Aggressive Periodontitis and a Prosthetic Solution: 7-Month Follow-Up
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Management of Rapidly Progressing Periodontitis: an Overview
Review Article Management of Rapidly Progressing Periodontitis: An Overview Sadat SMA1, Chowdhury NM2, Baten RBA3 Abstract causes rapid destruction of periodontal ligament and History of periodontal diseases recognition and alveolar bone which occurs in otherwise systemically treatment is ancient for at least 5000 years. There are healthy individuals generally of a younger age group but 1,8 different presentations of periodontal diseases. Rapidly patients may be older . A familial aggregation of the 9 progression periodontitis or aggressive periodontitis disease is also noted . It includes both localized and causes rapid destruction of the periodontium which leads generalized forms. It was previously known as early onset to early tooth loss. It may be generalized or localized. periodontitis that included three categories of periodontitis Periodontitis may be treated surgically or non-surgically - Pubertal, Juvenile and rapidly progressing but patients with rapidly progressing periodontitis do not periodontitis10,11. Early diagnosis and appropriate respond predictably to conventional therapy due to its treatment is necessary to prevent early tooth loss. multi factorial etiology. Successful management of the disease is difficult if not diagnosed early and treated Classification appropriately. Regenerative therapy, tissue engineering Classification of diseases helps the clinicians to develop a and genetic technologies are the new hope for the structure which can be used to identify diseases in relation treatment of rapidly progressing periodontitis. -
Probiotic Alternative to Chlorhexidine in Periodontal Therapy: Evaluation of Clinical and Microbiological Parameters
microorganisms Article Probiotic Alternative to Chlorhexidine in Periodontal Therapy: Evaluation of Clinical and Microbiological Parameters Andrea Butera , Simone Gallo * , Carolina Maiorani, Domenico Molino, Alessandro Chiesa, Camilla Preda, Francesca Esposito and Andrea Scribante * Section of Dentistry–Department of Clinical, Surgical, Diagnostic and Paediatric Sciences, University of Pavia, 27100 Pavia, Italy; [email protected] (A.B.); [email protected] (C.M.); [email protected] (D.M.); [email protected] (A.C.); [email protected] (C.P.); [email protected] (F.E.) * Correspondence: [email protected] (S.G.); [email protected] (A.S.) Abstract: Periodontitis consists of a progressive destruction of tooth-supporting tissues. Considering that probiotics are being proposed as a support to the gold standard treatment Scaling-and-Root- Planing (SRP), this study aims to assess two new formulations (toothpaste and chewing-gum). 60 patients were randomly assigned to three domiciliary hygiene treatments: Group 1 (SRP + chlorhexidine-based toothpaste) (control), Group 2 (SRP + probiotics-based toothpaste) and Group 3 (SRP + probiotics-based toothpaste + probiotics-based chewing-gum). At baseline (T0) and after 3 and 6 months (T1–T2), periodontal clinical parameters were recorded, along with microbiological ones by means of a commercial kit. As to the former, no significant differences were shown at T1 or T2, neither in controls for any index, nor in the experimental -
Aggressive Periodontitis and Its Multidisciplinary Focus: Review of the Literature
ODOVTOS-International Journal of Dental Sciences Frías et al: Aggressive Periodontitis and its Multidisciplinary Focus: Review of the Literature Aggressive Periodontitis and its Multidisciplinary Focus: Review of the Literature Periodontitis agresiva y su enfoque multidisciplinario: Revisión de literatura Maribel Frías-Muñoz DDS¹; Roxana Araujo-Espino DDS, MSc, PhD²; Víctor Manuel Martínez-Aguilar DDS, MSc³; Teresina Correa Alcalde DDS⁴; Luis Alejandro Aguilera-Galaviz DDS, MSc, PhD⁴; César Gaitán-Fonseca DDS, MSc, PhD⁴ 1. Estudiante, Maestría en Ciencias Biomédicas, Área Ciencias de la Salud, Universidad Autónoma de Zacatecas “Francisco García Salinas” (UAZ), Zacatecas, Zac., Mexico. 2. Docente-investigador, Unidad Académica de Enfermería, Área Ciencias de la Salud, Universidad Autónoma de Zacatecas “Francisco García Salinas” (UAZ), Zacatecas, Zac., Mexico. 3. Docente-investigador, Facultad de Odontología, Universidad Autónoma de Yucatán (UADY), Mérida, Yuc., Mexico. 