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Shigekazu Nagata
Shigekazu Nagata Is Better Known for His Work on Apoptosis
A Sublethal ATP11A Mutation Associated with Neurological Deterioration Causes Aberrant Phosphatidylcholine Flipping in Plasma Membranes
General Report on 8Th IDS
Enhanced and Accelerated Lymphoproliferation in Fas
World Premier International Research Center Initiative (WPI) FY2014 WPI Project Progress Report (Post-Interim Evaluation)
Tumor Apoptosis, Respectively by Fas-Fas Ligand- and Perforin-Induced Recombinant IL-12 Are Mediated Primarily Administration Of
Functional Expression of the P2X7 ATP
Mapping Multiple Mutations See Biobits Page 28
Engulfment of Apoptotic Cells Locus-1 in a Subset of Macrophages for Expression of Developmental Endothelial
DNA-Mediated Cyclic GMP−AMP Synthase− Dependent and −Independent Regulation of Innate Immune Responses
Swiss-Kyoto Symposium 21–22 November 2013 ETH Zurich and the University of Zurich Switzerland
Necrotic Death Pathway in Fas Receptor Signaling
ASBMB FELLOWS Planning a Scientific Conference? the ASBMB Is Here to Help
Lupus-Like Autoimmune Disease Caused by a Lack of Xkr8, a Caspase-Dependent Phospholipid Scramblase
Early Work on the Function of CD95, an Interview with Shige Nagata
20Th HFSP AWARDEES MEETING an Online Event 5 – 8 July 2021
The 5Th Kyoto University— Inamori Foundation Joint Kyoto Prize Symposium
Mice Lpr Disease in MRL- Lymphoproliferation but Not Autoimmune Transgenic Expression of Fas in T Cells Blocks
Top View
Drosophila EYA Regulates the Immune Response Against DNA Through An
AAI Expands Travel Awards and Outreach Programs
Suppressor of Cytokine Signaling-1 (SOCS-1) Suppresses Tumor Necrosis Factor ␣-Induced Cell Death in Fibroblasts
Announcement of the Keio Medical Science Prize 2013