Protecting Honey Bees from Pesticide Poisoning
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Global Insecticide Use for Vector-Borne Disease Control
WHO/CDS/NTD/WHOPES/GCDPP/2007.2 GLOBAL INSECTICIDE USE FOR VECTOR-BORNE DISEASE CONTROL M. Zaim & P. Jambulingam DEPARTMENT OF CONTROL OF NEGLECTED TROPICAL DISEASES (NTD) WHO PESTICIDE EVALUATION SCHEME (WHOPES) First edition, 2002 Second edition, 2004 Third edition, 2007 © World Health Organization 2007 All rights reserved. The designations employed and the presentation of the material in this publication do not imply the expression of any opinion whatsoever on the part of the World Health Organization concerning the legal status of any country, territory, city or area or of its authorities, or concerning the delimitation of its frontiers or boundaries. Dotted lines on maps represent approximate border lines for which there may not yet be full agreement. The mention of specific companies or of certain manufacturers’ products does not imply that they are endorsed or recommended by the World Health Organization in preference to others of a similar nature that are not mentioned. Errors and omissions excepted, the names of proprietary products are distinguished by initial capital letters. All reasonable precautions have been taken by the World Health Organization to verify the information contained in this publication. However, the published material is being distributed without warranty of any kind, either express or implied. The responsibility for the interpretation and use of the material lies with the reader. In no event shall the World Health Organization be liable for damages arising from its use. The named authors alone are responsible for the views expressed in this publication. CONTENTS Page Acknowledgements i Introduction 1 Collection of information 2 Data analysis and observations on reporting 3 All uses in vector control 6 Malaria vector control 22 Dengue vector control 38 Chagas disease vector control 48 Leishmaniasis vector control 52 Other vector-borne disease control 56 Selected insecticides – DDT 58 Selected insecticides – Insect growth regulators 60 Selected insecticides – Bacterial larvicides 62 Country examples 64 Annex 1. -
Honey Bee Immunity — Pesticides — Pests and Diseases
University of Nebraska - Lincoln DigitalCommons@University of Nebraska - Lincoln Distance Master of Science in Entomology Projects Entomology, Department of 2017 A GUIDEBOOK ON HONEY BEE HEALTH: Honey Bee Immunity — Pesticides — Pests and Diseases Joey Caputo Follow this and additional works at: https://digitalcommons.unl.edu/entodistmasters Part of the Entomology Commons This Article is brought to you for free and open access by the Entomology, Department of at DigitalCommons@University of Nebraska - Lincoln. It has been accepted for inclusion in Distance Master of Science in Entomology Projects by an authorized administrator of DigitalCommons@University of Nebraska - Lincoln. Photo by David Cappaert, Bugwood.org 1 A GUIDEBOOK ON HONEY BEE HEALTH Honey Bee Immunity — Pesticides— Pests and Diseases By Joey Caputo A graduate degree project submitted as partial fulfillment of the Option III requirements for the de- gree of Masters of Science in Entomology at the graduate school of the University of Nebraska- Lincoln, 2017. Last updated April 2017 — Version 1.2 i Contents Introduction 1 Honey Bee Immune System 2 Mechanical and Biochemical Immunity 2 Innate and Cell-Mediated Immunity 2 Humoral Immunity 2 Social Immunity 3 Detoxification Complexes 5 Problems in Beekeeping 5 Colony Collapse Disorder (CCD) 5 Bacterial, Fungal and Microsporidian Diseases 6 American foulbrood 6 European foulbrood 7 Nosemosis 8 Chalkbrood 10 Crithidia 10 Stonebrood 11 Varroa Mite and Viruses 11 Varroa Biology and Life Cycle 12 Varroa Mite Damage and Parasitic Mite -
2,4-Dichlorophenoxyacetic Acid
