Enteric Duplication Cyst Resembling Umbilical Cord Cyst

Total Page:16

File Type:pdf, Size:1020Kb

Enteric Duplication Cyst Resembling Umbilical Cord Cyst Journal of Perinatology (2006) 26, 368–370 r 2006 Nature Publishing Group All rights reserved. 0743-8346/06 $30 www.nature.com/jp PERINATAL/NEONATAL CASE PRESENTATION Enteric duplication cyst resembling umbilical cord cyst M Prada Arias, F Garc´ıa Lorenzo, M Montero Sa´nchez, R Muguerza Vellibre Service of Pediatric Surgery, University Hospital ‘Xeral-Cı´es’, Vigo, Spain with bowel was never suspected. Pregnancy was uncomplicated and We report a case of a neonatal male with a prenatal diagnosis of umbilical a 3220 g male infant was born at 39 weeks of gestation by normal cord cyst that was finally diagnosed as an ileal duplication cyst after post- vaginal delivery. Apgar scores were 7 and 8 at 1 and 5 min, natal surgical exploration of the umbilical mass. To our knowledge, a respectively. Post-natal examination showed an umbilical cystic similar case has never been reported previously. Although this form of mass separated of the abdominal wall which presented a small presentation of enteric duplication cyst is exceptional, it should be taken (< 2 cm) defect at the umbilicus level (Figure 1). The umbilical into consideration when evaluating suspected umbilical cord cysts. cord between abdominal wall and umbilical cyst was slightly Journal of Perinatology (2006) 26, 368–370. doi:10.1038/sj.jp.7211520 enlarged. A fluid level was detected by transilumination of the cyst. Keywords: enteric duplication cyst; umbilical cord cyst; prenatal Post-natal ultrasound examination showed a cystic mass with diagnosis echogenic content without evidence of communication or relation with the bowel. Abdominal X-ray showed a normal distribution of gas into the bowel without signs of intestinal obstruction. However, Introduction a surgical exploration was indicated to exclude relation with bowel. Enteric duplications (EDs) are a group of rare congenital Exploration revealed an ileal duplication cyst 6 cm in diameter anomalies that can occur along the entire length of the which shared muscular wall and communicated with the gastrointestinal (GI) system. Their most common location is the associated ileum (Figure 2). A segment comprising the ileal small intestine, in particular the terminal ileum, which represents duplication was resected and primary end-to-end anastomosis was more than 70% of small GI duplications. Their estimated incidence performed to maintain the intestinal continuity. Histopathological is 1:4500 newborns.1–3 Embryogenesis of these anomalies remains examination confirmed ileal duplication cyst with small intestinal unknown. Several theories have been proposed that do not mucosa without heterotopic gastric mucosa. The infant also had satisfactorily explain some of the pathological manifestations.2,4 choanal stenosis which required surgical treatment, atrial septal Here, we present a case of a male infant with a prenatal diagnosis defect (ostium secundum) and congenital hypothyroidism. The of umbilical cord cyst that was finally diagnosed as an ileal postoperative period was uneventful. duplication cyst during the neonatal period. Case report Routine sonographic evaluation of a 34-year-old woman, diagnosed and being treated for Graves disease, gravida 3, para 2, demonstrated an unilocular echogenic cystic mass within the umbilical cord that was 15 mm in diameter. The sonographic diagnosis was an umbilical cord cyst in a fetus at 20 weeks’ gestation. No other fetal anomalies were detected and the amount of amniotic fluid was normal. Genetic amniocentesis revealed normal fetal karyotype 46XY. Follow-up ultrasound examinations showed gradual enlargement of the umbilical cyst in proportion to the fetus’s growth and a relation or communication of the cyst Correspondence: Dr Marcos Prada Arias, Hoya del Enamorado, El Bosque, 5, 21 D, 35019 Las Palmas de Gran Canaria, Las Palmas, Spain. E-mail: [email protected] Received 16 February 2006; revised 6 April 2006; accepted 6 April 2006 Figure 1 Image shows umbilical cyst separated from abdominal wall. Enteric cyst resembling umbilical cyst M Prada Arias et al 369 peristaltic muscular contractions of the cyst wall and intimate contact of the cyst wall with the mesenteric border.8,9 However, the prenatal diagnosis of ED is difficult and rarely reported.3,7,8 In most cases, an abdominal cyst was seen but the exact diagnosis could only be ascertained after birth.8 Prenatal sonography only identifies approximately one in four of these lesions (20 to 30%).3 In our case, an umbilical cord cyst was diagnosed because prenatal sonography showed a cystic mass within the umbilical cord without evidence of relation or communication with the bowel. Umbilical cord cysts are classified into two types, true cysts and pseudocysts. True umbilical cord cysts originate from amniotic inclusions such as remnants of the omphalomesenteric duct, allantoids, or vascular malformations. Pseudocysts represent localized edema or