Anterior Interosseous Nerve Palsy Caused by Parsonage-Turner Syndrome
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Dental Plexopathy Vesta Guzeviciene, Ricardas Kubilius, Gintautas Sabalys
SCIENTIFIC ARTICLES Stomatologija, Baltic Dental and Maxillofacial Journal, 5:44-47, 2003 Dental Plexopathy Vesta Guzeviciene, Ricardas Kubilius, Gintautas Sabalys SUMMARY Aim and purpose of the study were: 1) to study and compare unfavorable factors playing role in the development of upper teeth plexitis and upper teeth plexopathy; 2) to study peculiarities of clinical manifestation of upper teeth plexitis and upper teeth plexopathy, and to establish their diagnostic value; 3) to optimize the treatment. The results of examination and treatment of 79 patients with upper teeth plexitis (UTP-is) and 63 patients with upper teeth plexopathy (UTP-ty) are described in the article. Questions of the etiology, pathogenesis and differential diagnosis are discussed, methods of complex medicamental and surgical treatment are presented. Keywords: atypical facial neuralgia, atypical odontalgia, atypical facial pain, vascular toothache. PREFACE Besides the common clinical tests, in order to ana- lyze in detail the etiology and pathogenesis of the afore- Usually the injury of the trigeminal nerve is re- mentioned disease, its clinical manifestation and pecu- lated to the pathology of the teeth neural plexuses. liarities, we performed specific examinations such as According to the literature data, injury of the upper orthopantomography of the infraorbital canals, mea- teeth neural plexuses makes more than 7% of all sured the velocity of blood flow in the infraorbital blood neurostomatologic diseases. Many terms are used in vessels (doplerography), examined the pain threshold literature to characterize the clinical symptoms com- of facial skin and oral mucous membrane in acute pe- plex of the above-mentioned pathology. Some authors riod and remission, and evaluated the role that the state (1, 2, 3) named it dental plexalgia or dental plexitis. -
Brachial-Plexopathy.Pdf
Brachial Plexopathy, an overview Learning Objectives: The brachial plexus is the network of nerves that originate from cervical and upper thoracic nerve roots and eventually terminate as the named nerves that innervate the muscles and skin of the arm. Brachial plexopathies are not common in most practices, but a detailed knowledge of this plexus is important for distinguishing between brachial plexopathies, radiculopathies and mononeuropathies. It is impossible to write a paper on brachial plexopathies without addressing cervical radiculopathies and root avulsions as well. In this paper will review brachial plexus anatomy, clinical features of brachial plexopathies, differential diagnosis, specific nerve conduction techniques, appropriate protocols and case studies. The reader will gain insight to this uncommon nerve problem as well as the importance of the nerve conduction studies used to confirm the diagnosis of plexopathies. Anatomy of the Brachial Plexus: To assess the brachial plexus by localizing the lesion at the correct level, as well as the severity of the injury requires knowledge of the anatomy. An injury involves any condition that impairs the function of the brachial plexus. The plexus is derived of five roots, three trunks, two divisions, three cords, and five branches/nerves. Spinal roots join to form the spinal nerve. There are dorsal and ventral roots that emerge and carry motor and sensory fibers. Motor (efferent) carries messages from the brain and spinal cord to the peripheral nerves. This Dorsal Root Sensory (afferent) carries messages from the peripheral to the Ganglion is why spinal cord or both. A small ganglion containing cell bodies of sensory NCS’s sensory fibers lies on each posterior root. -
Anatomical, Clinical, and Electrodiagnostic Features of Radial Neuropathies
