Remarks on Pelvic Peritonitis and Pelvic Cellulitis, with Illustrative Cases
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ICD10 Diagnoses FY2018 AHD.Com
ICD10 Diagnoses FY2018 AHD.com A020 Salmonella enteritis A5217 General paresis B372 Candidiasis of skin and nail A040 Enteropathogenic Escherichia coli A523 Neurosyphilis, unspecified B373 Candidiasis of vulva and vagina infection A528 Late syphilis, latent B3741 Candidal cystitis and urethritis A044 Other intestinal Escherichia coli A530 Latent syphilis, unspecified as early or B3749 Other urogenital candidiasis infections late B376 Candidal endocarditis A045 Campylobacter enteritis A539 Syphilis, unspecified B377 Candidal sepsis A046 Enteritis due to Yersinia enterocolitica A599 Trichomoniasis, unspecified B3781 Candidal esophagitis A047 Enterocolitis due to Clostridium difficile A6000 Herpesviral infection of urogenital B3789 Other sites of candidiasis A048 Other specified bacterial intestinal system, unspecified B379 Candidiasis, unspecified infections A6002 Herpesviral infection of other male B380 Acute pulmonary coccidioidomycosis A049 Bacterial intestinal infection, genital organs B381 Chronic pulmonary coccidioidomycosis unspecified A630 Anogenital (venereal) warts B382 Pulmonary coccidioidomycosis, A059 Bacterial foodborne intoxication, A6920 Lyme disease, unspecified unspecified unspecified A7740 Ehrlichiosis, unspecified B387 Disseminated coccidioidomycosis A080 Rotaviral enteritis A7749 Other ehrlichiosis B389 Coccidioidomycosis, unspecified A0811 Acute gastroenteropathy due to A879 Viral meningitis, unspecified B399 Histoplasmosis, unspecified Norwalk agent A938 Other specified arthropod-borne viral B440 Invasive pulmonary -
Womens Health Requisition Forms
PCR - WHI Implementation PLACE ON SWAB 10854 Midwest Industrial Blvd. St. Louis, MO 63132 MM DD YY Phone: (314) 200-3040 | Fax (314) 200-3042 (1) CLIA ID #26D0953866 JANE DOE v3 PCR MOLECULAR REQUISITION - WOMEN'S HEALTH INFECTION PRACTICE INFORMATION PATIENT INFORMATION *SPECIMEN INFORMATION (2) DOE JANE MM/DD/YY LAST NAME FIRST NAME DATE COLLECTED W O M E ' N S H E A L T H I F N E C T I O N SSN MM/DD/YY HH:MM AM SSN DATE OF BIRTH TIME COLLECTED REQUESTING PHYSICIAN: DR. SWAB TESTER (3) Sex: F X M (4) Diagnosis Codes X N76.0 Acute vaginitis B37.49 Other urogenital candidiasis A54.9 Gonococcal infection, unspecified N76.1 Subacute and chronic vaginitis N89.8 Other specified noninflammatory disorders of vagina A59.00 Urogenital trichomoniasis, unspecified N76.2 Acute vulvitis O99.820 Streptococcus B carrier state complicating pregnancy A64 Unspecified sexually transmitted disease N76.3 Subacute and chronic vulvitis O99.824 Streptococcus B carrier state complicating childbirth A74.9 Chlamydial infection, unspecified N76.4 Abscess of vulva B95.1 Streptococcus, group B, as the cause Z11.3 Screening for infections with a predmoninantly N76.5 Ulceration of vagina of diseases classified elsewhere sexual mode of trasmission N76.6 Ulceration of vulva Z22.330 Carrier of group B streptococcus Other: N76.81 Mucositis(ulcerative) of vagina and vulva N70.91 Salpingitis, unspecified N76.89 Other specified inflammation of vagina and vulva N70.92 Oophoritis, unspecified N95.2 Post menopausal atrophic vaginitis N71.9 Inflammatory disease of uterus, unspecified -
N35.12 Postinfective Urethral Stricture, NEC, Female N35.811 Other
N35.12 Postinfective urethral stricture, NEC, female N35.811 Other urethral stricture, male, meatal N35.812 Other urethral bulbous stricture, male N35.813 Other membranous urethral stricture, male N35.814 Other anterior urethral stricture, male, anterior N35.816 Other urethral stricture, male, overlapping sites N35.819 Other urethral stricture, male, unspecified site N35.82 Other urethral stricture, female N35.911 Unspecified urethral stricture, male, meatal N35.912 Unspecified bulbous urethral stricture, male N35.913 Unspecified membranous urethral stricture, male N35.914 Unspecified anterior urethral stricture, male N35.916 Unspecified urethral stricture, male, overlapping sites N35.919 Unspecified urethral stricture, male, unspecified site N35.92 Unspecified urethral stricture, female N36.0 Urethral fistula N36.1 Urethral diverticulum