Diagnosis and Treatment of Forefoot Disorders. Section 1: Digital Deformities
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Chief Complaint
Chief Complaint Please choose the primary reason you are coming to our office. Complaints are listed alphabetically. Please do not select more than 5 complaints. Upper Back: Thigh/Hip: Calf: o Asthma o Arterial insufficiency o Left calf pain o Bronchitis o Left hip pain o Left leg cramps o Emphysema o Left hip tendonitis o Left leg numbness o Left Flank Pain o Left leg cramps o Left leg pain o Midback pain o Left leg numbness o Left leg weakness o Left leg pain o Leg cramps Lower Back: o Left leg weakness o Leg numbness o Fatigue o Left post. thigh pain o Leg weakness o Left flank pain o Left thigh pain o Varicose veins o Low back pain o Sciatica o Venous insufficiency o Low back spasm o Venous insufficiency o Arterial insufficiency o Lumbar arthritis o Right hip pain o Right calf pain o Menstrual cramps o Right hip tendonitis Right leg o Right leg cramps o Nervousness cramps o Right leg numbness o Pain during BM o Right leg numbness o Right leg pain o Right flank pain o Right leg pain o Right leg weakness o Sacroiliac pain o Right leg weakness o Sciatica o Right post. thigh pain Neck: o Stiffness o Right thigh pain o Bronchitis o Whole body pain o Clavicular pain Head: Buttocks: o Cold o Agitation o Bleeding during BM o Coughing o Anxiety attack o Bursitis of hip o Dysphagia o Cold o Gluteal pain o Goiter o Diminished concentration o Hemorrhoids o Hoarseness o Dizziness o Left gluteal pain o Neck pain o Dysphagia o Left hip pain o Neck spasm o Ear pain o Left post. -
Medical Policy Ultrasound Accelerated Fracture Healing Device
Medical Policy Ultrasound Accelerated Fracture Healing Device Table of Contents Policy: Commercial Coding Information Information Pertaining to All Policies Policy: Medicare Description References Authorization Information Policy History Policy Number: 497 BCBSA Reference Number: 1.01.05 Related Policies Electrical Stimulation of the Spine as an Adjunct to Spinal Fusion Procedures, #498 Electrical Bone Growth Stimulation of the Appendicular Skeleton, #499 Bone Morphogenetic Protein, #097 Policy Commercial Members: Managed Care (HMO and POS), PPO, and Indemnity Members Low-intensity ultrasound treatment may be MEDICALLY NECESSARY when used as an adjunct to conventional management (i.e., closed reduction and cast immobilization) for the treatment of fresh, closed fractures in skeletally mature individuals. Candidates for ultrasound treatment are those at high risk for delayed fracture healing or nonunion. These risk factors may include either locations of fractures or patient comorbidities and include the following: Patient comorbidities: Diabetes, Steroid therapy, Osteoporosis, History of alcoholism, History of smoking. Fracture locations: Jones fracture, Fracture of navicular bone in the wrist (also called the scaphoid), Fracture of metatarsal, Fractures associated with extensive soft tissue or vascular damage. Low-intensity ultrasound treatment may be MEDICALLY NECESSARY as a treatment of delayed union of bones, including delayed union** of previously surgically-treated fractures, and excluding the skull and vertebra. 1 Low-intensity ultrasound treatment may be MEDICALLY NECESSARY as a treatment of fracture nonunions of bones, including nonunion*** of previously surgically-treated fractures, and excluding the skull and vertebra. Other applications of low-intensity ultrasound treatment are INVESTIGATIONAL, including, but not limited to, treatment of congenital pseudarthroses, open fractures, fresh* surgically-treated closed fractures, stress fractures, arthrodesis or failed arthrodesis. -
ICD-9CM Coding Achilles Bursitis Or Tendinitis 726.71 Adhesive
