VS-Regulated Livestock and Poultry Pathogens (Partial List)
Total Page：16
File Type：pdf, Size：1020Kb
Load more
Recommended publications
-
(Gladiolus Grandiflorus Hort.) Corm Rot in Mexico Revista Mexicana De Fitopatología, Vol
Revista Mexicana de Fitopatología ISSN: 0185-3309 [email protected] Sociedad Mexicana de Fitopatología, A.C. México González-Pérez, Enrique; Yáñez-Morales, María de Jesús; Ortega-Escobar, Héctor Manuel; Velázquez-Mendoza, Juan Comparative Analysis among Pathogenic Fungal Species that Cause Gladiolus (Gladiolus grandiflorus Hort.) Corm Rot in Mexico Revista Mexicana de Fitopatología, vol. 27, núm. 1, enero-junio, 2009, pp. 45-52 Sociedad Mexicana de Fitopatología, A.C. Texcoco, México Available in: http://www.redalyc.org/articulo.oa?id=61211414006 How to cite Complete issue Scientific Information System More information about this article Network of Scientific Journals from Latin America, the Caribbean, Spain and Portugal Journal's homepage in redalyc.org Non-profit academic project, developed under the open access initiative Revista Mexicana de FITOPATOLOGIA/ 45 Comparative Analysis among Pathogenic Fungal Species that Cause Gladiolus (Gladiolus grandiflorus Hort.) Corm Rot in Mexico Enrique González-Pérez1, María de Jesús Yáñez-Morales2, Héctor Manuel Ortega- Escobar1, and Juan Velázquez-Mendoza3, Colegio de Postgraduados, 1Hidrociencias, 2Fitopatología, and 3Forestal, Campus Montecillo, km 36.5 Carr. México-Texcoco, Montecillo, Edo. de México CP 56230. Correspondence to: [email protected] (Received: July 16, 2008 Accepted: February 10, 2009) González-Pérez, E., Yáñez-Morales, M.J., Ortega-Escobar, este cultivo. Todas las especies fueron patogénicas, se H.M., and Velázquez-Mendoza, J. 2009. Comparative analysis agruparon en tres categorías por su agresividad para causar among pathogenic fungal species that cause gladiolus la enfermedad, difirieron en características culturales. Los (Gladiolus grandiflorus Hort.) corm rot in Mexico. Revista análisis moleculares corroboraron las especies identificadas Mexicana de Fitopatología 27:45-52. -
Biofilm Formation by Moraxella Catarrhalis
BIOFILM FORMATION BY MORAXELLA CATARRHALIS APPROVED BY SUPERVISORY COMMITTEE Eric J. Hansen, Ph.D. ___________________________ Kevin S. McIver, Ph.D. ___________________________ Michael V. Norgard, Ph.D. ___________________________ Philip J. Thomas, Ph.D. ___________________________ Nicolai S.C. van Oers, Ph.D. ___________________________ BIOFILM FORMATION BY MORAXELLA CATARRHALIS by MELANIE MICHELLE PEARSON DISSERTATION Presented to the Faculty of the Graduate School of Biomedical Sciences The University of Texas Southwestern Medical Center at Dallas In Partial Fulfillment of the Requirements For the Degree of DOCTOR OF PHILOSOPHY The University of Texas Southwestern Medical Center at Dallas Dallas, Texas March, 2004 Copyright by Melanie Michelle Pearson 2004 All Rights Reserved Acknowledgements As with any grand endeavor, there was a large supporting cast who guided me through the completion of my Ph.D. First and foremost, I would like to thank my mentor, Dr. Eric Hansen, for granting me the independence to pursue my ideas while helping me shape my work into a coherent story. I have seen that the time involved in supervising a graduate student is tremendous, and I am grateful for his advice and support. The members of my graduate committee (Drs. Michael Norgard, Kevin McIver, Phil Thomas, and Nicolai van Oers) have likewise given me a considerable investment of time and intellect. Many of the faculty, postdocs, students and staff of the Microbiology department have added to my education and made my experience here positive. Many members of the Hansen laboratory contributed to my work. Dr. Eric Lafontaine gave me my first introduction to M. catarrhalis. I hope I have learned from his example of patience, good nature, and hard work. -
Kongre Kitabı