degeneration of the Wharton’s jelly that is a 10 Figure 2 Surgical image shows that the presumed umbilical cyst was matrix of the umbilical cord. It is rarely possible to prenatally in fact an ileal duplication cyst. diagnose the cyst as either a true cyst or a pseudocyst and exact differentiation between both can only be made by pathologic examination.10 Although intra-abdominal duplication cysts can give rise to Discussion symptoms at any point in life, they usually present during infancy Enteric duplications can be cystic or tubular in shape and present a and clinical presentation is extremely varied, depending on the well-developed coat of smooth muscle and intestinal epithelial location, size, and the presence or not of heterotopic gastric lining. They almost always lie on the mesenteric side of the bowel.1 mucosa. Almost 70% of ED that present with symptoms do so There appears to be a spectrum in ED regarding the blood supply within the first year of life and 85% by 2 years of age.1,2 They can and the area of attachment to the GI tract. The two ends of the manifest with a variety of signs and symptoms. The presence of spectrum would be: (1) the classical description with shared heterotopic gastric mucosa can cause bleeding or perforation by common wall and blood supply, which is the most frequent and peptic ulceration.3 Enteric duplications can serve as the focal point (2) the ED completely isolated from the bowel, which has an for volvulus or intussusceptions. Malignant development of this independent blood supply, found in extremely rare cases. An mucosal lining as well as cases of carcinoid tumors have been intermediate group would have part of the ED independent and reported.3 Many lesions, however, are asymptomatic and the part of it sharing a common wall with the bowel.1,5,6 diagnosis is often made incidentally during a surgical procedure.2 Communication between ED and intestinal lumen is much Reported cases of ED in an omphalocele are extremely rare.9,11 To rarer than non-communicating ones. In 1998, a vascular our knowledge, a case of ED resembling an umbilical cord cyst had classification was proposed to facilitate surgical management never been previously reported. of this disease: ED of small intestine is divided into parallel Full resection of the duplication with the adjacent bowel type (type I) and intra-mesenteric type (type II). In type I, has been recommended as the optimal treatment.2 Large the duplication is located on the border of one mesenteric leaf duplications, however, might be difficult to resect due to the and the straight artery to the duplication is separated from the risk of short-bowel syndrome. Mucosal stripping offers a good straight artery to the bowel. In type II, the duplication is located surgical option in these cases, eliminating the possibility of between the two leaves of the mesentery and the straight arteries subsequent peptic ulceration or carcinogenesis.2 According to the pass over both surfaces of the duplication to reach the adjacent proposed vascular classification, resection of the duplication bowel.4 without the adjacent bowel seems possible and advisable in some Enteric duplications may be suspected by sonographic types of duplication.4 In our case, type II according to this demonstration of an intra-abdominal cystic mass in the second or classification, segmental ileal resection including the ED seemed third trimester of gestation.7 The differential diagnosis of intra- more advisable as it would be only necessary to resect a short ileal abdominal cysts detected by prenatal sonography includes segment and it would be very difficult to separate the normal ileum ovarian cysts, enteric duplication cysts, renal cysts, choledochal from the duplication cyst. cysts, hepatic cysts, mesenteric or omental cysts, dilated bowel Although this form of presentation of enteric duplication cyst is loops of intestinal atresia and Meckel’s diverticulum.3 Sonographic exceptional, it should be taken into consideration when evaluating findings highly suggestive of an intestinal origin of the cyst are umbilical cord cysts. Journal of Perinatology Enteric cyst resembling umbilical cyst M Prada Arias et al 370 References 6 Norris RW, Brereton RJ, Wright WN, Cudmore, RE. A new surgical approach to duplications of the intestine. J Pediatr Surg 1986; 21: 167–170. 1 Bond SJ, Groff DB. Gastrointestinal duplications In: O’Neill JA, Jr Rowe MI, 7 Chen M, Lam YH, Lin CL, Chan KW, Hui PW, Tang MH. Sonographic Grosfeld JL, Fonkalsrud EW, Coran AG (eds). Pediatric Surgery. Mosby: St features of ileal duplication cyst al 12 weeks. Prenat Diagn 2002; 22: Louis, 1998 pp 1257–1267. 1067–1070. 2Ga´lvez Y, Ska´ba R, Kalousova´ J, Rouskova´ B, Hrˇı´balz Z, S˘najdauf J et al. 8 Gul A, Tekoglu G, Aslan H, Cebeci A, Erol O, Unal M et al. Prenatal Alimentary tract duplications in children: high incidence of associated sonographic features of esophageal and ileal duplications at 18 weeks of anomalies. Eur J Pediatr Surg 2004; 14: 79–84. gestation. Prenat Diagn 2004; 24: 969–971.
