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An Unusual Presentation of Subfrontal Meningioma: a Case Report and Literature Review for Foster Kennedy Syndrome
Intern Emerg Med (2011) 6:267–269 DOI 10.1007/s11739-010-0437-y CE - MEDICAL ILLUSTRATION An unusual presentation of subfrontal meningioma: a case report and literature review for Foster Kennedy syndrome Shahram Lotfipour • Kris Chiles • J. Akiva Kahn • Tareg Bey • Scott Rudkin Received: 17 December 2009 / Accepted: 13 July 2010 / Published online: 26 August 2010 Ó SIMI 2010 Introduction head trauma. She admitted to abusing crack cocaine for 13 years with her last use 4 months ago. She denied any Foster Kennedy syndrome, named after neurologist Robert trouble with ambulation, dizziness, and changes in hearing or Foster Kennedy (1884–1952), describes unilateral ipsilat- other alterations in sensation. She denied any suicidal or eral optic atrophy and contralateral papilledema from an homicidal ideation. The patient denied any auditory halluci- intracranial mass. This syndrome is unreliably associated nations, but did report that she had been experiencing with anosmia and ipsilateral proptosis [1]. It originates visual hallucinations and visual disturbances for at least from variety of intracranial pathologies, but most often a 6–8 months. She reported complete blindness in the left eye, subfrontal mass. We present a case of Foster Kennedy and shadow perception in her right for an unknown length of syndrome and review its etiology, pathology and incidence time. Her past medical history was notable for major in intracranial tumors. depression. The patient did not have a previous history of hallucinations or psychosis, and had never been hospitalized for psychiatric reasons. The patient was not on any medica- Case report tions, and was allergic to penicillin and codeine. -
An Algorithm to Detect Kayser-Fleischer Ring in Human Eye for Diagnosing Wilson Disease
ISSN: 2319-8753 International Journal of Innovative Research in Science, Engineering and Technology (An ISO 3297: 2007 Certified Organization) Vol. 3, Issue 5, May 2014 An Algorithm to Detect Kayser-Fleischer Ring in Human Eye for Diagnosing Wilson Disease 1S.Tharageshwari, 2 D.Sasikala 1PG scholar, Department Of ECE, Vivekanandha College of Engineering For Women, Namakkal , Tamil nadu, India. 2Associate Professor, Department Of ECE, Vivekanandha College of Engineering For Women, Namakkal, Tamil nadu, India. ABSTRACT-An eye image is segmented by JSEG (J measure based segmentation) algorithm without the manual parameter adjustment and simplifies texture and color for detecting the Kayser-Fleischer ring in diagnosing Wilson Disease. Segmentation with this algorithm passes through two major stages, namely color quantization and spatial segmentation as first stage and region growing and region merging as secondary stage. The biometric measurement provides information on the percentage of the extent of the cornea tissue affected from the copper accumulation. This algorithm detects the presence of symptoms reducing occurrence of false-negative diagnoses and improves accuracy of actual methods used in practice like slit lamp method. The described techniques reduces possible interpretation errors and assists doctor to diagnose the pathology. INDEX TERMS – Wilson disease, Kayser-Fleischer Ring, segmentation, Biometric measurement. I. INTRODUCTION Wilson disease is an autosomal recessive genetic disorder that prevent the body from getting rid of extra copper. A small amount of copper obtained from food is needed to stay healthy, but too much copper is poisonous. When the copper storage capacity of the liver is surpassed, copper is passed into the bloodstream and travels to the other organs-including the brain, kidney, and eyes. -
Cataract Influence on Iris Recognition Performance
