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Pediatric Chest Tubes and Pigtails
November 2015 Pediatric Chest Tubes And Volume 12, Number 11 Authors Pigtails: An Evidence-Based Jonathan Strutt, MD Pediatric Emergency Department, Children’s Hospital and Clinics of Minnesota, Minneapolis, MN Approach To The Management Anupam Kharbanda, MD, MSc Research Director, Associate Fellowship Director, Department of Pediatric Emergency Medicine, Children's Hospitals and Clinics of Of Pleural Space Diseases Minnesota, Minneapolis, MN Peer Reviewers Abstract Jennifer Mitzman, MD Assistant Professor of Emergency Medicine, The Ohio State Pediatric thoracostomy procedures are used in the emergency depart- University Wexner Medical Center; Assistant Professor of Pediatrics, Nationwide Children’s Hospital, Columbus, OH ment to treat diseases of the pleural space. As children have unique Emily Rose, MD, FAAP, FAAEM, FACEP thoracic anatomy and physiology, they may present with manage- Assistant Professor of Clinical Emergency Medicine, Keck School of ment challenges that the emergency clinician must consider. This Medicine of the University of Southern California, LA County + USC Medical Center, Los Angeles, CA issue reviews the use of chest tubes and pigtail catheters in pediatric CME Objectives patients, techniques and indications for placement, and possible complications. Diagnostic and treatment options for diseases of the Upon completion of this article, you should be able to: 1. Diagnose pleural space disease based on signs and pleural space, such as spontaneous pneumothorax, traumatic injury, symptoms. and parapneumonic effusions/empyema, are examined. Addition- 2. Choose the most effective imaging studies to aid in diagnosis. 3. Determine the types of procedural interventions necessary in ally, this issue discusses the use of imaging modalities to aid in the pleural space disease and when they should be performed. -
Caring for Yourself After Surgery: Preventing Surgical Site Infections
CARE AT HOME SERIES CARE AT HOME SERIES Caring for Yourself After Surgery Caring for Yourself After Surgery Preventing Surgical Site Infections Preventing Surgical Site Infections Wounds Canada has developed this simple guide that can be used by patients and their care partners when they are looking after a surgical wound. It provides guidance on things to do before and after surgery to help prevent infections and recognize the signs of infections if they do occur. In the past, people stayed in hospital for days or weeks following surgery. In those days, many of the complications that can occur soon after surgery (like infections, heart problems or bleeding) were treated when patients were still recovering in hospital. Today, patients having surgery get home Uninfected, healed surgical incision site. (See page 4 for faster than ever, often even the same day. Surgical uninfected and infected surgical incision sites.) site infections (SSIs) that were once treated in hospital are now being managed by patients at home. The good news is that there are actions you can take before and after your surgery to reduce the chances of developing a serious SSI. What is a surgical site infection? A surgical site infection is a problem where there are too many bacteria or really dangerous, active bacteria in your surgical incision. SSIs cause pain and delay wound healing. In more severe cases, SSIs can spread into the bloodstream (a condition called sepsis), which can lead to tissue loss, organ failure and death. A surgical site infection can be on the surface or deep. How much damage it does depends on how healthy you are as well as how strongly the bacteria affect your tissues. -
Causes of Surgical Wound Dehiscence: a Multicenter Study
