Diagnostic Classification of Temporomandibular Disorders (Part 2 of 3)I
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DTAP1007_14_16-17_GuzmanGremillion 30.10.2007 15:15 Uhr Seite 1 14 Trends & Applications DENTAL TRIBUNE Asia Pacific Edition TM Disorders: Diagnostic Classification of Temporomandibular Disorders (Part 2 of 3)i Ulises A. Guzman & Henry A. Gremillion, U.S.A. The head, face, masticatory while awake or sleep, are true soreness, autonomic effects tissue, and in this case arising in sytem, and cervical region are considerations in our patient and protective splinting (co- or involving the TM joint. Neo- Editorial Note common sites in which pain evaluation. contraction).8 Although there is plasms can be categorized as is experienced. Many condi- significant evidence that these benign, malignant or metastatic Some of the editorial corrections tions present with similar Myofascial pain is a regional conditions exists, there are few from a distant site. Approxi- intended for Part I of this article signs and characteristic pat- pain, usually dull and achy with reliable clinical characteristics mately 1% of malignant neopla- in Dental Tribune Asia Pacific, terns that may lead to diag- the presence of localized ten- that can be used to distinguish sia metastasize to the jaws.9,10 No. 9 Vol. 5, September 2007 did nostic confusion and ulti- derness in firm bands of mus- them from each other. Squamous cell carcinomas of not make into the final print. mately misdirected care. De- cle, tendons and/or fascia that the head and neck region, Please e-mail r.goodman@dental- fined, validated classification reproduce pain when palpated Myofibrotic contracture re- nasopharyngeal tumors, neo- tribune.com for a corrected PDF systems relating to the multi- and may produce a characteris- fers to the painless shortening plasm arising from the parotid of this article, which will also be plicity of painful entities can tic pattern of regional referred of a muscle. Previous terms gland (adenoid cystic carci- posted to our Web site for those simplify and enhance diag- pain and/or autonomic symp- used include chronic trismus, noma) and mucoepidermoid subscribers who have access. nostic outcomes. Due to the toms on provocation.3,4 Patients muscle fibrosis and muscle carcinomas have been reported The publisher sincerely regrets the errors. rapid advances in our knowl- to extend to the TMJ region edge regarding pain mecha- resulting in pain and alteration nisms and pathways, classifi- of normal function.11,12 Dys- cation systems must be ever “Presently an ideal system function is not usually caused Syndrome is an example of in- evolving, not rigid. Presently by neoplasm.13 complete development.20 Con- an ideal system related to related to masticatory dylar hypoplasia can occur sec- masticatory system disorders Primary tumors known to ondary to trauma, resulting does not exist. have involved the condyle in- from incomplete or underde- system disorders clude osteoma, benign os- velopment of the mandibular One set of diagnostic criteria teoblastoma14, chondroma and condyle. will not satisfy all circum- chondrosarcoma, benign giant stances to which it might be ap- does not exist.” cell tumor, ossifying fibroma, fi- Hyperplasia is the overde- plied. More importantly, many brous dysphasia and myxoma.15 velopment of the cranial bones classifications systems were may complain of muscle stiff- scarring. It is a chronic resist- Malignant neoplasm have been or the mandible. This can be developed for the purpose of ness, acute malocclusion, ear ance to a passive stretch as a re- reported originating from the developmental or acquired. enhancing the formation of symptoms, tinnitus, vertigo, sult of fibrosis of the supporting temporomandibular joint space Hyperplasia can occur as a study populations for clinical toothache, tension-type head- tendons, ligaments or muscle (fibrosarcoma, synovial sar- localized enlargement, such as research endeavors and are not ache and masticatory muscles fibers themselves. The patient coma).16,17,18 in condylar hyperplasia or co- absolutely applicable to every involvement. The most com- usually does not complain of ronoid hyperplasia, or as an clinical case presentation. mon differential diagnoses to pain unless the muscle is ex- Congenital or developmen- overdevelopment of the entire consider includes osteoarthri- tended beyond its functional tal disorders of the cranial mandible or side of the face. For example, the inclusion tis, myositis, myalgia, neoplasia length. There are two basic bones and mandible includes Fibrous dysplasia is a form of criteria for a clinical trial might and fibromyalgia. subcategories: myostatic (re- aplasia (agenesis), hypoplasia, hyperplasia due to a benign, require the presence of all crite- versible condition) and myofi- hyperplasia