Book of Abstracts International VETistanbul Group Congress 2014 28-30 April, 2014 Istanbul, Turkey Book of Abstracts www.vetistanbul2014.org International VETistanbul Group Congress 2014 28-30 April 2014 International VETistanbul Group Congress 2014 28-30 April, 2014 Istanbul, Turkey Organizing Committee Prof. Dr. Halil GÜNEŞ, Chair Prof. Dr. Bülent EKİZ Prof. Dr. Ali AYDIN Assoc. Prof. Dr. Serkan İKİZ Assoc. Prof. Dr. Hasret DEMİRCAN YARDİBİ Assoc. Prof. Dr. Gülsün PAZVANT Scientific Committee* Prof. Dr. Kemal AK, Turkey Prof. Dr. Anatoliy ALEXANDROVICH STEKOLNIKOV, Russia Prof. Dr. Bogdan AMINKOV, Bulgaria Prof. Dr. Geno ATASANOV ANGELOV, Bulgaria Prof. Dr. Hajrudin BESIROVIC, Bosnia and Herzegovina Prof. Dr. Nihad FEJZIC, Bosnia and Herzegovina Assoc. Prof. Dr. Plamen GEORGIEV, Bulgaria Prof. Dr. Zehra HAJRULAI MUSLIU, Macedonia Assoc. Prof. Dr. Afrim HAMIDI, Kosovo Prof. Dr. Telman ISKENDEROV, Azerbaijan Prof. Dr. Larisa KARPENKO, Russia Prof. Dr. Ismail KIRSAN, Turkey Prof. Dr. Mihni LYUTSKANOV, Bulgaria Assoc. Prof. Dr. Avni ROBAJ, Kosovo Prof. Dr. Velimir STOJKOVSKI, Macedonia Prof. Dr. Semsir VELIYEV, Azerbaijan *Alphabetically listed by the according to the family name Scientific Secreteria Prof. Dr. Bülent EKİZ, Turkey Dr. Karlo MURATOĞLU, Turkey International VETistanbul Group Congress 2014, 28-30 April, Istanbul, Turkey IV International VETistanbul Group Congress 2014 28-30 April 2014 Dear Respectable Colleagues and Guests, First of all, I greet you all with my heart. Also, I would like to thank you for taking place on our side due to the contribution given to the establishment of VETistanbul Group. Known as, VETistanbul Group was established, under the coordination of Istanbul University, with joint decision of Veterinary Faculty of the University of Sarajevo, Saint Petersburg State Academy of Veterinary Medicine, Stara Zagora Trakia University, Ss. -
JOURNAL of VIROLOGY Volume 18 Contents for May Number 2
JOURNAL OF VIROLOGY Volume 18 Contents for May Number 2 Animal Viruses Isolation and Properties of the Replicase of Encephalomyocarditis Virus. A. TRAUB,* B. DISKIN, H. ROSENBERG, AND E. KALMAR ...... ............... 375 Synthesis and Integration of Viral DNA in Chicken Cells at Different Times After Infection with Various Multiplicities of Avian Oncornavirus. ALLAN T. KHOURY* AND HIDESABURO HANAFUSA ........ .......................... 383 RNA Metabolism of Murine Leukemia Virus. III. Identification and Quantitation of Endogenous Virus-Specific mRNA in the Uninfected BALB/c Cell Line JLS-V9. HUNG FAN AND NIKOLAUS MUELLER-LANTZSCH* ..... ............ 401 Endogenous Ecotropic Mouse Type C Viruses Deficient in Replication and Produc- tion of XC Plaques. ULF W RAPP AND ROBERT C. NoWINSKI* ..... ....... 411 Further Characterization of the Friend Murine Leukemia Virus Reverse Tran- scriptase-RNase H Complex. KARIN MOELLING ...... ................... 418 State of the Viral DNA in Rat Cells Transformed by Polyoma Virus. I. Virus Res- cue and the Presence of Nonintegrated Viral DNA Molecules. ISHWARI PRASAD, DIMITRIS ZOUZIAS, AND CLAUDIO BASIUCO* ...... ................ 436 Inhibition of Infectious Rous Sarcoma Virus Production by a Rifamycin Deriva- tive. CHARLES SZABO,* MINA J. BISSELL, AND MELVIN CALVIN ..... ...... 445 Synthesis ofthe Adenovirus-Coded DNA Binding Protein in Infected Cells. ZVEE GILEAD,* KINJI SUGUWARA, G. SHANMUGAM, AND MAURICE GREEN ....... 454 Intracellular Distribution and Sedimentation Properties of Virus-Specific RNA in Two Clones of BHK Cells Transformed by Polyoma Virus. IAN H. MAX- WELL .............................................................. 461 Inherited Resistance to N- and B-Tropic Murine Leukemia Viruses In Vitro: Titration Patterns in Strains SIM and SIM.R Congenic at the Fv-1 Locus. VERA SCHUH, MARTIN E. BLACKSTEIN, AND ARTHUR A. -
Blocking Transmission of Vector-Borne Diseases