J Wound Manag Res 2018 September;14(2):74-79 pISSN 2586-0402 https://doi.org/10.22467/jwmr.2018.00374 eISSN 2586-0410 Journal of Wound Management and Research Causes of Surgical Wound Dehiscence: A Multicenter Study Jeong Jin Chun1, Seok Min Yoon1, Woo Jin Song1, Hyun Gyo Jeong1, Chang Yong Choi2, Syeo Young Wee1 1Department of Plastic and Reconstructive Surgery, College of Medicine, Soonchunhyang University, Gumi; 2Department of Plastic and Reconstructive Surgery, College of Medicine, Soonchunhyang University, Bucheon, Korea Abstract Surgical wound dehiscence is a postoperative complication involving breakdown of surgical incision site. Despite the in- creased knowledge of wound healing mechanism before and after surgery, wound dehiscence may increase the length of hospital stay, increase patient inconvenience and rates of re-operation. The purpose of this study was to analyze the causes of wound dehiscence in patients undergoing reoperation at 4 hospitals of Soonchunhyang Medical Center. The number of patients in each hospital and those operated previously were compared. In addition, other characteristics of patients were compared in patients who underwent reoperation. In 22 out of 1,026 patients consulted at the Seoul hos- pital, 32 cases out of 1,295 at Bucheon hospital, 14 cases out of 1,687 at Cheonan hospital and 15 cases out of 374 at Gumi hospital, wound revision was performed for wound dehiscence. Patients at the Department of Obstetrics and Gy- necology were the most common and included 33 patients (39.8%). The most common intervention before wound revi- sion was Cesarean section in 14 patients (19.3%). In this study, we retrospectively reviewed patients who underwent wound revision due to wound dehiscence and analyzed the underlying causes of the postoperative complication. -
VII. Wound and Fracture Healing
Journal of Rehabilitation Research and Development Rehabilitation R & D Progress Reports 1986 VII. Wound and Fracture Healing VII . Wound and Fracture Healing Electrical Stimulation for Augmentation of Wound Healing Scott R. Crowgey, M.D., and Steven M. Sharpe Veterans Administration Research and Development, Decatur, GA 30033 Sponsor: VA Rehabilitation Research and Development Service Purpose—This project will attempt to identify ing that could be influenced by electrical stimu- aspects of the wound healing process that may lation. Efforts will then be directed toward de- be augmented by the exogenous influence of veloping mathematical models of the possible electromagnetic fields. A theoretical analysis of electrical interaction of electromagnetic fields the possible effects of electromagnetic fields on with cells and cell structures to determine how wound healing will include analyses of the these interactions could be optimized to im- interaction of electromagnetic fields with cellu- prove wound healing. It is anticipated that the lar structures and of the deposition of heat in literature will not contain all the information damaged tissue via exogenously applied energy necessary to develop these models . Any gaps in fields. This analysis will then be used as a basis necessary information and data will be filled, if for developing a plan for future investigations practical, using tissue phantom modeling mate- into the potential application of electrical stim- rials, blood, and possibly even primitive tissue ulation for the augmentation of wound healing. culture exposed to a variety of known electro- The initial research will involve a review of magnetic environments, using easily construct- the literature to identify aspects of wound heal- ed exposure chambers. -
Wound Care: the Basics
Wound Care: The Basics Suzann Williams-Rosenthal, RN, MSN, WOC, GNP Norma Branham, RN, MSN, WOC, GNP University of Virginia May, 2010 What Type of Wound is it? How long has it been there? Acute-generally heal in a couple weeks, but can become chronic: Surgical Trauma Chronic -do not heal by normal repair process-takes weeks to months: Vascular-venous stasis, arterial ulcers Pressure ulcers Diabetic foot ulcers (neuropathic) Chronic Wounds Pressure Ulcer Staging Where is it? Where is it located? Use anatomical location-heel, ankle, sacrum, coccyx, etc. Measurements-in centimeters Length X Width X Depth • Length = greatest length (head to toe) • Width = greatest width (side to side) • Depth = measure by marking the depth with a Q- Tip and then hold to a ruler Wound Characteristics: Describe by percentage of each type of tissue: Granulation tissue: • red, cobblestone appearance (healing, filling in) Necrotic: • Slough-yellow, tan dead tissue (devitalized) • Eschar-black/brown necrotic tissue, can be hard or soft Evaluating additional tissue damage: Undermining Separation of tissue from the surface under the edge of the wound • Describe by clock face with patients head at 12 (“undermining is 1 cm from 12 to 4 o’clock”) Tunneling Channel that runs from the wound edge through to other tissue • “tunneling at 9 o’clock, measuring 3 cm long” Wound Drainage and Odor Exudate Fluid from wound • Document the amount, type and odor • Light, moderate, heavy • Drainage can be clear, sanguineous (bloody), serosanguineous (blood-tinged), -
