Nodular Episcleritis • Less Common Than Simple Episcleritis • May Take Longer to Resolve • Treatment - Similar to Simple Episcleritis
Total Page:16
File Type:pdf, Size:1020Kb
Load more
Recommended publications
-
Differentiate Red Eye Disorders
Introduction DIFFERENTIATE RED EYE DISORDERS • Needs immediate treatment • Needs treatment within a few days • Does not require treatment Introduction SUBJECTIVE EYE COMPLAINTS • Decreased vision • Pain • Redness Characterize the complaint through history and exam. Introduction TYPES OF RED EYE DISORDERS • Mechanical trauma • Chemical trauma • Inflammation/infection Introduction ETIOLOGIES OF RED EYE 1. Chemical injury 2. Angle-closure glaucoma 3. Ocular foreign body 4. Corneal abrasion 5. Uveitis 6. Conjunctivitis 7. Ocular surface disease 8. Subconjunctival hemorrhage Evaluation RED EYE: POSSIBLE CAUSES • Trauma • Chemicals • Infection • Allergy • Systemic conditions Evaluation RED EYE: CAUSE AND EFFECT Symptom Cause Itching Allergy Burning Lid disorders, dry eye Foreign body sensation Foreign body, corneal abrasion Localized lid tenderness Hordeolum, chalazion Evaluation RED EYE: CAUSE AND EFFECT (Continued) Symptom Cause Deep, intense pain Corneal abrasions, scleritis, iritis, acute glaucoma, sinusitis, etc. Photophobia Corneal abrasions, iritis, acute glaucoma Halo vision Corneal edema (acute glaucoma, uveitis) Evaluation Equipment needed to evaluate red eye Evaluation Refer red eye with vision loss to ophthalmologist for evaluation Evaluation RED EYE DISORDERS: AN ANATOMIC APPROACH • Face • Adnexa – Orbital area – Lids – Ocular movements • Globe – Conjunctiva, sclera – Anterior chamber (using slit lamp if possible) – Intraocular pressure Disorders of the Ocular Adnexa Disorders of the Ocular Adnexa Hordeolum Disorders of the Ocular -
Diagnosing, Treating, and Managing Scleritis in 2020 an Expert Panel Recommendation Panel Members Melissa Toyos, Md Stephen D
DIAGNOSING, TREATING, AND MANAGING SCLERITIS IN 2020 AN EXPERT PANEL RECOMMENDATION PANEL MEMBERS MELISSA TOYOS, MD STEPHEN D. ANESI, MD, FACS n Partner and Director of Research n Massachusetts Eye Research & Surgery Institution n Toyos Clinic n Waltham, MA n Nashville, TN DAVID S. CHU, MD n Medical Director, Metropolitan Eye Research THOMAS A. ALBINI, MD & Surgery Institute n Professor of Clinical Ophthalmology n Associate Professor of Clinical Ophthalmology n University of Miami Health n Rutgers University n Bascom Palmer Eye Institute n Newark, NJ n Miami, FL ROBERT C. WANG, MD n Texas Retina Associates n Dallas, TX Corresponding Author: Melissa Toyos, MD; Toyos Clinic, Nashville, TN; [email protected]. This work was supported by an unrestricted medical writing grant from Mallinckrodt Pharmaceuticals and is based on a virtual roundtable discussion hosted by Evolve Medical Education LLC. Although uncommon, scleritis is a dangerous immune-me- DIAGNOSING SCLERITIS diated disease that can potentially threaten the structural Melissa Toyos, MD: What percentage of patients in integrity of the eye and may be indicative of potentially your practice have scleritis? life-threatening systemic vasculitis.1,2 Data on the genetic factors of scleritis is lacking, but it is thought that genes affect- Stephen D. Anesi, MD, FACS: Scleritis accounts for 10% ing systemic autoimmune diseases impact scleritis as well.2 to 15% of the patients I see in my practice. Differentiating between episcleritis and scleritis and posterior and anterior scleritis can be challenging for physicians. An David S. Chu, MD: I agree; 10% to 15% sounds right to accurate diagnosis is critical to properly treat the disease and me as well. -
JMSCR Vol||08||Issue||02||Page 208-210||February 2020