4. Docente-investigador, Área Ciencias de la Salud, Maestría en Ciencias Biomédicas, Universidad Autónoma de Zacatecas “Francisco García Salinas” (UAZ), Zacatecas, Zac., Mexico. Correspondence to: Dr. César Gaitán-Fonseca - [email protected] Received: 1-V-2017 Accepted: 5-V-2017 Published Online First: 8-V-2017 DOI: http://dx.doi.org/10.15517/ijds.v0i0.28869 ABSTRACT The purpose of this review is to have a current prospect of periodontal diseases and, in particular, aggressive periodontitis. To know its classification and clinical characteristics, such as the extent and age group affected, as well as its distribution in the population, etiology, genetic variations, among other factors that could affect the development of this disease. Also, reference is made to different diagnostic options and, likewise, the current treatment options. KEYWORDS Aggressive periodontitis; Periodontal diseases; Diagnosis. -
Classification and Diagnosis of Aggressive Periodontitis
Received: 3 November 2016 Revised: 11 October 2017 Accepted: 21 October 2017 DOI: 10.1002/JPER.16-0712 2017 WORLD WORKSHOP Classification and diagnosis of aggressive periodontitis Daniel H. Fine1 Amey G. Patil1 Bruno G. Loos2 1 Department of Oral Biology, Rutgers School Abstract of Dental Medicine, Rutgers University - Newark, NJ, USA Objective: Since the initial description of aggressive periodontitis (AgP) in the early 2Department of Periodontology, Academic 1900s, classification of this disease has been in flux. The goal of this manuscript is to Center of Dentistry Amsterdam (ACTA), review the existing literature and to revisit definitions and diagnostic criteria for AgP. University of Amsterdam and Vrije Universiteit, Amsterdam, The Netherlands Study analysis: An extensive literature search was performed that included databases Correspondence from PubMed, Medline, Cochrane, Scopus and Web of Science. Of 4930 articles Dr. Daniel H. Fine, Department of Oral reviewed, 4737 were eliminated. Criteria for elimination included; age > 30 years old, Biology, Rutgers School of Dental Medicine, Rutgers University - Newark, NJ. abstracts, review articles, absence of controls, fewer than; a) 200 subjects for genetic Email: fi[email protected] studies, and b) 20 subjects for other studies. Studies satisfying the entrance criteria The proceedings of the workshop were were included in tables developed for AgP (localized and generalized), in areas related jointly and simultaneously published in the Journal of Periodontology and Journal of to epidemiology, microbial, host and genetic analyses. The highest rank was given to Clinical Periodontology. studies that were; a) case controlled or cohort, b) assessed at more than one time-point, c) assessed for more than one factor (microbial or host), and at multiple sites. -
Treatment of Localized Aggressive Periodontitis Regenerative Periodontal Therapy Offers Alternative to Extraction/Implant Placement Ahmad Soolari, DMD, MS
Inside PERIODONTICS PEER-REVIEWED Treatment of Localized Aggressive Periodontitis Regenerative periodontal therapy offers alternative to extraction/implant placement Ahmad Soolari, DMD, MS ue to the effect of im- with inconsistent margins, which could be was localized aggressive periodontitis. The plant-related compli- measured from the cementoenamel junc- patient’s medical history was unremarkable, cations, the longevity of tion or from the crest of the alveolar bone but a significant finding was the retention of implants is often ques- to the base of the defect. A periapical ra- primary teeth with premolar impaction on tionable, even when diograph showed angular bone loss on the the lower right side. The patient was later compared with that of distal aspect of tooth No. 24 with an angle referred to an orthodontist for consultation. compromised but suc- of almost 45°. The interproximal space be- The goal of regenerative periodontal cessfully treated natural teeth. This article de- tween tooth No. 24 and tooth No. 23 was the therapy is to improve a tooth’s prognosis by Dscribes a case of severe, localized periodontal only site with severe bone loss throughout regenerating the supporting structures. In disease in which the patient rejected a recom- the patient’s mouth; therefore, the diagnosis this case, the prognosis for tooth No. 24 was mendation to undergo extraction followed by implant placement and restoration in favor of receiving regenerative periodontal therapy to save the natural tooth. Case Report A 17-year-old female patient presented with bone loss associated with tooth No. 24. A clinical examination and periodontal eval- uation (ie, assessment of mobility, probing depth, bleeding on probing, plaque score, and clinical attachment loss) revealed severe horizontal and vertical bone loss, deep prob- ing depth with bleeding, class II mobility, a FIG. -