Recommended publications
  • Megaesophagus in Congenital Diaphragmatic Hernia
    Megaesophagus in congenital diaphragmatic hernia M. Prakash, Z. Ninan1, V. Avirat1, N. Madhavan1, J. S. Mohammed1 Neonatal Intensive Care Unit, and 1Department of Paediatric Surgery, Royal Hospital, Muscat, Oman For correspondence: Dr. P. Manikoth, Neonatal Intensive Care Unit, Royal Hospital, Muscat, Oman. E-mail: [email protected] ABSTRACT A newborn with megaesophagus associated with a left sided congenital diaphragmatic hernia is reported. This is an under recognized condition associated with herniation of the stomach into the chest and results in chronic morbidity with impairment of growth due to severe gastro esophageal reflux and feed intolerance. The infant was treated successfully by repair of the diaphragmatic hernia and subsequently Case Report Case Report Case Report Case Report Case Report by fundoplication. The megaesophagus associated with diaphragmatic hernia may not require surgical correction in the absence of severe symptoms. Key words: Congenital diaphragmatic hernia, megaesophagus How to cite this article: Prakash M, Ninan Z, Avirat V, Madhavan N, Mohammed JS. Megaesophagus in congenital diaphragmatic hernia. Indian J Surg 2005;67:327-9. Congenital diaphragmatic hernia (CDH) com- neonate immediately intubated and ventilated. His monly occurs through the posterolateral de- vital signs improved dramatically with positive pres- fect of Bochdalek and left sided hernias are sure ventilation and he received antibiotics, sedation, more common than right. The incidence and muscle paralysis and inotropes to stabilize his gener- variety of associated malformations are high- al condition. A plain radiograph of the chest and ab- ly variable and may be related to the side of domen revealed a left sided diaphragmatic hernia herniation. The association of CDH with meg- with the stomach and intestines located in the left aesophagus has been described earlier and hemithorax (Figure 1).
    [Show full text]
  • Pediatric Gastroesophageal Reflux Clinical Practice
    SOCIETY PAPER Pediatric Gastroesophageal Reflux Clinical Practice Guidelines: Joint Recommendations of the North American Society for Pediatric Gastroenterology, Hepatology, and Nutrition and the European Society for Pediatric Gastroenterology, Hepatology, and Nutrition ÃRachel Rosen, yYvan Vandenplas, zMaartje Singendonk, §Michael Cabana, jjCarlo DiLorenzo, ôFrederic Gottrand, #Sandeep Gupta, ÃÃMiranda Langendam, yyAnnamaria Staiano, zzNikhil Thapar, §§Neelesh Tipnis, and zMerit Tabbers ABSTRACT This document serves as an update of the North American Society for Pediatric INTRODUCTION Gastroenterology, Hepatology, and Nutrition (NASPGHAN) and the European n 2009, the joint committee of the North American Society for Society for Pediatric Gastroenterology, Hepatology, and Nutrition (ESPGHAN) Pediatric Gastroenterology, Hepatology, and Nutrition (NASP- 2009 clinical guidelines for the diagnosis and management of gastroesophageal GHAN)I and the European Society for Pediatric Gastroenterology, refluxdisease(GERD)ininfantsandchildrenandisintendedtobeappliedin Hepatology, and Nutrition (ESPGHAN) published a medical posi- daily practice and as a basis for clinical trials. Eight clinical questions addressing tion paper on gastroesophageal reflux (GER) and GER disease diagnostic, therapeutic and prognostic topics were formulated. A systematic (GERD) in infants and children (search until 2008), using the 2001 literature search was performed from October 1, 2008 (if the question was NASPGHAN guidelines as an outline (1). Recommendations were addressed
    [Show full text]
  • Abdominal Wall Defects—Current Treatments