2,4-Dichlorophenoxyacetic acid 2,4-Dichlorophenoxyacetic acid IUPAC (2,4-dichlorophenoxy)acetic acid name 2,4-D Other hedonal names trinoxol Identifiers CAS [94-75-7] number SMILES OC(COC1=CC=C(Cl)C=C1Cl)=O ChemSpider 1441 ID Properties Molecular C H Cl O formula 8 6 2 3 Molar mass 221.04 g mol−1 Appearance white to yellow powder Melting point 140.5 °C (413.5 K) Boiling 160 °C (0.4 mm Hg) point Solubility in 900 mg/L (25 °C) water Related compounds Related 2,4,5-T, Dichlorprop compounds Except where noted otherwise, data are given for materials in their standard state (at 25 °C, 100 kPa) 2,4-Dichlorophenoxyacetic acid (2,4-D) is a common systemic herbicide used in the control of broadleaf weeds. It is the most widely used herbicide in the world, and the third most commonly used in North America.[1] 2,4-D is also an important synthetic auxin, often used in laboratories for plant research and as a supplement in plant cell culture media such as MS medium. History 2,4-D was developed during World War II by a British team at Rothamsted Experimental Station, under the leadership of Judah Hirsch Quastel, aiming to increase crop yields for a nation at war.[citation needed] When it was commercially released in 1946, it became the first successful selective herbicide and allowed for greatly enhanced weed control in wheat, maize (corn), rice, and similar cereal grass crop, because it only kills dicots, leaving behind monocots. Mechanism of herbicide action 2,4-D is a synthetic auxin, which is a class of plant growth regulators. -
Common and Chemical Names of Herbicides Approved by the WSSA
Weed Science 2010 58:511–518 Common and Chemical Names of Herbicides Approved by the Weed Science Society of America Below is the complete list of all common and chemical of herbicides as approved by the International Organization names of herbicides approved by the Weed Science Society of for Standardization (ISO). A sponsor may submit a proposal America (WSSA) and updated as of September 1, 2010. for a common name directly to the WSSA Terminology Beginning in 1996, it has been published yearly in the last Committee. issue of Weed Science with Directions for Contributors to A herbicide common name is not synonymous with Weed Science. This list is published in lieu of the selections a commercial formulation of the same herbicide, and in printed previously on the back cover of Weed Science. Only many instances, is not synonymous with the active ingredient common and chemical names included in this complete of a commercial formulation as identified on the product list should be used in WSSA publications. In the absence of label. If the herbicide is a salt or simple ester of a parent a WSSA-approved common name, the industry code number compound, the WSSA common name applies to the parent as compiled by the Chemical Abstracts Service (CAS) with compound only. CAS systematic chemical name or the systematic chemical The chemical name used in this list is that preferred by the name alone may be used. The current approved list is also Chemical Abstracts Service (CAS) according to their system of available at our web site (www.wssa.net). -
Carbamate Pesticides Aldicarb Aldicarb Sulfoxide Aldicarb Sulfone
Connecticut General Statutes Sec 19a-29a requires the Commissioner of Public Health to annually publish a list setting forth all analytes and matrices for which certification for testing is required. Connecticut ELCP Drinking Water Analytes Revised 05/31/2018 Microbiology Total Coliforms Fecal Coliforms/ E. Coli Carbamate Pesticides Legionella Aldicarb Cryptosporidium Aldicarb Sulfoxide Giardia Aldicarb Sulfone Carbaryl Physicals Carbofuran Turbidity 3-Hydroxycarbofuran pH Methomyl Conductivity Oxamyl (Vydate) Minerals Chlorinated Herbicides Alkalinity, as CaCO3 2,4-D Bromide Dalapon Chloride Dicamba Chlorine, free residual Dinoseb Chlorine, total residual Endothall Fluoride Picloram Hardness, Calcium as Pentachlorophenol CaCO3 Hardness, Total as CaCO3 Silica Chlorinated Pesticides/PCB's Sulfate Aldrin Chlordane (Technical) Nutrients Dieldrin Endrin Ammonia Heptachlor Nitrate Heptachlor Epoxide Nitrite Lindane (gamma-BHC) o-Phosphate Metolachlor Total Phosphorus Methoxychlor PCB's (individual aroclors) Note 1 PCB's (as decachlorobiphenyl) Note 1 Demands Toxaphene TOC Nitrogen-Phosphorus Compounds Alachlor Metals Atrazine Aluminum Butachlor Antimony Diquat Arsenic Glyphosate Barium Metribuzin Beryllium Paraquat Boron Propachlor Cadmium Simazine Calcium Chromium Copper SVOC's Iron Benzo(a)pyrene Lead bis-(2-ethylhexyl)phthalate Magnesium bis-(ethylhexyl)adipate Manganese Hexachlorobenzene Mercury Hexachlorocyclopentadiene Molybdenum Nickel Potassium Miscellaneous Organics Selenium Dibromochloropropane (DBCP) Silver Ethylene Dibromide (EDB) -