JMSCR Vol||08||Issue||02||Page 208-210||February 2020 http://jmscr.igmpublication.org/home/ ISSN (e)-2347-176x ISSN (p) 2455-0450 DOI: https://dx.doi.org/10.18535/jmscr/v8i2.40 Keratomalacia - A Case Report Authors Deepthi Pullepu1*, Deepthi Janga2, V Murali Krishna3 *Corresponding Author Deepthi Pullepu Department of Ophthalmology, Rangaraya Medical College, Kakinada, Andhra Pradesh, India Abstract Xerophthalmia refers to an ocular condition of destructive dryness of conjunctiva and cornea caused due to severe vitamin A deficiency. Usually affects infants, children and women of reproductive age group. It is estimated to affect about one-third of children under the age of five around the world. Keratomalacia means dry and cloudy cornea which melts and perforates, caused due to severe vitamin A deficiency. Here we describe a case of 21 year old female with neurofibromatosis type 1 presenting with keratomalacia. Keywords: Vitamin A deficiency, Xerophthalmia, Keratomalacia. Introduction blindness is a significant contributor Vitamin A is essential for the functioning of the In India, it is estimated that there are immune system, healthy growth and development approximately 6.8 million people who have vision of body and is usually acquired through diet. less than 6/60 in at least one eye due to corneal Globally, 190 million children under five years of diseases; of these, about a million have bilateral age are affected by vitamin A deficiency involvement. They suffer from an increased risk of visual Corneal blindness resulting due to this disease can impairment, illness and death from childhood be completely prevented by institution of effective infections such as measles and those causing preventive or prophylactic measures at the diarrhoea community level. -
CAUSES, COMPLICATIONS &TREATMENT of A“RED EYE”
CAUSES, COMPLICATIONS & TREATMENT of a “RED EYE” 8 Most cases of “red eye” seen in general practice are likely to be conjunctivitis or a superficial corneal injury, however, red eye can also indicate a serious eye condition such as acute angle glaucoma, iritis, keratitis or scleritis. Features such as significant pain, photophobia, reduced visual acuity and a unilateral presentation are “red flags” that a sight-threatening condition may be present. In the absence of specialised eye examination equipment, such as a slit lamp, General Practitioners must rely on identifying these key features to know which patients require referral to an Ophthalmologist for further assessment. Is it conjunctivitis or is it something more Iritis is also known as anterior uveitis; posterior uveitis is serious? inflammation of the choroid (choroiditis). Complications include glaucoma, cataract and macular oedema. The most likely cause of a red eye in patients who present to 4. Scleritis is inflammation of the sclera. This is a very rare general practice is conjunctivitis. However, red eye can also be presentation, usually associated with autoimmune a feature of a more serious eye condition, in which a delay in disease, e.g. rheumatoid arthritis. treatment due to a missed diagnosis can result in permanent 5. Penetrating eye injury or embedded foreign body; red visual loss. In addition, the inappropriate use of antibacterial eye is not always a feature topical eye preparations contributes to antimicrobial 6. Acid or alkali burn to the eye resistance. The patient history will usually identify a penetrating eye injury Most general practice clinics will not have access to specialised or chemical burn to the eye, but further assessment may be equipment for eye examination, e.g. -
Abstract: a 19 Year Old Male Was Diagnosed with Vitamin a Deficiency
Robert Adam Young Abstract: A 19 year old male was diagnosed with vitamin A deficiency (VAD). Clinical examination shows conjunctival changes with central and marginal corneal ulcers. Patient history and lab testing were used to confirm the diagnosis. I. Case History 19 year old Hispanic Male Presents with chief complaint of progressive blur at distance and near in both eyes, foreign body sensation, ocular pain, photophobia, and epiphora; signs/symptoms are worse in the morning upon wakening. Started 6 months to 1 year ago, and has progressively gotten worse. Patient reports that the right eye is worse than the left eye. Ocular history of Ocular Rosacea Medical history of Hypoaldosteronism, Pernicious Anemia, and Type 2 Polyglandular Autoimmune Syndrome Last eye examination was two weeks ago at a medical center Presenting topical/systemic medications - Tobramycin TID OU, Prednisolone Acetate QD OU (has used for two weeks); Fludrocortisone (used long-term per PCP) Other pertinent info includes reports that patient cannot gain weight, although he has a regular appetite. Patient presents looking very slim, malnourished, and undersized for his age. II. Pertinent findings Entering unaided acuities are 20/400 OD with no improvement with pinhole; 20/50 OS that improves to 20/25 with pinhole. Pupil testing shows (-)APD; Pupils are 6mm in dim light, and constrict to 4mm in bright light. Ocular motilities are full and smooth with no reports of diplopia or pain. Tonometry was performed with tonopen and revealed intraocular pressures of 12 mmHg OD and 11 mmHG OS. Slit lamp examination shows 2+ conjunctival injection with trace-mild bitot spots both nasal and temporal, OU. -
