Discharge Advice After Atrial Fibrillation Ablation
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Incidence of Post Cross Clamp Ventricular Fibrillation in Isolated Coronary Artery Bypass Surgery Using Del Nido Cardioplegia and Conventional Blood Cardioplegia
Jemds.com Original Research Article Incidence of Post Cross Clamp Ventricular Fibrillation in Isolated Coronary Artery Bypass Surgery Using del Nido Cardioplegia and Conventional Blood Cardioplegia Biju Kambil Thyagarajan1, Anandakuttan Sreenivasan2, Ravikrishnan Jayakumar3, Ratish Radhakrishnan4 1, 2, 3, 4 Department of Cardiovascular and Thoracic Surgery, Government T D Medical College, Alappuzha, Kerala, India. ABSTRACT BACKGROUND The cardiac surgical procedures and surgical outcomes witnessed a dramatic Corresponding Author: improvement with the introduction of cardiopulmonary bypass and cardioplegia Dr. Anandakuttan Sreenivasan, techniques. Ventricular fibrillation immediately after removal of aortic cross clamp is Department of Cardiovascular and an energy consuming process leading to myocardial injury in an already energy Thoracic Surgery, Government T D Medical College, Alappuzha, Kerala, India, depleted heart. Electrical cardioversion, which itself causes myocardial injury, is E-mail: [email protected] required to regain normal rhythm. Prevention of ventricular fibrillation is important in preventing myocardial injury. We retrospectively analysed the incidence of post DOI: 10.14260/jemds/2020/797 cross clamp ventricular fibrillation requiring electrical defibrillation in isolated coronary artery bypass surgery using del Nido cardioplegia and conventional blood How to Cite This Article: cardioplegia. Thyagarajan BK, Sreenivasan A, Jayakumar R, et al. Incidence of post cross clamp METHODS ventricular fibrillation in isolated coronary -
Cardiac Arrhythmia and Catheter Ablation UK
Media backgrounder Cardiac Arrhythmia and Catheter Ablation Technique Cardiac arrhythmia – when the heart falls out of rhythm Cardiac arrhythmias (CA) represent a group of conditions with an abnormal heart rhythm or heart rate. This may involve the heart beating too fast (over 100 bpm), too slow (less than 60 bpm) or irregularly.1 Arrhythmias are often caused by problems with the electrical system that regulates the steady heartbeat and can originate in the lower chambers of the heart (ventricles) or in the upper chambers (atria).2 There are various types of arrhythmia; the most common forms include atrial fibrillation (AF), atrial flutter and atrioventricular nodal re-entrant tachycardia. Noticeable warning symptoms include fluttering in the chest, shortness of breath, fatigue, chest pain, dizziness or fainting.3 Many people experience irregular heartbeats at some point in their lives. Most of the time they are harmless, especially when not associated with other heart conditions. However, some arrhythmias can be serious and even life-threatening if left untreated. For example, atrial fibrillation is the most common cardiac arrhythmia and an independent contributor to mortality, morbidity and impaired quality of life.4,5 According to data from the long-term, ongoing Framingham heart study, AF is associated with a 1.5- to 1.9-fold higher risk of death. This is in part attributed to the strong association between AF and thromboembolic events (where the blood in the heart can clot).6 Overall, AF is estimated to be responsible for approximately 15 percent of all strokes7,8,9 and 20 percent of all ischemic strokes.10 Cardiac arrhythmia – a growing global burden • Anyone can develop arrhythmias, even young adults without previous heart problems. -
Radiofrequency Catheter Ablation of Persistent Atrial Fibrillation with Myotonic Dystrophy and Achalasia-Like Esophageal Dilatation Bong Joon Kim
