Epiphora (Excessive Tearing)
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Low Level Light Therapy for the Treatment of Recalcitrant Chalazia: a Sample Case Summary
Clinical Ophthalmology Dovepress open access to scientific and medical research Open Access Full Text Article ORIGINAL RESEARCH Low level light therapy for the treatment of recalcitrant chalazia: a sample case summary This article was published in the following Dove Press journal: Clinical Ophthalmology Karl Stonecipher1 Purpose: To evaluate the effects of low-level light therapy (LLLT) on the resolution of Richard Potvin 2 recalcitrant chalazia. Patients and Methods: This was a single-site retrospective chart review of patients with 1Physicians Protocol, Greensboro, NC, USA; 2Science in Vision, Akron, NY, USA chalazia, all of whom were unresponsive to previous pharmaceutical therapy or surgical intervention, who received a 15 min LLLT treatment in conjunction with a standard phar- maceutical regimen. A second treatment was applied 24 hrs to as late as 2 months if there was no evidence of progression of resolution in appearance. Results: A total of 26 eyes of 22 patients with relevant history and treatment were reviewed, all with a history of prior pharmaceutical treatment for their chalazia. After a single 15 min LLLT treatment, followed by a standard pharmaceutical regimen, 46% of eyes (12/26) showed resolution of their chalazia. Resolution was noted from 3 days to one-month post- treatment. With a second treatment, the chalazia resolved in 92% of eyes (24/26). Only two For personal use only. eyes of the 26 (8%) required incision and curettage after LLLT treatment. Conclusion: The use of LLLT for the treatment of recalcitrant chalazia appears to be beneficial in patients who have failed topical and/or systemic therapy, significantly reducing the likelihood of requiring surgical intervention. -
Altered Hyaluronic Acid Content in Tear Fluid of Patients with Adenoviral Conjunctivitis
Anais da Academia Brasileira de Ciências (2015) 87(1): 455-462 (Annals of the Brazilian Academy of Sciences) Printed version ISSN 0001-3765 / Online version ISSN 1678-2690 http://dx.doi.org/10.1590/0001-3765201520140122 www.scielo.br/aabc Altered hyaluronic acid content in tear fluid of patients with adenoviral conjunctivitis JULIANA L. DREYFUSS1,2*, CAIO V. REGATIERI1,3,5*, BRUNO COELHO1, JOSÉ B. BARBOSA3, DENISE DE FREITAS3, HELENA B. NADER1 and JOÃO R. MARTINS1,4 1Departamento de Bioquímica, Disciplina de Biologia Molecular, Universidade Federal de São Paulo, Rua Três de Maio, 100, 4º andar, 04044-020 São Paulo, SP, Brasil 2Departamento de Informática em Saúde, Grupo Interdisciplinar de Ciências Exatas em Saúde, Universidade Federal de São Paulo, Rua Botucatu, 862, 04023-062 São Paulo, SP, Brasil 3Departamento de Oftalmologia, Universidade Federal de São Paulo, Rua Botucatu, 821, 04023-062 São Paulo, SP, Brasil 4Departamento de Medicina, Disciplina de Endocrinologia, Universidade Federal de São Paulo, Rua Borges Lagoa, 800, 04038-001 São Paulo, SP, Brasil 5Department of Ophthalmology, New England Eye Center, Tufts Medical Center, 800 Washington Street, 02111 Boston, MA, USA Manuscript received on March 13, 2014; accepted for publication on October 24, 2014 ABSTRACT The adenoviral conjunctivitis is one of the biggest causes of conjunctival infection in the world. Conjunctivitis causes relatively nonspecific symptoms, as hyperaemia and chemosis. Even after biomicroscopy, complex laboratory tests, such as viral culture, are necessary to identify the pathogen or its etiology. To contribute to the better understanding of the pathobiology of the adenoviral conjunctivitis, the tear fluids of patients with unilateral acute adenovirus conjunctivitis (UAAC), normal donors (control) and patients with allergic conjunctivitis were analyzed. -
Epiphora During the First Year of Life
