Pressure Ulcers and Wound Care 2 Objectives
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A Brief History of Pressure Ulcer Measurement in England: the Last 20 Years
EDITORIAL A brief history of pressure ulcer measurement in England: the last 20 years ressure ulcers (PU) have long been Measuring pressure ulcers: recognised as a challenge to healthcare, are prevalence and incidence a cause of significant pain and distress for During this time, the most reported forms of Ppatients and are costly for healthcare providers measurement have been prevalence and incidence. in term of finances and use of human resource Prevalence is the proportion of a population (Guest et al, 2020). who have a specific characteristic in a given This paper, the first in a series of three, describes time period. Therefore, the prevalence of PUs is the predominant methods that have been used to the proportion of a defined patient population JACQUI FLETCHER OBE capture the prevalence of PUs in England over the with pressure damage during a specified time Clinical Lead Pressure Ulcer last 20 years. The second paper will describe the period. Incidence is the number of specified new Workstream National Wound proposed system for national PU measurement events, during a specified period in a specified Care Strategy Clinical Editor, Wounds UK in England and the third paper will outline population. Therefore, the incidence of pressure proposals for implementing this system to drive damage is the number of people in a defined quality improvement. patient population who develop a new PU during a specified time period. BACKGROUND Prevalence of pressure damage is a good Measurement of the occurrence of PUs (also indicator of the overall burden and clinical known as pressure sores, decubitus ulcers, workload related to pressure damage but does pressure injury and bed sores) has been a part of not identify when and where the PU occurred, nursing activity for many years. -
The Pressure Sore Case: a Medical Perspective
Marquette Elder's Advisor Volume 2 Article 7 Issue 2 Fall The rP essure Sore Case: A Medical Perspective Jeffrey M. Levine Follow this and additional works at: http://scholarship.law.marquette.edu/elders Part of the Elder Law Commons Repository Citation Levine, Jeffrey M. (2000) "The rP essure Sore Case: A Medical Perspective," Marquette Elder's Advisor: Vol. 2: Iss. 2, Article 7. Available at: http://scholarship.law.marquette.edu/elders/vol2/iss2/7 This Featured Article is brought to you for free and open access by the Journals at Marquette Law Scholarly Commons. It has been accepted for inclusion in Marquette Elder's Advisor by an authorized administrator of Marquette Law Scholarly Commons. For more information, please contact [email protected]. The Pressure Sore Case: A Medical Perspective Although bedsores sometimes result dents.7 As a result, malpractice litigation related to pressure sores exploded in the 1990s.1 from inadequate care, not all cases Clinical Practice Guidelines involving pressure ulcers merit a law- Define Pressure Sores suit. This article adopts a medical per- Clinical practice guidelines define a pressure ulcer as "[a]ny skin lesion, usually over a bony promi- spective in its consideration of the pres- nence, caused by unrelieved pressure resulting in damage of underlying tissue." 9 The incidence of sure sore case, particularlywhen evalu- pressure ulcers in nursing homes is 0.20 to 0.56 per 1,000 resident-days, which may increase to 14 per ating deviation from the standard of 1,000 resident-days among those at high risk.' ° Commonly affected sites, comprising approximate- care. -
Skin As a Mirror of Gastrointestinal Diseases