ICD-9CM CODING OF COMMON CHIROPRACTIC CONDITIONS CONDITION ICD-9CM coding Achilles bursitis or tendinitis 726.71 Adhesive capsulitis of shoulder 726 Anklyosing Spondylitis (spine only) 720 Anterior cruciate ligament (old disruption) 717.83 Bicipital tenosynovitis 726.12 Boutonniere deformity 736.21 Brachial neuritis or radiculitis 723.4 Bunion 727.1 Bursitis of knee 726.6 Calcaneal spur 726.73 Carpal tunnel syndrome 354 Cervical radiculitis 723.4 Cervical spondylosis with myelopathy 721.1 Cervical spondylosis without myelopathy 721 Cervicalgia 723.1 Chondromalacia of patella 717.7 Claw hand (acquired) 736.06 Claw toe (acquired) 735.5 Coccygodynia 724.79 Coxa plana 732.1 Coxa valga (acquired) 736.31 Coxa vara (acquired) 736.32 de Quervain's disease 727.04 Degeneration of cervical intervertebral disc 722.4 Degeneration of thoracic or lumbar intervertebral disc 722.5 Disc Degeneration (Cervical with myelopathy) 722.71 Disc Degeneration (Lumbar with myelopathy) 722.73 Disc Degeneration (Thoracic) 722.51 Disc Degeneration (Cervical) 722.4 Disc Degeneration (Lumbar) 722.52 Disc Degeneration (Thoracic with myelopathy) 722.72 Disc displacement without myelopathy (Thoracic) 722.11 Disc displacement of without myelopathy (Cervical ) 722 Disc displacement without myelopathy (Lumbar) 722.1 Dupuytren's contracture 728.6 Entrapment syndromes 354.0-355.9 Epicondylitis (Lateral) 726.32 Epicondylitis (Medial) 726.31 Flat foot-Pes planus (acquired) 734 Fracture (Lumbar) 805.4 Ganglion of tendon sheath 727.42 Genu recurvatum (acquired) 736.5 Genu valgum -
Knee Osteoarthritis
BRIGHAM AND WOMEN’S HOSPITAL Department of Rehabilitation Services Standard of Care: _Osteoarthritis of the Knee Case Type / Diagnosis: Knee Osteoarthritis. ICD-9: 715.16, 719.46 Osteoarthritis/Osteoarthrosis (OA) is the most common joint disease causing disability, affecting more than 7 million people in the United States 1. OA is a disease process of axial and peripheral joints. It is characterized by progressive deterioration and loss of articular cartilage and by reactive bone changes at the margins of the joints and in the subchondral bone. Clinical manifestations are characterized by slowly developing joint pain, stiffness, and joint enlargement with limitations of motion. Knee osteoarthritis (OA) results from mechanical and idiopathic factors that alter the balance between degradation and synthesis of articular cartilage and subchondral bone. The etiology of knee OA is not entirely clear, yet its incidence increases with age and in women. 1 The etiology may have genetic factors affecting collagen, or traumatic factors, such as fracture or previous meniscal damage. Obesity is a risk factor for the development and progression of OA. Early degenerative changes predict progression of the disease. Underlying biomechanical factors, such as varum or valgum of the tibial femoral joint may predispose people to OA. However Hunter et al 2reported knee alignment did not predict OA, but rather was a marker of the disease severity. Loss of quadriceps muscle strength is associated with knee pain and disability in OA. Clinical criteria for the diagnosis of OA of the knee has been established by Altman3 Subjects with examination finding consistent with any of the three categories were considered to have Knee OA. -
A Case Report on Charcot-Marie-Tooth Disease with a Novel Periaxin Gene Mutation
Open Access Case Report DOI: 10.7759/cureus.5111 A Case Report on Charcot-Marie-Tooth Disease with a Novel Periaxin Gene Mutation Sorabh Datta 1 , Saurabh Kataria 1 , Raghav Govindarajan 1 1. Neurology, University of Missouri, Columbia, USA Corresponding author: Sorabh Datta, [email protected] Abstract Charcot-Marie-Tooth (CMT) disease is one of the most common primary hereditary neuropathies causing peripheral neuropathies. More than 60 different gene mutations are causing this disease. The PRX gene codes for Periaxin proteins that are expressed by Schwann cells and are necessary for the formation and maintenance of myelination of peripheral nerves. Dejerine-Sottas neuropathy and Charcot-Marie-Tooth type 4F (CMT4F) are the two different clinical phenotypes observed in association with PRX gene mutation. This article describes a case of an elderly male with a novel mutation involving the PRX gene. Categories: Genetics, Internal Medicine, Neurology Keywords: neurology, sensorimotor neuropathy, congenital, gene expression, genetic mutation, protein, pes cavus, demyelinating diseases, charcot-marie-tooth, autosomal recessive disorder Introduction As per the Dyck classification in the year 