    children Review Abdominal Wall Defects—Current Treatments Isabella N. Bielicki 1, Stig Somme 2, Giovanni Frongia 3, Stefan G. Holland-Cunz 1 and Raphael N. Vuille-dit-Bille 1,* 1 Department of Pediatric Surgery, University Children’s Hospital of Basel (UKBB), 4056 Basel, Switzerland; [email protected] (I.N.B.); [email protected] (S.G.H.-C.) 2 Department of Pediatric Surgery, University Children’s Hospital of Colorado, Aurora, CO 80045, USA; [email protected] 3 Section of Pediatric Surgery, Department of General, Visceral and Transplantation Surgery, 69120 Heidelberg, Germany; [email protected] * Correspondence: [email protected]; Tel.: +41-61-704-27-98 Abstract: Gastroschisis and omphalocele reflect the two most common abdominal wall defects in newborns. First postnatal care consists of defect coverage, avoidance of fluid and heat loss, fluid administration and gastric decompression. Definitive treatment is achieved by defect reduction and abdominal wall closure. Different techniques and timings are used depending on type and size of defect, the abdominal domain and comorbidities of the child. The present review aims to provide an overview of current treatments. Keywords: abdominal wall defect; gastroschisis; omphalocele; treatment 1. Gastroschisis Citation: Bielicki, I.N.; Somme, S.; 1.1. Introduction Frongia, G.; Holland-Cunz, S.G.; Gastroschisis is one of the most common congenital abdominal wall defects in new- Vuille-dit-Bille, R.N. Abdominal Wall borns. Children born with gastroschisis have a full-thickness paraumbilical abdominal Defects—Current Treatments. wall defect, which is associated with evisceration of bowel and sometimes other organs Children 2021, 8, 170.
    [Show full text]
  • Guideline for the Evaluation of Cholestatic Jaundice
    CLINICAL GUIDELINES Guideline for the Evaluation of Cholestatic Jaundice in Infants: Joint Recommendations of the North American Society for Pediatric Gastroenterology, Hepatology, and Nutrition and the European Society for Pediatric Gastroenterology, Hepatology, and Nutrition ÃRima Fawaz, yUlrich Baumann, zUdeme Ekong, §Bjo¨rn Fischler, jjNedim Hadzic, ôCara L. Mack, #Vale´rie A. McLin, ÃÃJean P. Molleston, yyEzequiel Neimark, zzVicky L. Ng, and §§Saul J. Karpen ABSTRACT Cholestatic jaundice in infancy affects approximately 1 in every 2500 term PREAMBLE infants and is infrequently recognized by primary providers in the setting of holestatic jaundice in infancy is an uncommon but poten- physiologic jaundice. Cholestatic jaundice is always pathologic and indicates tially serious problem that indicates hepatobiliary dysfunc- hepatobiliary dysfunction. Early detection by the primary care physician and tion.C Early detection of cholestatic jaundice by the primary care timely referrals to the pediatric gastroenterologist/hepatologist are important physician and timely, accurate diagnosis by the pediatric gastro- contributors to optimal treatment and prognosis. The most common causes of enterologist are important for successful treatment and an optimal cholestatic jaundice in the first months of life are biliary atresia (25%–40%) prognosis. The Cholestasis Guideline Committee consisted of 11 followed by an expanding list of monogenic disorders (25%), along with many members of 2 professional societies: the North American Society unknown or multifactorial (eg, parenteral nutrition-related) causes, each of for Gastroenterology, Hepatology and Nutrition, and the European which may have time-sensitive and distinct treatment plans. Thus, these Society for Gastroenterology, Hepatology and Nutrition. This guidelines can have an essential role for the evaluation of neonatal cholestasis committee has responded to a need in pediatrics and developed to optimize care.