International Journal for Parasitology: Drugs and Drug Resistance 7 (2017) 90e109 Contents lists available at ScienceDirect International Journal for Parasitology: Drugs and Drug Resistance journal homepage: www.elsevier.com/locate/ijpddr Review Blocking transmission of vector-borne diseases * Sandra Schorderet-Weber a, Sandra Noack b, Paul M. Selzer b, Ronald Kaminsky c, a Sablons 30, 2000 Neuchatel,^ Switzerland b Boehringer Ingelheim Animal Health GmbH, Binger Str. 173, 55216 Ingelheim, Germany c ParaC Consulting for Parasitology and Drug Discovery, Altenstein 13, 79685 Haeg-Ehrsberg, Germany article info abstract Article history: Vector-borne diseases are responsible for significant health problems in humans, as well as in companion Received 14 November 2016 and farm animals. Killing the vectors with ectoparasitic drugs before they have the opportunity to pass Accepted 22 January 2017 on their pathogens could be the ideal way to prevent vector borne diseases. Blocking of transmission Available online 30 January 2017 might work when transmission is delayed during blood meal, as often happens in ticks. The recently described systemic isoxazolines have been shown to successfully prevent disease transmission under Keywords: conditions of delayed pathogen transfer. However, if the pathogen is transmitted immediately at bite as it Vector-borne diseases is the case with most insects, blocking transmission becomes only possible if ectoparasiticides prevent Transmission blocking Drug discovery the vector from landing on or, at least, from biting the host. Chemical entities exhibiting repellent activity Speed of kill in addition to fast killing, like pyrethroids, could prevent pathogen transmission even in cases of im- mediate transfer. Successful blocking depends on effective action in the context of the extremely diverse life-cycles of vectors and vector-borne pathogens of medical and veterinary importance which are summarized in this review. -
Biological Diversity in the Patent System
Biological Diversity in the Patent System Paul Oldham1,2*, Stephen Hall1,3, Oscar Forero1,4 1 ESRC Centre for Economic and Social Aspects of Genomics (Cesagen), Lancaster University, Lancaster, United Kingdom, 2 Institute of Advanced Studies, United Nations University, Yokohama, Japan, 3 One World Analytics, Lancaster, United Kingdom, 4 Centre for Development, Environment and Policy, SOAS, University of London, London, United Kingdom Abstract Biological diversity in the patent system is an enduring focus of controversy but empirical analysis of the presence of biodiversity in the patent system has been limited. To address this problem we text mined 11 million patent documents for 6 million Latin species names from the Global Names Index (GNI) established by the Global Biodiversity Information Facility (GBIF) and Encyclopedia of Life (EOL). We identified 76,274 full Latin species names from 23,882 genera in 767,955 patent documents. 25,595 species appeared in the claims section of 136,880 patent documents. This reveals that human innovative activity involving biodiversity in the patent system focuses on approximately 4% of taxonomically described species and between 0.8–1% of predicted global species. In this article we identify the major features of the patent landscape for biological diversity by focusing on key areas including pharmaceuticals, neglected diseases, traditional medicines, genetic engineering, foods, biocides, marine genetic resources and Antarctica. We conclude that the narrow focus of human innovative activity and ownership of genetic resources is unlikely to be in the long term interest of humanity. We argue that a broader spectrum of biodiversity needs to be opened up to research and development based on the principles of equitable benefit-sharing, respect for the objectives of the Convention on Biological Diversity, human rights and ethics. -
Downloads/Global-Burden-Report.Pdf (Accessed on 20 December 2017)