1970, primary hereditary neuropathies are divided into hereditary motor sensory neuropathy (HMSN) and hereditary sensory autonomic neuropathy (HSAN) [1]. Charcot- Marie-Tooth (CMT) disease is a type of HMSN with an estimated prevalence of 1 in 2,500 [2]. CMT can follow autosomal recessive (ARCMT), X-linked recessive, and also an autosomal dominant pattern. CMT type 4 is a rapidly increasing ARCMT disease form in HMSN, although CMT type 1 and 2 still account for the most substantial proportion of the patient population [3]. CMT4F is a severe, demyelinating subtype of CMT type 4 and is characterized by childhood onset of slowly progressing weakness in the distal muscles associated with atrophy. -
Knee Evaluation
Evaluation of Knee Injuries Dr. Alan A. Zakaria, D.O., M.S. 1080 Kirts Blvd., Suite 400 Troy, Mi., 48084 Team Physician United States Soccer Federation University of Michigan Men’s and Women’s Soccer Objective Identify main anatomic components of the knee Perform basic knee exam along with special tests Identify common knee injury patterns and their physical exam findings. Anatomy ➢ Bony Anatomy ➢ Ligaments ➢ Cartilage ➢ Musculature ➢ Other Soft Tissue Knee Anatomy Two functional joints – Femorotibial – Femoropatellar Femoral condyles – Flex/extend Knee Anatomy Patella – Sesamoid with two concave surfaces and vertical ridge – Increases efficiency of extension Knee Anatomy: Anterior Cruciate Ligament (ACL) Run inferior, anterior, and medially Arises from medial aspect lateral femoral condyle Insert lateral to medial tibial eminence Restrains anterior subluxation of tibia on femur Knee Anatomy: Posterior Cruciate Ligament (PCL) Arises from the posterior intercondylar area of the tibia Inserts at the medial condyle of the femur Restrains posterior subluxation of the tibia on the femur Knee Anatomy: Medial Collateral Ligament (MCL) Postero-superior medial femoral condyle to proximal end of tibia Maximum tension at full extension Restraint to valgus stress Knee Anatomy: Lateral Collateral Ligament (LCL) Posterosuperior lateral femoral condyle to lateral head of fibula Restraint to varus stress Knee Anatomy: Meniscus Load bearing, joint stability, shock absorption Peripheral third vascularized Knee Anatomy: Articular Cartilage Hyaline cartilage -
Cavus Foot, from Neonates to Adolescents
Orthopaedics & Traumatology: Surgery & Research (2012) 98, 813—828 Available online at www.sciencedirect.com REVIEW ARTICLE ଝ Cavus foot, from neonates to adolescents P. Wicart Paris Descartes University, Necker—Sick Children Hospital (AP—HP), 149, rue de Sèvres, Paris 75015, France Accepted: 10 July 2012 KEYWORDS Summary Pes cavus, defined as a high arch in the sagittal plane, occurs in various clinical situa- Pes cavus; tions. A cavus foot may be a variant of normal, a simple morphological characteristic, seen in Charcot-Marie-Tooth healthy individuals. Alternatively, cavus may occur as a component of a foot deformity. When it disease; is the main abnormality, direct pes cavus should be distinguished from pes cavovarus. In direct Neurology; pes cavus, the deformity occurs only in the sagittal plane (in the forefoot, hindfoot, or both). Morphological types Direct pes cavus may be related to a variety of causes, although neurological diseases predom- inate in posterior pes cavus. Pes cavovarus is a three-dimensional deformity characterized by rotation of the calcaneopedal unit (the foot minus the talus). This deformity is caused by palsy of the intrinsic foot muscles, usually related to Charcot-Marie-Tooth disease. The risk of pro- gression during childhood can be eliminated by appropriate conservative treatment (orthosis to realign the foot). Extra-articular surgery is indicated when the response to orthotic treatment is inadequate. Muscle transfers have not been proven effective. Triple arthrodesis (talocalcanear, talonavicular, and calcaneocuboid) accelerates the mid-term development of osteoarthritis in the adjacent joints and should be avoided. © 2012 Published by Elsevier Masson SAS. Introduction The cavus may be either one of the components or the main component of the deformity (Tables 1 and 2). -
Supplemental Information