Review article Skin as a mirror of gastrointestinal diseases Sunil Kumar Gupta1, Amanjot Kaur Arora1, Jasveen Kaur1, Veenu Gupta2, Deepinder Kaur2 Department of Dermatology, Venerology and Leprology1, Department of Microbiology2, Dayanand Medical College and Hospital, Ludhiana, Punjab, India ABSTRACT The closely related origins of the skin and the gastrointestinal system make for a fascinating grouping of diseases with concomitant involvement of these two important organ systems. The dermatologic manifestations may precede clinically evident gastrointestinal disease and help in early diagnosis, saving unnecessary wastage of time and resources. In this overview, we review the cutaneous manifestations of various hereditary, neoplastic and inflammatory gastrointestinal diseases including the various polyposis syndromes, hereditary colorectal cancers, inflammatory bowel diseases, etc. Dermatologic manifestations of acute and chronic hepatic and pancreatic diseases have also been discussed. This review underscores the importance of collaboration between dermatologists and gastroenterologists for better and efficient management of the affected patients. Keywords: Gastrointestinal diseases, Genodermatoses, Hepatitis, Inflammatory bowel disease, Pancreatic diseases, Polyposis syndromes INTRODUCTION diseases with skin and GIT involvement, genodermatoses, and the various hereditary Skin is the largest organ of the body. With a wide surface gastrointestinal cancers. area and being the outermost covering of the human body, skin has proven to be of good diagnostic help or atleast II.Cutaneous manifestations of liver diseases raise the index of suspicion that something inside the covering is wrong. Like many other systemic diseases, III.Cutaneous manifestations of pancreatic diseases. ,gastrointestinal tract (GIT) diseases may present with I. CUTANEOUS MANIFESTATIONS OF cutaneous symptoms, either primarily or secondarily as a GASTROINTESTINAL DISEASES sequalae to gut pathology, embryologic development being responsible for such clinically important associations. -
Wound Classification
Wound Classification Presented by Dr. Karen Zulkowski, D.N.S., RN Montana State University Welcome! Thank you for joining this webinar about how to assess and measure a wound. 2 A Little About Myself… • Associate professor at Montana State University • Executive editor of the Journal of the World Council of Enterstomal Therapists (JWCET) and WCET International Ostomy Guidelines (2014) • Editorial board member of Ostomy Wound Management and Advances in Skin and Wound Care • Legal consultant • Former NPUAP board member 3 Today We Will Talk About • How to assess a wound • How to measure a wound Please make a note of your questions. Your Quality Improvement (QI) Specialists will follow up with you after this webinar to address them. 4 Assessing and Measuring Wounds • You completed a skin assessment and found a wound. • Now you need to determine what type of wound you found. • If it is a pressure ulcer, you need to determine the stage. 5 Assessing and Measuring Wounds This is important because— • Each type of wound has a different etiology. • Treatment may be very different. However— • Not all wounds are clear cut. • The cause may be multifactoral. 6 Types of Wounds • Vascular (arterial, venous, and mixed) • Neuropathic (diabetic) • Moisture-associated dermatitis • Skin tear • Pressure ulcer 7 Mixed Etiologies Many wounds have mixed etiologies. • There may be both venous and arterial insufficiency. • There may be diabetes and pressure characteristics. 8 Moisture-Associated Skin Damage • Also called perineal dermatitis, diaper rash, incontinence-associated dermatitis (often confused with pressure ulcers) • An inflammation of the skin in the perineal area, on and between the buttocks, into the skin folds, and down the inner thighs • Scaling of the skin with papule and vesicle formation: – These may open, with “weeping” of the skin, which exacerbates skin damage. -
Pressure Ulcer Staging Cards and Skin Inspection Opportunities.Indd
Pressure Ulcer Staging Pressure Ulcer Staging Suspected Deep Tissue Injury (sDTI): Purple or maroon localized area of discolored Suspected Deep Tissue Injury (sDTI): Purple or maroon localized area of discolored intact skin or blood-fi lled blister due to damage of underlying soft tissue from pressure intact skin or blood-fi lled blister due to damage of underlying soft tissue from pressure and/or shear. The area may be preceded by tissue that is painful, fi rm, mushy, boggy, and/or shear. The area may be preceded by tissue that is painful, fi rm, mushy, boggy, warmer or cooler as compared to adjacent tissue. warmer or cooler as compared to adjacent tissue. Stage 1: Intact skin with non- Stage 1: Intact skin with non- blanchable redness of a localized blanchable redness of a localized area usually over a bony prominence. area usually over a bony prominence. Darkly pigmented skin may not have Darkly pigmented