Red Eye Sub-Conjunctival Hemorrhage
AEP Course 1 Friday 1:00 - 2:00 pm Disclosures Red Eye • Consultant for Alcon Cecelia Koetting, OD FAAO Virginia Eye Consultants Norfolk, VA VOA Conference Norfolk 2018 Nothing makes the office more Multiple Causes of “Red Eye” nervous than when this walks in. • Sub-Conjunctival hemorrhage • Allergic Conjunctivitis • Viral Conjunctivitis • Bacterial Conjunctivitis • Episcleritis • Scleritis • Ocular Surface Disease How to Triage Sub-Conjunctival • On the phone • Get as much information as possible about which eye, Hemorrhage when, how, recent surgeries, recent trauma and what medications have been used • In the room • Gather the same information as well as details on symptoms • Help to avoid possible spread of bacterial and viral conjunctivitis 1 AEP Course 1 Friday 1:00 - 2:00 pm Causes • Straining from: • Broken blood vessel releasing blood into the • Coughing • Vomiting surrounding sub conjunctival tissue • Constipation • A bruise • Diarrhea • Lifting • More common in patients on blood thinners • Sneezing • Bruise more easily and will take longer to resolve • Rubbing the eyes • Contact lens • Severe dryness • Trauma, surgery, injections • Blood clotting disorders and blood thinners • Occasionally, high blood pressure High BP • Remember to ask if the patient is on blood thinners or blood pressure meds Allergic Conjunctivitis • Last time took BP meds • Possibly check BP in office if haven't taken • Inflammation in the lining of the conjunctiva from allergies • IgE and Mast Cell release • Symptoms: itching, redness, tearing, foreign body -
Current Health Issues in the Caribbean BLINDNESS in THE
Current Health Issues in the Caribbean BLINDNESS IN THE CARIBBEAN Alfred L. Anduze, M.D. St. Croix Vision Center St. Croix Hospital St. Croix U.S. Virgin Islands Caribbean Studies Association Merida, Mexico May 26, 1994 Abstract: Blindness in the Caribbean Background: The prevailing of blindness in the Caribbean region are reviewed in the context of world blindness statistics to identify differences and similarities that might exist. Method: A review of the status of blindness in the U.S. Virgin Islands, Barbados, Jamaica, Puerto Rico, Trinidad, and Mexico; individually with regard to causal etiology, epidemiology, treatment and possible future research. Results: Blindness in the Caribbean is the result of genetics, tropical environment and cultural habits of the inhabitants and consist of Age-related macular disease, Infectious diseases, Diabetes mellitus, Glaucoma, Congenital defects, Xerophthalmia, Trachoma, Trauma and Cataracts. Conclusion: There are almost 50 million people who are legally blind worldwide (i.e. with a vision of 20/200 or less) 2-3 million in the Caribbean region. The social and economic consequences are serious additional deterrents in developing countries. Outline: Causes of Blindness in the Caribbean I. Age-related macular disease a. Vascular insufficiency b. Senile macular degeneration II. Cataracts III. Glaucoma IV. Diabetes mellitus V. Infectious diseases a. Trachoma b. Onchocerciasis c. Leprosy d. Toxoplasmosis e. Toxocariasis f. AIDS VI. Trauma a. industrial/work-related b. sports c. home accidents VII. Nutritional a. Xerophthalmia/keratomalacia b. Iron-deficiency anemia c. Tobacco/Alcohol Retinopathy VIII. Congenital defects a. genetic syndromes b. strabismus Legal blindness is acceptably defined as vision 20/200 (6/60) or less. -
2002 Samel 10 Most Common Systemic Health Conditions with A