Risk Factors for Surgical Wound Dehiscence by Hazim Ibrahim DPM 1
! The Northern Ohio Foot and Ankle Journal Official Publication of the NOFA Foundation A Literature Review of Causes and Risk Factors for Surgical Wound Dehiscence by Hazim Ibrahim DPM 1 The Northern Ohio Foot and Ankle Journal 4(26): 1-5 Abstract: Postoperative wound healing plays a significant role in facilitating a patient’s recovery and rehabilitation. Surgical wound dehiscence (SWD) impacts mortality and morbidity rates and significantly contributes to prolonged hospital stays and associated psychosocial stressors on individuals and their families. Most common risk factors associated with SWD include obesity and wound infections, particularly in the case of orthopedic surgery. There is limited reporting of variables associated with SWD across other surgical domains and a lack of risk assessment tools. Furthermore, there was a lack of clarity in the definition of SWD in the literature. This review provides an overview of the available research and provides a basis for more rigorous analysis of factors that contribute to SWD. Key words: Dehiscence, wound infection, obesity This is an Open Access article distributed under the terms of the Creative Commons Attribution License. It permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ©The Northern Ohio Foot and Ankle Foundation Journal. (www.nofafoundation.org) 2016. All rights reserved. time. The third week after surgery the durability equals 20% of the initial strength, and after 6-12 Wound dehiscence is one of the most weeks it reaches 70-80% (1). Sutures placed during surgery allow the tissues the necessary time to regain common complications of surgical incision sites. -
Corticosteroids and Wound Healing: Clinical Considerations in the Perioperative Period
The American Journal of Surgery (2013) 206, 410-417 Review Corticosteroids and wound healing: clinical considerations in the perioperative period Audrey S. Wang, M.D.a,*, Ehrin J. Armstrong, M.D., M.Sc.b, April W. Armstrong, M.D., M.P.H.a aDepartment of Dermatology, University of California, Davis, 3301 C Street, Suite 1400, Sacramento, CA 95816; bDepartment of Internal Medicine, Division of Cardiovascular Medicine, University of California, Davis, 4860 Y Street, Suite 2820, Sacramento, CA 95817 KEYWORDS: Abstract Corticosteroids; BACKGROUND: Determining whether systemic corticosteroids impair wound healing is a clinically Wound healing; relevant topic that has important management implications. Perioperative METHODS: We reviewed literature on the effects of corticosteroids on wound healing from animal and human studies searching MEDLINE from 1949 to 2011. RESULTS: Some animal studies show a 30% reduction in wound tensile strength with perioperative corticosteroids at 15 to 40 mg/kg/day. The preponderance of human literature found that high-dose cor- ticosteroid administration for ,10 days has no clinically important effect on wound healing. In patients taking chronic corticosteroids for at least 30 days before surgery, their rates of wound complications may be increased 2 to 5 times compared with those not taking corticosteroids. Complication rates may vary depending on dose and duration of steroid use, comorbidities, and types of surgery. CONCLUSIONS: Acute, high-dose systemic corticosteroid use likely has no clinically significant effect on wound healing, whereas chronic systemic steroids may impair wound healing in susceptible individuals. Ó 2013 Elsevier Inc. All rights reserved. The effects of corticosteroids on wound healing have perioperative corticosteroid administration, namely, dosing, been a topic of great interest among surgeons, internists, and chronicity, and timing relative to surgery. -
Resistant Staphylococcus Aureus Infection in a Diabetic Patient with Femorotibial Vascular Bypass Occlusion