viruses Review The Interactions between Host Glycobiology, Bacterial Microbiota, and Viruses in the Gut Vicente Monedero 1, Javier Buesa 2 and Jesús Rodríguez-Díaz 2,* ID 1 Department of Food Biotechnology, Institute of Agrochemistry and Food Technology (IATA, CSIC), Av Catedrático Agustín Escardino, 7, 46980 Paterna, Spain; [email protected] 2 Departament of Microbiology, Faculty of Medicine, University of Valencia, Av. Blasco Ibañez 17, 46010 Valencia, Spain; [email protected] * Correspondence: [email protected]; Tel.: +34-96-386-4903; Fax: +34-96-386-4960 Received: 31 January 2018; Accepted: 22 February 2018; Published: 24 February 2018 Abstract: Rotavirus (RV) and norovirus (NoV) are the major etiological agents of viral acute gastroenteritis worldwide. Host genetic factors, the histo-blood group antigens (HBGA), are associated with RV and NoV susceptibility and recent findings additionally point to HBGA as a factor modulating the intestinal microbial composition. In vitro and in vivo experiments in animal models established that the microbiota enhances RV and NoV infection, uncovering a triangular interplay between RV and NoV, host glycobiology, and the intestinal microbiota that ultimately influences viral infectivity. Studies on the microbiota composition in individuals displaying different RV and NoV susceptibilities allowed the identification of potential bacterial biomarkers, although mechanistic data on the virus–host–microbiota relation are still needed. The identification of the bacterial and HBGA interactions that are exploited by RV and NoV would place the intestinal microbiota as a new target for alternative therapies aimed at preventing and treating viral gastroenteritis. Keywords: rotavirus; norovirus; secretor; fucosyltransferase-2 gene (FUT2); histo-blood group antigens (HBGAs); microbiota; host susceptibility 1. -
Abstract Betaproteobacteria Alphaproteobacteria
Abstract N-210 Contact Information The majority of the soil’s biosphere containins biodiveristy that remains yet to be discovered. The occurrence of novel bacterial phyla in soil, as well as the phylogenetic diversity within bacterial phyla with few cultured representatives (e.g. Acidobacteria, Anne Spain Dr. Mostafa S.Elshahed Verrucomicrobia, and Gemmatimonadetes) have been previously well documented. However, few studies have focused on the Composition, Diversity, and Novelty within Soil Proteobacteria Department of Botany and Microbiology Department of Microbiology and Molecular Genetics novel phylogenetic diversity within phyla containing numerous cultured representatives. Here, we present a detailed University of Oklahoma Oklahoma State University phylogenetic analysis of the Proteobacteria-affiliated clones identified in a 13,001 nearly full-length 16S rRNA gene clones 770 Van Vleet Oval 307 LSE derived from Oklahoma tall grass prairie soil. Proteobacteria was the most abundant phylum in the community, and comprised Norman, OK 73019 Stillwater, OK 74078 25% of total clones. The most abundant and diverse class within the Proteobacteria was Alphaproteobacteria, which comprised 405 325 5255 405 744 6790 39% of Proteobacteria clones, followed by the Deltaproteobacteria, Betaproteobacteria, and Gammaproteobacteria, which made Anne M. Spain (1), Lee R. Krumholz (1), Mostafa S. Elshahed (2) up 37, 16, and 8% of Proteobacteria clones, respectively. Members of the Epsilonproteobacteria were not detected in the dataset. [email protected] [email protected] Detailed phylogenetic analysis indicated that 14% of the Proteobacteria clones belonged to 15 novel orders and 50% belonged (1) Dept. of Botany and Microbiology, University of Oklahoma, Norman, OK to orders with no described cultivated representatives or were unclassified. -
Anaplasmosis: an Emerging Tick-Borne Disease of Importance in Canada