Eye (1991) 5, 596--600 Epiphora During the First Year of Life C. J. MACEWEN and J. D. H. YOUNG Dundee Summary A cohort of 4,792 infants was observed in order to determine the incidence and natu ral history of epiphora during the first year of life. Evidence of defective lacrimal drainage was present in 964 (20%) at some time during the year. 9S�/o became symp tomatic during the first month of life. Spontaneous remission occurred throughout the year and 96% had resolved before the age of one. This study provides no evidence to support probing before the age of one year. Infants with epiphora are a common problem haps the most reliable estimate of the inci in clinical ophthalmology. It is generally dence is 6%. This comes from a follow-up accepted that the condition is the result of a study of 200 consecutive, unselected newborn congenital abnormality of the lacrimal drain infants.7 age system, in the form of a membranous Information on the rate of spontaneous obstruction at the lower end of the naso-lac remission is also limited. The studies that are rimal duct (NLD). I In addition it is recognised available were based on small clinic popula that there is a high rate of spontaneous resol tions, referred for treatment of their epiphora ution.2•3 However, despite it's frequency, rela and probing was usually undertaken in a tively little is known about the incidence or number of cases before the end of the year.2,3 natural history of epiphora in young children. -
Ν Prefit Examination
Cosmetics and the Contact Lens Wearer - What and Why By Diane F. Drake, LDO, ABOM, FCLSA Mission Statement Better Quality of Life Through Better Vision Course Description This presentation discusses cosmetics offered for the male and female contact lens wearers. Obvious and not so obvious cosmetics such as hairsprays, aftershaves, colognes, and soaps will be discussed. Proper handling and hygiene of contact lenses and the various structures of the tear layer and ocular structure will be included. Discover how improper cosmetic use can affect tear layers and the success or lack of success of the contact lens wearer. Introduction ν Tear layers ν Cosmetic usage ν Importance of tear film ν Patient instructions ν Types of cosmetics ν Conclusion - Communication ν Prefit examination Anatomy of the Eye Cornea Eyelids ν Five distinct layers ν Important in health of eye – Epithelium – Help to keep eye moist – Bowman’s layer – Help to distribute tears, oxygen and – Stroma nutrients – Descemets membrane – Protects the eye from light and injury – Endothelium – Lids are elastic ν Lose elasticity with age Tissues of Eyelids Conjunctiva ν Conjunctiva ν Thin mucous membrane, running ν Termed palpebral aperture continuous from lid to corneal limbus – While opened – Not always same size – Palpebral - lids – Bulbar - Globe of eye ν Contain Meibomian glands ν Contain Sebaceous glands ν Muscles of eyelid – Levator Muscle ν Pumps tears away – Orbicularis Oculi Muscle Contact lenses cannot get lost behind the eye Lacrimal Apparatus - Tear Production ν The conjunctival -
Olivia Steinberg ICO Primary Care/Ocular Disease Resident American Academy of Optometry Residents Day Submission
Olivia Steinberg ICO Primary Care/Ocular Disease Resident American Academy of Optometry Residents Day Submission The use of oral doxycycline and vitamin C in the management of acute corneal hydrops: a case comparison Abstract- We compare two patients presenting to clinic with an uncommon complication of keratoconus, acute corneal hydrops. Management of the patients differs. One heals quickly, while the other has a delayed course to resolution. I. Case A a. Demographics: 40 yo AAM b. Case History i. CC: red eye, tearing, decreased VA x 1 day OS ii. POHx: (+) keratoconus OU iii. PMHx: depression, anxiety, asthma iv. Meds: Albuterol, Ziprasidone v. Scleral CL wearer for approximately 6 months OU vi. Denies any pain OS, denies previous occurrence OU, no complaints OD c. Pertinent Findings i. VA cc (CL’s)- 20/25 OD, 20/200 PH 20/60+2 OS ii. Slit Lamp 1. Inferior corneal thinning and Fleisher ring OD, central scarring OD, 2+ diffuse microcystic edema OS, Descemet’s break OS (photos and anterior segment OCT) 2. 2+ diffuse injection OS 3. D&Q A/C OU iii. Intraocular Pressures: deferred OD due to CL, 9mmHg OS (tonopen) iv. Fundus Exam- unremarkable OU II. Case B a. Demographics: 39 yo AAM b. Case History i. CC: painful, red eye, tearing, decreased VA x 1 day OS ii. POHx: unremarkable iii. PMHx: hypertension iv. Meds: unknown HTN medication v. Wears Soflens toric CL’s OU; reports previous doctor had difficulty achieving proper fit OU; denies diagnosis of keratoconus OU vi. Denies any injury OS, denies previous occurrence OU, no complaints OD c. -
