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Births, Marriages, and Deaths
DEC. 31, 1955 MEDICAL NEWS MEDICALBRrsIJOURNAL. 1631 Lead Glazes.-For some years now the pottery industry British Journal of Ophthalmology.-The new issue (Vol. 19, has been forbidden to use any but leadless or "low- No. 12) is now available. The contents include: solubility" glazes, because of the risk of lead poisoning. EXPERIENCE IN CLINIcAL EXAMINATION OP CORNEAL SENsITiVrry. CORNEAL SENSITIVITY AND THE NASO-LACRIMAL REFLEX AFTER RETROBULBAR However, in some teaching establishments raw lead glazes or ANAES rHESIA. Jorn Boberg-Ans. glazes containing a high percentage of soluble lead are still UVEITIS. A CLINICAL AND STATISTICAL SURVEY. George Bennett. INVESTIGATION OF THE CARBONIC ANHYDRASE CONTENT OF THE CORNEA OF used. The Ministry of Education has now issued a memo- THE RABBIT. J. Gloster. randum to local education authorities and school governors HYALURONIDASE IN OCULAR TISSUES. I. SENSITIVE BIOLOGICAL ASSAY FOR SMALL CONCENTRATIONS OF HYALURONIDASE. CT. Mayer. (No. 517, dated November 9, 1955) with the object of INCLUSION BODIES IN TRACHOMA. A. J. Dark. restricting the use of raw lead glazes in such schools. The TETRACYCLINE IN TRACHOMA. L. P. Agarwal and S. R. K. Malik. APPL IANCES: SIMPLE PUPILLOMETER. A. Arnaud Reid. memorandum also includes a list of precautions to be ob- LARGE CONCAVE MIRROR FOR INDIRECT OPHTHALMOSCOPY. H. Neame. served when handling potentially dangerous glazes. Issued monthly; annual subscription £4 4s.; single copy Awards for Research on Ageing.-Candidates wishing to 8s. 6d.; obtainable from the Publishing Manager, B.M.A. House, enter for the 1955-6 Ciba Foundation Awards for research Tavistock Square, London, W.C.1. -
Blood Plasma Bromide Levels in Bromoderma' Lester W
BLOOD PLASMA BROMIDE LEVELS IN BROMODERMA' LESTER W. KIMBERLY, M.D.2 (Received for publication May 16, 1939) The ordinary cutaneous manifestations of bromide eruptions are well known. Numerous reports have appeared in the litera- ture supporting various theories as to the development of cutane- ous lesions due to bromides but practically no one has studied the plasma bromide levels with relation to bromoderma. Hanes and Yates (1) found approximately 0.9 per cent of the total admissions to Duke Hospital had increased amounts of bromide in their plasma. They also found that 28 per cent of 64 patients with blood serum bromides above 200 mgm. had bromoderma. The percentage of patients with significantly elevated plasma bromides is even higher among patients admitted to psychopathic hospitals. Szadek (2) believed that the cutaneous eruption originated from an irritation of the sebaceous glands. Laudenheimer (3) and Von Wyss (4) thought that the ingestion of bromide led to a gradual retention of the drug in the tissues and the replacement of the chloride. Engman and Mook (5) felt that the lesions were more apt to occur at the site of previous inflammation and that trauma might play a part, thereby explaining the predilection of a bromoderma for old seborrheic or acne areas. Wile (6) stated that he was unable to demonstrate the drug in the content of the lesions and he believed that it was not present unless there was an admixture of blood serum. Bloch and Tenchio (7) considered bromoderma to be an idiosyncratic phenome- non of the skin and mucous membranes brought about by sensitization. -
Nonbacterial Pus-Forming Diseases of the Skin Robert Jackson,* M.D., F.R.C.P[C], Ottawa, Ont
Nonbacterial pus-forming diseases of the skin Robert Jackson,* m.d., f.r.c.p[c], Ottawa, Ont. Summary: The formation of pus as a Things are not always what they seem Fungus result of an inflammatory response Phaedrus to a bacterial infection is well known. North American blastomycosis, so- Not so well appreciated, however, The purpose of this article is to clarify called deep mycosis, can present with a is the fact that many other nonbacterial the clinical significance of the forma¬ verrucous proliferating and papilloma- agents such as certain fungi, viruses tion of pus in various skin diseases. tous plaque in which can be seen, par- and parasites may provoke pus Usually the presence of pus in or on formation in the skin. Also heat, the skin indicates a bacterial infection. Table I.Causes of nonbacterial topical