Cataract influence on iris recognition performance Mateusz Trokielewicz1,2, Adam Czajka2,1, Piotr Maciejewicz3 1Biometrics Laboratory, Research and Academic Computer Network, Wawozowa 18, 02-796 Warsaw, Poland; 2Institute of Control and Computation Engineering, Warsaw University of Technology, Nowowiejska 15/19, 00-665 Warsaw, Poland; 3Department of Ophthalmology, Medical University of Warsaw, Lindleya 4, 02-005 Warsaw, Poland ABSTRACT This paper presents the experimental study revealing weaker performance of the automatic iris recognition methods for cataract-affected eyes when compared to healthy eyes. There is little research on the topic, mostly incorporating scarce databases that are often deficient in images representing more than one illness. We built our own database, acquiring 1288 eye images of 37 patients of the Medical University of Warsaw. Those images represent several common ocular diseases, such as cataract, along with less ordinary conditions, such as iris pattern alterations derived from illness or eye trauma. Images were captured in near-infrared light (used in biometrics) and for selected cases also in visible light (used in ophthalmological diagnosis). Since cataract is a disorder that is most populated by samples in the database, in this paper we focus solely on this illness. To assess the extent of the performance deterioration we use three iris recognition methodologies (commercial and academic solutions) to calculate genuine match scores for healthy eyes and those influenced by cataract. Results show a significant degradation in iris recognition reliability manifesting by worsening the genuine scores in all three matchers used in this study (12% of genuine score increase for an academic matcher, up to 175% of genuine score increase obtained for an example commercial matcher). -
Annual Report Research Activity 2019
Annual Report Research Activity 2019 Division of Clinical Neuroscience University of Oslo and Oslo University Hospital 0 Contents Oslo University Hospital and the University of Oslo .................................................................................... 4 From Division Director Eva Bjørstad ........................................................................................................... 4 Division of Clinical Neuroscience (NVR) Organizational Chart ..................................................................... 5 Department of Physical Medicine and Rehabilitation Rehabilitation after trauma....................................................................................................................... 6 Group Leader: Nada Andelic Painful musculoskeletal disorders .............................................................................................................. 9 Group Leader: Cecilie Røe Department of Refractory Epilepsy - National Centre for Epilepsy Complex epilepsy .................................................................................................................................... 11 Group Leader: Morten Lossius Department of Neurosurgery Neurovascular-Hydrocephalus Research Group ..................................................................................... 16 Group Leader: Per Kristian Eide Oslo Neurosurgical Outcome Study Group (ONOSG) ................................................................................. 19 Group Leaders: Eirik Helseth and Torstein -
References Briskly Compared with the Fellow Eye
LETTERS TO THE JOURNAL 367 frontal tumour. The optic atrophy is commonly felt to Sir, result from optic nerve compression and the contralateral Apraclonidine in the Management of Glaucomatocy 1.2 papilloedema from increased intracranial pressure. clitic crisis Another mechanism suggests that Foster Kennedy syn Glaucomatocyclitic cnSlS (Posner-Schlossman syn drome is due to bilateral direct optic nerve compression by drome) is a unilateral inflammation of the uveal tract in a midline basal mass or less commonly by long-standing which signs of an acute increase in intraocular pressure increased intracranial pressure without direct com predominate. As the aetiology is doubtful, numerous treat pression of either nerve.3 ments have been suggested, the main aim being to reduce Since the early cases of Foster Kennedy syndrome, the exceptionally high intraocular pressure which, left many cases have been reported in the literature caused by untreated, will cause permanent optic nerve damage. other tumours, especially meningiomas such as olfactory Apraclonidine hydrochloride I %, a clonidine deriva groove and sphenoid ridge meningiomas, with gliomas tive and a peripheral alpha-adrenergic agonist. was devel occasionally reported.�-7 To our knowledge, nasopharyn oped to lower intraocular pressure while minimising geal carcinoma is rarely reported in the literature as a systemic side effects. It has specificrecept or-binding and cause of Foster Kennedy syndrome. physico chemical properties that limit its access to the cen Other terms have been used in the literature to describe tral nervous system. In normal human volunteers it pro atypical cases of Foster Kennedy syndrome. 'Pseudo Fos duces a significant fall in intraocular pressure. -