American Academy of Periodontology Task Force Report on the Update to the 1999 Classification of Periodontal Diseases and Conditions*
J Periodontol • July 2015 American Academy of Periodontology Task Force Report on the Update to the 1999 Classification of Periodontal Diseases and Conditions* The American Academy of Periodontology (AAP) peri- 4 mm CAL, and Severe =‡5 mm CAL.’’ Numerous odically publishes reports, statements, and guidelines important studies since 1999 have used similar pa- on a variety of topics relevant to periodontics. These rameters to define periodontitis. For example, the papers are developed by an appointed committee of recent epidemiologic studies outlining the prevalence experts, and the documents are reviewed and ap- of periodontitis in the United States used attachment proved by the AAP Board of Trustees. loss parameters to define various severities of peri- odontitis.2,3 It is recognized that CAL is of importance for the scientific advancement of the knowledge of n 2014, the American Academy of Periodontology periodontitis. However, in clinical practice, measure- Board of Trustees charged a Task Force to develop ment of CAL has proven to be challenging, and is time Ia clinical interpretation of the 1999 Classification consuming. Measuring the location of the cemento- of Periodontal Diseases and Conditions to address enamel junction (CEJ) when the gingival margin is concerns expressed by the education community, the located coronal to the CEJ is difficult and may involve American Board of Periodontology, and the practic- some guesswork when the CEJ is not readily evident ing community that the current Classification pres- via tactile sensation. These issues can result in ex- ents challenges for the education of dental students aminations being performed in which, rather than and implementation in clinical practice. -
Idiopathic Gingival Fibromatosis Associated with Generalized Aggressive Periodontitis: a Case Report
Clinical P RACTIC E Idiopathic Gingival Fibromatosis Associated with Generalized Aggressive Periodontitis: A Case Report Contact Author Rashi Chaturvedi, MDS, DNB Dr. Chaturvedi Email: rashichaturvedi@ yahoo.co.in ABSTRACT Idiopathic gingival fibromatosis, a benign, slow-growing proliferation of the gingival tissues, is genetically heterogeneous. This condition is usually part of a syndrome or, rarely, an isolated disorder. Aggressive periodontitis, another genetically transmitted disorder of the periodontium, typically results in severe, rapid destruction of the tooth- supporting apparatus. The increased susceptibility of the host population with aggressive periodontitis may be caused by the combined effects of multiple genes and their inter- action with various environmental factors. Functional abnormalities of neutrophils have also been implicated in the etiopathogenesis of aggressive periodontitis. We present a rare case of a nonsyndromic idiopathic gingival fibromatosis associated with general- ized aggressive periodontitis. We established the patient’s diagnosis through clinical and radiologic assessment, histopathologic findings and immunologic analysis of neutrophil function with a nitro-blue-tetrazolium reduction test. We describe an interdisciplinary approach to the treatment of the patient. For citation purposes, the electronic version is the definitive version of this article: www.cda-adc.ca/jcda/vol-75/issue-4/291.html diopathic gingival fibromatosis is a rare syndromic, have been genetically linked to hereditary condition -
Idiopathic Gingival Enlargement With
CODSJOD KL Vandana et al. 10.5005/jp-journals-10063-0030 CASE REPORT Idiopathic Gingival Enlargement with Aggressive Periodontitis Treated with Surgical Gingivectomy and 0.2% Hyaluronic Acid Gel (Gengigel®) 1Kharidhi L Vandana, 2Priyanka Dalvi, 3Neha Mahajan ABSTRACT INTRODUCTION Background: Idiopathic gingival fibromatosis is known to be In inflammatory periodontal diseases, gingival enlarge- a benign slow growing proliferation of the gingival tissue. It is ment will be a common finding. Based on the etiologic genetically heterogeneous, associated with syndromes and rarely presents as an isolated disorder. Aggressive periodontitis factors and histopathologic findings, there are several (AP) is a disorder that results in severe rapid destruction of the kinds of gingival