CASE REPORTS International Journal of Arrhythmia 2017;18(4):205-208 doi: http://dx.doi.org/10.18501/arrhythmia.2017.031 Radiofrequency Catheter Ablation of Persistent Atrial Fibrillation with Myotonic Dystrophy and Achalasia-like Esophageal Dilatation Bong Joon Kim Bong-Joon Kim, MD; Tae-Joon Cha, ABSTRACT MD, PhD, FHRS Myotonic dystrophy, a multi-systemic disease with cardiac involve- Division of Cardiology, Kosin University Gospel ment, is the most common inherited neuromuscular disease. Here, Hospital, Busan, Republic of Korea we report the results of radiofrequency catheter ablation of persis- tent atrial fibrillation in a patient with myotonic dystrophy and acha- Received: September 25, 2017 Accepted: September 29, 2017 lasia-like esophageal dilatation. Correspondence: Tae-Joon Cha, MD, PhD, FHRS Division of Cardiology, Kosin University Gospel Hospital, 262, Gamchen-Ro, Seo-Gu, Busan 49267, Republic of Korea Tel: +82-51-990-6105, Fax: +82-51-990-3047 E-mail: [email protected] Copyright © 2017 The Official Journal of Korean Heart Rhythm Society Editorial Board and EUM & Communications Key Words: ■Atrial Fibrillation ■Myotonic Dystrophy Introduction Case Myotonic dystrophy (dystrophia myotonica [DM]) is an On March 7, 2009, a 46-year-old man presented with autosomal-dominant, multi-systemic disease for which the clinical palpitation that had persisted for several years. He had undergone presentation includes myotonia, muscular dystrophy, cardiac DC cardioversion for AF 8 months prior. He did not have a involvement, posterior iridescent cataracts, and endocrine history of hypertension, diabetes mellitus, dyslipidemia, or disorders.1 The incidence of myotonic dystrophy type 1 (DM1) ischemic heart disease; however, he had a stroke 4 years prior. -
CT Coronary Angiography for Detection of Coronary Artery Obstruction, Without the Need for Further Imaging Studies Or Additional Radiation Exposure
IAEA Department of Technical Cooperation And Nuclear Medicine Section RAS 6/063 Strengthening the Application of Nuclear Medicine in the Management of Cardiovascular Diseases Cardiac Imaging CT and MR Prof Lin Tun Tun Head of Department of Radiology University of Medicine (1) Yangon General Hospital CARDIAC CT Advances in CT Technology • Increased image quality • Improvements in hardware and software such as refined image reconstruction methods. • lower radiation exposure of cardiac CT especially for coronary CTA INDICATIONS FOR CARDIAC CT • Chest pain with intermediate pretest probability of CAD • Acute coronary syndrome with intermediate pretest probability of CAD (no ECG changes and negative serial enzymes negative) Evaluation of bypass grafts and coronary anatomy • Evaluation of complex congenital heart disease (anomalies of coronary circulation, great vessels, and cardiac chambers and valves) • Evaluation of cardiac masses or pericardial conditions • Evaluation of pulmonary vein anatomy before radiofrequency ablation, coronary vein • Evaluation of suspected aortic dissection, aortic aneurysm, or pulmonary embolism. History • EBCT – mid 1990 – 1.5 to 3mm slice thickness • 4 MSCT -2000- 1mm s thickness (30 heart beats minimum over all image-acquisition time) • 4-16-64 MSCT - 2004 -0.5 to 0.75 mm (4-8 heart beats) EBCT- Electron Beam CT Evolution of CT - 40 years ‘72. …85…… 89…..91 …….95 ……98.99…01..02..04…05…2006----2012 1st CT Slip Ring Spiral Twin ub sec ½ sec 64 DSCT 256 / ……640/ CT detector MSCT /FPD DSCT/DECT 0.33sec/ritation Multidetector, Multisource CT New detector technologies Data Handling, Dose management Dual source CT 2005- Dual Source CT 2 X-ray sources and 2 detectors Faster than every beating heart DSCT became available around the year 2005, with 2 64 simultaneously acquired slices and a rotation time of 33 milliseconds and More recently, with 2 128 simultaneously acquired slices and a gantry rotation time of 0.28 seconds. -
Pulmonary Vein Isolation Induces Changes in Vectorcardiogram P-Wave Loops
Pulmonary Vein Isolation Induces Changes in Vectorcardiogram P-wave Loops Nuria Ortigosa1, Oscar´ Cano2, Frida Sandberg3 1 I.U. Matematica´ Pura y Aplicada, Universitat Politecnica` de Valencia,` Spain 2 Servicio de Cardiolog´ıa, Hospital Universitari i Politecnic` La Fe. Valencia, Spain 3 Department of Biomedical Engineering, Lund University, Sweden Abstract ful AF ablation has been the durable PVI [6] and it is there- fore desirable to study changes induced by the procedure Pulmonary vein isolation (PVI) is considered a stan- that may be linked to durability of the PVI. dard treatment of paroxysmal atrial fibrillation aiming to Previous studies have analysed how PVI changes the restore and maintain sinus rhythm. The purpose of this surface ECG signal by