Etiology and Management of Epiphora in the Adult Patient: a Retrospective Study in an Interdisciplinary Epiphora Clinic
Research Article Journal of Clinical Ophthalmology and Eye Disorders Published: 26 Feb, 2021 Etiology and Management of Epiphora in the Adult Patient: A Retrospective Study in an Interdisciplinary Epiphora Clinic Vincent Q1*, Roxane F1, Aurelie L1 and Nicolas M2 1Department of Ophthalmology, UCLouvain, UCLouvain Catholic University of Louvain, Belgium 2Department of Otorhinolaryngology, UCLouvain, UCLouvain Catholic University of Louvain, Belgium Abstract Background: Epiphora in adults is a frequent ophthalmological condition with multiple etiologies, and requires a multidisciplinary approach for management, diagnosis and treatment, combining ophthalmologists, ENTs, nuclear medicine specialists, radiologists. Few centers possess a truly multi-specialty method for analyzing and treating epiphora in adults. Materials and Methods: We have conducted a retrospective study on 57 patients with a follow-up of 12 months, to examine the different etiologies and treatments in a multidisciplinary epiphora clinic in a tertiary care setting. Patients were systematically examined by an ophthalmologist and an ENT specialist, in addition to a full epiphora clinical workup. If needed, they were then referred for additional examinations in radiology (dacryo cone beam scanner) or scintigraphy. Results: Obstruction at any stage of the lacrimal drainage system was the most common cause of epiphora (48%), followed by ocular surface disease (28%), then eyelid malposition or laxity (26%), and finally functional causes. Regarding treatments, 10.5% (n=6) of patients underwent 3-snip punctoplasty, 8% (n=5) underwent canalicular repermeabilization through sharp catheterization, 21% (n=12) underwent DCR, 42% (n=24) were prescribed lid hygiene or ocular lubrication, 21% OPEN ACCESS (n=12) underwent eyelid surgery through canthopexy, 1% (n=1) had a combined treatment and 19% (n=11) had no treatment. -
Abstract: a 19 Year Old Male Was Diagnosed with Vitamin a Deficiency
Robert Adam Young Abstract: A 19 year old male was diagnosed with vitamin A deficiency (VAD). Clinical examination shows conjunctival changes with central and marginal corneal ulcers. Patient history and lab testing were used to confirm the diagnosis. I. Case History 19 year old Hispanic Male Presents with chief complaint of progressive blur at distance and near in both eyes, foreign body sensation, ocular pain, photophobia, and epiphora; signs/symptoms are worse in the morning upon wakening. Started 6 months to 1 year ago, and has progressively gotten worse. Patient reports that the right eye is worse than the left eye. Ocular history of Ocular Rosacea Medical history of Hypoaldosteronism, Pernicious Anemia, and Type 2 Polyglandular Autoimmune Syndrome Last eye examination was two weeks ago at a medical center Presenting topical/systemic medications - Tobramycin TID OU, Prednisolone Acetate QD OU (has used for two weeks); Fludrocortisone (used long-term per PCP) Other pertinent info includes reports that patient cannot gain weight, although he has a regular appetite. Patient presents looking very slim, malnourished, and undersized for his age. II. Pertinent findings Entering unaided acuities are 20/400 OD with no improvement with pinhole; 20/50 OS that improves to 20/25 with pinhole. Pupil testing shows (-)APD; Pupils are 6mm in dim light, and constrict to 4mm in bright light. Ocular motilities are full and smooth with no reports of diplopia or pain. Tonometry was performed with tonopen and revealed intraocular pressures of 12 mmHg OD and 11 mmHG OS. Slit lamp examination shows 2+ conjunctival injection with trace-mild bitot spots both nasal and temporal, OU. -
Canine Red Eye Elizabeth Barfield Laminack, DVM; Kathern Myrna, DVM, MS; and Phillip Anthony Moore, DVM, Diplomate ACVO