REVIEW ARTICLE Supplemental Information SEARCH STRATEGIES 7. exp Congenital Abnormalities/ or remifentanil or sufentanil or 8. (defect or cleft or heart defect tapentadol or tramadol or heroin Database: Ovid MEDLINE(R) In- or nalmefene or naloxone or Process and Other Nonindexed or gastroschisis or cryptorchidism or atresia or congenital or clubfoot naltrexone).mp. Citations and Ovid MEDLINE(R), or renal or craniosynostosis or 4. 1 or 2 or 3 1946 to Present hypospadias or malformation or 5. exp pregnancy/or exp pregnancy spina bifida or neural tube defect). outcome/ mp. 1. exp Analgesics, Opioid/ 6. exp teratogenic agent/ 9. 5 or 6 or 7 or 8 2. (opioid* or opiate*).mp. 7. exp congenital disorder/ 10. 4 and 9 3. (alfentanil or alphaprodine or 11. Limit 10 to (English language and 8. (defect or cleft or heart defect buprenorphine or butorphanol humans) or gastroschisis or cryptorchidism or codeine or dezocine or or atresia or congenital or clubfoot dihydrocodeine or fentanyl or Database: Ovid Embase, 1988– or renal or craniosynostosis or hydrocodone or hydromorphone 2016, Week 7 hypospadias or malformation or or levomethadyl or levorphanol spina bifida or neural tube defect). or meperidine or methadone or mp. 1. exp opiate/ morphine or nalbuphine or opium 9. 5 or 6 or 7 or 8 or oxycodone or oxymorphone 2. (opioid* or opiate*).mp. or pentazocine or propoxyphene 10. 4 and 9 3. (alfentanil or alphaprodine or or remifentanil or sufentanil or buprenorphine or butorphanol 11. Limit 10 to (human and English tapentadol or tramadol or heroin or codeine or dezocine or language and (article or book or or nalmefene or naloxone or book series or conference paper dihydrocodeine or fentanyl or “ ” naltrexone).mp. -
ICD-9 to ICD-10 Mapping Tool Courtesy Of: the Paperwork Project
ICD-9 to ICD-10 Mapping Tool Courtesy of: The Paperwork Project Spinal Subluxation ICD-9 ICD-10 M99.00 Segmental and somatic dysfunction, Head region (occipito-cervical) 739.0 Segmental and somatic dysfunction, Head region (occipito-cervical) M99.10 Subluxation complex (vertebral), Head region M99.01 Segmental and somatic dysfunction, Cervical region 739.1 Segmental and somatic dysfunction, Cervical region M99.11 Subluxation complex (vertebral), Cervical region M99.02 Segmental and somatic dysfunction, Thoracic region 739.2 Segmental and somatic dysfunction, Thoracic region M99.12 Subluxation complex (vertebral), Thoracic region M99.03 Segmental and somatic dysfunction, Lumbar region 739.3 Segmental and somatic dysfunction, Lumbar region M99.13 Subluxation complex (vertebral), Lumbar region M99.04 Segmental and somatic dysfunction, Sacral region 739.4 Segmental and somatic dysfunction, Sacral region M99.14 Subluxation complex (vertebral), Sacral region M99.05 Segmental and somatic dysfunction, Sacroiliac, hip, pubic regions 739.5 Segmental and somatic dysfunction, Sacroiliac, hip, pubic regions M99.15 Subluxation complex (vertebral), Pelvic region 839.08 Closed dislocation, Multiple cervical vertebra (injury) S13.101_ Dislocation of unspecified cervical vertebra (injury) ** 839.20 Closed dislocation, Lumbar vertebra (injury) S33.101_ Dislocation of unspecified lumbar vertebra (injury) ** 839.21 Closed dislocation, Thoracic vertebra (injury) S23.101_ Dislocation of unspecified thoracic vertebra (injury) ** 839.42 Closed dislocation, Sacrum, -
Billing and Coding: Injections - Tendon, Ligament, Ganglion Cyst, Tunnel Syndromes and Morton's Neuroma (A57079)
Local Coverage Article: Billing and Coding: Injections - Tendon, Ligament, Ganglion Cyst, Tunnel Syndromes and Morton's Neuroma (A57079) Links in PDF documents are not guaranteed to work. To follow a web link, please use the MCD Website. Contractor Information CONTRACTOR NAME CONTRACT TYPE CONTRACT JURISDICTION STATE(S) NUMBER Noridian Healthcare Solutions, A and B MAC 01111 - MAC A J - E California - Entire State LLC Noridian Healthcare Solutions, A and B MAC 01112 - MAC B J - E California - Northern LLC Noridian Healthcare Solutions, A and B MAC 01182 - MAC B J - E California - Southern LLC Noridian Healthcare Solutions, A and B MAC 01211 - MAC A J - E American Samoa LLC Guam Hawaii Northern Mariana Islands Noridian