ORIGINALACUTE MEDICAL RESEARCH CARE ClinicalClinical Medicine Medicine 2020 2017 Vol 20, Vol No 17, 1: No 98–100 6: 98–8 Surgical wound dehiscence complicated by methicillin- resistant Staphylococcus aureus infection in a diabetic patient with femorotibial vascular bypass occlusion Authors: Enrico M Zardi,A Nunzio Montelione,B Rossella C Vigliotti,C Camilla Chello,D Domenico M Zardi,E Francesco SpinelliF and Francesco StiloG Diabetic patients with critical limb ischaemia may be affected combined with endarterectomy of the superficial femoral artery. A by severe wound and skin ulcer infections. We report a case of month later he underwent the calcaneal skin ulcers debridement. a patient with bilateral femorotibial occlusion and methicillin- The wound specimen for bacterial culture was positive for resistant Staphylococcus aureus infection. The patient was Staphylococcus haemolyticus, Corynebacterium aurimucosum treated with femoroperoneal vascular bypass, debridement and Corynebacterium simulans, he was treated with intravenous ABSTRACT of wound dehiscence and targeted antimicrobial therapy for teicoplanin at 400 mg once per day and intravenous meropenem symptom resolution and healing of the wound. at 500 mg every 8 hours. In June and July, he was admitted to our hospital for rest pain and persistence of bilateral calcaneal KEYWORDS: Diabetes, infection, skin ulcer, therapy, vascular bypass ulcers; he was treated with left femorotibial posterior bypass and right femorotibial anterior bypass, using the greater saphenous vein. After an additional -
Biology of Bone Repair
Biology of Bone Repair J. Scott Broderick, MD Original Author: Timothy McHenry, MD; March 2004 New Author: J. Scott Broderick, MD; Revised November 2005 Types of Bone • Lamellar Bone – Collagen fibers arranged in parallel layers – Normal adult bone • Woven Bone (non-lamellar) – Randomly oriented collagen fibers – In adults, seen at sites of fracture healing, tendon or ligament attachment and in pathological conditions Lamellar Bone • Cortical bone – Comprised of osteons (Haversian systems) – Osteons communicate with medullary cavity by Volkmann’s canals Picture courtesy Gwen Childs, PhD. Haversian System osteocyte osteon Picture courtesy Gwen Childs, PhD. Haversian Volkmann’s canal canal Lamellar Bone • Cancellous bone (trabecular or spongy bone) – Bony struts (trabeculae) that are oriented in direction of the greatest stress Woven Bone • Coarse with random orientation • Weaker than lamellar bone • Normally remodeled to lamellar bone Figure from Rockwood and Green’s: Fractures in Adults, 4th ed Bone Composition • Cells – Osteocytes – Osteoblasts – Osteoclasts • Extracellular Matrix – Organic (35%) • Collagen (type I) 90% • Osteocalcin, osteonectin, proteoglycans, glycosaminoglycans, lipids (ground substance) – Inorganic (65%) • Primarily hydroxyapatite Ca5(PO4)3(OH)2 Osteoblasts • Derived from mesenchymal stem cells • Line the surface of the bone and produce osteoid • Immediate precursor is fibroblast-like Picture courtesy Gwen Childs, PhD. preosteoblasts Osteocytes • Osteoblasts surrounded by bone matrix – trapped in lacunae • Function -
Spinal Cord Injury Guidelines 2018
SPINAL CORD INJURY GUIDELINES 2018 Department of Physical Medicine and Rehabilitation/Trauma Rehabilitation Resources Program TELE-REHABILITATION GUIDELINE Pressure Ulcers Author(s): Peer Reviewed: Finalized: April 2018 Drafted: February 2014 Date: Published: May 2018 I. Definition, assessment, and diagnosis A. Definition 1. Pressure ulcers are sores caused by ischemia due to elevated or prolonged pressure to the skin and underlying tissue, and occur most often over bony prominences. 2. The injury occurs as a result of intense and/or prolonged pressure or pressure in combination with shear. 3. The tolerance of soft tissue for pressure and shear may also be affected by microclimate, nutrition, perfusion, co-morbidities and condition of the soft tissue. 4. There are numerous grading systems but one widely used system is the National Pressure Ulcer Advisory Panel (NPUAP) pressure ulcer stages. 1 In this system the stage is determined by the depth of the tissue damage observed and is used primarily for initial assessment of a pressure ulcer. a. Stage 1 Pressure Injury: Non-blanchable erythema of intact skin: Intact skin with a localized area of non-blanchable erythema, which may appear differently in darkly pigmented skin. Presence of blanchable erythema or changes in sensation, temperature, or firmness may precede visual changes. Color changes do not include purple or maroon discoloration; these may indicate deep tissue pressure injury. b. Stage 2 Pressure Injury: Partial-thickness skin loss with exposed dermis: Partial-thickness loss of skin with exposed dermis. The wound bed is viable, pink or red, moist, and may also present as an intact or ruptured serum-filled blister. -