    [Show full text]
  • Intestinal Malrotation: a Diagnosis to Consider in Acute Abdomen In
    Submitted on: 05/20/2018 Approved on: 08/07/2018 CASE REPORT Intestinal malrotation: a diagnosis to consider in acute abdomen in newborns Antônio Augusto de Andrade Cunha Filho1, Paula Aragão Coimbra2, Adriana Cartafina Perez-Bóscollo3, Robson Azevedo Dutra4, Katariny Parreira de Oliveira Alves5 Keywords: Abstract Intestinal obstruction, Intestinal malrotation is an anomaly of the midgut, resulting from an embryonic defect during the phases of herniation, Acute abdomen, rotation, and fixation. The objective is to report a case of complex diagnostics and approach. The diagnosis was made Gastrointestinal tract. surgically in a patient presenting with hemodynamic instability, abdominal distension, signs of intestinal obstruction, and pneumoperitoneum on abdominal X-ray, with suspected grade III necrotizing enterocolitis. During surgery, a volvulus resulting from poor intestinal rotation was found at a distance of 12 cm from the ileocecal valve. Hemodynamic instability and abdominal distension recurred, and another exploratory laparotomy was required to correct new intestinal perforations. Therefore, early diagnosis with surgical correction before a volvulus appears is essential. Abdominal Doppler ultrasonography has been promising for early diagnosis. 1 Academic in Medicine - Federal University of the Triângulo Mineiro - Uberaba - Minas Gerais - Brazil 2 Resident in Pediatric Intensive Care - Federal University of the Triângulo Mineiro - Uberaba - Minas Gerais - Brazil 3 Associate Professor - Federal University of the Triângulo Mineiro - Uberaba - Minas Gerais - Brazil. 4 Adjunct Professor - Federal University of the Triângulo Mineiro - Uberaba - Minas Gerais - Brazil 5 Academic in Medicine - Federal University of the Triângulo Mineiro - Uberaba - Minas Gerais - Brazil Correspondence to: Antônio Augusto de Andrade Cunha Filho. Universidade Federal do Triângulo Mineiro, Acadêmico de Medicina - Uberaba - Minas Gerais - Brasil.
    [Show full text]
  • Statistical Analysis Plan
    Cover Page for Statistical Analysis Plan Sponsor name: Novo Nordisk A/S NCT number NCT03061214 Sponsor trial ID: NN9535-4114 Official title of study: SUSTAINTM CHINA - Efficacy and safety of semaglutide once-weekly versus sitagliptin once-daily as add-on to metformin in subjects with type 2 diabetes Document date: 22 August 2019 Semaglutide s.c (Ozempic®) Date: 22 August 2019 Novo Nordisk Trial ID: NN9535-4114 Version: 1.0 CONFIDENTIAL Clinical Trial Report Status: Final Appendix 16.1.9 16.1.9 Documentation of statistical methods List of contents Statistical analysis plan...................................................................................................................... /LQN Statistical documentation................................................................................................................... /LQN Redacted VWDWLVWLFDODQDO\VLVSODQ Includes redaction of personal identifiable information only. Statistical Analysis Plan Date: 28 May 2019 Novo Nordisk Trial ID: NN9535-4114 Version: 1.0 CONFIDENTIAL UTN:U1111-1149-0432 Status: Final EudraCT No.:NA Page: 1 of 30 Statistical Analysis Plan Trial ID: NN9535-4114 Efficacy and safety of semaglutide once-weekly versus sitagliptin once-daily as add-on to metformin in subjects with type 2 diabetes Author Biostatistics Semaglutide s.c. This confidential document is the property of Novo Nordisk. No unpublished information contained herein may be disclosed without prior written approval from Novo Nordisk. Access to this document must be restricted to relevant parties.This
    [Show full text]
  • Appendix 3.1 Birth Defects Descriptions for NBDPN Core, Recommended, and Extended Conditions Updated March 2017
    Appendix 3.1 Birth Defects Descriptions for NBDPN Core, Recommended, and Extended Conditions Updated March 2017 Participating members of the Birth Defects Definitions Group: Lorenzo Botto (UT) John Carey (UT) Cynthia Cassell (CDC) Tiffany Colarusso (CDC) Janet Cragan (CDC) Marcia Feldkamp (UT) Jamie Frias (CDC) Angela Lin (MA) Cara Mai (CDC) Richard Olney (CDC) Carol Stanton (CO) Csaba Siffel (GA) Table of Contents LIST OF BIRTH DEFECTS ................................................................................................................................................. I DETAILED DESCRIPTIONS OF BIRTH DEFECTS ...................................................................................................... 1 FORMAT FOR BIRTH DEFECT DESCRIPTIONS ................................................................................................................................. 1 CENTRAL NERVOUS SYSTEM ....................................................................................................................................... 2 ANENCEPHALY ........................................................................................................................................................................ 2 ENCEPHALOCELE ..................................................................................................................................................................... 3 HOLOPROSENCEPHALY.............................................................................................................................................................