applications, systemically However, by no means is this always pus-forming skin diseases administered drugs and some injected true. From a diagnostic and therapeutic Fungus materials can do likewise. Numerous point of view it is important that physi¬ skin diseases of unknown etiology cians be aware of the nonbacterial such as pustular acne vulgaris, causes of pus-forming skin diseases. North American blastomycosis pustular psoriasis and pustular A few definitions are required. Pus dermatitis herpetiformis can have is a yellowish [green]-white, opaque, lymphangitic sporotrichosis bacteriologically sterile pustules. The somewhat viscid matter (S.O.E.D.). Pus- cervicofacial actinomycosis importance of considering nonbacterial forming diseases are those in which Intermediate causes of pus-forming conditions of pus can be seen macroscopicaily. -
Clinical Spectrum of Nail Disorders Derma 2020; 3(2): 48-54 Received: 19-05-2020 Accepted: 22-07-2020 Dr
International Journal of Dermatology, Venereology and Leprosy Sciences. 2020; 3(2): 48-54 E-ISSN: 2664-942X P-ISSN: 2664-9411 www.dermatologypaper.com/ Clinical spectrum of nail disorders Derma 2020; 3(2): 48-54 Received: 19-05-2020 Accepted: 22-07-2020 Dr. Kotha Raghupathi Reddy, Dr. Munnaluri Mohan Rao and Dr. Dr. Kotha Raghupathi Reddy Chittla Sravan Associate Professor, Department of DVL, Gandhi DOI: https://doi.org/10.33545/26649411.2020.v3.i2a.46 Medical College, Secunderabad, Telangana, Abstract India Background: The nail disorders comprise approximately 10% of all dermatological conditions. The nail unit may reflect dermatological disorder by its own and may show specific changes that are Dr. Munnaluri Mohan Rao Associate Professor, markers for a wide range of systemic disorders. Department of DVL, Great Aims: To study the clinical spectrums of nail disorders including congenital, developmental, Eastern Medical School and infectious, neoplastic, degenerative, dermatologic and systemic diseases affecting the nail unit. Hospital, Ragolu Srikakulam, Materials and Methods: 200 consecutive cases with nail disorders attending to the Dermatology. Andhra Pradesh, India Complete dermatologic and systemic examinations were carried out. Hematological investigations, including hemoglobin, total leukocyte count, differential leukocyte count and urine examination were Dr. Chittla Sravan carried out in all patients. Assistant Professor, Results: In this study, the involvement of nail was more common among males when compared to Department of DVL, MNR females. Onychomycosis was the commonest finding (27%) followed by psoriatic nail change (14.5%), Medical College and Hospital, Onycholysis (5.5%), Pitting (4.5%), Onychogryphosis (4.5%), Trachyonychia (4.5%), Chronic Sangareddy, Telangana, India Paronychia (4.5%), Clubbing (4%), Subungual Warts (3.5%), Clubbing with resorption of terminal fingers (2.5%) and Ingrowing toe nail (2%). -
Studies on the Breaking Pattern in Man at Rest and During Sleep
STUDIES ON THE BREAKING PATTERN IN MAN AT REST AND DURING SLEEP by Steven Andrew Shea A thesis submitted to the Faculty of Science, University of London for the degree of Doctor of Philosophy 1988 Department of Medicine, Charing Cross and Westminster Medical School, London. 2 ABSTRACT . This thesis quantifies the breathing pattern and the extent of the reproducibility of this pattern within an individual at rest and during sleep. From breath-by-breath measurements of respiratory frequency, tidal volune and end-tidal POO2 made under standardised conditions of relaxed wakefulness - with a minimum of sensory stimulation - the results show that differences between individuals are highly significantly greater than differences seen on repeated measurements within an individual: people tend to breathe in a reproducible and characteristic fashion. The basic respiratory pattern is shown to have long-term reproducibility for periods of up to 5 years and may be, to some extent, inherited since it is shown to be similar between identical twins. The individual’s ’respiratory personality’ also persists during deep non-rapid eye movement (non- REM) sleep when any forebrain influences upon breathing are minimal. Further studies, using similar techniques, examine the effect upon this basic respiratory pattern of some behavioural, metabolic and pulmonary reflex control mechanisms. These studies reveal that visual, and auditory stimulation, and altered cognitive activity (performing mental arithmetic) affects the pattern of breathing; principally by increasing respiratory frequency. However, these changes in breathing which occur between the different ’states’ are not solely behavioural responses since they are also related to increases in cerebral and/or somatic metabolism. -