Anatomical, Clinical, and Electrodiagnostic Features of Radial Neuropathies a, b Leo H. Wang, MD, PhD *, Michael D. Weiss, MD KEYWORDS Radial Posterior interosseous Neuropathy Electrodiagnostic study KEY POINTS The radial nerve subserves the extensor compartment of the arm. Radial nerve lesions are common because of the length and winding course of the nerve. The radial nerve is in direct contact with bone at the midpoint and distal third of the humerus, and therefore most vulnerable to compression or contusion from fractures. Electrodiagnostic studies are useful to localize and characterize the injury as axonal or demyelinating. Radial neuropathies at the midhumeral shaft tend to have good prognosis. INTRODUCTION The radial nerve is the principal nerve in the upper extremity that subserves the extensor compartments of the arm. It has a long and winding course rendering it vulnerable to injury. Radial neuropathies are commonly a consequence of acute trau- matic injury and only rarely caused by entrapment in the absence of such an injury. This article reviews the anatomy of the radial nerve, common sites of injury and their presentation, and the electrodiagnostic approach to localizing the lesion. ANATOMY OF THE RADIAL NERVE Course of the Radial Nerve The radial nerve subserves the extensors of the arms and fingers and the sensory nerves of the extensor surface of the arm.1–3 Because it serves the sensory and motor Disclosures: Dr Wang has no relevant disclosures. Dr Weiss is a consultant for CSL-Behring and a speaker for Grifols Inc. and Walgreens. He has research support from the Northeast ALS Consortium and ALS Therapy Alliance. -
An Unusual Cause of Pseudomedian Nerve Palsy
Hindawi Publishing Corporation Case Reports in Neurological Medicine Volume 2011, Article ID 474271, 3 pages doi:10.1155/2011/474271 Case Report An Unusual Cause of Pseudomedian Nerve Palsy Zina-Mary Manjaly, Andreas R. Luft, and Hakan Sarikaya Department of Neurology, University Hospital Zurich, Frauenklinikstraße 26, 8091 Zurich,¨ Switzerland Correspondence should be addressed to Zina-Mary Manjaly, [email protected] Received 20 July 2011; Accepted 9 August 2011 Academic Editors: J. L. Gonzalez-Guti´ errez,´ V. Rajajee, and Y. Wakabayashi Copyright © 2011 Zina-Mary Manjaly et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. We describe a patient who presented with an acute paresis of her distal right hand suggesting a peripheral median nerve lesion. However, on clinical examination a peripheral origin could not be verified, prompting further investigation. Diffusion-weighted magnetic resonance imaging revealed an acute ischaemic lesion in the hand knob area of the motor cortex. Isolated hand palsy in association with cerebral infarction has been reported occasionally. However, previously reported cases presented predominantly as ulnar or radial palsy. In this case report, we present a rather rare finding of an acute cerebral infarction mimicking median never palsy. 1. Case median nerve, which was normal (Figure 1(c)). Magnetic resonance imaging (MRI) on the same day revealed a small A 60-year-old woman presented to the emergency depart- diffusion restriction in a part of the left precentral gyrus that ffi ment with di culty in moving the thumb, index, and middle is known as “the hand knob” area (Figure 1(d))[2]. -
New Insights in Lumbosacral Plexopathy
New Insights in Lumbosacral Plexopathy Kerry H. Levin, MD Gérard Said, MD, FRCP P. James B. Dyck, MD Suraj A. Muley, MD Kurt A. Jaeckle, MD 2006 COURSE C AANEM 53rd Annual Meeting Washington, DC Copyright © October 2006 American Association of Neuromuscular & Electrodiagnostic Medicine 2621 Superior Drive NW Rochester, MN 55901 PRINTED BY JOHNSON PRINTING COMPANY, INC. C-ii New Insights in Lumbosacral Plexopathy Faculty Kerry H. Levin, MD P. James. B. Dyck, MD Vice-Chairman Associate Professor Department of Neurology Department of Neurology Head Mayo Clinic Section of Neuromuscular Disease/Electromyography Rochester, Minnesota Cleveland Clinic Dr. Dyck received his medical degree from the University of Minnesota Cleveland, Ohio School of Medicine, performed an internship at Virginia Mason Hospital Dr. Levin received his bachelor of arts degree and his medical degree from in Seattle, Washington, and a residency at Barnes Hospital and Washington Johns Hopkins University in Baltimore, Maryland. He then performed University in Saint Louis, Missouri. He then performed fellowships at a residency in internal medicine at the University of Chicago Hospitals, the Mayo Clinic in peripheral nerve and electromyography. He is cur- where he later became the chief resident in neurology. He is currently Vice- rently Associate Professor of Neurology at the Mayo Clinic. Dr. Dyck is chairman of the Department of Neurology and Head of the Section of a member of several professional societies, including the AANEM, the Neuromuscular Disease/Electromyography at Cleveland Clinic. Dr. Levin American Academy of Neurology, the Peripheral Nerve Society, and the is also a professor of medicine at the Cleveland Clinic College of Medicine American Neurological Association. -