    [Show full text]
  • Duplication Cyst with Intestinal Volvulus Causing Intestinal Atresia/Stenosis in Neonates
    Journal of Neonatal Surgery 2018; 7:43 DOI: 10.21699/jns.v7i4.820. CASE SERIES Duplication Cyst with Intestinal Volvulus Causing Intestinal Atresia/Stenosis in Neonates Muhammad Bilal Mirza*, Imran Hashim, Shabbir Ahmad, Nabila Talat, M Zubair Shaukat, Muhammad Saleem Department of Pediatric Surgery, The Children’s Hospital and The Institute of Child Health, Lahore, Pakistan How to cite: Mirza B, Hashim I, Ahmad S, Talat N, Shaukat MZ, Saleem M. Duplication cyst with intestinal volvulus causing intestinal atresia/ stenosis in neonates. J Neonatal Surg. 2018; 7:43. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. ABSTRACT Duplication cysts of small bowel seldom present in newborns and usually represent the development of complications. In utero complications may lead to mesenteric vascular accidents and thus resulting in intestinal atresias. We report three neonates with duplication cyst of small bowel causing localized intestinal volvulus, leading to small bowel intestinal atresia/stenosis. The neonates underwent excision of the duplication cyst and resection anastomosis of the small bowel. Post-operative recovery was uneventful in all three of them. Key words: Atresia; Duplication cyst; Stenosis; Volvulus INTRODUCTION sound of abdomen showed dilated bowel loops. After adequate resuscitation, the patient was operated with Volvulus due to duplication cyst is a rare cause of suspicion of intestinal atresia. At surgery, a 3 cm × neonatal intestinal obstruction [1]. In utero volvulus 5 cm sized non-communicating cyst was encountered due to duplication cyst may also produce intestinal with 3–4 twists causing localized volvulus.
    [Show full text]
  • Congenital Pyloric Atresia: Clinical Features, Diagnosis, Associated Anomalies, Management and Outcome Osama A
    188 Original article Congenital pyloric atresia: clinical features, diagnosis, associated anomalies, management and outcome Osama A. Bawazira,b and Ahmed H. Al-Salemc Background Congenital pyloric atresia (CPA) is very rare period; however, 10 died later giving an overall survival of and usually seen as an isolated anomaly, which has an 40%. Sepsis was the main cause of death. excellent prognosis. CPA can be associated with other Conclusion CPA is a very rare malformation that can be anomalies or familial and these are usually associated with familial and inherited as an autosomal recessive. It can other hereditary conditions with poor prognosis. This either occur as an isolated lesion with an excellent review is based on our experience with 20 infants with CPA. prognosis, or be associated with other anomalies. Patients and methods This is a review of CPA, The overall prognosis of CPA, however, is still poor, and this highlighting its clinical features; associated anomalies; and is due to the frequent-and often fatal-associated aspects of diagnosis, management and outcome. anomalies. Ann Pediatr Surg 13:188–193 c 2017 Annals of Pediatric Surgery. Results This review is based on our experience with 20 patients with CPA (nine male and 11 female). Their mean Annals of Pediatric Surgery 2017, 13:188–193 birth weight was 2.1 kg (1.1-3.9 kg). Polyhydramnios was Keywords: aplasia cutis congenita, congenital pyloric atresia, seen in 13 (65%) patients. Seven patients were full-term epidermolysis bullosa, hereditary multiple intestinal atresia and the remaining 13 were premature. Two were brothers aDepartment of Surgery, Faculty of Medicine, Umm Al Qura University, Makkah, and four were members of the same family.
    [Show full text]
  • Nova Scotia Atlee Perinatal Database Coding Manual 15 Edition (Version
    Nova Scotia Atlee Perinatal Database Coding Manual 15th Edition (Version 15.0.0) April 2011 TABLE OF CONTENTS LISTINGS OF HOSPITALS 11 ADMISSION INFORMATION 16 DELIVERED ADMISSION 26 Routine Information – Delivered Admission 26 Routine Information – Labour 57 Routine Information – Infant 77 UNDELIVERED ADMISSION 94 Routine information – undelivered 94 POSTPARTUM ADMISSIONS 104 Routine Information – Postpartum Admission 104 NEONATAL ADMISSIONS 112 Routine Information – Neonatal Admissions 112 ADULT RCP CODES 124 INFANT RCP CODES 143 INDEX OF MATERNAL DISEASES AND PROCEDURES 179 INDEX OF NEONATAL DISEASES AND PROCEDURES 195 1 INDEX FOR ADMISSION INFORMATION Admission date .............................................................................................................................. 17 Admission time .............................................................................................................................. 17 Admission process status ............................................................................................................... 25 Admission type .............................................................................................................................. 17 A/S/D number ................................................................................................................................ 18 Birth date ....................................................................................................................................... 18 Care provider attending ................................................................................................................