N36.2 Urethral caruncle N36.41 Hypermobility of urethra N36.42 Intrinsic sphincter deficiency (ISD) N36.43 Combined hypermobility of urethra and intrns sphincter defic N36.44 Muscular disorders of urethra N36.5 Urethral false passage N36.8 Other specified disorders of urethra N36.9 Urethral disorder, unspecified N37 Urethral disorders in diseases classified elsewhere N39.0 Urinary tract infection, site not specified N39.3 Stress incontinence (female) (male) N39.41 Urge incontinence N39.42 Incontinence without sensory awareness N39.43 Post-void dribbling N39.44 Nocturnal enuresis N39.45 Continuous leakage N39.46 Mixed incontinence N39.490 Overflow incontinence N39.491 Coital incontinence N39.492 Postural -
Traveler's Diarrhea
Traveler’s Diarrhea JOHNNIE YATES, M.D., CIWEC Clinic Travel Medicine Center, Kathmandu, Nepal Acute diarrhea affects millions of persons who travel to developing countries each year. Food and water contaminated with fecal matter are the main sources of infection. Bacteria such as enterotoxigenic Escherichia coli, enteroaggregative E. coli, Campylobacter, Salmonella, and Shigella are common causes of traveler’s diarrhea. Parasites and viruses are less common etiologies. Travel destination is the most significant risk factor for traveler’s diarrhea. The efficacy of pretravel counseling and dietary precautions in reducing the incidence of diarrhea is unproven. Empiric treatment of traveler’s diarrhea with antibiotics and loperamide is effective and often limits symptoms to one day. Rifaximin, a recently approved antibiotic, can be used for the treatment of traveler’s diarrhea in regions where noninvasive E. coli is the predominant pathogen. In areas where invasive organisms such as Campylobacter and Shigella are common, fluoroquinolones remain the drug of choice. Azithromycin is recommended in areas with qui- nolone-resistant Campylobacter and for the treatment of children and pregnant women. (Am Fam Physician 2005;71:2095-100, 2107-8. Copyright© 2005 American Academy of Family Physicians.) ILLUSTRATION BY SCOTT BODELL ▲ Patient Information: cute diarrhea is the most com- mised and those with lowered gastric acidity A handout on traveler’s mon illness among travelers. Up (e.g., patients taking histamine H block- diarrhea, written by the 2 author of this article, is to 55 percent of persons who ers or proton pump inhibitors) are more provided on page 2107. travel from developed countries susceptible to traveler’s diarrhea. -
Amoebic Dysentery
University of Nebraska Medical Center DigitalCommons@UNMC MD Theses Special Collections 5-1-1934 Amoebic dysentery H. C. Dix University of Nebraska Medical Center This manuscript is historical in nature and may not reflect current medical research and practice. Search PubMed for current research. Follow this and additional works at: https://digitalcommons.unmc.edu/mdtheses Part of the Medical Education Commons Recommended Citation Dix, H. C., "Amoebic dysentery" (1934). MD Theses. 320. https://digitalcommons.unmc.edu/mdtheses/320 This Thesis is brought to you for free and open access by the Special Collections at DigitalCommons@UNMC. It has been accepted for inclusion in MD Theses by an authorized administrator of DigitalCommons@UNMC. For more information, please contact [email protected]. A MOE B leD Y SEN T E R Y By H. c. Dix University of Nebraska College of Medicine Omaha, N~braska April 1934 Preface This paper is presented to the University of Nebraska College of MediCine to fulfill the senior requirements. The subject of amoebic dysentery wa,s chosen due to the interest aroused from the previous epidemic, which started in Chicago la,st summer (1933). This disea,se has previously been considered as a tropical disease, B.nd was rarely seen and recognized in the temperate zone. Except in indl vidu8,ls who had been in the tropics previously. In reviewing the literature, I find that amoebio dysentery may be seen in any part of the world, and from surveys made, the incidence is five in every hun- dred which harbor the Entamoeba histolytlca, it being the only pathogeniC amoeba of the human gastro-intes tinal tract. -
Diagnostic Tests for Vaginosis/Vaginitis