Eye Disease 1 Eye Disease
Eye disease 1 Eye disease Eye disease Classification and external resources [1] MeSH D005128 This is a partial list of human eye diseases and disorders. The World Health Organisation publishes a classification of known diseases and injuries called the International Statistical Classification of Diseases and Related Health Problems or ICD-10. This list uses that classification. H00-H59 Diseases of the eye and adnexa H00-H06 Disorders of eyelid, lacrimal system and orbit • (H00.0) Hordeolum ("stye" or "sty") — a bacterial infection of sebaceous glands of eyelashes • (H00.1) Chalazion — a cyst in the eyelid (usually upper eyelid) • (H01.0) Blepharitis — inflammation of eyelids and eyelashes; characterized by white flaky skin near the eyelashes • (H02.0) Entropion and trichiasis • (H02.1) Ectropion • (H02.2) Lagophthalmos • (H02.3) Blepharochalasis • (H02.4) Ptosis • (H02.6) Xanthelasma of eyelid • (H03.0*) Parasitic infestation of eyelid in diseases classified elsewhere • Dermatitis of eyelid due to Demodex species ( B88.0+ ) • Parasitic infestation of eyelid in: • leishmaniasis ( B55.-+ ) • loiasis ( B74.3+ ) • onchocerciasis ( B73+ ) • phthiriasis ( B85.3+ ) • (H03.1*) Involvement of eyelid in other infectious diseases classified elsewhere • Involvement of eyelid in: • herpesviral (herpes simplex) infection ( B00.5+ ) • leprosy ( A30.-+ ) • molluscum contagiosum ( B08.1+ ) • tuberculosis ( A18.4+ ) • yaws ( A66.-+ ) • zoster ( B02.3+ ) • (H03.8*) Involvement of eyelid in other diseases classified elsewhere • Involvement of eyelid in impetigo -
Pseudomonas Skin Infection Clinical Features, Epidemiology, and Management
Am J Clin Dermatol 2011; 12 (3): 157-169 THERAPY IN PRACTICE 1175-0561/11/0003-0157/$49.95/0 ª 2011 Adis Data Information BV. All rights reserved. Pseudomonas Skin Infection Clinical Features, Epidemiology, and Management Douglas C. Wu,1 Wilson W. Chan,2 Andrei I. Metelitsa,1 Loretta Fiorillo1 and Andrew N. Lin1 1 Division of Dermatology, University of Alberta, Edmonton, Alberta, Canada 2 Department of Laboratory Medicine, Medical Microbiology, University of Alberta, Edmonton, Alberta, Canada Contents Abstract........................................................................................................... 158 1. Introduction . 158 1.1 Microbiology . 158 1.2 Pathogenesis . 158 1.3 Epidemiology: The Rise of Pseudomonas aeruginosa ............................................................. 158 2. Cutaneous Manifestations of P. aeruginosa Infection. 159 2.1 Primary P. aeruginosa Infections of the Skin . 159 2.1.1 Green Nail Syndrome. 159 2.1.2 Interdigital Infections . 159 2.1.3 Folliculitis . 159 2.1.4 Infections of the Ear . 160 2.2 P. aeruginosa Bacteremia . 160 2.2.1 Subcutaneous Nodules as a Sign of P. aeruginosa Bacteremia . 161 2.2.2 Ecthyma Gangrenosum . 161 2.2.3 Severe Skin and Soft Tissue Infection (SSTI): Gangrenous Cellulitis and Necrotizing Fasciitis. 161 2.2.4 Burn Wounds . 162 2.2.5 AIDS................................................................................................. 162 2.3 Other Cutaneous Manifestations . 162 3. Antimicrobial Therapy: General Principles . 163 3.1 The Development of Antibacterial Resistance . 163 3.2 Anti-Pseudomonal Agents . 163 3.3 Monotherapy versus Combination Therapy . 164 4. Antimicrobial Therapy: Specific Syndromes . 164 4.1 Primary P. aeruginosa Infections of the Skin . 164 4.1.1 Green Nail Syndrome. 164 4.1.2 Interdigital Infections . 165 4.1.3 Folliculitis . -
In the Prevention of Occupational Diseases 94 7.1 Introduction