Products & Technology Wound Inflammation and the Role of A
Products & technology Wound inflammation and the role of a multifunctional polymeric dressing Temporary inflammation is a normal response in acute wound healing. However, in chronic wounds, the inflammatory phase is dysfunctional in nature. This results in delayed healing, and causes further problems such as increased pain, odour and Intro high levels of exudate production. It is important to choose a dressing that addresses all of these factors while meeting the patient’s needs. Multifunctional polymeric Authors: Keith F Cutting membrane dressings (e.g. PolyMem®, Ferris) can help to simplify this choice and Authors: Peter Vowden assist healthcare professionals in chronic wound care. The unique actions of xxxxx Cornelia Wiegand PolyMem® have been proven to reduce and prevent inflammation, swelling, bruising and pain to promote rapid healing, working in the deep tissues beneath the skin[1,2]. he mechanism of acute wound healing — the vascular and cellular stages. During is a well-described complex cellular vascular response, immediately on injury there is T interaction[3] that can be divided into an initial transient vasoconstriction that can be several integrated processes: haemostasis, measured in seconds. This is promptly followed inflammation, proliferation, epithelialisation by vasodilation under the influence of histamine and tissue remodeling. Inflammation is a key and nitric oxide (NO) that cause an inflow of blood. component of acute wound healing, clearing An increase in vascular permeability promotes damaged extracellular matrix, cells and debris leakage of serous fluid (protein-rich exudate) into from zones of tissue damage. This is normally a the extravascular compartment, which in turn time-limited orchestrated process. Successful increases the concentration of cells and clotting progression of the inflammatory phase allows factors. -
Integumentary Changes and Considerations Impacting People
Integumentary Changes and Considerations Impacting People with Spinal Cord Injury Authors: Eddie Monroy, PT, DPT, CLW, CWS Why should PTs be concerned about skin for Fact Sheet patients with spinal cord injury? Changes to the skin in patients with SCI (listed below), combined with skin changes common due to aging and other co-morbidities, can make the SCI population at a Produced by very high risk for skin breakdown and pressure injury. Patients with sensory and motor complete injuries are more susceptible to breakdown; however people with 4 incomplete injuries can still be at high risk, especially when medically unstable. o Decreased blood flow, supply, pressure o Decreased amino acid concentration o Decreased enzymes for biosynthesis o Potential change in gene expression o Decreased Adrenergic receptors o (norepinephrine, epinephrine) o Decreased proportion of Type I to Type III collagen fibers o Increased collagen catabolism (dermis) o Decreased Partial Pressure of Oxygen (PO2) a Special Interest What types of skin injuries are most common in SCI? Group of Pressure ulcers/injuries are the most common types of wounds for patients with SCI. They form over bony prominences such the sacrum, ischial tuberosities, greater trochanters, and heels secondary to lack of mobility and blood flow to the areas. It’s important to perform manual skin checks (palpation) in addition to visual skin checks, to note a change in tissue quality (induration / boggy); as signs of potential breakdown may show up before visual signs are noted. Teaching staff and caregivers how to perform these manual and visual checks daily is an integral part of the patient’s overall SCI education and future health.