enlargement. Gingival enlargement tooth-supporting apparatus and is also a genetically transmitted induced by drugs such as phenytoin, cyclosporine, and disorder of the periodontium. In the case of gingival enlarge- calcium channel blockers associated with systemic dis- ment there will be an excessive display of gingiva affecting the esthetic and functional problems. Gingival enlargement in orders produced by hormonal factors, leukemia, vitamin association with generalized AP is very rare. C deficiency, and idiopathic gingival fibromatosis have Case description: A 23-year-old female was reported with a been reported.1 recurrence of gingival enlargement along with generalized tooth Aggressive periodontitis (AP) is a rare condition of mobility. On detailed history, clinical and laboratory findings, periodontitis which can be localized (LAP) or generali- it was diagnosed as recurrent idiopathic gingival enlargement with generalized aggressive periodontitis. This patient has been zed (GAP) based on the clinical and laboratory findings. followed up for nearly 9 years ever since she first reported to A case may be diagnosed as AP if it presents with the us in the year 2004 with a similar finding. -
Pub 100-04 Medicare Claims Processing
Department of Health & CMS Manual System Human Services (DHHS) Pub 100-04 Medicare Claims Centers for Medicare & Processing Medicaid Services (CMS) Transmittal 990 Date: JUNE 23, 2006 Change Request 5142 Subject: Medicare Contractor Annual Update of the International Classification of Diseases, Ninth Revision, Clinical Modification (ICD-9-CM) I. SUMMARY OF CHANGES: This instruction is CMS' annual reminder to the contractors of the ICD-9-CM update that is effective for the dates of service on and after October 1, 2006, as well as discharges on or after October 1, 2006 for institutional providers. New / Revised Material Effective Date: October 1, 2006 Implementation Date: October 2, 2006 Disclaimer for manual changes only: The revision date and transmittal number apply only to red italicized material. Any other material was previously published and remains unchanged. However, if this revision contains a table of contents, you will receive the new/revised information only, and not the entire table of contents. II. CHANGES IN MANUAL INSTRUCTIONS: (N/A if manual is not updated) R=REVISED, N=NEW, D=DELETED-Only One Per Row. R/N/D Chapter / Section / Subsection / Title N/A III. FUNDING: No additional funding will be provided by CMS; Contractor activities are to be carried out within their FY 2006 operating budgets. IV. ATTACHMENTS: Recurring Update Notification *Unless otherwise specified, the effective date is the date of service. Attachment – Recurring Update Notification Pub. 100-04 Transmittal: 990 Date: June 23, 2006 Change Request 5142 SUBJECT: Medicare Contractor Annual Update of the International Classification of Diseases, Ninth Revision, Clinical Modification (ICD-9-CM) I. -
Auxiliary Chemical Therapies in the Treatment of Aggressive Periodontitis: Current Aspects
http://dx.doi.org/10.1590/1981-863720150002000091903 REVISÃO | REVIEW Auxiliary chemical therapies in the treatment of aggressive periodontitis: current aspects Terapias químicas auxiliares para o tratamento da periodontite agressiva: aspectos atuais Lauro Garrastazu AYUB1 Arthur Belém NOVAES JÚNIOR1 Márcio Fernando de Moraes GRISI1 Sérgio Luís Scombatti de SOUZA1 Daniela Bazan PALIOTO1 Mário TABA JÚNIOR1 ABSTRACT Aggressive periodontitis, a distinct clinical entity of periodontal disease, is characterized by a pronounced episodic and rapid destruction of periodontal tissues and may result in rapid and early loss of teeth. Some studies have shown that conventional mechanical debridement together with oral hygiene is often not sufficient to disease control. Recent studies of this condition have shown beneficial effects of auxiliary therapies or adjuncts such as the administration of systemic and locally antimicrobials. Among the local adjuncts, the literature presents antiseptics, antibiotics and photodynamic therapy. Antibiotics and anti-inflammatory represent systemic adjuncts. Regardless of the results presented by each of them, the difficulty of establishing a single protocol for all cases is recognized depending on the individual response shown by each patient. The aim of the present study was to review the current results about chemical adjuncts administration associated with conventional treatment in cases of aggressive periodontitis and suggest clinical protocols. Indexing terms: Aggressive periodontitis. Anti-bacterial agents. Anti-infective agents, local. Anti-inflammatory agentes. Mouthwashes. Photochemotherapy. RESUMO A periodontite agressiva, uma entidade clínica distinta da doença periodontal, é caracterizada por uma pronunciada destruição episódica e rápida dos tecidos periodontais e pode resultar em perda rápida e precoce dos dentes. Alguns trabalhos têm mostrado que o debridamento mecânico convencional juntamente com higiene oral muitas vezes não é suficiente para o controle da doença. -
