogene by Targeting the Tumor Protein 53-Induced Nuclear Protein 1 in Esophageal Squamous Cell Carcinoma
Microrna-15A-5P Down-Regulation Inhibits Cervical Cancer by Targeting TP53INP1 in Vitro
Tp53inp1 Gene Is Implicated in Early Radiation Response in Human Fibroblast Cells
Chemico-Biological Interactions 196 (2012) 89–95
Open Moore Sarah P53network.Pdf
TP53INP1 (H-110): Sc-68919
Table S1. 103 Ferroptosis-Related Genes Retrieved from the Genecards
Downloaded from the Protein Data Bank (4O2B, PDB)[99], (Figure S6)
Gene-Wide Analysis Detects Two New Susceptibility Genes for Alzheimerâ
Pulmonary Deregulation of Expression of Mir-155 and Two of Its Putative
ONCOGENOMICS a Genome-Wide Study of the Repressive Effects of Estrogen Receptor Beta on Estrogen Receptor Alpha Signaling in Breast Cancer Cells
Genome-Wide Mrna and Microrna Profiling of the NCI 60 Cell-Line Screen and Comparison of Fdump[10] with Fluorouracil, Floxuridine, and Topoisomerase 1 Poisons
Top View
A Positive Feedback Loop of P53/Mir-19/TP53INP1 Modulates Pancreatic Cancer Cell Proliferation and Apoptosis
TP53INP1 Downregulation Activates a P73- Dependent DUSP10/ERK
Microrna-155-5P Promotes Tumor Progression and Contributes Paclitaxel Resistance to Chemotherapy Via TP53INP1 in Human Breast Cancer
Pathophysiological Mechanisms Underlying Phenotypic Differences in Pulmonary Radioresponse Received: 15 June 2016 Isabel L
Tumor Protein 53–Induced Nuclear Protein 1 Is a Major Mediator of P53 Antioxidant Function Carla E
Meaning of Tumor Protein 53-Induced Nuclear Protein 1 in the Molecular Mechanism of Gemcitabine Sensitivity
CCAR1 5′ UTR As a Natural Mirancer of Mir-1254 Overrides Tamoxifen Resistance
Gene Section Mini Review
The Mir-17B92 Family Regulates the Response to Toll-Like Receptor 9 Triggering of CLL Cells with Unmutated IGHV Genes
Original Article Mir-155 Promotes Proliferation of Human Non-Small Cell Lung Cancer H460 Cells Via Targeting TP53INP1
Mir-524-5P of the Primate-Specific C19MC Mirna Cluster Targets TP53IPN1- and EMT-Associated Genes to Regulate Cellular Reprogram
Integrative P53, Micro-RNA and Cathepsin Protease Co-Regulatory Expression Networks in Cancer
Roles for Micrornas, Mir-93 and Mir-130B, and Tumor Protein 53
Genomic Innovation of ATD Alleviates Mistranslation Associated With
Identification by High-Throughput in Silico Screening of Radio-Protecting Compounds Targeting the DNA-Binding Domain of the Tumor Suppressor P53
Mir-106B Regulates the Proliferation and Differentiation of Neural Stem/Progenitor Cells Through Tp53inp1-Tp53-Cdkn1a Axis
1 Tumour Protein 53-Induced Nuclear Protein 1 (TP53INP1) Enhances
Microrna-155-5P Contributes to 5-Fluorouracil Resistance Through Down-Regulating TP53INP1 in Oral Squamous Cell Carcinoma