Report on the current situation in relation to occupational diseases' systems in EU Member States and EFTA/EEA countries, in particular relative to Commission Recommendation 2003/670/EC concerning the European Schedule of Occupational Diseases and gathering of data on relevant related aspects ‘Report on the current situation in relation to occupational diseases’ systems in EU Member States and EFTA/EEA countries, in particular relative to Commission Recommendation 2003/670/EC concerning the European Schedule of Occupational Diseases and gathering of data on relevant related aspects’ Table of Contents 1 Introduction 4 1.1 Foreword .................................................................................................... 4 1.2 The burden of occupational diseases ......................................................... 4 1.3 Recommendation 2003/670/EC .................................................................. 6 1.4 The EU context .......................................................................................... 9 1.5 Information notices on occupational diseases, a guide to diagnosis .................................................................................................. 11 1.6 Objectives of the project ........................................................................... 11 1.7 Methodology and sources ........................................................................ 12 1.8 Structure of the report .............................................................................. 15 2 Developments -
Dr. Keesha Ewers Foreword by Dr
The Woman’s Guide to Reclaiming Emotional Freedom and Vibrant Health Dr. Keesha Ewers Foreword by Dr. Tom O’Bryan www.DrKeesha.com • Solving the Autoimmune Puzzle 1 Bestselling Author of The Autoimmune Fix and the Betrayal Docuseries Praise for Solving the Autoimmune Puzzle “Solving the Autoimmune Puzzle is functional medicine at its best. Dr. Keesha Ewers integrates her wealth of knowledge from behavioral science, Ayurve- dic medicine, and functional medicine to create an easy to use system that gets to the core of real healing. This book is full of practical information and tools that will free all who use it from pain and suffering of any kind, including autoimmune disease.” —Dr. Mark Hyman, New York Times bestselling author of Eat Fat, Get Thin. Director, Cleveland Clinic Center for Functional Medicine. “I applaud Dr. Ewers for bringing to light two very often overlooked root causes for those suffering with autoimmunity: stress and trauma. In her book, Solving the Autoimmune Puzzle: The Woman’s Guide to Reclaiming Emotional Freedom and Vibrant Health, Dr. Ewers dives deep into emotional aspects of those with autoimmunity and shows the reader how to come to peace with themselves and their trauma allowing them to heal from autoimmunity.” —Amy Myers, MD, New York Times bestselling author of The Autoimmune Solution and The Thyroid Connection. “Dr. Keesha Ewers lays out a clear, easy-to-follow roadmap to break free from inflammation and autoimmune disease. Her insightful, well-researched plan uncovers the missing pieces of the autoimmune puzzle and shows how to reverse this century’s greatest health challenge for women.” —JJ Virgin, CNS, CHFS, NYT bestselling author of The Virgin Diet & Sugar Impact Diet “Solving the Autoimmune Puzzle provides you with a straight forward way to understand the root causes of complex diseases of all kinds, including auto- immunity. -
Mineral Makeup and Its Role with Acne and Rosacea Jane Iredale, MA; Jennifer Linder, MD
REVIEW Mineral Makeup and Its Role With Acne and Rosacea Jane Iredale, MA; Jennifer Linder, MD Rosacea and acne have been the cause of physical and emotional distress for patients worldwide. Part of the distress has originated from the inability to find products that provide coverage without exacerbating the conditions. This includes understanding the role of certain ingredients with their attendant negative and positive effects. Fifteen years of experience has shown that mineral makeup can play a large part in helping to repair patients’ self-esteem as well as playing a meaningful role in skin improvement. IDENTIFYING AUTHENTIC For physicians to assess mineral makeup and its ben- MINERAL MAKEUP efits for their patients with rosacea and acne, it is neces- Patients with acne and rosacea frequently seek options to sary to explore the chemical composition of authentic cover what they consider toCOS be visually frustrating condi- DERMmineral powder. Many makeup brands are now mar- tions. Regrettably, they often make choices that are not keting products they call mineral