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MEF2C
Prox1regulates the Subtype-Specific Development of Caudal Ganglionic
And Represses TNF Gene Expression Activating Transcription Factor-2
Targeting Endothelial Kruppel-Like Factor 2 (KLF2) in Arteriovenous
Global Mef2 Target Gene Analysis in Skeletal and Cardiac Muscle
Cortical Foxp2 Supports Behavioral Flexibility and Developmental Dopamine D1 Receptor Expression
Foxp2 Loss of Function Increases Striatal Direct Pathway Inhibition Via Increased GABA Release
Transcription Factor MEF2C Influences Neural Stem/Progenitor Cell Differentiation and Maturation in Vivo
A Mef2 Gene That Generates a Muscle-Specific Isoform Via Alternative Mrna Splicing JAMES F
Rb and N-Ras Function Together to Control Differentiation in the Mouse
Integrative Analysis Identifies Bhlh Transcription Factors As Contributors to Parkinson's Disease Risk Mechanisms
Glucocorticoid Receptor and Myocyte Enhancer Factor 2 Cooperate to Regulate the Expression of C-JUN in a Neuronal Context
Foxp2 Regulates Anatomical Features That May Be Relevant for Vocal Behaviors and Bipedal Locomotion
Research Article ERK5 Contributes to VEGF Alteration in Diabetic Retinopathy
Inflammation Novel Mechanism to Cope
Transcriptional Factor Binding Sites and Disease
MEF2 Transcription Factors Are Key Regulators of Sprouting Angiogenesis
ISL1 Loss-Of-Function Mutation Contributes to Congenital Heart Defects
Supplementary Appendix
Top View
MEF2C Analysis
Egr/Mef2c Transcription Factors Regulate The
Altered Expression of the Transcription Factor Mef2c During Retinal Degeneration in Rpe65–/– Mice
Similarities of Retinoblastoma Function Between Plants and Animals, from Developmental Processes to Epigenetic Regulation
Foxp2 Controls Synaptic Wiring of Corticostriatal Circuits and Vocal Communication by Opposing Mef2c
NKL-Code in Normal and Aberrant Hematopoiesis
MEF2C Regulates Outflow Tract Alignment and Transcriptional Control of Tdgf1 Ralston M
Gene Activation and Repression by the Glucocorticoid Receptor Are Mediated by Sequestering Ep300 and Two Modes of Chromatin Binding
Transcription Factors in Cancer: When Alternative Splicing Determines Opposite Cell Fates
VU Research Portal
MEF2C Haploinsufficiency Syndrome, the Changes That Cause MEF2C Websites, Facebook Groups and Other Links the Syndrome Have Occurred out of the Blue (De Novo )
Epigenetic Regulation of the Lineage Specificity of Primary Human Dermal
MEF2C Phosphorylation Is Required for Chemotherapy Resistance in Acute Myeloid Leukemia
Myocyte Enhancer Factor 2C in Hematopoiesis and Leukemia
Positional Specificity of Different Transcription Factor Classes Within Enhancers
Landscape and Dynamics of the Transcriptional Regulatory Network
Data Set 1. Biological Analysis of the Genes Found to Be Significant in the Endotoxin Study
The LIM Domain Protein Ntrip6 Acts As a Co-Repressor for the Transcription Factor MEF2C in Myoblasts