    [Show full text]
  • The Gastrointestinal Tract Frank A
    91731_ch13 12/8/06 8:55 PM Page 549 13 The Gastrointestinal Tract Frank A. Mitros Emanuel Rubin THE ESOPHAGUS Bezoars Anatomy THE SMALL INTESTINE Congenital Disorders Anatomy Tracheoesophageal Fistula Congenital Disorders Rings and Webs Atresia and Stenosis Esophageal Diverticula Duplications (Enteric Cysts) Motor Disorders Meckel Diverticulum Achalasia Malrotation Scleroderma Meconium Ileus Hiatal Hernia Infections of the Small Intestine Esophagitis Bacterial Diarrhea Reflux Esophagitis Viral Gastroenteritis Barrett Esophagus Intestinal Tuberculosis Eosinophilic Esophagitis Intestinal Fungi Infective Esophagitis Parasites Chemical Esophagitis Vascular Diseases of the Small Intestine Esophagitis of Systemic Illness Acute Intestinal Ischemia Iatrogenic Cancer of Esophagitis Chronic Intestinal Ischemia Esophageal Varices Malabsorption Lacerations and Perforations Luminal-Phase Malabsorption Neoplasms of the Esophagus Intestinal-Phase Malabsorption Benign tumors Laboratory Evaluation Carcinoma Lactase Deficiency Adenocarcinoma Celiac Disease THE STOMACH Whipple Disease Anatomy AbetalipoproteinemiaHypogammaglobulinemia Congenital Disorders Congenital Lymphangiectasia Pyloric Stenosis Tropical Sprue Diaphragmatic Hernia Radiation Enteritis Rare Abnormalities Mechanical Obstruction Gastritis Neoplasms Acute Hemorrhagic Gastritis Benign Tumors Chronic Gastritis Malignant Tumors MénétrierDisease Pneumatosis Cystoides Intestinalis Peptic Ulcer Disease THE LARGE INTESTINE Benign Neoplasms Anatomy Stromal Tumors Congenital Disorders Epithelial Polyps
    [Show full text]
  • Duodenal Stenosis: a Diagnostic Challenge in a Neonate with Poor Weight Gain
    Open Access Case Report DOI: 10.7759/cureus.8559 Duodenal Stenosis: A Diagnostic Challenge in a Neonate With Poor Weight Gain Ma Khin Khin Win 1 , Carole Mensah 1 , Kunal Kaushik 1 , Louisdon Pierre 1 , Adebayo Adeyinka 1 1. Pediatrics, The Brooklyn Hospital Center, Brooklyn, USA Corresponding author: Carole Mensah, [email protected] Abstract Cases of isolated duodenal stenosis in the neonatal period are minimally reported in pediatric literature. Causes of small bowel obstruction such as duodenal atresia or malrotation with midgut volvulus have been well documented and are often diagnosed due to their acute clinical presentation. Duodenal stenosis, however, causes an incomplete intestinal obstruction with a more indolent and varying clinical presentation thus making it a diagnostic challenge. We present a neonate with a unique case of congenital duodenal stenosis. The neonate presented with poor weight gain and frequent "spit-ups" as per the mother at the initial newborn visit. The clinical presentation was masked as the patient was being fed infrequently and with concentrated formula. We postulate that this may be due to the fact that the mother was an adolescent and relatively inexperienced with newborn care. During the hospital course, the patient had recurrent episodes of emesis with notable electrolyte abnormalities including hypochloremia and metabolic alkalosis. Further investigation with an abdominal X-ray showed dilated loops of bowel. Pyloric stenosis was ruled out via abdominal ultrasound. An upper gastrointestinal (GI) series ultimately confirmed a diagnosis of duodenal stenosis and the infant underwent surgical repair with full recovery. Congenital duodenal stenosis may have atypical presentations in neonates requiring pediatricians to have a high index of suspicion for diagnosis and to ensure timely therapy.
    [Show full text]