Avanti Samel / March 15, 2002 10 Most Common Systemic Health Conditions with a Listing of Frequently Prescribed Medications and their Ocular Side Effects HYPERTENSION ACE Inhibitors: Captopril (Capoten}- blurred vision Enalapril (Vasotec) - Blurred vision, conjunctivitis, dry eyes, tearing Quinipril (Accupril)- amblyopia Benazepril (Lotensin)- N/A Lisinopril (Zestril) - Visual loss, diplopia, blurred vision, photophobia Beta-Blockers: Propranolol (Inderal)- visual disturbances, dry eyes Atenolol (Tenormin)- blurred vision, dry eyes, visual disturbances Metoprolol (Lopressor) - blurred vision, dry eyes calcium Channel Blockers: Diltiazem (Cardizem)- Amblyopia, eye irritation Amlodipine (Norvasc)- abnormal vision, conjunctivitis, diplopia, eye pain Verapamil (Calan)- Blurred vision Nifedipine (Procardia) - blurred vision, Transient blindness at the peak of plasma level · Diuretics: Thiazides: Chlorothiazide (Diuril) - Transient blurred vision, xanthopsia Loop: Furosemide (Lasix) - Blurred vision, Xanthopsia Potassium Sparing: Amiloride (Midamor) - Visual disturbances, Increased lOP Triamterene (Dyrenium)- N/A HYPERLIPIDEMIA Statins: Lovastatin (Mevacor) - Blurred vision, Eye irritation Simvastatin (Zocor)- Cataracts Atorvastatin (Lipitor)- Amblyopia, dry eyes, refraction disorder, eye hemorrhage, glaucoma Resins: Cholestyramine (Questran)- Uveitis Fibrates: Gemfibrozil (Lopid)- Blurred vision Niacin: Niacin (Niacor) - Toxic amblyopia, cystoid macular edema ASTHMA ( ) Beta 2 Agonists: Albuterol (Proventil) - N/A ( Salmeterol (Serevent)- -
© Alan Macfarlane and Gerry Martin, 2002 1 MYOPIA: ITS
© Alan Macfarlane and Gerry Martin, 2002 MYOPIA: ITS COMPARATIVE INCIDENCE AND PROBABLE CAUSES What Rasmussen encountered in his many years of working with Chinese eye problem astonished him. He summarized his impressions thus. 'Three facts stand out clearly in both ancient and modern records of Chinese eyesight: one that they are a highly myopic nation; two that keratomalacia is the major cause of ocular disease; and three that presbyopia is earlier than in the West by about five years. They stand out against an historical background of neglect and abuse, malnutrition, and centuries of State indifference.' (p.48) Moving on to the statistics of myopia, he summarized the research of a quarter of a century by saying that 'only 20 per cent. of all lenses supplied by the ancients, and similar amount by the moderns (excluding high astigmatic) were for old-sight lenses. It is the other 80 per cent. that matters; the 65 per cent. for myopia and the 15 per cent. for glare and therapeutics.' (p.20) Elsewhere he gives slightly different statistics, which applied to those aged under forty years, showed that approximately 75 per cent. of the lenses were supplied for myopia. In a detailed table he presents the figures; in the 'Ancient Chinese Sight Records', 65% were for myopia; in the 'Modern Chinese Sight Records' myopia (simple) or with astigmatism combined accounted for 70% of the lenses.(p.48) The second figure of 70% was based on 120,000 case histories collected by Rasmussen and his colleagues over a period of 25 years. (p.49) The significance of the degree of myopia only comes out if we compare the Chinese figures with those for other nationalities. -
The Main Causes of Blindness and Low Vision in School for Blind
26ORIGINAL ARTICLE DOI 10.5935/0034-7280.20160006 The main causes of blindness and low vision in school for blind As principais causas de cegueira e baixa visão em escola para deficientes visuais Abelardo Couto Junior1, Lucas Azeredo Gonçalves de Oliveira2 ABSTRACT Objective: Identify and analyze the main causes of blindness and low vision in school for blind. Methods: One hundred sixty-five medical records of visually impaired students were reviewed in an institution specialized in teaching the blind, treated between august 2013 and may 2014. The variables analyzed were age, sex, visual acuity, primary and secondary diagnoses, treatment, optical prescription features and prognosis. Results: 165 students were evaluated, 91 students (55%) are legally blind and only 74 (45%) of the students are classified as low vision. The main causes of blindness were: retinopathy of prematurity (21%), optic nerve atrophy (18%), congenital glaucoma (16%), retinal dystrophy (11%) and cancer (8%). The causes