Bilateral Brachial Plexopathy As an Initial Presentation in a Newly-Diagnosed, Uncontrolled Case of Diabetes Mellitus Pica E C, Verma K K
Case Report Singapore Med J 2008; 49(2) : e29 Bilateral brachial plexopathy as an initial presentation in a newly-diagnosed, uncontrolled case of diabetes mellitus Pica E C, Verma K K ABSTRACT A 55-year-old Indian woman with newly-diagnosed diabetes mellitus presented with acute onset right upper limb proximal weakness. This was followed three weeks later by pain, weakness and sensory loss in the left upper limb. Electrodiagnosis showed patchy multiple proximal and distal axonal neuropathies in both upper limbs, consistent with bilateral brachial neuritis. Laboratory investigations, cerebrospinal fluid analysis, and imaging studies were normal except for an anti- nuclear antibody titre of 1:640. Sural nerve and quadriceps biopsy did not show vasculitis. Fig. 1 Photograph shows the patient’s inability to flex the Brachial plexopathy has seldom been associated interphalangeal joint of the thumb and the distal interphalangeal with diabetes mellitus and could represent a rare joint of the index finger (pinch or OK sign) implying a median subtype of the diabetic neuropathies. nerve lesion proximal to the wrist bilaterally. Keywords: bilateral brachial plexopathy, brachial with signs of dehydration. HbA1c at the time was 14.2%. plexus neuropathies, diabetes mellitus, diabetic She was hydrated and blood sugars were optimised. She neuropathies was discharged after one week to continue medication Singapore Med J 2008; 49(2): e29-e32 (Metformin) at home. Upon reaching home, while taking a shower, she INTRODUCTION found that she could not lift her right shoulder to wash her Neuropathy is a frequent complication of diabetes mellitus hair. Elbow, wrist and hand movements were unaffected. -
Brachial Plexopathy Following High-Dose Melphalan and Autologous Peripheral Blood Stem Cell Transplantation
Bone Marrow Transplantation (2010) 45, 951–952 & 2010 Macmillan Publishers Limited All rights reserved 0268-3369/10 $32.00 www.nature.com/bmt LETTER TO THE EDITOR Brachial plexopathy following high-dose melphalan and autologous peripheral blood stem cell transplantation Bone Marrow Transplantation (2010) 45, 951–952; Tone and power were normal throughout, lower limb deep doi:10.1038/bmt.2009.243; published online 21 September 2009 tendon reflexes were absent and plantars were downgoing. Magnetic resonance imaging of the whole spine at this point revealed widespread myelomatous involvement of the Neuromuscular pathologies are a recognized complication bony spine but no cord compromise. Thalidomide was of SCT, often occurring as a result of infection or continued with no alteration in dosage. The patient haemorrhage, but also in association with GVHD follow- subsequently underwent a PBSCT with high-dose melpha- ing allogeneic SCT.1 The published literature on post- lan as the conditioning regime. transplant peripheral nervous system pathologies includes Within 14 days of stem-cell infusion the patient devel- descriptions of myasthenia gravis, Guillain-Barre´ oped progressive proximal weakness affecting predomi- syndrome, polymyositis and peripheral neuropathy.2–5 nantly the upper limbs. There was no neck pain or Ocular toxicity, radiculopathy and plexopathy have also involvement of bladder or bowel. Examination revealed rarely been reported. We report three cases of brachial bilateral wrist-drop with grade 3–4 weakness of the small plexopathy occurring after autologous peripheral blood muscles of the hand, elbow flexors and extensors and stem cell transplantation (PBSCT). shoulder abduction. Upper limb reflexes were absent. The neurological findings in the lower limbs were unchanged. -
Is the Diagnosis Written in the Palm?