Alberta Heritage Foundation for Medical Research Diagnostic tests for vaginosis/vaginitis Christa Harstall and Paula Corabian October 1998 HTA12 Diagnostic tests for vaginosis/vaginitis Christa Harstall and Paula Corabian October 1998 © Copyight Alberta Heritage Foundation for Medical Research, 1998 This Health Technology Assessment Report has been prepared on the basis of available information of which the Foundation is aware from public literature and expert opinion, and attempts to be current to the date of publication. The report has been externally reviewed. Additional information and comments relative to the Report are welcome, and should be sent to: Director, Health Technology Assessment Alberta Heritage Foundation for Medical Research 3125 Manulife Place, 10180 - 101 Street Edmonton Alberta T5J 3S4 CANADA Tel: 403-423-5727, Fax: 403-429-3509 This study is based, in part, on data provided by Alberta Health. The interpretation of the data in the report is that of the authors and does not necessarily represent the views of the Government of Alberta. ISBN 1-896956-15-7 Alberta's health technology assessment program has been established under the Health Research Collaboration Agreement between the Alberta Heritage Foundation for Medical Research and the Alberta Health Ministry. Acknowledgements The Alberta Heritage Foundation for Medical Research is most grateful to the following persons for their comments on the draft report and for provision of information. The views expressed in the final report are those of the Foundation. Dr. Jane Ballantine, Section of General Practice, Calgary Dr. Deirdre L. Church, Microbiology, Calgary Laboratory Services, Calgary Dr. Nestor N. Demianczuk, Royal Alexandra Hospital, Edmonton Dr. -
Ulcerative Post-Dysenteric Colitis
Gut: first published as 10.1136/gut.7.5.438 on 1 October 1966. Downloaded from Gut, 1966, 7, 438 Ulcerative post-dysenteric colitis S. J. POWELL AND A. J. WILMOT From the Amoebiasis Research Unit' and the Department ofMedicine, University ofNatal, Durban, South Africa EDITORIAL COMMENT Better treatment is resulting in more severe cases of amoebic colitis surviving and these patients may have severe residual damage to the bowel resulting in ulcerative post-dysenteric colitis. This is considered to be a distinct entity. The term 'post-dysenteric colonic irritability' was thousand patients who attend this hospital annually introduced by Sir Arthur Hurst (1943) to describe with acute amoebic dysentery complications are persistent irritability of the bowel following an acute common and we have had the opportunity to study attack of bacillary or amoebic dysentery. The early them (Wilmot, 1962). It is from this material that we symptoms were attributed to a non-specific chronic have based the following report of ulcerative post- colitis occurring after the specific infection had died dysenteric colitis in 33 African patients observed in out, but in the later stages were thought to be due to recent years. 'functional irritability' of the colon. Stewart (1950) found that post-dysenteric colitis was more common- CLINICAL FINDINGS ly a sequel to acute amoebic dysentery and was able All patients presented initially with severe amoebic to recognize two forms in his patients: 1 Those with dysentery, sigmoidoscopic examination showing a mild symptoms and no colonic ulceration, which he congested, oedematous mucosa with extensive rectal named 'functional post-dysenteric colitis', and (2) ulcers the surfaces of which were covered by sloughs http://gut.bmj.com/ Those with colonic ulceration and more severe and exudate. -
The Relative Risk
Clinical Expert Series Pelvic Inflammatory Disease David E. Soper, MD Obstet Gynecol 2010;116(2) ACCME Accreditation The American College of Obstetricians and Gynecologists (ACOG) is accredited by the Accreditation Council for Continuing Medical Education (ACCME) to provide continuing medical education for physicians. (Continuing medical education credit for “Pelvic Inflammatory Disease” will be available through August 2013.) AMA PRA Category 1 CreditTM and ACOG Cognate Credit The American College of Obstetricians and Gynecologists (ACOG) designates this educational activity for a maximum of 2 AMA PRA Category 1 CreditsTM or up to a maximum of 2 Category 