IDCases 14 (2018) xxx–xxx Contents lists available at ScienceDirect IDCases journal homepage: www.elsevier.com/locate/idcr Case report Anaplasmosis: An emerging tick-borne disease of importance in Canada a, b,c d,e e,f Kelsey Uminski *, Kamran Kadkhoda , Brett L. Houston , Alison Lopez , g,h i c c Lauren J. MacKenzie , Robbin Lindsay , Andrew Walkty , John Embil , d,e Ryan Zarychanski a Rady Faculty of Health Sciences, Max Rady College of Medicine, Department of Internal Medicine, University of Manitoba, Winnipeg, MB, Canada b Cadham Provincial Laboratory, Government of Manitoba, Winnipeg, MB, Canada c Rady Faculty of Health Sciences, Max Rady College of Medicine, Department of Medical Microbiology and Infectious Diseases, University of Manitoba, Winnipeg, MB, Canada d Rady Faculty of Health Sciences, Max Rady College of Medicine, Department of Internal Medicine, Section of Medical Oncology and Hematology, University of Manitoba, Winnipeg, MB, Canada e CancerCare Manitoba, Department of Medical Oncology and Hematology, Winnipeg, MB, Canada f Rady Faculty of Health Sciences, Max Rady College of Medicine, Department of Pediatrics and Child Health, Section of Infectious Diseases, Winnipeg, MB, Canada g Rady Faculty of Health Sciences, Max Rady College of Medicine, Department of Internal Medicine, Section of Infectious Diseases, University of Manitoba, Winnipeg, MB, Canada h Rady Faculty of Health Sciences, Max Rady College of Medicine, Department of Community Health Sciences, University of Manitoba, Winnipeg, MB, Canada i Public Health Agency of Canada, National Microbiology Laboratory, Zoonotic Diseases and Special Pathogens, Winnipeg, MB, Canada A R T I C L E I N F O A B S T R A C T Article history: Human Granulocytic Anaplasmosis (HGA) is an infection caused by the intracellular bacterium Received 11 September 2018 Anaplasma phagocytophilum. -
TNF Decoy Receptors Encoded by Poxviruses
pathogens Review TNF Decoy Receptors Encoded by Poxviruses Francisco Javier Alvarez-de Miranda † , Isabel Alonso-Sánchez † , Antonio Alcamí and Bruno Hernaez * Centro de Biología Molecular Severo Ochoa, Consejo Superior de Investigaciones Científicas, Campus de Cantoblanco, Universidad Autónoma de Madrid, Nicolás Cabrera 1, 28049 Madrid, Spain; [email protected] (F.J.A.-d.M.); [email protected] (I.A.-S.); [email protected] (A.A.) * Correspondence: [email protected]; Tel.: +34-911-196-4590 † These authors contributed equally. Abstract: Tumour necrosis factor (TNF) is an inflammatory cytokine produced in response to viral infections that promotes the recruitment and activation of leukocytes to sites of infection. This TNF- based host response is essential to limit virus spreading, thus poxviruses have evolutionarily adopted diverse molecular mechanisms to counteract TNF antiviral action. These include the expression of poxvirus-encoded soluble receptors or proteins able to bind and neutralize TNF and other members of the TNF ligand superfamily, acting as decoy receptors. This article reviews in detail the various TNF decoy receptors identified to date in the genomes from different poxvirus species, with a special focus on their impact on poxvirus pathogenesis and their potential use as therapeutic molecules. Keywords: poxvirus; immune evasion; tumour necrosis factor; tumour necrosis factor receptors; lymphotoxin; inflammation; cytokines; secreted decoy receptors; vaccinia virus; ectromelia virus; cowpox virus Citation: Alvarez-de Miranda, F.J.; Alonso-Sánchez, I.; Alcamí, A.; 1. TNF Biology Hernaez, B. TNF Decoy Receptors TNF is a potent pro-inflammatory cytokine with a broad range of biological effects, Encoded by Poxviruses. Pathogens ranging from the activation of inflammatory gene programs to cell differentiation or 2021, 10, 1065. -
Ehrlichiosis and Anaplasmosis Are Tick-Borne Diseases Caused by Obligate Anaplasmosis: Intracellular Bacteria in the Genera Ehrlichia and Anaplasma