decreasing the P-wave duration [7, study is to analyse how the PVI treatment affects the elec- 8], and P-wave amplitude and duration dispersion [9–11]. trical conduction pattern in the atria. We have compared In this study we propose to analyse the vectorcardio- the morphology of P wave loops extracted from the vector- gram (VCG) to assess how PVI affects the electrical con- cardiogram (VCG) before and after PVI. duction pattern in the atria. Based on the 12-lead surface Ten patients suffering from paroxysmal atrial fibrillation ECG of patients with paroxysmal AF in sinus rhythm, we who underwent PVI were included in the study. All patients have extracted the VCG, and then compared the morphol- were in sinus rhythm before as well as after the procedure. ogy of P-wave loops before and after PVI to reveal changes Vectorcardiogram was obtained using the Kors matrix and induced by the procedure. -
Flecainide Considerations For
Flecainide (Tambocor) Considerations for Use* US/FDA Approved Indications: Heart Rhythm Control for Atrial Fibrillation Black Box Warning* Proarrhythmic. Increased mortality in patients with non-life-threatening ventricular arrhythmias, structural heart disease (ie, MI, LV dysfunction); not recommended for use with chronic atrial fibrillation. Mechanism of Action Depresses phase 0 depolarization significantly, slows cardiac conduction significantly (Class 1C). Dosing† Cardioversion: 200 to 300 mg PO‡1 Maintenance: 50 to 150 mg PO every 12 hrs Hepatic Impairment: Reduce initial dosage. Monitor serum level frequently. Allow at least 4 days after dose changes to reach steady state level before adjusting dosage. Renal Impairment: CrCl > 35 ml/min: No dosage adjustment is required. CrCl <= 35 ml/min: Initially, 100 mg PO once daily or 50 mg PO twice daily. Adjust dosage at intervals > 4 days, since steady-state conditions may take longer to achieve in these patient Contraindications cardiogenic shock sick sinus syndrome or significant conduction delay 2nd/3rd degree heart block or bundle brand block without pacemaker acquired/congenital QT prolongation patients with history of torsade de pointes Major Side Effects hypotension, atrial flutter with high ventricular rate, ventricular tachycardia, HF Dosage forms and Strengths PO: 50, 100, 150mg tablets Special Notes Close monitoring of this drug is required. When starting a patient on flecainide, it is prudent to do a treadmill stress test after the patient is fully loaded.4 Do not use in patients with ischemic heart disease or LV dysfunction; increases risk of arrhythmias. Additional AV nodal blocking agent may be required to maintain rate control when AF recurs. -
Resuscitation and Defibrillation
AARC GUIDELINE: RESUSCITATION AND DEFIBRILLATION AARC Clinical Practice Guideline Resuscitation and Defibrillation in the Health Care Setting— 2004 Revision & Update RAD 1.0 PROCEDURE: signs, level of consciousness, and blood gas val- Recognition of signs suggesting the possibility ues—included in those conditions are or the presence of cardiopulmonary arrest, initia- 4.1 Airway obstruction—partial or complete tion of resuscitation, and therapeutic use of de- 4.2 Acute myocardial infarction with cardio- fibrillation in adults. dynamic instability 4.3 Life-threatening dysrhythmias RAD 2.0 DESCRIPTION/DEFINITION: 4.4 Hypovolemic shock Resuscitation in the health care setting for the 4.5 Severe infections purpose of this guideline encompasses all care 4.6 Spinal cord or head injury necessary to deal with sudden and often life- 4.7 Drug overdose threatening events affecting the cardiopul- 4.8 Pulmonary edema monary system, and involves the identification, 4.9 Anaphylaxis assessment, and treatment of patients in danger 4.10 Pulmonary embolus of or in frank arrest, including the high-risk de- 4.11 Smoke inhalation livery patient. This includes (1) alerting the re- 4.12 Defibrillation is indicated when cardiac suscitation team and the managing physician; (2) arrest results in or is due to ventricular fibril- using adjunctive equipment and special tech- lation.1-5 niques for establishing, maintaining, and moni- 4.13 Pulseless ventricular tachycardia toring effective ventilation and circulation; (3) monitoring the electrocardiograph and recogniz- -