skin may not have visible blanching; its color may differ visible blanching; its color may differ from surrounding area. from surrounding area. Stage 2: Partial thickness loss of Stage 2: Partial thickness loss of dermis presenting as a shallow open dermis presenting as a shallow open ulcer with a red pink wound bed, ulcer with a red pink wound bed, without slough. May also present as without slough. May also present as an intact or open/ruptured serum- an intact or open/ruptured serum- fi lled blister. fi lled blister. Stage 3: Full thickness tissue loss. Stage 3: Full thickness tissue loss. Subcutaneous fat may be visible but Subcutaneous fat may be visible but bone, tendon or muscle are not exposed. -
Angina Bullosa Haemorrhagica (Oral Blood Blister) (PDF)
Patient Information Maxillo-facial Angina Bullosa Haemorrhagica (Oral Blood Blister) What is Angina Bullosa Haemorrhagica? Angina Bullosa Hemorrhagica (ABH) is a condition where an often painful, but benign blood-filled blister suddenly develops in the mouth. The blisters are generally not due to a blood clotting disorder or any other medical disorder. It is a fairly common, sudden onset and benign blood blistering oral (mouth) disorder. It mainly affects people over 45 years and both males and females are equally affected. Usually there is no family history of the condition. It may be associated with Type 2 Diabetes, a family history of diabetes or Hyperglycaemia. What are the signs and symptoms of ABH? The first indication is a stinging pain or burning sensation just before the appearance of a blood blister The blisters last only a few minutes and then spontaneously rupture (burst), leaving a shallow ulcer that heals without scarring, discomfort or pain They can reach an average size of one to three centimetres in diameter The Soft Palate (back of the mouth) is the most affected site If they occur on the palate and are relatively big, they may need to be de-roofed (cut and drained) to ease the sensation of choking Patient Information Occasionally blisters can occur in the buccal mucosa (cheek) and tongue Approximately one third of the patients have blood blisters in more than one location. What are the causes of ABH? More than 50% of cases are related to minor trauma caused by: hot foods, restorative dentistry (fillings, crowns etc) or Periodontal Therapy (treatment of gum disease). -
The Role of Pressure Ulcers in the Fight Against Antimicrobial Resistance
The role of pressure ulcer prevention in the fight against antimicrobial resistance Every year over 25,000 patients die in the EU alone as a result of infections caused by antibiotic- resistant bacteria. Globally the number of deaths due to antimicrobial resistance (AMR) was estimated to be 700,0001 in 2014 and that number has been calculated to rise to at least 10 million by 2050. The continuing emergence of AMR has become a recurring topic in the international health agenda as the increasingly serious threat to cross-border public health is recognised. From WHO to OECD, international bodies are constantly monitoring, reporting and formulating strategies to contain AMR. AMR is defined by WHO as the ability of microorganisms to survive antimicrobial treatments; consequently, prophylactic and therapeutic regimens are ineffective in controlling infections caused by resistant bacteria, fungi, parasites and viruses.2 The situation has deteriorated dramatically in the past decade with AMR reaching levels of 80% in some countries.3 How has this happened? Whereas greater investment and skill in reporting of AMR may be one reason, an important consideration is that AMR is a natural and inevitable process which is aggravated by the inappropriate use of antimicrobial agents. Healthcare authorities have been aware of the consequences of overuse of antibiotics in animal and human health, yet relatively few actions have been implemented to slow the process down.4 The good news is that the EU has made a significant step forward to gain a global lead in the fight against AMR. In June 2017 the Commission adopted the ambitious EU One Health Action Plan against AMR5 (as requested by the Member States in the Council Conclusions of 17 June 2016). -
Clinical Case of the Month Pressure Ulcer Treatment