The Buzz About Bees: Honey Bee Biology and Behavior
4-H Honey Bee Leaders Guide Book I The Buzz About Bees: 18 U.S.C. 707 Honey Bee Biology and Behavior Publication 380-071 2009 To the 4-H Leader: The honey bee project (Books Grade 5 1 - 4) is intended to teach young people the basic biology and behavior of honey bees in addition to Living Systems 5.5 hands-on beekeeping management skills. The honey The student will investigate and understand that bee project books begin with basic honey bee and organisms are made up of cells and have distin- insect information (junior level) and advance to guishing characteristics. Key concepts include: instruction on how to rear honey bee colonies and • vertebrates and invertebrates extract honey (senior level). These project books are intended to provide in-depth information related Grade 6 to honey bee management, yet they are written for the amateur beekeeper, who may or may not have Life Science 5 previous experience in rearing honey bees. The student will investigate and understand how organisms can be classified. Key concepts include: Caution: • characteristics of the species If anyone in your club is known to have severe Life Science 8 allergic reactions to bee stings, they should not The student will investigate and understand that participate in this project. interactions exist among members of a population. The honey bee project meets the following Vir- Key concepts include: ginia State Standards of Learning (SOLs) for the • competition, cooperation, social hierarchy, and fourth, fifth, and sixth grades: territorial imperative Grade 4 Acknowledgments Authors: Life Processes 4.4 Dini M. -
Pesticides May Reduce Lettuce Yield Frank V
High-value crops such as strawberries, tain classes of insecticides on lettuce photo- usually clearly visible. Insecticides applied at broccoli, and iceberg lettuce often receive synthesis, transpiration, and productivity. normal rates and under the right environ- “preventive” or “insurance” pesticide treat- mental conditions may subtly damage a plant ments, which may result in weekly scheduled Insecticides but remain unobserved, because symptoms applications of insecticides. Many times such Insecticides in the various “classes,” such are not visible. treatments are unwarranted economically as chlorinated hydrocarbons (DDT, endrin, During the last few years, plant physiolo- and may reduce yields by detrimental effects and methoxychlor), organophosphates gists at University of California, Riverside, on the plants. Decreases in strawberry yields (guthion, parathion, and methyl-parathion), have developed the dual isotope porometer, due to preventive insecticide treatments in the carbamates (malathion and methomyl), and which provides accurate, simultaneous mea- absence of economically significant pest synthetic pyrethroids (fenvalerate and per- surements of a plant’s photosynthesis and populations have been reported. Research methrin), differ in their effects on plants. Ad- transpiration rates in the field. Entomologists supported by the California Iceberg Lettuce ditionally, the rates, number, and timing of have used the instrument to measure effects Research Advisory Board indicated that head applications may alter a compound’s effect -
Impact of Pesticide Use on Health in Developing Countries