Atrial Fibrillation Ablation
Atrial Fibrillation Ablation Atrial Fibrillation Ablation This handout will help you learn about atrial fibrillation ablation, also called pulmonary vein isolation. © Hamilton Health Sciences, 2008 PD 6062 – 02/2012 dpc/pted/LrgBk/AtrialFibrillationAblation-trh.doc dt/February 27, 2012 2 15 Atrial Fibrillation Ablation Atrial Fibrillation Ablation How does the heart work? Notes: To understand atrial fibrillation, you need to know how the heart’s electrical system works. __________________________________________________________ The sinoatrial node (SA node) is a natural pacemaker. It starts the __________________________________________________________ electrical signal that travels across the upper 2 chambers or atria of the heart to the atrioventricular node (AV node). __________________________________________________________ The AV node transfers the electrical signal from the upper part of the heart to the lower 2 pumping chambers or ventricles. The bundle branches are __________________________________________________________ specialized tissue that help send electrical impulses through the ventricles. This makes a normal heart beat, called normal sinus rhythm. __________________________________________________________ __________________________________________________________ __________________________________________________________ SA node __________________________________________________________ Bundle branches __________________________________________________________ AV node __________________________________________________________ -
MANAGING ATRIAL FIBRILLATION (AF) (MODULE 2) MODULE 2: MANAGING ATRIAL FIBRILLATION Ii CONTENTS
YOUR COMPLETE GUIDE TO ATRIAL FIBRILLATION MANAGING ATRIAL FIBRILLATION (AF) (MODULE 2) MODULE 2: MANAGING ATRIAL FIBRILLATION ii CONTENTS iii Overview of managing AF 28 How are blood thinners used to reduce the risk of stroke in AF? 1 What are the goals of managing AF and atrial flutter? 31 What are the signs of a stroke? 2 Rate Control Strategy: 32 Being realistic when managing AF How is the heart rate controlled? (a) Medicine (b) Procedures 9 Rhythm Control Strategy: How is the heart rhythm controlled? (a) Medicine (b) Procedures 25 Reducing the risk of stroke: How is my stroke risk assessed? HEARTANDSTROKE.CA/AFGUIDE Published: February 2014 © 2014 Canadian Cardiovascular Society and Heart and Stroke Foundation of Canada. All rights reserved. Unauthorized use prohibited. MODULE 2: MANAGING ATRIAL FIBRILLATION iii OVERVIEW OF MANAGING AF AF Diagnosed MODULE 1 Find and Treat Common Causes What is it? Is it harmful? MODULE 2 Manage Arrhythmia Symptoms Assess Stroke Risk (CHADS2) Rate Control Rhythm Control • Medicine • Medicine Blood Thinner, Aspirin, or Nothing • Procedures • Procedures MODULE 3 Living Well with AF What to Expect Understanding Following a from Your AF Common Responses Patient Stories Healthy Lifestyle Management Plan and Finding Support FACT SHEET: OVERVIEW OF MANAGING AF HEARTANDSTROKE.CA/AFGUIDE Published: February 2014 © 2014 Canadian Cardiovascular Society and Heart and Stroke Foundation of Canada. All rights reserved. Unauthorized use prohibited. MODULE 2: MANAGING ATRIAL FIBRILLATION iv WHAT ARE TWO WAYS TO MANAGE AF? While AF is a chronic condition, it can be managed by: • medicine • procedures Medicine is usually tried first to manage symptoms caused by AF. -
Cardiac Ablation: Types and Outcomes
CARDIAC ABLATION: TYPES AND OUTCOMES CARDIOVERSION AND ABLATION JASSIN M. JOURIA Dr. Jassin M. Jouria is a practicing Emergency Medicine physician, professor of academic medicine, and medical author. He graduated from Ross University School of Medicine and has completed his clinical clerkship training in various teaching hospitals throughout New York, including King’s County Hospital Center and Brookdale Medical Center, among others. Dr. Jouria has passed all USMLE medical board exams, and has served as a test prep tutor and instructor for Kaplan. He has developed several medical courses and curricula for a variety of educational institutions. Dr. Jouria has also served on multiple levels in the academic field including faculty member and Department Chair. Dr. Jouria continues to serve as a Subject Matter