Ehrlichiosis and Importance Ehrlichiosis and anaplasmosis are tick-borne diseases caused by obligate Anaplasmosis: intracellular bacteria in the genera Ehrlichia and Anaplasma. These organisms are widespread in nature; the reservoir hosts include numerous wild animals, as well as Zoonotic Species some domesticated species. For many years, Ehrlichia and Anaplasma species have been known to cause illness in pets and livestock. The consequences of exposure vary Canine Monocytic Ehrlichiosis, from asymptomatic infections to severe, potentially fatal illness. Some organisms Canine Hemorrhagic Fever, have also been recognized as human pathogens since the 1980s and 1990s. Tropical Canine Pancytopenia, Etiology Tracker Dog Disease, Ehrlichiosis and anaplasmosis are caused by members of the genera Ehrlichia Canine Tick Typhus, and Anaplasma, respectively. Both genera contain small, pleomorphic, Gram negative, Nairobi Bleeding Disorder, obligate intracellular organisms, and belong to the family Anaplasmataceae, order Canine Granulocytic Ehrlichiosis, Rickettsiales. They are classified as α-proteobacteria. A number of Ehrlichia and Canine Granulocytic Anaplasmosis, Anaplasma species affect animals. A limited number of these organisms have also Equine Granulocytic Ehrlichiosis, been identified in people. Equine Granulocytic Anaplasmosis, Recent changes in taxonomy can make the nomenclature of the Anaplasmataceae Tick-borne Fever, and their diseases somewhat confusing. At one time, ehrlichiosis was a group of Pasture Fever, diseases caused by organisms that mostly replicated in membrane-bound cytoplasmic Human Monocytic Ehrlichiosis, vacuoles of leukocytes, and belonged to the genus Ehrlichia, tribe Ehrlichieae and Human Granulocytic Anaplasmosis, family Rickettsiaceae. The names of the diseases were often based on the host Human Granulocytic Ehrlichiosis, species, together with type of leukocyte most often infected. -
Identification of Acremonium Isolates from Grapevines and Evaluation of Their Antagonism Towards Plasmopara Viticola
Identification of Acremonium isolates from grapevines and evaluation of their antagonism towards Plasmopara viticola Sandra Lo Piccolo, Antonio Alfonzo, Selene Giambra, Gaetano Conigliaro, Luis V. Lopez-Llorca & Santella Burruano Annals of Microbiology ISSN 1590-4261 Ann Microbiol DOI 10.1007/s13213-015-1082-5 1 23 Your article is protected by copyright and all rights are held exclusively by Springer- Verlag Berlin Heidelberg and the University of Milan. This e-offprint is for personal use only and shall not be self-archived in electronic repositories. If you wish to self- archive your article, please use the accepted manuscript version for posting on your own website. You may further deposit the accepted manuscript version in any repository, provided it is only made publicly available 12 months after official publication or later and provided acknowledgement is given to the original source of publication and a link is inserted to the published article on Springer's website. The link must be accompanied by the following text: "The final publication is available at link.springer.com”. 1 23 Author's personal copy Ann Microbiol DOI 10.1007/s13213-015-1082-5 ORIGINAL ARTICLE Identification of Acremonium isolates from grapevines and evaluation of their antagonism towards Plasmopara viticola Sandra Lo Piccolo1 & Antonio Alfonzo1 & Selene Giambra1 & Gaetano Conigliaro1 & Luis V. Lopez-Llorca2 & Santella Burruano 1 Received: 8 July 2014 /Accepted: 1 April 2015 # Springer-Verlag Berlin Heidelberg and the University of Milan 2015 Abstract Some endophytic fungal genera in Vitis vinifera, Keywords Fungal endophytes . Phylogeny . RAPD . including Acremonium,havebeenreportedasantagonistsof Inhibition . Sporangia germination . Vitis vinifera Plasmopara viticola.EndophyticAcremonium isolates from an asymptomatic grapevine cultivar Inzolia from Italy were identified by morphological features and multigene phyloge- Introduction nies of ITS, 18S and 28S genes, and their intra-specific geno- mic diversity was analyzed by RAPD analysis.