1 ACOG cognate credits. Physicians should only claim credit commensurate with the extent of their participation in the activity. Disclosure Statement Current guidelines state that continuing medical education (CME) providers must ensure that CME activities are free from the control of any commercial interest. All authors, reviewers, and contributors have disclosed to the College all relevant financial relationships with any commercial interests. The authors, reviewers, and contributors declare that neither they nor any business associate nor any member of their immediate families has financial interest or other relationships with any manufacturer of products or any providers of services discussed in this program. Any conflicts have been resolved through group and outside review of all content. Before submitting this form, please print a completed copy as confirmation of your program participation. ACOG Fellows: To obtain credits, complete and return this form by clicking on “Submit” at the bottom of the page. Credit will be automatically recorded upon receipt and online transcripts will be updated twice monthly. -
Molecular Characterization of Enterotoxigenic Escherichia Coli
iolog ter y & c P a a B r f a o s i l t o a l n o r Yameen et al., J Bacteriol Parasitol 2018, 9:3 g u y o J Bacteriology and Parasitology DOI: 10.4172/2155-9597.1000339 ISSN: 2155-9597 Research Article Open Access Molecular Characterization of Enterotoxigenic Escherichia coli: Effect on Intestinal Nitric Oxide in Diarrheal Disease Muhammad Arfat Yameen1, Ebuka Elijah David2*, Humphrey Chukwuemeka Nzelibe3, Muhammad Nasir Shuaibu3, Rabiu Abdussalam Magaji4, Amakaeze Jude Odugu5 and Ogamdi Sunday Onwe6 1Department of Pharmacy, COMSATS Institute of Information Technology, Abbottabad, Pakistan 2Department of Biochemistry, Federal University, Ndufu-Alike, Ikwo, Ebonyi State, Nigeria 3Department of Biochemistry, Ahmadu Bello University, Zaria, Kaduna State, Nigeria 4Department of Human Physiology, Ahmadu Bello University, Zaria, Kaduna State, Nigeria 5Medical Laboratory, Ahmadu Bello University, Teaching Hospital, Zaria, Kaduna State, Nigeria 6Laboratory Service Unit, Federal Teaching Hospital, Abakiliki, Ebonyi State, Nigeria *Corresponding author: Ebuka Elijah David, Department of Biochemistry, Federal University, Ndufu-Alike, Ikwo, Ebonyi State, Nigeria, Tel: +2348033188823; E-mail: [email protected] Received date: May 01, 2018; Accepted date: May 25, 2018; Published date: May 30, 2018 Copyright: ©2018 Yameen MA, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Abstract This study was aimed to investigate the effect of enterotoxigenic E. coli (ETEC)-induced diarrhea on fecal nitric oxide (NO) and intestinal inducible nitric oxide synthase (iNOS) expression in rats. -
The Global View of Campylobacteriosis
FOOD SAFETY THE GLOBAL VIEW OF CAMPYLOBACTERIOSIS REPORT OF AN EXPERT CONSULTATION UTRECHT, NETHERLANDS, 9-11 JULY 2012 THE GLOBAL VIEW OF CAMPYLOBACTERIOSIS IN COLLABORATION WITH Food and Agriculture of the United Nations THE GLOBAL VIEW OF CAMPYLOBACTERIOSIS REPORT OF EXPERT CONSULTATION UTRECHT, NETHERLANDS, 9-11 JULY 2012 IN COLLABORATION WITH Food and Agriculture of the United Nations The global view of campylobacteriosis: report of an expert consultation, Utrecht, Netherlands, 9-11 July 2012. 1. Campylobacter. 2. Campylobacter infections – epidemiology. 3. Campylobacter infections – prevention and control. 4. Cost of illness I.World Health Organization. II.Food and Agriculture Organization of the United Nations. III.World Organisation for Animal Health. ISBN 978 92 4 156460 1 _____________________________________________________ (NLM classification: WF 220) © World Health Organization 2013 All rights reserved. Publications of the World Health Organization are available on the WHO web site (www.who.int) or can be purchased from WHO Press, World Health Organization, 20 Avenue Appia, 1211 Geneva 27, Switzerland (tel.: +41 22 791 3264; fax: +41 22 791 4857; e-mail: [email protected]). Requests for permission to reproduce or translate WHO publications –whether for sale or for non-commercial distribution– should be addressed to WHO Press through the WHO web site (www.who.int/about/licensing/copyright_form/en/index. html). The designations employed and the presentation of the material in this publication do not imply the expression of any opinion whatsoever on the part of the World Health Organization concerning the legal status of any country, territory, city or area or of its authorities, or concerning the delimitation of its frontiers or boundaries. -