Impact of pesticide use on health in developing countries Proceedings of a symposium held in Ottawa, Canada, 1 7-20 September 1990 IDRC CRDI International Development Research Centre Centre de recherches pour le devetoppement international 1 March 1993 Dear Reader/Librarian, IDRC is a public corporation created by the Canadian parliament in 1970 to help developing countries find viable solutions to their problems through research. At the 1992 Earth Summit, IDRC's mandate was broadened to emphasize sustainable development issues. As part of IDRC's strengthened commitment to global action and harüony, we are pleased to send you a complimentary copy of our most recent publication: The impact of pesticide use on health in developing countries (March 1993, 352 pages, 0-88936-560-1, $17.95). The first part of this book presents a brief survey of the global situation and the results of twelve epidemiological studies carried out by researchers from Africa, Latin America, Asia and the Middle East. These focus on poisonings resulting from organophosphates, herbicides, and pyrethroids. The second part illustrates the role of the process of development, production, spraying techniques and legislation in protecting the health of workers. A discussion of the benefits and modalities of access to pertinent information for the prevention of pesticide poisonings is provided in the third section. Finally, in the fourth section, consideration is given to the advantages and disadvantages of certain alternatives to the use of synthetic pesticides in agriculture and public health, such as botanical pesticides and integrated pest management strategies. We hope this book is a valuable addition to your collection. -
Organophosphate Poisoning : a Review
120 Sinha and Sharma Med J Indones Organophosphate poisoning : A review Parmod K. Sinha, Ashok Sharma Abstrak Pestisida organofosfat digunakan secara luas di seluruh dunia. Keracunan oleh bahan ini merupakan masalah kesehatan masyarakat, terutama di negara berkembang. Zat neurotoksik organofosfat merupakan bahan yang dianggap mengancam dalam bidang militer dan terorisme. Mekanisme toksisitas bahan ini adalah dengan cara menghambat asetilkolinesterase yang mengakibatkan menumpuknya neurotransmitor asetilkolin dan terjadi rangsangan terus-menerus pada reseptor asetilkolin pada sistem saraf sentral maupun perifer. Selain krisis kolinergik, organofosfat dapat menimbulkan berbagai sindrom neurologis, baik akut maupun kronik. Sedangkan gejala peralihan ( intermediate) terjadi 1-4 hari setelah krisis kolinergik teratasi. Pengobatan standar terdiri dari reaktivasi asetilkolinesterase dengan antidot golongan oksim (prolidoksim, oksidoksime, HI-6 dan HLo7), dan pengendalian efek biokimia asetilkolin dengan menggunakan atropin. Golongan oksim yang baru HI-6 dan Hlo7 merupakan reaktivator asetilkolinesterase yang lebih cocok dan efektif untuk keracunan akut dan berat dibandingkan dengan prolidoksim dan obidoksim. Penderita yang mendapat pengobatan segera, biasanya dapat sembuh dari toksisitas akut, namun gejala neurologis ikutan dapat saja terjadi. (Med J Indones 2003; 12: 120-6) Abstract Organophosphate pesticides are used extensively worldwide, and poisoning by these agents, particularly in developing nations is a public health problem. Organophosphorous -
162998571.Pdf
View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by University of Liverpool Repository Please do not adjust margins Investigating the breakdown of the nerve agent simulant methyl paraoxon and chemical warfare agents GB and VX using nitrogen containing bases Received 00th January 20xx, Accepted 00th January 20xx Craig Wilson,a Nicholas J. Cooper,b Michael E. Briggs,a Andrew I. Cooper,*a and Dave J. Adams*c DOI: 10.1039/x0xx00000x A range of nitrogen containing bases was tested for the hydrolysis of a nerve agent simulant, methyl paraoxon (MP), and www.rsc.org/ the chemical warfare agents, GB and VX. The product distribution was found to be highly dependant on the basicity of the base and the quantity of water used for the hydrolysis. This study is important in the design of decontamination technology, which often involve mimics of CWAs. production of EA-2192 Introduction (S-(2-diisopropylaminoethyl) methylphosphonothioic acid), which exhibits roughly