Recent Development of Active Ingredients in Mouthwashes and Toothpastes for Periodontal Diseases
molecules Review Recent Development of Active Ingredients in Mouthwashes and Toothpastes for Periodontal Diseases Meenakshi Rajendiran 1, Harsh M Trivedi 2, Dandan Chen 2, Praveen Gajendrareddy 1,* and Lin Chen 1,* 1 The Center for Wound Healing and Tissue Regeneration, Department of Periodontics, College of Dentistry, University of Illinois at Chicago, Chicago, IL 60612, USA; [email protected] 2 Colgate-Palmolive Company, Piscataway, NJ 08854, USA; [email protected] (H.M.T.); [email protected] (D.C.) * Correspondence: [email protected] (P.G.); [email protected] (L.C.); Tel.: +1-312-413-8405 (P.G.); +1-312-413-5387 (L.C.) Abstract: Periodontal diseases like gingivitis and periodontitis are primarily caused by dental plaque. Several antiplaque and anti-microbial agents have been successfully incorporated into toothpastes and mouthwashes to control plaque biofilms and to prevent and treat gingivitis and periodontitis. The aim of this article was to review recent developments in the antiplaque, anti-gingivitis, and anti-periodontitis properties of some common compounds in toothpastes and mouthwashes by evaluating basic and clinical studies, especially the ones published in the past five years. The com- mon active ingredients in toothpastes and mouthwashes included in this review are chlorhexidine, cetylpyridinium chloride, sodium fluoride, stannous fluoride, stannous chloride, zinc oxide, zinc chloride, and two herbs—licorice and curcumin. We believe this comprehensive review will provide useful up-to-date information for dental care professionals and the general public regarding the Citation: Rajendiran, M.; Trivedi, major oral care products on the market that are in daily use. H.M; Chen, D.; Gajendrareddy, P.; Chen, L. -
Periodontitis Have Struggled to Decide If There Are Different Diseases Or Variations of a Single Disease
• Attempts to classify periodontitis have struggled to decide if there are different diseases or variations of a single disease. • There is no evidence to support differentiating “chronic” and “aggressive” periodontitis. • Three forms of periodontitis have been identified: (1) periodontitis, (2) necrotising periodontitis, (3) periodontitis as a direct manifestation of systemic diseases. • A classification system must include complexity and risk factors as well as disease severity. • Individual cases of periodontitis should be characterised according to the stage and grade of the disease. Authors Mariano Sanz and Maurizio Tonetti Published March 2019 European Federation of © European Federation of Periodontology Periodontology Introduction: classifying periodontitis Previous attempts to classify periodontitis have revolved around the question of whether phenotypically different case presentations represent different diseases or variations of a single disease. The internationally accepted classification of periodontitis, published in 1999, provided a workable framework that has been used extensively in both clinical practice and scientific investigation. But this system suffers from significant shortcomings, including substantial overlap, the lack of a clear pathobiology- based distinctions between categories, diagnostic imprecision, and difficulties in implementation. The New Classification from the 2017 World Workshop on Periodontal and Peri- implant Disease and Conditions (“the World Workshop”) reviewed the scientific evidence and reached four main conclusions: 1. There is no evidence of a specific pathophysiology that enables the differentiation of cases as “aggressive” or “chronic” periodontitis or provides guidance for different kinds of intervention. 2. There is little consistent evidence that aggressive and chronic periodontitis are different diseases. But there is evidence that multiple factors, and the interactions between them, influence clinically observable disease outcomes (phenotypes) at the individual level.