20-OPHTHALMOLOGY Cataract-Ds Brushfield-Down Synd Christmas
20-OPHTHALMOLOGY cataract-ds BrushfielD-Down synd christmas tree-myotonic dystrophy coronaRY-pubeRtY cuneiform-cortical(polyopia) cupuliform-post subcapsular(max vision loss) Elschnig pearl, ring of Soemmering-after(post capsule) experimenTal-Tyr def glassworker-infrared radiation grey(soft), yellow, amber, red(cataracta rubra), brown(cataracta brunescence), black(cat nigrans)(GYARBB)-nuclear(hard) heat-ionising radiation Membranous-HallerMan Streiff synd morgagnian-hypermature senile oildrop(revers)-galactossemia(G1PUT def) post cortical/bread crumb/polychromatic lustre/rainbow-complicated post polar-PHPV(persistent hyperplastic prim vitreous) radiational-post subcapsular riders-zonular/lamellar(vitD def, hypoparathy) roseTTe(ant cortex)-Trauma, concussion shield-atopic dermatitis snowstorm/flake-juvenile DM(aldose reductase def, T1>T2, sorbitol accumulat) star-electrocution sunflower/flower of petal-Wilson ds, chalcosis, penetrating trauma syndermatotic-atopic ds total-cong rubella zonular-galactossemia(galactokinase def) stage of cataract lamellar separation incipient/intumescence(freq change of glass) immature mature hypermature Aim4aiims.inmorgagnian sclerotic lens layer ant capsule ant epithelium lens fibre[66%H2O, 34%prot-aLp(Largest), Bet(most aBundant), γ(crystalline, soluble)] nucleus embryonic(0-3mthIUL) fetal(3-8mthIUL)-Y shape(suture) infantile(8mthIUL-puberty) adult(>puberty) cortex post capsule thinnest-post pole>ant pole thickest, most active cell-equator vitA absent in lens vitC tpt in lens by myoinositol H2O tpt in lens -
Fluorescein Angiography Reference Number: OC.UM.CP.0028 Coding Implications Last Review Date: 05/2020 Revision Log
Clinical Policy: Fluorescein Angiography Reference Number: OC.UM.CP.0028 Coding Implications Last Review Date: 05/2020 Revision Log See Important Reminder at the end of this policy for important regulatory and legal information. Description Intravenous Fluorescein Angiography (IVFA) or fluorescent angiography is a technique for examining the circulation of the retina and choroid using a fluorescent dye and a specialized camera. It involves injection of sodium fluorescein into the systemic circulation, and then an angiogram is obtained by photographing the fluorescence emitted after illumination of the retina with blue light at a wavelength of 490 nanometers. This policy describes the medical necessity guidelines for fluorescein angiography. Policy/Criteria I. It is the policy of health plans affiliated with Envolve Vision, Inc.® that fluorescein angiography is medically necessary for the following indications: A. Initial evaluation of a patient with abnormal findings of the fundus / retina on ophthalmoscopy exam including one of the following: 1. Choroidal Neovascular Membranes (CNVM) 2. Lesions of the Retinal Pigment Epithelium (RPE) a. Serous Detachment of the RPE b. Tears or rips of the RPE c. Hemorrhagic detachment 3. Fibrovascular Disciform Scar 4. Vitreous Hemorrhage (patient presents with sudden loss of vision) 5. Drusen 6. Diabetic Retinopathy B. Evaluation of patient presenting with symptoms of sudden vision loss (especially central vision), blurred vision, distortion, etc., which may suggest a subretinal neovascularization and abnormal findings of the fundus / retina on ophthalmoscopy exam. C. Evaluation of patients with nonproliferative (background) and proliferative diabetic retinopathy without macular edema. Frequency is determined by disease progression and the treatment performed. Fluorescein angiography may be performed on the treated eye only at 6 weeks post-treatment and as often as every 8-12 weeks to assist in management of the retinopathy. -
A Case of Foster Kennedy Syndrome in a Pregnant Lady Presenting with Unilateral Deterioration of Vision