CLINICAL Is the diagnosis written in the palm? Compression neuropathy from a walking frame Anupam Datta Gupta ANSWER 1 cause significant functional limitations. The diagnosis is compression neuropathy In late cases where the hand muscles of the right ulnar nerve and bilateral have already undergone atrophy, the CASE carpal tunnel syndrome at the wrist. motor recovery of those muscles, even A man aged 72 years requires a walking Pigmentation, callosity and atrophy on the after surgical decompression, may frame for mobility because of weakness ulnar side (hypothenar) of the right hand be incomplete. For early diagnosis of of both legs secondary to poliomyelitis. are indicative of ulnar nerve compression compression neuropathies, it is important He presents to the rehabilitation around the Guyon’s tunnel. This is either to routinely look at the hands of patients medicine outpatient clinic with soreness caused or exacerbated by the excessive who are taking increased weight through and weakness of both hands, which he pressure around the wrist during walking their hands because of a lower extremity developed following the use of the walking with the frame. Wasting of the first web problem and using mobility aids. If not frame. He also complains of loss of grip space caused by denervation of the picked up early, compression neuropathies strength and tingling of his hands. He is first dorsal interosseous and adductor can compound the disability. using the heel of the hand to manipulate pollicis muscles is a telltale sign of ulnar objects. Examination reveals skin neuropathy. On the left hand, the pressure ANSWER 3 pigmentation and callosities on the ulnar areas are around the carpal tunnel, causing To establish a diagnosis, the patient side of both palms, distal to the wrist crease median nerve compression. -
Electrodiagnosis of Brachial Plexopathies and Proximal Upper Extremity Neuropathies
Electrodiagnosis of Brachial Plexopathies and Proximal Upper Extremity Neuropathies Zachary Simmons, MD* KEYWORDS Brachial plexus Brachial plexopathy Axillary nerve Musculocutaneous nerve Suprascapular nerve Nerve conduction studies Electromyography KEY POINTS The brachial plexus provides all motor and sensory innervation of the upper extremity. The plexus is usually derived from the C5 through T1 anterior primary rami, which divide in various ways to form the upper, middle, and lower trunks; the lateral, posterior, and medial cords; and multiple terminal branches. Traction is the most common cause of brachial plexopathy, although compression, lacer- ations, ischemia, neoplasms, radiation, thoracic outlet syndrome, and neuralgic amyotro- phy may all produce brachial plexus lesions. Upper extremity mononeuropathies affecting the musculocutaneous, axillary, and supra- scapular motor nerves and the medial and lateral antebrachial cutaneous sensory nerves often occur in the context of more widespread brachial plexus damage, often from trauma or neuralgic amyotrophy but may occur in isolation. Extensive electrodiagnostic testing often is needed to properly localize lesions of the brachial plexus, frequently requiring testing of sensory nerves, which are not commonly used in the assessment of other types of lesions. INTRODUCTION Few anatomic structures are as daunting to medical students, residents, and prac- ticing physicians as the brachial plexus. Yet, detailed understanding of brachial plexus anatomy is central to electrodiagnosis because of the plexus’ role in supplying all motor and sensory innervation of the upper extremity and shoulder girdle. There also are several proximal upper extremity nerves, derived from the brachial plexus, Conflicts of Interest: None. Neuromuscular Program and ALS Center, Penn State Hershey Medical Center, Penn State College of Medicine, PA, USA * Department of Neurology, Penn State Hershey Medical Center, EC 037 30 Hope Drive, PO Box 859, Hershey, PA 17033. -
Tendon Transfer for Triple Nerve Paralysis of the Hand in Leprosy