of low vision were: congenital cataract (18%), congenital glaucoma (15%) and retinochoroidal scarring (12%). Conclusion: The main causes of blindness and low vision in the Benjamin Constant Institute are from preventable diseases. Keywords: Blindness; Low vision; Visual acuity; Child; Health school RESUMO Objetivo: Identificar e analisar as principais causas de cegueira e baixa visão em escola para deficientes visuais. Métodos: Foram revisados 165 prontuários de alunos portadores de deficiência visual em instituição especializada no ensino de cegos, atendidos no período de agosto de 2013 a maio de 2014. As variáveis analisadas foram: idade, gênero, acuidade visual, diagnóstico principal e secundário, tratamento, recursos ópticos prescritos e prognóstico. Resultados: Dos 165 alunos avaliados, 91 alunos (55%) são legalmente cegos e apenas 74 (45%) dos alunos são enquadrados como baixa visão. -
Ophthaproblem
Pratique clinique Clinical Pract ice Ophthaproblem Shaun Segal Sanjay Sharma, MD, MSC, MBA, FRCSC : Ophthalmic Photography : Ophthalmic Photography Photo credit Photo Dieu Hotel University, Laboratory of Queen’s Kingston, Ont. Hospital, 30-year-old woman had been admitted to hospital in infancy with diarrhea, steatorrhea, and failure to thrive. During late childhood, she developed atypi- Acal retinitis pigmentosa involving the retina and a progressive ataxic neuropathy. Investigations showed her serum lacked beta-lipoprotein and her red corpuscles had a spiky shape. Recent ophthalmic examination revealed substantial choroidal atrophy around the disk and a speckled peripheral fundus. Which vitamins should this patient be given as therapy for her disorder? 1. Vitamin A 2. Vitamin E 3. Vitamin C 4. Vitamin B Answer on page 1085 Mr Segal is a medical student at Queen’s University in Kingston, Ont. Dr Sharma is an Associate Professor of Ophthalmology and an Assistant Professor of Epidemiology at Queen’s University VOL 5: AUGUST • AOÛT 2005 d Canadian Family Physician • Le Médecin de famille canadien 1079 Pratique clinique Clinical Pract ice Answer to Ophthaproblem continued from page 1079 1. Vitamin A and 2. vitamin E Th is patient has abetalipoproteinemia (ABL), some- times called Bassen-Kornzweig syndrome,1 a dis- ease characterized by malabsorption of fat. The condition is treated with supplements of the two fat-soluble vitamins A and E. Abetalipoproteinemia manifests itself through signs of lipid malabsorp- tion, dense contracted red blood -
Retina Associated with Systemic Diseases*
MACULAR AND PARAMACULAR DETACHMENT OF THE NEUROSENSORY RETINA ASSOCIATED WITH SYSTEMIC DISEASES* BY Careen Y. Lowder, PhD, MD (BY INVITATION), Froncie A. Gutman, MD, Hernando Zegarra, MD (BY INVITATION), Z. Nicholas Zakov, MD (BY INVITATION)P James N. Lowder, MD (BY INVITATION), AND (BY INVITATION) John D. Clough, MD INTRODUCTION SEROUS RETINAL DETACHMENT (SRD) IN THE MACULAR REGION MAY DEVELOP AS A result of trauma, uveitis, optic neuritis, optic pit, and other conditions.1"2 In addition to these well-recognized disorders, there is a group ofpatients in whom transient episodes of SRD of the macula develop that are not presently associated with any recognized ocular or systemic disease. This group represents the clinical syndrome known as idiopathic central serous choroidopathy (CSC).2 The systemic implications of CSC are not known. Many reports have dealt with this without satisfactory evidence that there is a specific association between CSC and a systemic disease.1"3'4 This paper is a retrospective review of 168 cases of angiographically proved SRD of the macular region of unknown etiology that were seen at The Cleveland Clinic Foundation between 1971 and 1980. The associated systemic diseases were identified. Nine patients who had SRD of the posterior pole and subjective visual symptoms that appeared to correlate with an exacerbation oftheir system- ic diseases were included in the category of autoimmune disorders. Im- *From the Cleveland Clinic Foundation, Department of Ophthalmology, (Drs C. Lowder, Gutman, Zegarra, and Zakov), Department of Rheumatic and Immunologic Disease, (Dr Clough) and Department of Internal Medicine, University Hospitals,. (Dr J. Lowder) Cleveland, Ohio.