and neoplasia. Le- slow growing swelling of the ria for a specific disease, while Myositis is inflammation of brotic (irreversible condition). sions and disorders of the jaws mandible and/or maxilla. It is a clinical diagnosis might re- a muscle due to local causes Clinical characteristics include can be either odontogenic or characterized by the presence quire the presence of only a few. such as infection or injury. Pain a limited range of motion, un- non-odontogenic in origin and of fibrous connective tissue. These criteria are meant only is usually acute and in a local- yielding firmness on passive generalized or metastatic in na- to provide clinical guidance for ized area with localized tender- The disease occurs in chil- diagnosis. Final diagnostic de- ness over the entire region of dren and young adults and be- cisions must be based on the the muscle. The inflammation “two basic categories comes inactive when they reach clinical judgment of the health can occur also in the tendinous skeletal maturity. Radiographi- care professional. This article attachment of the muscle, cally the lesion may appear will provide the reader with “tendonitis or tendomyositis”. of TMD exist, extracapsular from an opaque ground-glass to a review of the most accepted Increased pain with mandibu- a lucent appearance, depend- diagnostic classification system lar activity with alteration in (myogenous) and intracapsular ing on the ratio of fibrous tissue related to temporomandibular function due to inflammation to bone. Clinically, usually there disorder (TMD). or pain. Swelling, tissue red- (arthrogenous)” is no displacement of teeth and dening and an increase in tem- the cortical bone and occlusion It is generally recognized perature over the entire muscle remain intact. that two basic categories of can be noticed. The most com- stretch and a history of trauma ture. Most congenital or devel- TMD exist, extracapsular (myo- mon differential diagnoses to or infection is usually reported opmental disorders primarily Disc derangement disorders genous) and intracapsular consider includes myositis, lo- by the patient. The most com- cause problems with esthetics are an abnormal arrangement (arthrogenous). The majority cal myalgia-unclassified and mon differential diagnoses to or function and are rarely accom- of intra-capsular joint parts of TMDs are extracapsular in myofascial pain. consider includes TMJ ankylo- panied by orofacial pain unless causing interference with the nature; however, it is not un- sis and coronoid hypertrophy. associated with Neoplasia (eg, structural relation during common for these two basic Myospasm is an involuntary, osteomyelitis, multiple myeloma, mandibular condyle translation categories to co-exist. sudden, continuous (fascicula- Masticatory muscle neopla- Paget’s disease). Complete age- with mouth opening and clos- tion) tonic contraction of the sia can be benign or malignant nesis is extremely rare.19 ing. In the TM joint this alter- Masticatory muscle-related muscle. Previously used terms and may be associated with pain ation can relate to the elon- conditions are found to be the are trismus, “cramp”. A muscle or not. Neoplasia is defined as Aplasia is a faulty or incom- gation, tear or rupture of the most common subgroup of in spasm is acutely shortened. a new, abnormal or uncon- plete development of the cra- capsule or ligaments causing TMD.1,2 The patient experiences acute trolled growth of muscle tissue nial bones or mandible. Most of a disruption in the disc position pain, a limited range of motion (eg, myxoma). Confirmation the aplasias conditions of the or morphology. The subclassi- The current understanding and often acute malocclusion. must be obtained by biopsy and mandible are categorized un- fication of disc displacement of the complexity and the dy- EMG studies verify sustained imaging. der hemifacial microsomia represents a disc-condyle mis- namic relationship between muscle contraction even at syndromes. The auditory sys- alignment and is subdivided the masticatory and cervical rest.5 The most common dif- Congenital or developmen- tem is frequently affected in into disc displacement with musculature enables the practi- ferential diagnoses to consider tal disorders Most congenital or these syndromes reduction or disc displacement tioner to better assess the condi- includes myositis, local myal- developmental disorders are without reduction.21–23 tion(s) possible etiology(ies). gia-unclassified and neoplasia. not associated with orofacial Hypoplasia is the incom- The individual variations and pain. They can be categorized plete development or underde- Disc displacement with re- demands placed on the system, Local Myalgia-Unclassified as agenesis, hypoplasia, hyper- velopment of cranial bones or duction is characterized by the as well as normal function