Lepr Rev (2002) 73, 319±325 Tendon transfer for triple nerve paralysis of the hand in leprosy ELAINE MCEVITT & RICHARD SCHWARZ Green Pastures Hospital, Box 5, Pokhara, Nepal Accepted for publication 27June 2002 Summary Paralysis of ulnar, median and radial nerves is seen in less than 1% of those affected with leprosy. This condition is a particular challenge for the surgeon, physiotherapist, and patient. A retrospective chart review was conducted at the Green Pastures Hospital and Rehabilitation Centre (GPHRC) and Anandaban Leprosy Hospital (ALH) in Nepal, and results were graded by the system outlined by Sundararaj in 1984. Thirty-one patients were identi®ed, and 21 charts were available for review. Excellent or good results were obtained in 93% of patients for wrist extension, 85% of patients for ®nger extension, 90% of patients for thumb extension, 71% of patients for intrinsic reconstruction, and 63% of patients for thumb opposition reconstruction. These results are reasonable but inferior to those obtained by Sundararaj in his study. Surgical intervention offers a very signi®cant improvement in function in these very dif®cult hands. Intensive physiotherapy is required both pre- and postoperatively. Introduction Hansen's disease results from infection with Mycobacterium leprae with subsequent involvement of skin, nerve, and mucosal tissue. Nerve damage occurs in 20±25% of patients.1 In the upper limb the nerve paralysis most frequently affects the ulnar nerve. Median nerve dysfunction may occur later or develop simultaneously, most frequently affecting the distal innervation (simian hand).2 High radial nerve involvement is least common (wrist drop), with 1% of patients having combined ulnar, median, and radial paralysis (triple nerve palsy).1,2 The typical pattern is that of high radial nerve palsy combined with high ulnar nerve and low median nerve loss. -
Painful Brachial Plexopathy: an Unusual Presentation of Polyarteritis Nodosa S
Postgrad Med J: first published as 10.1136/pgmj.58.679.311 on 1 May 1982. Downloaded from Postgraduate Medical Journal (May 1982) 58, 311-313 Painful brachial plexopathy: an unusual presentation of polyarteritis nodosa S. G. ALLAN* H. M. A. TOWLA* M.R.C.P. B. Med. Biol., M.B. C. C. SMITH* A. W. DOWNIE* F.R.C.P. F.R.C.P. J. C. CLARKt M.B., Ch.B. *Department ofMedicine, The Royal Infirmary, Foresterhill, Aberdeen tDepartment ofPathology, University ofAberdeen, Foresterhill Summary intact save for evidence of a right recurrent laryn- An elderly man was admitted to hospital with an geal nerve palsy on indirect laryngoscopy. There unusual painful bilateral brachial neuropathy, which was a flicker of left biceps contraction and grade 2/5 progressed in spite ofhigh dose corticosteroid therapy. power in the deltoids and adductors of the shoulders, Polyarteritis nodosa, characterized by widespread but otherwise total paralysis of the upper limbs. arteriolar involvement, was shown at post-mortem. Marked tenderness was noted over each brachial copyright. plexus, but there was neither muscle tenderness nor fasciculation. 'Glove' loss of all sensory modalities Introduction was present up to the elbows bilaterally, with hypo- The clinical presentations of polyarteritis nodosa tonicity and areflexia. There was minimal hip flexor are classically diverse, reflecting variable vascular weakness and absent left knee and both ankle involvement of many organs and tissues. Mono- reflexes without sensory abnormality or calf tender- neuritis multiplex is the most common neurological ness. Bladder and bowel functions were unaffected sequel (Ford and Siekert, 1965), but asymmetrical and examination otherwise unremarkable. -
Brachial Plexus Injuries: an Interactive Teaching and Learning Academic Model
International Journal of ChemTech Research CODEN (USA): IJCRGG, ISSN: 0974-4290, ISSN(Online):2455-9555 Vol.11 No.03, pp 01-08, 2018 Brachial plexus injuries: An interactive teaching and learning academic model Tarek M. El-gohary1,2*, Samiha M. Abdelkader3 1) Biomechanics Department, Faculty of Physical Therapy, Cairo University, Egypt 1) Board Certified Orthopedic Clinical Specialist, USA 1) Mechanical Diagnosis& Therapy, McKenzie Institute, USA 1) Pediatric Physical Therapy Consultant, NY,NY,USA 2) College of Medical Rehabilitation Sciences, Taibah University, Saudi Arabia 3) Physical Therapy Department, College of Applied Medical Science, King Saud University, Saudi Arabia Abstract : The purpose of this educational paper is to report the feedback from academics and students regarding newly introduced interactive teaching- learning model aiming to master brachial plexus injuries. An interactive questions and answers format was presented to number of academics and students at college of medical rehabilitation sciences. All academics and 90% of students reported that the newly introduced interactive teaching- learning model was helpful. It has been concluded that the interactive teaching- learning model is feasible and self- explanatory to be used and adopted by students and academics to facilitate the educational process. Keywords : Brachial plexus, injuries, teaching, learning, educational model. Introduction Brachial plexus is a group of intertwined nerves that emerge from the spinal cord in the cervical region and travel down the