Spinal Cord (1997) 35, 641 ± 646 1997 International Medical Society of Paraplegia All rights reserved 1362 ± 4393/97 $12.00 Clinical Case of the Month Pressure ulcer treatment F Biering-Sùrensen1, JL Sùrensen2, TEP Barros3, AA Monteiro3, I Nuseibeh4, SM Shenaq5 and K Shibasaki6 1Centre for Spinal Cord Injured, the Neuroscience Centre, and 2Department of Plastic Surgery, Rigshospitalet, Copenhagen University Hospital, Denmark; 3Spinal Injury Unit, Department of Orthopaedics, University of Sao Paulo, Brazil; 4National Spinal Injuries Centre, Stoke Mandeville, England; 5Department of Surgery, Baylor College of Medicine, The Methodist Hospital, Houston, Texas, USA; 6Spinal Cord Division, Department of Orthopedic Surgery, National Murayama Hospital, Japan Keywords: spinal cord injury; paraplegia; pressure ulcer; management; treatment; surgery Introduction The complicated case story below was presented to and found. Over the sacrum there was an ulcer measuring discussed by senior colleagues from Brazil, England, 2.561.5 cm with bone at the base. The plastic surgeon Japan and USA. None of the the responders knew that concluded, not least because of the patient's mental the patient had sustained his injury in 1981, and the habitus, not to operate, but to try conservative initial report stopped at the New Year 1986/87. A treatment. X-ray gave no indication of osteitis. Three review of the patient's medical history for the following weeks later the left trochanteric ulcer measured 3.5 cm years up to date is presented. Finally a short discussion in diameter and 3.5 cm in depth with necrosis and an of the possibilities of treatment is given. iliotibial tract at the base. -
Pressure Ulcers By: Esther Hattler BS,RN,WCC
Pressure Ulcers By: Esther Hattler BS,RN,WCC Staging Objectives The attendee will be able to list the 6 stages of pressure ulcers. Stage I Definition Intact skin with non-blanchable redness of a localized area usually over a bony prominence. Darkly pigmented skin may not have visible blanching. Its color may differ from surrounding area. Description Stage I The area may be painful, firm, soft, warmer or cooler as compared to adjacent tissue. Stage I may be difficult to detect in individuals with dark skin tones. May indicate “at risk” persons (a heralding sign of risk). Pictures stage I Stage II Definition Partial thickness loss of dermis presenting as a shallow open ulcer with a red/pink wound bed, WITHOUT slough. May also present as an intact or open ruptured serum filled blister. Description stage II Presents as a shiny or dry shallow ulcer WITHOUT slough or bruising. The stage II should NOT be used to describe skin tears, tape burns, perineal dermatitis, maceration or excoriation. Pictures stage II Stage II Stage III Definition Full thickness tissue loss. Subcutaneous fat may be visible but bone, tendon, or muscle are not exposed. Slough may be present but does not obscure the depth of tissue loss. May include undermining and tunneling. Description stage III The depth of a a stage III pressure ulcer varies by anatomical location. The bridge of the nose, ear, occiput and malleolus do not have subcutaneous tissue and stage III ulcers can be shallow. In contrast, areas of significant adiposity can develop extremely deep stage III pressure ulcers. -
Pressure Injuries Caused by Medical Devices and Other Objects: a Clinical Update
WOUND WISE 1.5 HOURS CE Continuing Education A series on wound care in collaboration with the World Council of Enterostomal Therapists Pressure Injuries Caused by Medical Devices and Other Objects: A Clinical Update A review of practical resources, including mnemonics, to aid in prevention and identification. ABSTRACT: At the April 2016 National Pressure Ulcer Advisory Panel (NPUAP) consensus conference, termi- nology and staging definitions were updated and two definitions were revised to describe pressure injuries (PIs) caused by medical devices or other items on the skin or mucosa. Here, the authors discuss the etiology and prevention of PIs resulting from medical and other devices, the frequency of such injuries, and the bodily sites at which they most often occur. They provide an overview of the current NPUAP guideline, highlight im- portant risk factors, and explain why mucosal PIs cannot be staged. Keywords: medical device–related pressure injuries, mucosal pressure injuries, National Pressure