Unusual Presentation of Shigellosis: Acute Perforated Appendicitis And
Case Report 45 Unusual Presentation of Shigellosis: Acute Perforated Appendicitis and Peritonitis Gülsüm İclal Bayhan1, Gönül Tanır1, Haşim Ata Maden2, Şengül Özkan3 1Pediatric Infection Clinic, Dr. Sami Ulus Gynecology, Child Care and Treatment Training and Research Hospital, Ankara, Turkey 2Department of Pediatric Surgery, Dr. Sami Ulus Gynecology, Child Care and Treatment Training and Research Hospital, Ankara, Turkey 3Microbiology Clinic. Dr. Sami Ulus Gynecology, Child Care and Treatment Training and Research Hospital, Ankara, Turkey Abstract Shigella spp. is one of the most common agents that cause bacterial diarrhea and dysentery in developing coun- tries. Clinical presentation of shigellosis may vary over a wide spectrum from mild diarrhea to severe dysentery. We report the case of 5.5-year-old previously healthy boy, who presented to our clinic with abdominal pain, vomiting, and constipation. On examination, we noticed abdominal tenderness with guarding at the right lower quadrant. With the diagnosis of acute appendicitis, open appendectomy was performed. Exploration of the abdominal cavity revealed perforated appendicitis and generalized peritonitis. Shigella sonnei was isolated from the peritoneal fluid culture. The patient completely recovered without any complications. Surgical complications, including appendicitis, could have developed during shigellosis. There are few reported cases of perforated appendicitis associated with Shigella. Prompt surgical intervention can be beneficial to prevent morbidity and mortality if it is performed early in the course of the disease. (J Pediatr Inf 2015; 9: 45-8) Keywords: Shigella spp., acute appendicitis, peritonitis, surgical complication Introduction intestinal and extra-intestinal complications. There are few reported cases of perforated Shigella spp., a group of Gram-negative, appendicitis complicated with peritonitis due to Received: 04.10.2013 Accepted: 03.02.2014 small, non-motile, non-spore forming, and rod- Shigella spp. -
Bacillary Dysentery
Bacillary dysentery by Sudhir Chandra Pal uring the first half of 1984, a infectious dose. It requires only 10 to drome and leukaemoid reactions were severe epidemic of bacillary 100 shigella bacteria to produce dys also reported. dysentery swept through the entery, whereas one million to ten Similar epidemics due to the districts of West Bengal and a few million germs may need to be swal multiple-drug-resistant S. shigae have other eastern Indian States, affecting lowed to cause cholera. also occurred in Somalia (1976), three over 350,000 people and leaving By 1920, dysentery due to the most villages in South India (1976), Sri about 3,500, mostly children, dead. It virulent variety, the Shiga bacillus, Lanka (1978-80), Central Africa was like a nightmare as the disease had almost disappeared from Europe (1980-82), Eastern India, Nepal, Bhu stubbornly refused to respond to con and North America. However, it con tan and the Maldives (1984) and Bur ventional treatment, and its galloping tinued to be reported from the de ma (1984-85). The pattern was more spread could not be contained by all veloping countries in the form of local or less the same everywhere. The available public health measures. ised outbreaks. During the late sixties, disease spread with terrific speed in People became confused and panicky, Shiga's bacillus reappeared with a big spite of all available public health not knowing what to do. bang as the main culprit of a series of measures, attacking over 10 per cent Bacillary dysentery, characterised devastating epidemics of dysentery of the population and killing between by frequent passage of blood and in a number of countries in Latin two and 10 per cent even of the mucus in the stools accompanied by America, Asia and Africa.