Healthcare Solutions, A and B MAC 01212 - MAC B J - E American Samoa LLC Guam Hawaii Northern Mariana Islands Noridian Healthcare Solutions, A and B MAC 01311 - MAC A J - E Nevada LLC Noridian Healthcare Solutions, A and B MAC 01312 - MAC B J - E Nevada LLC Noridian Healthcare Solutions, A and B MAC 01911 - MAC A J - E American Samoa LLC California - Entire State Guam Hawaii Nevada Northern Mariana Created on 09/28/2019. Page 1 of 33 CONTRACTOR NAME CONTRACT TYPE CONTRACT JURISDICTION STATE(S) NUMBER Islands Article Information General Information Original Effective Date 10/01/2019 Article ID Revision Effective Date A57079 N/A Article Title Revision Ending Date Billing and Coding: Injections - Tendon, Ligament, N/A Ganglion Cyst, Tunnel Syndromes and Morton's Neuroma Retirement Date N/A Article Type Billing and Coding AMA CPT / ADA CDT / AHA NUBC Copyright Statement CPT codes, descriptions and other data only are copyright 2018 American Medical Association. -
5Th Metatarsal Fracture
FIFTH METATARSAL FRACTURES Todd Gothelf MD (USA), FRACS, FAAOS, Dip. ABOS Foot, Ankle, Shoulder Surgeon Orthopaedic You have been diagnosed with a fracture of the fifth metatarsal bone. Surgeons This tyPe of fracture usually occurs when the ankle suddenly rolls inward. When the ankle rolls, a tendon that is attached to the fifth metatarsal bone is J. Goldberg stretched. Because the bone is weaker than the tendon, the bone cracks first. A. Turnbull R. Pattinson A. Loefler All bones heal in a different way when they break. This is esPecially true J. Negrine of the fifth metatarsal bone. In addition, the blood suPPly varies to different I. PoPoff areas, making it a lot harder for some fractures to heal without helP. Below are D. Sher descriPtions of the main Patterns of fractures of the fifth metatarsal fractures T. Gothelf and treatments for each. Sports Physicians FIFTH METATARSAL AVULSION FRACTURE J. Best This fracture Pattern occurs at the tiP of the bone (figure 1). These M. Cusi fractures have a very high rate of healing and require little Protection. Weight P. Annett on the foot is allowed as soon as the Patient is comfortable. While crutches may helP initially, walking without them is allowed. I Prefer to Place Patients in a walking boot, as it allows for more comfortable walking and Protects the foot from further injury. RICE treatment is initiated. Pain should be exPected to diminish over the first four weeks, but may not comPletely go away for several months. Follow-uP radiographs are not necessary if the Pain resolves as exPected. -
Clinical Guidelines: Foot / Ankle
Clinical Guidelines: Foot / Ankle Plantar Fasciitis/Heel spurs: Initial Evaluation: History includes usually atraumatic plantar medial heel pain, worst first thing in the morning or after prolonged sitting. Exam includes tenderness with deep palpation of the plantar medial heel. Squeezing the heel bone side to side is NOT tender, but if present could represent a calcaneal stress fracture. X-rays may or may not reveal a heel spur, but the spur is NOT the source of the pain despite podiatry frequently referring to this as “heel spur syndrome.” Follow-up: The plantar fascia is the soft tissue under our foot that runs from the heel to the toes, much like the palm of our hand; it is the sole of our foot. The plantar fascia stretch includes crossing your legs and dorsiflexing the ankle and stretching the toes into extension. This is the most effective stretch. A night splint is imperative for improvement and should be used at night for 6 weeks. Cortisone injections and physical therapy can be helpful. NSAIDS and a frozen water bottle rolled on the plantar foot could be used with the above treatment, but the most effective treatment is a night splint. Referral: 90% of heel pain resolves with non-op treatment, but make a referral to a foot / ankle ortho surgeon with any atypical heel pain or failure of 6-8 weeks of non-operative treatment. Atypical heel pain usually gets an MRI, but classic plantar fasciitis does not. Bunion (hallux valgus): Initial Treatment: Bunion deformity includes a bump on the medial side of the big toe, the big toe going the wrong way, and a widened forefoot.