Expert for several continuing education organizations covering multiple basic medical sciences. He has also developed several continuing medical education courses covering various topics in clinical medicine. Recently, Dr. Jouria has been contracted by the University of Miami/Jackson Memorial Hospital’s Department of Surgery to develop an e-module training series for trauma patient management. Dr. Jouria is currently authoring an academic textbook on Human Anatomy & Physiology. ABSTRACT Atrial arrhythmias are serious disorders that can cause an irregular and/or rapid heartbeat, which can lead to serious clinical sequelae. Atrial fibrillation is an example of an atrial arrhythmia that can lead to blood clots, stroke, or heart failure. Electrical cardioversion and ablation are two procedures that can minimize these risks and treat atrial arrhythmia. Each of these treatments have risks and neither offers a complete success rate, but they can be very effective in providing greater quality of life, and extending the life expectancy of patients. -
Stress, Protocols, and Tracers
ASNC IMAGING GUIDELINES ASNC imaging guidelines for SPECT nuclear cardiology procedures: Stress, protocols, and tracers a b c Milena J. Henzlova, MD, W. Lane Duvall, MD, Andrew J. Einstein, MD, d e Mark I. Travin, MD, and Hein J. Verberne, MD a Mount Sinai Medical Center, New York, NY b Hartford Hospital, Hartford, CT c New York Presbyterian Hospital, Columbia University Medical Center, New York, NY d Montefiore Medical Center, Albert Einstein College of Medicine, Bronx, NY e Academic Medical Center, Amsterdam, The Netherlands doi:10.1007/s12350-015-0387-x Abbreviations LEHR Low energy high resolution A2A Adenosine 2a LVEF Left ventricular ejection fraction AHA American Heart Association MBq Megabecquerels ALARA As low as reasonably achievable mCi Millicuries AV Atrioventricular MPI Myocardial perfusion imaging BP Blood pressure MRI Magnetic resonance imaging CBF Coronary blood flow mSv Millisievert CAD Coronary artery disease NE Norepinephrine CPET Cardiopulmonary exercise testing NET1 Norepinephrine transporter-1 DSP Deconvolution of septal penetration NPO Nil per os (nothing by mouth) ECG Electrocardiogram NYHA New York Heart Association EF Ejection fraction PET Positron emission tomography ESRD End-stage renal disease ROI Region of interest HF Heart failure SPECT Single-photon emission computed HFrEF Heart failure (with) reduced ejection tomography fraction TAVR Transcatheter aortic valve replacement HMR Heart-to-mediastinum ratio WR Washout rate ICD Implantable cardioversion defibrillator WPW Wolff-Parkinson White IV Intravenous -
Cardiopulmonary Resuscitation in Prone Position
International Journal of Cardiovascular Sciences. 2021; 34(3):315-318 315 REVIEW ARTICLE Cardiopulmonary Resuscitation in Prone Position Leandro Menezes Alves da Costa,1 Rafael Amorim Belo Nunes,1,2 Thiago Luis Scudeler3 Hospital Alemão Oswaldo Cruz,1 de São Paulo, SP - Brazil Hospital das Clínicas da Faculdade de Medicina da Universidade de São Paulo,2 São Paulo, SP - Brazil Instituto do Coração (InCor),3 São Paulo, SP - Brazil Abstract effectively reduce the difference between transpulmonary dorsal and ventral pressures. In addition, PPV reduces Mechanical ventilation in prone position is an pulmonary compression and improves corporeal alternative strategy for patients with acute respiratory perfusion.6,7 Prone ventilation should be initiated in the discomfort syndrome (ARDS) to improve oxygenation first 36 hours of mechanical ventilation and ideally be in situations when traditional ventilation modalities maintained for 12 to 16 hours.4 have failed. However, due to the significant increase During this period of treatment, the patient may become in ARDS cases during the SARS-CoV-2 pandemic vulnerable to emergency situations, with cardiac arrest and the experimental therapeutic use of potentially as the diagnosis with the worst prognosis. Moreover, arrhythmogenic drugs, cardiopulmonary resuscitation the current use of experimental drugs may increase the in this unusual position could be needed. Therefore, occurrence of malignant arrhythmia. The combination of we will review the available scientific evidence of hydroxychloroquine and azithromycin