Ulcer Ad- visory Panel, pressure injury, pressure injury staging, pressure ulcer, SORE mnemonic, DEVICE mnemonic ressure injuries (PIs), formerly known as bed- pressure ulcer was replaced by pressure injury to sores, decubiti, pressure sores, or pressure ul- underscore the fact that PIs may be present even Pcers, have been a nursing concern since the when the skin is intact, and the definitions of medi- time of Florence Nightingale. In April 2016, the Na- cal device–related PIs (MDRPIs) and mucosal mem- tional Pressure Ulcer Advisory Panel (NPUAP) shone brane PIs were revised.1 (See Figure 1 for a depiction a spotlight on this issue by convening a consensus of intact, undamaged skin.) The NPUAP currently conference in which associated terminology and defines MDRPIs as PIs that “result from the use of staging definitions were updated. -
CLINICAL RESEARCH PROJECT Protocol #11-H-0134 Drug Name: Eltrombopag (Promacta®) IND Number: 104,877 IND Holder: NHLBI OCD Date: January 2, 2019
CLINICAL RESEARCH PROJECT Protocol #11-H-0134 Drug Name: eltrombopag (Promacta®) IND number: 104,877 IND holder: NHLBI OCD Date: January 2, 2019 Title: A Pilot Study of a Thrombopoietin-receptor Agonist (TPO-R agonist), Eltrombopag, in Moderate Aplastic Anemia Patients Other Identifying Words: Hematopoiesis, autoimmunity, thrombocytopenia, neutropenia, anemia, stem cells, cytokine, Promacta® (eltrombopag) Protocol Principal Investigator: *Cynthia E. Dunbar, M.D., TSCBB, NHLBI (E) Medically and Scientifically Responsible Investigator: *Cynthia E. Dunbar, M.D., TSCBB, NHLBI (E) Associate Investigators: *Georg Aue, M.D., OCD, NHLBI (E) *Neal S. Young, M.D., Chief, HB, NHLBI (E) *André Larochelle, M.D., Ph.D., CMTB, NHLBI (E) David Young, M.D., TSCBB, NHLBI (E) Susan Soto, M.S.N., R.N., Research Nurse, OCD, NHLBI(E) Olga Rios, RN, Research Nurse, OCD, NHLBI (E) Evette Barranta, R.N, Research Nurse, OCD, NHLBI (E) Jennifer Jo Kyte, DNP, Research Nurse, OCD, NHLBI (E) Colin Wu, PhD, Biostatistician, OBR, NHLBI (E) Xin Tian, PhD, Biostatistician, OBR/NHLBI (E) *Janet Valdez, MS, PAC, OCD, NHLBI (E) *Jennifer Lotter, MSHS, PA-C., OCD, NHLBI (E) Qian Sun, Ph.D., DLM, CC (F) Xing Fan, M.D., HB, NHLBI (F) Non-NIH, Non-Enrolling Engaged Investigators: Thomas Winkler, M.D., NHLBI, HB (V)# # Covered under the NIH FWA Independent Medical Monitor: John Tisdale, MD, NHLBI, OSD 402-6497 Bldg. 10, 9N116 * asterisk denotes who can obtain informed consent on this protocol Subjects of Study: Number Sex Age-range 38 Either ≥ 2 years and weight >12 kg Project Involves Ionizing Radiation? No (only when medically indicated) Off-Site Project? No Multi center trial? No DSMB Involvement? Yes 11-H-0134 1 Cynthia E. -
Pressure Ulcer Staging Guide
Pressure Ulcer Staging Guide Pressure Ulcer Staging Guide STAGE I STAGE IV Intact skin with non-blanchable Full thickness tissue loss with exposed redness of a localized area usually Reddened area bone, tendon, or muscle. Slough or eschar may be present on some parts Epidermis over a bony prominence. Darkly Epidermis pigmented skin may not have of the wound bed. Often includes undermining and tunneling. The depth visible blanching; its color may Dermis of a stage IV pressure ulcer varies by Dermis differ from the surrounding area. anatomical location. The bridge of the This area may be painful, firm, soft, nose, ear, occiput, and malleolus do not warmer, or cooler as compared to have subcutaneous tissue and these adjacent tissue. Stage I may be Adipose tissue ulcers can be shallow. Stage IV ulcers Adipose tissue difficult to detect in individuals with can extend into muscle and/or Muscle dark skin tones. May indicate "at supporting structures (e.g., fascia, Muscle risk" persons (a heralding sign of Bone tendon, or joint capsule) making risk). osteomyelitis possible. Exposed bone/ Bone tendon is visible or directly palpable. STAGE II DEEP TISSUE INJURY Partial thickness loss of dermis Blister Purple or maroon localized area of Reddened area presenting as a shallow open ulcer discolored intact skin or blood-filled Epidermis with a red pink wound bed, without Epidermis blister due to damage of underlying soft slough. May also present as an tissue from pressure and/or shear. The intact or open/ruptured serum-filled Dermis area may be preceded by tissue that is Dermis blister.