Mehmood A, et al., J Ophthalmic Clin Res 2021, 8: 076 DOI: 10.24966/OCR-8887/100076 HSOA Journal of Ophthalmology & Clinical Research Case Report last 4 months but over the last week before presentation, it had fallen A Case of Foster Kennedy precipitously to counting fingers. The vision in her right eye has also deteriorated over the preceding week together with frontal headaches Syndrome in a Pregnant Lady but not associated with nausea and vomiting. On examination, she was generally well, afebrile, alert, and oriented. Visual acuities were Presenting with Unilateral 6/18 in her right eye and counting fingers in the left eye not improving with pinhole or refraction. On slit lamp, anterior segment examination Deterioration of Vision of both eyes was unremarkable and fundal examination revealed Asif Mehmood1, Farooq Ul Abidin2* and Sharjeel Khan3 marked papilloedema on the right eye and optic atrophy on the left eye (Figure 1A,1B). There was no relative afferent pupillary defect, 1Consultant Ophthalmologist, Rehman medical institute, Peshawar, Pakistan no proptosis, no bruit, and extraocular movements of both the eyes 2Resident Ophthalmology, AFIO, Rawalpindi, Pakistan were full range. There were no other focal or generalized neurological 3Department of Ophthalmology, Dera Ismail Khan, Pakistan signs. Abstract Foster Kennedy syndrome is a rare neurological entity that includes ipsilateral optic atrophy, contralateral papilledema, and sometimes anosmia. The syndrome has been described in association with a variety of intracranial pathologies such as a large frontal lobe tumor, olfactory groove meningioma, or medial third sphenoidal wing meningioma. In this report, we present a case of sphenoidal wing meningioma with Foster Kennedy syndrome in a 25-year-old pregnant female. -
Neuroradiology for Ophthalmologists
Eye (2020) 34:1027–1038 https://doi.org/10.1038/s41433-019-0753-z REVIEW ARTICLE Neuroradiology for ophthalmologists 1 2 1 1,3,4,5,6,7 Bayan Al Othman ● Jared Raabe ● Ashwini Kini ● Andrew G. Lee Received: 3 June 2019 / Revised: 29 October 2019 / Accepted: 24 November 2019 / Published online: 2 January 2020 © The Author(s), under exclusive licence to The Royal College of Ophthalmologists 2020 Abstract This article will review the best approaches to neuroimaging for specific ophthalmologic conditions and discuss characteristic radiographic findings. A review of the current literature was performed to find recommendations for the best approaches and characteristic radiographic findings for various ophthalmologic conditions. Options for imaging continue to grow with modern advances in technology, and ophthalmologists should stay current on the various radiographic techniques available to them, focusing on their strengths and weaknesses for different clinical scenarios. Introduction Computed tomography (CT) 1234567890();,: 1234567890();,: Modern imaging technology continues to advance the CT imaging reconstructs a three-dimensional image made boundaries and increase the options available to physicians of many conventional x-ray images. The conventional x-ray with respect to neuroradiology. In ophthalmology, the most images are ordered in tomographic slices that have been common studies employed are computed tomography (CT) computerized so a viewer can scroll through the images, and magnetic resonance imaging (MRI). There are several analyzing sequential cross-sections [1]. Density is the pri- different types of protocols that provide unique advantages mary characteristic that determines image appearance on a and disadvantages depending on the clinical scenario. This CT scan. As with traditional x-rays, tissues appear on a grey article endeavours to review those options and discuss how scale ranging from white (i.e., hyperdense tissues such as they are best employed to evaluate a variety of specific bone) to black (i.e., hypodense materials such as air). -
Astrocytic Tumors
Neuro-ophthalmologic Examination of the Neurosurgical Patients Amgad Hanna, MD and Peter Savino, MD Departments of Neurosurgery and Ophthalmology Thomas Jefferson University December 2005 Agenda • Abnormal pupils: miosis and mydriasis • Trochlear N palsy • Fundi: NL and abnormal Abnormal pupils Miosis Not all constricted pupils are caused by carotid dissection V1 Postganglionic (3rd order) Horner’s Long Cil Nn Central (1st order) Horner’s Preganglionic (2nd order) Horner’s Causes of Horner’s Syndrome (usually have associated signs and symptoms) Cocaine NO Mydriasis response NE NE Blocks reuptake NE NE Of NE (90%) NO NE outside vesicles True Pseudo-Horner’s Horner’s (physiologic anisocoria) (MRI/A H/N – CT Chest) True Horner’s Hydroxyamphetamine test No response Mydriasis NE NE Release