PEER REVIEWED Clinical Approach to the CANINE RED EYE Elizabeth Barfield Laminack, DVM; Kathern Myrna, DVM, MS; and Phillip Anthony Moore, DVM, Diplomate ACVO he acute red eye is a common clinical challenge for tion of the deep episcleral vessels, and is characterized general practitioners. Redness is the hallmark of by straight and immobile episcleral vessels, which run Tocular inflammation; it is a nonspecific sign related 90° to the limbus. Episcleral injection is an external to a number of underlying diseases and degree of redness sign of intraocular disease, such as anterior uveitis and may not reflect the severity of the ocular problem. glaucoma (Figures 3 and 4). Occasionally, episcleral Proper evaluation of the red eye depends on effective injection may occur in diseases of the sclera, such as and efficient diagnosis of the underlying ocular disease in episcleritis or scleritis.1 order to save the eye’s vision and the eye itself.1,2 • Corneal Neovascularization » Superficial: Long, branching corneal vessels; may be SOURCE OF REDNESS seen with superficial ulcerative (Figure 5) or nonul- The conjunctiva has small, fine, tortuous and movable vessels cerative keratitis (Figure 6) that help distinguish conjunctival inflammation from deeper » Focal deep: Straight, nonbranching corneal vessels; inflammation (see Ocular Redness algorithm, page 16). indicates a deep corneal keratitis • Conjunctival hyperemia presents with redness and » 360° deep: Corneal vessels in a 360° pattern around congestion of the conjunctival blood vessels, making the limbus; should arouse concern that glaucoma or them appear more prominent, and is associated with uveitis (Figure 4) is present1,2 extraocular disease, such as conjunctivitis (Figure 1). -
Dry Eye Syndrome: Excessive Tear Evaporation
IOWA CITY DEPARTMENT OF VETERANS AFFAIRS (VA) MEDICAL CENTER Medical Center 601 Highway 6 West, Iowa City, IA 52246-2208 Community-Based Outpatient Clinics 2979 Victoria Street, Bettendorf, IA 52722-2784 200 Mercy Drive, Suite 106, Dubuque, IA 52201-7343 387 E. Grove Street, Galesburg, IL 61401-3728 721 Broadway, Quincy, IL 62301-2708 1015 S. Hackett, Waterloo, IA 50701-3500 Coralville Clinic: 520 10th Avenue, Suite 200, Coralville, IA 52241-1923 Dry Eye Syndrome: Excessive Tear Evaporation Dry eye syndromes can be caused by excessive tear evaporation. The root cause of this condition is obstruction and poor quality of the eyelid (meibomian) oil glands. These glands are responsible for coating the tears with a layer of oil that helps prevent evaporation. Meibomian gland dysfunction, or, more simply, MGD, is the most common dry eye syndrome, accounting for approximately 85% of cases. MGD may occur as a consequence of getting older and may be become symptomatic in 10% to 15% of adults over the age of 50. It can also occur in patients with acne rosacea (where it is almost universally present), in patients with multiple allergies (especially those with strong family histories of allergy and/or eczema), and after eye surgery (especially LASIK, but also after cataract or glaucoma surgery). The MOST common symptom of MGD is fluctuation of vision. This is most noticeable during visual tasks that are associated with decreased blinking, such as reading, using the computer, watching television, and driving. It is aggravated during exposure to “evaporative conditions,” such as low environmental humidity and blowing air, especially ceiling and floor fans and heaters or air conditioners. -
Sympathy Crying: Insights from Infrared Thermal Imaging on a Female Sample
View metadata, citation and similar papers at core.ac.uk brought to you by CORE provided by Portsmouth University Research Portal (Pure) RESEARCH ARTICLE Sympathy Crying: Insights from Infrared Thermal Imaging on a Female Sample Stephanos Ioannou1*, Paul Morris2, Samantha Terry2, Marc Baker2, Vittorio Gallese3,4, Vasudevi Reddy2 1 Alfaisal University, Department of Physiological Sciences, College of Medicine, Riyadh, Kingdom of Saudi Arabia, 2 Department of Psychology-Centre for Situated Action and Communication, University of Portsmouth, Portsmouth, United Kingdom, 3 Parma University, Department of Neuroscience, Section of Human Physiology, Parma, Italy, 4 Institute of Philosophy, School of Advanced Study, University of London, London, United Kingdom * [email protected] a11111 Abstract Sympathy crying is an odd and complex mixture of physiological and emotional phenom- ena. Standard psychophysiological theories of emotion cannot attribute crying to a single subdivision of the autonomic nervous system (ANS) and disagreement exists regarding the emotional origin of sympathy crying. The current experiment examines sympathy crying OPEN ACCESS using functional thermal infrared imaging (FTII), a novel contactless measure of ANS activ- Citation: Ioannou S, Morris P, Terry S, Baker M, ity. To induce crying female participants were given the choice to decide which film they Gallese V, Reddy V (2016) Sympathy Crying: Insights wanted to cry to. Compared to baseline, temperature started increasing on the forehead, from Infrared Thermal Imaging on a Female Sample. PLoS ONE 11(10): e0162749.doi:10.1371/journal. the peri-orbital region, the cheeks and the chin before crying and reached even higher tem- pone.0162749 peratures during crying. The maxillary area showed the opposite pattern and a gradual tem- Editor: Alessio Avenanti, University of Bologna, perature decrease was observed compared to baseline as a result of emotional sweating. -
Eye External Anatomy of Eye Accessory Structures
4/22/16 Eye Bio 40B Dr. Kandula External Anatomy of Eye Accessory Structures l Eyebrows l Levator Palpebrae Superioris - opens eye l Eyelashes l Ciliary glands – modified sweat glands l Small sebaceous glands l Sty is inflamed ciliary glands or small sebaceous glands 1 4/22/16 Terms: Lacrimal gland and duct Surface of eye Lacrimal puncta Lacrimal sac Nasolacrimal duct Nasal cavity Tears / Lacrimal fluid l a watery physiologic saline, with a plasma-like consistency, l contains the bactericidal enzyme lysozyme; l it moistens the conjunctiva and cornea, l provides nutrients and dissolved O2 to the cornea. Extrinsic Muscles of the Eye: Lateral/medial rectus Important to know Superior/inferior rectus actions and nerve Superior/inferior oblique supply in table 2 4/22/16 Extrinsic Eye Muscles • Eye movements controlled by six extrinsic eye muscles Four recti muscles § Superior rectus – moves eyeball superiorly supplied by Cranial Nerve III § Inferior rectus - moves eyeball inferiorly supplied by Cranial Nerve III § Lateral rectus - moves eyeball laterally supplied by Cranial Nerve VI § Medial rectus - moves eyeball medially supplied by Cranial Nerve III Extrinsic Eye Muscles Two oblique muscles rotate eyeball on its axis § Superior oblique rotates eyeball inferiorly and laterally and is supplied by Cranial Nerve IV § Inferior oblique rotates superiorly and laterally and is supplied by Cranial Nerve III Convergence of the Eyes l Binocular vision in humans has both eyes looking at the same object l As you look at an object close to your face, -
TEAR PRODUCTION and DRAINAGE the Lacrimal Gland Is Located in the Superolateral Aspect of the Eyelid Below the Eyebrow(S)
Anatomy and Physiology 9 The conjunctiva is a mucous membrane that lines the upper and lower eyelids and extends over the sclera to the corneal margin. It contains lym- phoid tissue, which provides some immunology protection. It is innervated by CN V, the trigeminal nerve. The portion of the conjunctiva that covers the sclera is termed the bulbar conjunctiva; the portion covering the inner surface of the eyelids is termed the palpebral conjunctiva. Figure 1. Eyelid Muscles TEAR PRODUCTION AND DRAINAGE The lacrimal gland is located in the superolateral aspect of the eyelid below the eyebrow(s). It secretes watery (aqueous) tears and produces about 0.2 ml of tears in 24 hours. Aqueous tears flow downward and inward toward the tear drainage system at the inner canthus. In addition to aqueous tears, several glands located in the conjunctiva and eyelid margins secrete oily and sticky (mucous) tears. The meibomian glands are located within the tarsal plate of the eyelid and secrete oily tears. The glands of Zeiss, Moll, Wolfing, and Krause secrete sticky tears. These three types of tears provide moisture and protection to the surface of the eye(s). With each blink, tears are pushed across the eye toward the puncta located at the medial junction of the upper and lower eyelids. From the puncta, tears are pushed into the canaliculi and then into the lacrimal sac. 10 Essentials of Ophthalmic Nursing They are drained from the lacrimal sac and nasolacrimal duct to the inside of the nose and down the throat (see Figure 2). Figure 2. Lacrimal System TEAR FILM The tear film has three distinct layers.