the same toxicity as VX itself.12 The Chemical warfare agents (CWAs) have a devastating effect on blister agent HD (Fig. 1d) can also undergo deactivation via the body and will disable or kill on exposure. Blister agents, such hydrolysis.13 However, its poor water solubility reduces the as sulfur mustard (HD, bis(2-chloroethyl) sulfide), target the skin efficiency of this decontamination method.14 As a result, and respiratory system causing severe pain and damage to the oxidation to the sulfoxide, or the addition of a co-solvent is most body whereas nerve agents, such as sarin (GB, isopropyl commonly used for HD deactivation.15-17 methylphosphonofluoridate) and VX (O-ethyl The high toxicity of CWAs means that research into their S-[2-(diisopropylamino)ethyl] methylphosphonothioate), target deactivation is often carried out using simulants. -
2018 Treated Water Undetected Chemical Contaminant List
2018 Treated Water Undetected Chemical Contaminant List ESTROGENS AND OTHER HORMONES Diethylstilbestrol (DES) Estrone 17alpha-Estradiol 17alpha-Ethynal estradiol 17beta-Estradiol Progesterone Estriol cis-Testosterone trans-Testosterone INORGANIC CHEMICALS Antimony Niobium Arsenic Osmium Beryllium Palladium Cadmium Platinum Cerium Praseodymium Cesium Rhenium Cobalt Rhodium Cyanide Ruthenium Dysprosium Samarium Erbium Selenium Europium Silver Gadolinium Tantalum Gallium Tellurium Germanium Thallium Gold Thorium Hafnium Thulium Holmium Tin Iridium Titanium Lanthanum Tungsten Lead Uranium Lutetium Vanadium Mercury Ytterbium Molybdenum Zinc Neodymium Zirconium Nickel NITROSAMINES N-Nitropyrrolidine (NPYR) N-Nitrosomorpholine (NMOR) N-Nitrosodi-N-butylamine (NDBA) N-Nitrosodiphenylamine (NDPhA) N-Nitrosodiethylamine (NDEA) N-Nitrosodi-N-propylamine (NDPA) N-Nitrosodimethylamine (NDMA) N-Nitrosomethylethylamine (NMEA) N-Nitrosopiperidine (NPIP) 1 ORGANIC CHEMICALS Acenaphthene Butylbenzylphthalate Acenaphthylene Butyraldehyde (Butanal) Acetaldehyde Carbaryl Acetochlor Carbofuran Acetone Carbon disulfide Acrylamide Carbophenothion Acrylonitrile Carbon tetrachloride Alachlor Carboxin Aldicarb (Temik) Chlordane Aldicarb sulfone Chlordane, alpha Aldicarb sulfoxide Chlordane, gamma Aldrin Chlorfenvinphos Allyl chloride Chloroacetonitrile Tert-Amyl Methyl ether Chlorobenzene Ametryn Chlorobenzilate Anilizine 2-Chlorobiphenyl Anthracene 1-Chlorobutane Aspon Chloroethane Atraton Chloromethane Atrazine Chloroneb Azinphos-ethyl Chloroprene Azinphos-methyl -
2002 NRP Section 6, Tables 6.1 Through
Table 6.1 Scoring Table for Pesticides 2002 FSIS NRP, Domestic Monitoring Plan } +1 0.05] COMPOUND/COMPOUND CLASS * ) (EPA) (EPA) (EPA) (EPA) (EPA) (FSIS) (FSIS) PSI (P) TOX.(T) L-1 HIST. VIOL. BIOCON. (B) {[( (2*R+P+B)/4]*T} REG. CON. (R) * ENDO. DISRUP. LACK INFO. (L) LACK INFO. {[ Benzimidazole Pesticides in FSIS Benzimidazole MRM (5- 131434312.1 hydroxythiabendazole, benomyl (as carbendazim), thiabendazole) Carbamates in FSIS Carbamate MRM (aldicarb, aldicarb sulfoxide, NA44234416.1 aldicarb sulfone, carbaryl, carbofuran, carbofuran 3-hydroxy) Carbamates NOT in FSIS Carbamate MRM (carbaryl 5,6-dihydroxy, chlorpropham, propham, thiobencarb, 4-chlorobenzylmethylsulfone,4- NT 4 1 3 NV 4 4 13.8 chlorobenzylmethylsulfone sulfoxide) CHC's and COP's in FSIS CHC/COP MRM (HCB, alpha-BHC, lindane, heptachlor, dieldrin, aldrin, endrin, ronnel, linuron, oxychlordane, chlorpyrifos, nonachlor, heptachlor epoxide A, heptachlor epoxide B, endosulfan I, endosulfan I sulfate, endosulfan II, trans- chlordane, cis-chlordane, chlorfenvinphos, p,p'-DDE, p, p'-TDE, o,p'- 3444NV4116.0 DDT, p,p'-DDT, carbophenothion, captan, tetrachlorvinphos [stirofos], kepone, mirex, methoxychlor, phosalone, coumaphos-O, coumaphos-S, toxaphene, famphur, PCB 1242, PCB 1248, PCB 1254, PCB 1260, dicofol*, PBBs*, polybrominated diphenyl ethers*, deltamethrin*) (*identification only) COP's and OP's NOT in FSIS CHC/COP MRM (azinphos-methyl, azinphos-methyl oxon, chlorpyrifos, coumaphos, coumaphos oxon, diazinon, diazinon oxon, diazinon met G-27550, dichlorvos, dimethoate, dimethoate