of NE from No NE NE NE inside vesicles synaptic vesicles NE Postganglionic Central or Horner’s Preganglionic Horner’s Physiologic Anisocoria Central or Preganglionic R Horner’s Postganglionic R Horner’s Case • 40 y/o Woman • C/O R swollen eyelid and H/A Upper lid (Muller’s m), lower lid (Lid retractis) Dim Light Post-Cocaine; True Horner’s Post-Hydroxyamphetamine; Postganglionic Horner’s Carotid Dissection Mydriasis Not all dilated pupils are caused by P Com aneurysms Parasympathetic and light reflex Posterior commissure III Inf div Short Cil Nn N to inf obl Physiologic anisocoria • Normal reaction to light • The size difference between both eyes remains the same with dim and bright light • 0.3 – 0.4 mm difference found in 50% of the normal population • Up -
Fibrillary Lines of the Cornea a Clinical Sign in Keratoconus
Brit. J. Ophthal. (I975) 59, I 36 Br J Ophthalmol: first published as 10.1136/bjo.59.3.136 on 1 March 1975. Downloaded from Fibrillary lines of the cornea A clinical sign in keratoconus A. J. BRON,* D. J. LOBASCHER,t W. S. DIXON,: S. N. DAS,¶ AND M. RUBENt From The Oxford Eye Hospital,* Moorfield's Eye Hospital,t The University of Torontot, and the Royal Surrey County Hospital¶ In the course of studying keratoconus patients, mented on a proforma which recorded clinical history, fibrillar structures lying immediately inside Fleischer's vision, refraction, ocular pressure, keratometry, and bio- ring have been observed. Their arrangement is microscopical and ophthalmoscopical findings. Photo-slit thought to be characteristic of this condition. They photographs were taken of all subjects and macrophoto- lie at the level of the subepithelium and are identical graphs (Brown, I970) were taken some cases. Drawings were made of the corneal changes. The dimensions of the in form but not in arrangement to similar structures fibrillary structures observed were gauged by measure- in the normal cornea. These are described in the ments from selected macrophotographs. The findings in 77 preceding paper (Bron, 1975). The present paper newly referred patients are presented in this paper. describes the distribution of fibrillary lines in kerato- conus and how they differ from those in normal eyes. Clinical findings copyright. Material and methods The fibrillary lines of keratoconus are fine, white, Observations were made in the Keratoconus Clinic of curved, and slightly wavy, and lie in concentric Moorfields Eye Hospital. Clinical findings were docu- bundles at the internal margin of Fleischer's ring (Fig. -
North American Neuro-Ophthalmology Society 42Nd Annual Meeting February 27 - March 3, 2016 JW Starr Pass Marriott • Tucson, Arizona
North American Neuro-Ophthalmology Society 42nd Annual Meeting February 27 - March 3, 2016 JW Starr Pass Marriott • Tucson, Arizona Poster Session I: Clinical Highlights in Neuro-Ophthalmology Sunday, February 28, 2016 • 12:30 pm – 2:00 pm Authors will be standing by their posters during the following hours: Odd-Numbered Posters: 12:30 - 1:15 pm Even-Numbered Posters: 1:15 - 2:00 pm *Please note that all abstracts are published as submitted. Poster # Presenting Author Category: Disorders of the Anterior Visual Pathway (Retina, Optic Nerve, and Chiasm) 1 Compressive Optic Neuropathy from Salivary Gland Tumor of Sphenoid Sinus Nafiseh Hashemi 2 Unexpected Pathologic Diagnosis of Primary Dural B Cell Marginal Zone Lymphoma Alberto G. Distefano 3 Bilateral Epstein-Barr Virus Optic Neuritis in a Lung Transplant Patient Yen C. Hsia 4 Purtscher's Retinopathy as a Manifestation of Hemophagocytic Lymphohistiocytosis Dov B. Sebrow 5 Central Retinal Vein Occlusion, Paracentral Acute Middle Maculopathy, and Dov B. Sebrow Cilioretinal Vein Sparing with Acquired Shunt in a Patient with Antiphospholipid Syndrome and Cryoglobulinemia 6 A Case of Wyburn-Mason Syndrome Dae Hee Kim 7 Debulking Optic Nerve Gliomas for Disfiguring Proptosis: A Globe-Sparing Approach Faisal Y. Althekair by Lateral Orbitotomy Alone 8 Longitudinally Extensive Spinal Cord Lesion in Leber’s Hereditary Optic Neuropathy Faisal Y. Althekair Due to the M.3460A Mitochondrial DNA Mutation Longitudinally Extensive Spinal Cord Lesion in Leber’s Hereditary Optic Neuropathy due to the M.3460A Mitochondrial DNA Mutation 9 Growth of an Optic Disc Vascular Anomaly for Twelve Years John E. Carter 10 Retreatment with Ethambutol After Toxic Optic Neuropathy Marc A.