makeup, but they effective and potentially detrimental to their situation. do not utilize authentic minerals in their formulations. To serve these patients better, physicians should educate The incorrect use of the word mineral as a marketing themselves and their staffs about camouflaging options. term confuses patients and can lead to the use of prod- Mineral makeup can beDo a satisfactory solutionNot as it is a ucts Copythat can potentially worsen their condition due to healthy, skin-friendly alternative to traditional makeup. problematic ingredients. Mineral makeup not only provides superior coverage and The original definition of mineral makeup is a makeup is easy to use, but it is also UV protective, noncomedo- that eliminates talc, potential skin irritants, and comedo- genic, and anti-inflammatory. -
Dermatopathology
Dermatopathology Clay Cockerell • Martin C. Mihm Jr. • Brian J. Hall Cary Chisholm • Chad Jessup • Margaret Merola With contributions from: Jerad M. Gardner • Talley Whang Dermatopathology Clinicopathological Correlations Clay Cockerell Cary Chisholm Department of Dermatology Department of Pathology and Dermatopathology University of Texas Southwestern Medical Center Central Texas Pathology Laboratory Dallas , TX Waco , TX USA USA Martin C. Mihm Jr. Chad Jessup Department of Dermatology Department of Dermatology Brigham and Women’s Hospital Tufts Medical Center Boston , MA Boston , MA USA USA Brian J. Hall Margaret Merola Department of Dermatology Department of Pathology University of Texas Southwestern Medical Center Brigham and Women’s Hospital Dallas , TX Boston , MA USA USA With contributions from: Jerad M. Gardner Talley Whang Department of Pathology and Dermatology Harvard Vanguard Medical Associates University of Arkansas for Medical Sciences Boston, MA Little Rock, AR USA USA ISBN 978-1-4471-5447-1 ISBN 978-1-4471-5448-8 (eBook) DOI 10.1007/978-1-4471-5448-8 Springer London Heidelberg New York Dordrecht Library of Congress Control Number: 2013956345 © Springer-Verlag London 2014 This work is subject to copyright. All rights are reserved by the Publisher, whether the whole or part of the material is concerned, specifi cally the rights of translation, reprinting, reuse of illustrations, recitation, broadcasting, reproduction on microfi lms or in any other physical way, and transmission or information storage and retrieval, electronic adaptation, computer software, or by similar or dissimilar methodology now known or hereafter developed. Exempted from this legal reservation are brief excerpts in connection with reviews or scholarly analysis or material supplied specifi cally for the purpose of being entered and executed on a computer system, for exclusive use by the purchaser of the work. -
Copyrighted Material
Part 1 General Dermatology GENERAL DERMATOLOGY COPYRIGHTED MATERIAL Handbook of Dermatology: A Practical Manual, Second Edition. Margaret W. Mann and Daniel L. Popkin. © 2020 John Wiley & Sons Ltd. Published 2020 by John Wiley & Sons Ltd. 0004285348.INDD 1 7/31/2019 6:12:02 PM 0004285348.INDD 2 7/31/2019 6:12:02 PM COMMON WORK-UPS, SIGNS, AND MANAGEMENT Dermatologic Differential Algorithm Courtesy of Dr. Neel Patel 1. Is it a rash or growth? AND MANAGEMENT 2. If it is a rash, is it mainly epidermal, dermal, subcutaneous, or a combination? 3. If the rash is epidermal or a combination, try to define the SIGNS, COMMON WORK-UPS, characteristics of the rash. Is it mainly papulosquamous? Papulopustular? Blistering? After defining the characteristics, then think about causes of that type of rash: CITES MVA PITA: Congenital, Infections, Tumor, Endocrinologic, Solar related, Metabolic, Vascular, Allergic, Psychiatric, Latrogenic, Trauma, Autoimmune. When generating the differential, take the history and location of the rash into account. 4. If the rash is dermal or subcutaneous, then think of cells and substances that infiltrate and associated diseases (histiocytes, lymphocytes, mast cells, neutrophils, metastatic tumors, mucin, amyloid, immunoglobulin, etc.). 5. If the lesion is a growth, is it benign or malignant in appearance? Think of cells in the skin and their associated diseases (keratinocytes, fibroblasts, neurons, adipocytes, melanocytes, histiocytes, pericytes, endothelial cells, smooth muscle cells, follicular cells, sebocytes, eccrine